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1 -nine house dust mite-sensitised adults were skin prick and allergen patch tested against a sterile s
2 herapies (elemental, 6-food elimination, and skin prick and atopy patch-directed elimination diets) a
6 tization in an unsupervised manner, based on skin prick and sIgE tests taken throughout childhood and
8 ) LEN) survey, we measured the prevalence of skin prick positivity to a panel of allergens, and geome
12 lected at age 12 months: food sensitization (skin prick test >/= 2 mm) and allergy (oral food challen
18 wheeze in last year, atopy assessed both by skin prick test (SPT) and by the measurement of allergen
21 in had the highest AUC (0.79), comparable to skin prick test (SPT) and sIgE to soy extract (0.76 and
23 sought to determine the association between skin prick test (SPT) and specific IgE (sIgE) to egg pro
24 ized nonallergic (n = 25) children underwent skin prick test (SPT) and specific IgE (sIgE) to peanut
25 ercise, were all negative.The results of the skin prick test (SPT) for Citrus unshiu and specific IgE
26 1 spiking of the ImmunoCAP, and size of the skin prick test (SPT) for hazelnut were determined, also
27 lergic sensitization was determined based on skin prick test (SPT) of five mites, three molds, and ni
29 ng cows' milk-specific IgE antibodies (IgE), skin prick test (SPT) reactivity and double-blind, place
30 HDE) bioactivity were predictive of allergen skin prick test (SPT) reactivity for infants at high ris
31 pecific IgE against aeroallergens (sIgE) and skin prick test (SPT) reactivity for the most common loc
35 action to egg, milk, or both with a positive skin prick test (SPT) response to the trigger food and/o
36 lergy, milk allergy, or both with a positive skin prick test (SPT) response to the trigger food and/o
37 nd 4 years of age and develop thresholds for skin prick test (SPT) results and specific IgE (sIgE) le
38 ping peanut allergy, and the implications of skin prick test (SPT) screening before peanut introducti
40 protein levels in household dust and peanut skin prick test (SPT) sensitization and likely allergy.
41 nfantile atopic dermatitis and preceding egg skin prick test (SPT) sensitization, we found a strong a
43 of 5276 one-year-old children who underwent skin prick test (SPT) to 4 food allergens and those with
46 eczema, egg allergy, or both but 0-mm peanut skin prick test (SPT) wheal responses (n = 542); group I
47 ty-two poly-sensitized athletes according to skin prick test (SPT) with different allergic phenotypes
48 fe (EQ-5D) health questionnaire, spirometry, skin prick test (SPT), exhaled nitric oxide (FeNO), smel
54 pecific IgE was 10.1% (95% CI: 9.4-10.8) and skin prick test 2.7% (95% CI: 2.4-3.0), food challenge p
55 nates, and allergic disease was evaluated by skin prick test and clinical examination at 12 months of
57 le NSAID in history were tested first with a skin prick test and if negative challenged with the culp
60 nical-demographic questionnaire, spirometry, skin prick test and specific IgE were evaluated yearly.
64 pplied for selected cases where the history, skin prick test and/or specific IgE are not definitive f
65 e positive ELISA results correlated with the skin prick test areas with the whole body and the setae
71 f the outcomes examined: at least 1 positive skin prick test from the panel of 10 allergens (21.7% vs
72 extracts was evaluated by IgE immunoblot and skin prick test in patients with clinical allergy to pea
74 s not appear to contain factors that enhance skin prick test or atopy patch test responses to house d
75 including detection of milk-specific IgE (by skin prick test or serum assay), diagnostic elimination
76 aluation, milk-specific IgE levels, and milk skin prick test performed at enrollment, 6 months, 12 mo
77 wanted to measure geographical variation in skin prick test positivity and assess whether it was exp
78 Geographical variation in the prevalence of skin prick test positivity in Europe is unlikely to be e
79 re fitted for allergic sensitization (either skin prick test positivity or serum-specific IgE >/= 0.3
82 Associations of NVAS and atopy (defined as skin prick test reaction of >/=3 mm) were analysed using
84 ion between a chronic helminth infection and skin prick test reactivity even in a developed country.
85 e reactions to peanut were reported in 1.5%, skin prick test reactivity in 2.0%, and IgE sensitizatio
87 studies demonstrated that exercise increases skin prick test reactivity to and bioavailability of the
88 Total IgE, grass pollen-specific IgE, and skin prick test reactivity to grass pollen were all redu
89 children with eczema, wheeze, or a positive skin prick test response before ending exclusive breast-
90 SAFS (n = 38) was defined as specific IgE or skin prick test response positivity to Aspergillus fumig
92 ible mold at age 1 year and child's positive skin prick test response to aeroallergens and molds at a
93 ID-independent anaphylaxis to LTPs, positive skin prick test response to LTPs, and serum LTP IgE.
95 PPOIT was associated with reduced peanut skin prick test responses and peanut-specific IgE levels
98 tifiable by using routinely available peanut skin prick test responses or specific IgE levels, but th
100 serum total IgE levels, specific IgE levels, skin prick test responses to common aeroallergens, and I
101 ves with allergic disease) but with negative skin prick test responses to common allergens at randomi
104 th a history of ragweed allergy and positive skin prick test responses to ragweed were randomized and
106 exposure and sensitization (as determined by skin prick test responses) was analyzed in more than 100
107 ith peanut- and Ara h 2-specific IgE levels, skin prick test responses, basophil activation, and TH2
108 eanut but have peanut-specific IgE, positive skin prick test responses, or both represents a signific
111 random sample of participants with negative skin prick test results attended a hospital-based food c
112 asing their judgment on clinical history and skin prick test results before and after obtaining the I
114 th antivenoms and cetuximab induced positive skin prick test results in patients with sIgE to alpha-g
116 higher IgG4 values (P = .001) and lower egg skin prick test scores (P = .0002) over time and a lower
117 among M+ participants tracked the following skin prick test sensitization statuses: M+P+C- > M+P+C+
118 lacebo-controlled oral food challenge (OFC), skin prick test titration (SPTT), and basophil histamine
119 e in peanut-specific basophil activation and skin prick test titration compared with nonresponders.
120 iral culture for varicella-zoster virus, and skin prick test to common food and animal allergens were
121 icipants aged 18 to 65 years with a positive skin prick test to Dactylis glomerata pollen were expose
122 easonal allergic rhinitis (SAR) and positive skin prick test to grass and olive pollens and evaluate
123 ty reaction after peanut ingestion, positive skin prick test to peanuts, and positive by double-blind
129 E 33.3 kUA /l (7.2-120.2), and median peanut skin prick test wheal 11.3 mm (6.5-18)]; four experience
130 In subjects ingesting baked egg, EW-induced skin prick test wheal diameter and EW-, ovalbumin-, and
133 thma, atopy (grass, house dust mite, and cat skin prick test) and atopic vs. non-atopic asthma at the
136 iagnostic testing, 47.3% was assessed with a skin prick test, 39.9% with a serum specific IgE test, a
137 ondary outcomes were desensitization, peanut skin prick test, and specific IgE and specific IgG4 meas
139 nical-demographic questionnaire, spirometry, skin prick test, and specific IgE to aeroallergens were
142 nly 4 predictors of the original model: sex, skin prick test, peanut sIgE, and total IgE minus sIgE.
143 shed, using 6 predictors: sex, age, history, skin prick test, peanut specific immunoglobulin E (sIgE)
149 gE/4 allergens: OR = 1.81, 95% CI 0.80-4.24; skin prick test/4+ allergens: OR = 2.27, 95% CI 1.34-3.9
152 on who initially had negative results on the skin-prick test, the prevalence of peanut allergy at 60
153 ract, which was determined with the use of a skin-prick test--one consisting of participants with no
154 dence of "atopic eczema," "any positive SPT [skin-prick test]," "sensitization to egg," and "sensitiz
155 dence and severity of atopic dermatitis, and skin-prick-test positivity at 6 mo of age were not diffe
159 tern blot and IgE-ELISA were complemented by Skin Prick Testing (SPT) and mediator release assay to d
163 orm beta lactam testing with 17% undertaking skin prick testing (SPT) only, 77% SPT followed by intra
164 5276 infants (HealthNuts), infants underwent skin prick testing (SPT) to egg white at 12 months of ag
165 ng hay fever, eczema, food allergy, positive skin prick testing (SPT), or elevated allergen-specific
168 intervention (structured allergy history and skin prick testing and appropriate advice on allergy avo
169 gy intervention (structured allergy history, skin prick testing and appropriate allergy avoidance adv
171 s defined as one or more positive results on skin prick testing and clinically relevant symptoms of r
172 stic correlates included end point titration skin prick testing and measurement of CM-specific IgE an
173 vited for a standardized physician exam with skin prick testing and parental interview at age 2 years
176 re, taking a structured allergy history with skin prick testing and tailored advice on allergy avoida
182 ly relevant sensitizations are elucidated by skin prick testing or by the determination of specific I
183 ulture and a sputum cell differential count; skin prick testing to both common aeroallergens and an e
184 ample of 5276 one-year-old infants underwent skin prick testing to peanut, egg, sesame, and cow's mil
187 grass pollen allergic individuals underwent skin prick testing with allergen alone, allergen plus Be
188 subjected to topical cowhage provocation and skin prick testing with histamine and assessed for diffe
190 minth Opishorchis felineus and specific IgE, skin prick testing, and atopic symptoms in Western Siber
191 e an interviewer-administered questionnaire, skin prick testing, and measurement of lung function fro
192 similar in children positive and negative on skin prick testing, and were not appreciably altered by
195 post-bronchodilator spirometry (n = 1,389), skin prick testing, lung volumes, and diffusing capacity
196 ths for scorings of symptoms and medication, skin prick testing, total IgE, specific IgE, and Der p 1
204 e markers measured, small wheal diameters on skin-prick testing and increases in egg-specific IgG4 an
211 y against common allergens was determined by skin prick tests (SPT); specific immunoglobulin E (sIgE)
215 ich they answered a questionnaire, underwent skin prick tests (SPTs) for common aeroallergens, and pr
216 were invited to a parental questionnaire and skin prick tests (SPTs) to ten airborne allergens, and 2
217 ous reactions was obtained, and standardized skin prick tests (SPTs) using finely ground tree-nut sol
218 erial 10-fold dilutions of milk protein, and skin prick tests (SPTs) were performed to commercial mil
220 logical work-up included a detailed history, skin prick tests (SPTs) with IVIP, and basophil activati
221 e following outcomes at age 2 years: eczema, skin prick tests (SPTs), increased allergen-specific IgE
222 7 with mollusc tolerance) were studied using skin prick tests (SPTs), specific IgEs (sIgEs) and SDS-P
224 tic sensitivity to 65% compared with 20% for skin prick tests and 46% ImmunoCAP using kiwi extract.
225 nonallergic subjects (group 4) by performing skin prick tests and APTs with rBet v 1 and hypoallergen
228 re, taking a structured allergy history with skin prick tests and giving tailored advice on allergy a
234 reened 5276 infants (74% participation) with skin prick tests and sensitized infants underwent food c
236 ust mites was diagnosed longitudinally using skin prick tests and specific IgE measurements at (1/2),
237 icult, because of the limited sensitivity of skin prick tests and specific IgE tests to meat extracts
239 ergenicity of Ory c 3 was confirmed by using skin prick tests and the basophil activation assay.
240 formed consent; evidencing of an allergen by skin prick tests and/or serum-specific IgE dosages; bein
241 ldren (5-17 years old) with asthma underwent skin prick tests at baseline and had clinical data colle
242 sensitization (FAS) was identified by using skin prick tests conducted between 1 and 18 years of age
245 hensive set of recommendations on the use of skin prick tests in allergic rhinitis-conjunctivitis and
246 to determine whether C+ assayed by means of skin prick tests influenced AR symptom severity in contr
247 es to TG and DM allergen based on results of skin prick tests or nasal disk challenges (P < .01 and P
248 Two hundred eighty-one children had valid skin prick tests performed, and 14% (39/281) were atopic
250 population-based HealthNuts study underwent skin prick tests to determine peanut sensitization and s
253 ciation between 10 loci and specific IgE and skin prick tests to individual allergens and poly-sensit
256 easurements of eczema, asthma, rhinitis, and skin prick tests were available for all follow-ups.
261 Food-specific serum IgE measurements and skin prick tests were performed before initiating the di
262 n, IgE inhibition, ImmunoCAP inhibition, and skin prick tests were performed using samples from selec
266 meat, we studied the possibility to perform skin prick tests with cetuximab, which carries the alpha
269 llenges with standardized doses of rMal d 1, skin prick tests with recombinant allergens, and measure
271 apnic voluntary hyperpnea challenge, allergy skin prick tests, and bronchoscopy with bronchial biopsi
272 ecific immunoglobulin E-antibodies in serum, skin prick tests, and double-blind, placebo-controlled f
274 te use of testing (specific IgE measurement, skin prick tests, and oral food challenges), and the tim
275 food-allergic sensitization were measured by skin prick tests, and physician-diagnosed inhalant and f
278 -of-function samples, we performed histamine skin prick tests, investigated the contribution of STAT3
280 for food allergy by standardized interviews, skin prick tests, prick-to-prick tests and ImmunoCAP.
295 co-factor enhanced food allergy, assessed by skin-prick tests, specific IgE and oral challenges.
299 At Day 85, 6 weeks after the last dose, skin prick wheal responses to allergen were suppressed b
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