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1 o standard diagnostic methods (ImmunoCAP and skin prick test).
2 n infants with allergic mothers positive for skin prick test.
3 nd ELISA inhibition, basophil activation and skin prick test.
4 85; P = 0.02), and with similar findings for skin prick tests.
5 he patient's allergic triggers or performing skin prick tests.
6 en Asthma Study completed questionnaires and skin prick tests.
7 Atopy was assessed by using skin prick tests.
8 Atopy was assessed using skin prick tests.
9 n observed pasteurized raw egg challenge and skin prick tests.
10 and 6 years by specific IgE assessments and skin prick tests.
11 inical examinations including serum IgEs and skin prick tests.
12 saline challenge tests, questionnaires, and skin prick tests.
13 t basophils and elicit positive responses in skin prick tests.
14 ystematic reintroduction analysis but not by skin-prick tests.
15 en patients assigned to immediate or delayed skin prick testing.
16 pic status were determined by spirometry and skin prick testing.
17 ng a rat basophil leukaemia cell line and by skin prick testing.
18 ed with IL-31 and NaCl (negative control) by skin prick testing.
19 s underwent a clinical examination including skin prick testing.
20 of rhinitis but without positive results on skin prick testing.
21 ldren were examined for eczema and underwent skin prick testing.
22 ly to attend research clinics and consent to skin-prick testing.
23 re invited for measurement of spirometry and skin-prick testing.
24 pecific IgE was 10.1% (95% CI: 9.4-10.8) and skin prick test 2.7% (95% CI: 2.4-3.0), food challenge p
26 iagnostic testing, 47.3% was assessed with a skin prick test, 39.9% with a serum specific IgE test, a
27 gE/4 allergens: OR = 1.81, 95% CI 0.80-4.24; skin prick test/4+ allergens: OR = 2.27, 95% CI 1.34-3.9
29 nates, and allergic disease was evaluated by skin prick test and clinical examination at 12 months of
31 le NSAID in history were tested first with a skin prick test and if negative challenged with the culp
34 nical-demographic questionnaire, spirometry, skin prick test and specific IgE were evaluated yearly.
38 pplied for selected cases where the history, skin prick test and/or specific IgE are not definitive f
39 intervention (structured allergy history and skin prick testing and appropriate advice on allergy avo
40 gy intervention (structured allergy history, skin prick testing and appropriate allergy avoidance adv
42 s defined as one or more positive results on skin prick testing and clinically relevant symptoms of r
43 stic correlates included end point titration skin prick testing and measurement of CM-specific IgE an
44 vited for a standardized physician exam with skin prick testing and parental interview at age 2 years
47 re, taking a structured allergy history with skin prick testing and tailored advice on allergy avoida
48 tic sensitivity to 65% compared with 20% for skin prick tests and 46% ImmunoCAP using kiwi extract.
49 nonallergic subjects (group 4) by performing skin prick tests and APTs with rBet v 1 and hypoallergen
52 re, taking a structured allergy history with skin prick tests and giving tailored advice on allergy a
58 reened 5276 infants (74% participation) with skin prick tests and sensitized infants underwent food c
60 ust mites was diagnosed longitudinally using skin prick tests and specific IgE measurements at (1/2),
61 icult, because of the limited sensitivity of skin prick tests and specific IgE tests to meat extracts
64 formed consent; evidencing of an allergen by skin prick tests and/or serum-specific IgE dosages; bein
66 e markers measured, small wheal diameters on skin-prick testing and increases in egg-specific IgG4 an
68 thma, atopy (grass, house dust mite, and cat skin prick test) and atopic vs. non-atopic asthma at the
69 ondary outcomes were desensitization, peanut skin prick test, and specific IgE and specific IgG4 meas
71 nical-demographic questionnaire, spirometry, skin prick test, and specific IgE to aeroallergens were
73 20 children, respectively; 700 children were skin prick tested, and FEV(1) was measured in 478 and ex
74 minth Opishorchis felineus and specific IgE, skin prick testing, and atopic symptoms in Western Siber
76 e an interviewer-administered questionnaire, skin prick testing, and measurement of lung function fro
77 similar in children positive and negative on skin prick testing, and were not appreciably altered by
79 apnic voluntary hyperpnea challenge, allergy skin prick tests, and bronchoscopy with bronchial biopsi
80 ecific immunoglobulin E-antibodies in serum, skin prick tests, and double-blind, placebo-controlled f
82 te use of testing (specific IgE measurement, skin prick tests, and oral food challenges), and the tim
83 food-allergic sensitization were measured by skin prick tests, and physician-diagnosed inhalant and f
87 er operating characteristic curves comparing skin prick tests (area under the curve [AUC], 0.87; 95%
88 e positive ELISA results correlated with the skin prick test areas with the whole body and the setae
90 ldren (5-17 years old) with asthma underwent skin prick tests at baseline and had clinical data colle
91 creased incidence of allergic sensitization (skin prick test) at the age of 4 years in a cohort of 98
93 sensitization (FAS) was identified by using skin prick tests conducted between 1 and 18 years of age
104 f the outcomes examined: at least 1 positive skin prick test from the panel of 10 allergens (21.7% vs
105 lected at age 12 months: food sensitization (skin prick test >/= 2 mm) and allergy (oral food challen
108 extracts was evaluated by IgE immunoblot and skin prick test in patients with clinical allergy to pea
109 hensive set of recommendations on the use of skin prick tests in allergic rhinitis-conjunctivitis and
111 to determine whether C+ assayed by means of skin prick tests influenced AR symptom severity in contr
113 -of-function samples, we performed histamine skin prick tests, investigated the contribution of STAT3
116 post-bronchodilator spirometry (n = 1,389), skin prick testing, lung volumes, and diffusing capacity
120 ract, which was determined with the use of a skin-prick test--one consisting of participants with no
121 s not appear to contain factors that enhance skin prick test or atopy patch test responses to house d
122 including detection of milk-specific IgE (by skin prick test or serum assay), diagnostic elimination
123 ly relevant sensitizations are elucidated by skin prick testing or by the determination of specific I
124 es to TG and DM allergen based on results of skin prick tests or nasal disk challenges (P < .01 and P
125 nly 4 predictors of the original model: sex, skin prick test, peanut sIgE, and total IgE minus sIgE.
126 shed, using 6 predictors: sex, age, history, skin prick test, peanut specific immunoglobulin E (sIgE)
127 aluation, milk-specific IgE levels, and milk skin prick test performed at enrollment, 6 months, 12 mo
128 Two hundred eighty-one children had valid skin prick tests performed, and 14% (39/281) were atopic
130 wanted to measure geographical variation in skin prick test positivity and assess whether it was exp
131 t birth order, is a significant predictor of skin prick test positivity at age 4 (IgE below detection
132 Geographical variation in the prevalence of skin prick test positivity in Europe is unlikely to be e
133 re fitted for allergic sensitization (either skin prick test positivity or serum-specific IgE >/= 0.3
135 dence and severity of atopic dermatitis, and skin-prick-test positivity at 6 mo of age were not diffe
137 for food allergy by standardized interviews, skin prick tests, prick-to-prick tests and ImmunoCAP.
140 Associations of NVAS and atopy (defined as skin prick test reaction of >/=3 mm) were analysed using
142 ion between a chronic helminth infection and skin prick test reactivity even in a developed country.
143 e reactions to peanut were reported in 1.5%, skin prick test reactivity in 2.0%, and IgE sensitizatio
145 studies demonstrated that exercise increases skin prick test reactivity to and bioavailability of the
146 Total IgE, grass pollen-specific IgE, and skin prick test reactivity to grass pollen were all redu
147 rtive evidence for linkage between a general skin prick test reactivity trait (but not with total ser
148 children with eczema, wheeze, or a positive skin prick test response before ending exclusive breast-
149 SAFS (n = 38) was defined as specific IgE or skin prick test response positivity to Aspergillus fumig
151 ible mold at age 1 year and child's positive skin prick test response to aeroallergens and molds at a
152 ID-independent anaphylaxis to LTPs, positive skin prick test response to LTPs, and serum LTP IgE.
154 PPOIT was associated with reduced peanut skin prick test responses and peanut-specific IgE levels
157 tifiable by using routinely available peanut skin prick test responses or specific IgE levels, but th
159 serum total IgE levels, specific IgE levels, skin prick test responses to common aeroallergens, and I
160 ves with allergic disease) but with negative skin prick test responses to common allergens at randomi
163 th a history of ragweed allergy and positive skin prick test responses to ragweed were randomized and
165 exposure and sensitization (as determined by skin prick test responses) was analyzed in more than 100
166 ith peanut- and Ara h 2-specific IgE levels, skin prick test responses, basophil activation, and TH2
167 eanut but have peanut-specific IgE, positive skin prick test responses, or both represents a signific
170 random sample of participants with negative skin prick test results attended a hospital-based food c
171 asing their judgment on clinical history and skin prick test results before and after obtaining the I
173 th antivenoms and cetuximab induced positive skin prick test results in patients with sIgE to alpha-g
176 higher IgG4 values (P = .001) and lower egg skin prick test scores (P = .0002) over time and a lower
178 among M+ participants tracked the following skin prick test sensitization statuses: M+P+C- > M+P+C+
179 dence of "atopic eczema," "any positive SPT [skin-prick test]," "sensitization to egg," and "sensitiz
184 co-factor enhanced food allergy, assessed by skin-prick tests, specific IgE and oral challenges.
187 wheeze in last year, atopy assessed both by skin prick test (SPT) and by the measurement of allergen
190 in had the highest AUC (0.79), comparable to skin prick test (SPT) and sIgE to soy extract (0.76 and
192 sought to determine the association between skin prick test (SPT) and specific IgE (sIgE) to egg pro
193 ized nonallergic (n = 25) children underwent skin prick test (SPT) and specific IgE (sIgE) to peanut
194 ercise, were all negative.The results of the skin prick test (SPT) for Citrus unshiu and specific IgE
195 1 spiking of the ImmunoCAP, and size of the skin prick test (SPT) for hazelnut were determined, also
196 lergic sensitization was determined based on skin prick test (SPT) of five mites, three molds, and ni
198 ng cows' milk-specific IgE antibodies (IgE), skin prick test (SPT) reactivity and double-blind, place
199 HDE) bioactivity were predictive of allergen skin prick test (SPT) reactivity for infants at high ris
200 pecific IgE against aeroallergens (sIgE) and skin prick test (SPT) reactivity for the most common loc
204 action to egg, milk, or both with a positive skin prick test (SPT) response to the trigger food and/o
205 lergy, milk allergy, or both with a positive skin prick test (SPT) response to the trigger food and/o
206 nd 4 years of age and develop thresholds for skin prick test (SPT) results and specific IgE (sIgE) le
207 ping peanut allergy, and the implications of skin prick test (SPT) screening before peanut introducti
209 protein levels in household dust and peanut skin prick test (SPT) sensitization and likely allergy.
210 nfantile atopic dermatitis and preceding egg skin prick test (SPT) sensitization, we found a strong a
212 of 5276 one-year-old children who underwent skin prick test (SPT) to 4 food allergens and those with
215 eczema, egg allergy, or both but 0-mm peanut skin prick test (SPT) wheal responses (n = 542); group I
216 ty-two poly-sensitized athletes according to skin prick test (SPT) with different allergic phenotypes
217 fe (EQ-5D) health questionnaire, spirometry, skin prick test (SPT), exhaled nitric oxide (FeNO), smel
223 tern blot and IgE-ELISA were complemented by Skin Prick Testing (SPT) and mediator release assay to d
227 orm beta lactam testing with 17% undertaking skin prick testing (SPT) only, 77% SPT followed by intra
228 5276 infants (HealthNuts), infants underwent skin prick testing (SPT) to egg white at 12 months of ag
229 ng hay fever, eczema, food allergy, positive skin prick testing (SPT), or elevated allergen-specific
236 y against common allergens was determined by skin prick tests (SPT); specific immunoglobulin E (sIgE)
240 ich they answered a questionnaire, underwent skin prick tests (SPTs) for common aeroallergens, and pr
241 were invited to a parental questionnaire and skin prick tests (SPTs) to ten airborne allergens, and 2
242 ous reactions was obtained, and standardized skin prick tests (SPTs) using finely ground tree-nut sol
243 erial 10-fold dilutions of milk protein, and skin prick tests (SPTs) were performed to commercial mil
245 logical work-up included a detailed history, skin prick tests (SPTs) with IVIP, and basophil activati
246 al physical examinations, children underwent skin prick tests (SPTs), eczema was diagnosed by a clini
247 e following outcomes at age 2 years: eczema, skin prick tests (SPTs), increased allergen-specific IgE
248 7 with mollusc tolerance) were studied using skin prick tests (SPTs), specific IgEs (sIgEs) and SDS-P
250 on who initially had negative results on the skin-prick test, the prevalence of peanut allergy at 60
251 lacebo-controlled oral food challenge (OFC), skin prick test titration (SPTT), and basophil histamine
252 e in peanut-specific basophil activation and skin prick test titration compared with nonresponders.
253 iral culture for varicella-zoster virus, and skin prick test to common food and animal allergens were
254 icipants aged 18 to 65 years with a positive skin prick test to Dactylis glomerata pollen were expose
255 easonal allergic rhinitis (SAR) and positive skin prick test to grass and olive pollens and evaluate
256 ty reaction after peanut ingestion, positive skin prick test to peanuts, and positive by double-blind
257 ulture and a sputum cell differential count; skin prick testing to both common aeroallergens and an e
258 ample of 5276 one-year-old infants underwent skin prick testing to peanut, egg, sesame, and cow's mil
260 population-based HealthNuts study underwent skin prick tests to determine peanut sensitization and s
263 ciation between 10 loci and specific IgE and skin prick tests to individual allergens and poly-sensit
266 ths for scorings of symptoms and medication, skin prick testing, total IgE, specific IgE, and Der p 1
272 rdiac surgery (age, 1 week to 14 years), and skin prick testing was performed from 12 months of age.
276 easurements of eczema, asthma, rhinitis, and skin prick tests were available for all follow-ups.
281 Food-specific serum IgE measurements and skin prick tests were performed before initiating the di
282 n, IgE inhibition, ImmunoCAP inhibition, and skin prick tests were performed using samples from selec
286 E 33.3 kUA /l (7.2-120.2), and median peanut skin prick test wheal 11.3 mm (6.5-18)]; four experience
287 In subjects ingesting baked egg, EW-induced skin prick test wheal diameter and EW-, ovalbumin-, and
291 grass pollen allergic individuals underwent skin prick testing with allergen alone, allergen plus Be
292 subjected to topical cowhage provocation and skin prick testing with histamine and assessed for diffe
294 meat, we studied the possibility to perform skin prick tests with cetuximab, which carries the alpha
297 llenges with standardized doses of rMal d 1, skin prick tests with recombinant allergens, and measure
300 E, eosinophil count in peripheral blood, and skin-prick tests with three different allergens (cockroa
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