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1 ant to the development of chemically induced skin papillomas.
2 biting DMBA-initiated and TPA-promoted mouse-skin papillomas.
3 zygocity, they also developed characteristic skin papillomas.
4 decreased the number of mutant Hras-induced skin papillomas.
5 in A431 cells and during ablation of murine skin papillomas.
6 tiation led to the development of additional skin papillomas.
7 generate oncogenic mutations found in mouse skin papillomas.
8 r capacity to prevent the induction of mouse skin papillomas.
9 keratinocytes, which are precursor cells to skin papillomas.
10 rather promotes the malignant conversion of skin papillomas.
12 e decreases protein levels of Pdcd4 in mouse skin papillomas and keratinocytes as well as in human HE
13 analogs were effective in the prevention of skin papillomas and selective agonists for nuclear recep
14 cle are resistant to the development of both skin papillomas and squamous cell carcinoma (SCC) elicit
15 binding activity in adult SENCAR mouse skin, skin papillomas, and squamous cell carcinomas (SCC) gene
18 rosis factor alpha-mediated killing of MT1/2 skin papilloma cells where p50 homodimer activity is ele
19 tion-promotion protocol, substantially fewer skin papillomas developed in Odc(+/-) mice compared with
21 bearing chemically or UVB radiation-induced skin papillomas, followed by light application, led to i
23 n in tumor promoter-treated epidermis and in skin papillomas may occur, at least in part, via interac
24 te (TPA), a marked increase in the number of skin papillomas occurred in Tg.AC mice, which carry the
26 either a v-Ha-ras transgene to induce benign skin papillomas or p53 deficiency to induce spontaneous
27 expression was also shown to be elevated in skin papillomas relative to normal skins, and further in
30 using lysates from TPA-treated epidermis and skin papillomas showed enhanced interaction between the
31 se of TPA, 58% of K5-PKCalpha mice developed skin papillomas that progressed to carcinoma, whereas wi
33 as allele dramatically reduced the number of skin papillomas with Hras mutations, consistent with Hra
34 12-O-tetradecanoylphorbol-13-acetate-induced skin papillomas, with increased latency and greatly redu
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