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   1  discontinued treatment because of toxicity (skin rash).                                             
     2 AL-PEG (0.100 mg/kg) developed a generalised skin rash.                                              
     3 bexarotene-induced hypertriglyceremia and/or skin rash.                                              
     4 xarotene-induced hypertriglyceridemia and/or skin rash.                                              
     5 ose reductions of erlotinib due to grade 2/3 skin rash.                                              
     6  prophylactic treatment of erlotinib-induced skin rash.                                              
     7 oxicities were mild hypertriglyceridemia and skin rash.                                              
     8  is a commonly occurring, intensely pruritic skin rash.                                              
     9 tion of viremia and developed a desquamating skin rash.                                              
    10  syndrome, and two patients (5%) had grade 3 skin rash.                                              
    11 atient experienced a reactivation-associated skin rash.                                              
    12 w transplant (seroconversion, HHV-6 viremia, skin rash); 18 of 20 had increased peripheral blood mono
    13 he most common grade 3-4 adverse events were skin rash (21 [27%] of 77 patients vs 20 [22%] of 92 pat
    14  fever (43%), anorexia (33%), fatigue (33%), skin rash (21%), stomatitis (14%), and allergic reaction
    15 pulmonary toxicity were common, and included skin rash (38%), peripheral eosinophilia (38%), liver dy
  
    17    The most common grade 3 or 4 toxicity was skin rash (45%), followed by neutropenia (21%) without f
  
  
  
  
  
    23  on 2 girls with periodic episodes of fever, skin rash, abdominal pain, and arthralgia, of whom 1 had
    24 de female predominance, presence of fever or skin rash, absence of subcutaneous nodules or finger clu
  
    26 ghlights successful management of telaprevir skin rash and anal discomfort by switching to boceprevir
  
    28 ght (53%) experienced grade 1 to 2 acne-like skin rash and diarrhea, but no grade 3 or 4 toxicity occ
  
    30 1-blind MV, viremia was short-lived, and the skin rash and other clinical signs observed with wild-ty
    31 All responding patients had mild (grade 1/2) skin rash and two patients had positive tumoral HER1/EGF
    32 rile neutropenia, hypotension, myalgias, and skin rash and were removed from treatment more often as 
    33 itis, sensorineural hearing loss, urticarial skin rash, and a characteristic deforming arthropathy.  
    34 f emergency department visits for asthma and skin rash, and Culex quinquefasciatus species-specific v
    35 en patients had normal liver function tests, skin rash, and diagnosis of GvHD histologically confirme
    36 vere infantile-onset IBD, failure to thrive, skin rash, and perirectal abscesses refractory to medica
  
    38  phenotype includes granulomatous arthritis, skin rash, and uveitis and probably represents a subtype
    39 r, sterile peritonitis, arthralgia, myalgia, skin rash, and/or conjunctivitis; some patients also dev
  
  
    42 ned with the terms "psoriasis," "pustular," "skin," "rash," and "palmoplantar." All relevant articles
  
  
    45  -4.03 [95% CI, -13.76 to 5.70]; P = .42) or skin rash (beta = -1.00 [95% CI, -6.92 to 4.92]; P = .74
    46 netic disorder characterized by a congenital skin rash, birth defects of the skeleton, genomic instab
    47 and hypersensitivity, diarrhea and vomiting, skin rash, clinical deterioration, and patient's wishes 
    48 include GI (diarrhea, anorexia, and nausea), skin rash, cytopenias, pleural effusions, and fatigue.  
    49 re to industrial phenolics is known to cause skin rash, dermal inflammation, contact dermatitis, leuc
  
  
  
    53 ecipients with a clinical suspicion of GvHD (skin rash, diarrhea, pyrexia, pancytopenia, or anemia, w
  
    55 tion (three [8%]), hypertension (six [16%]), skin rash (eight [22%]), and pancreatitis (six [16%] pat
    56 mptoms; however, all 3 children did manifest skin rash, fatigue, and biopsy-proven glomerulonephritis
  
  
  
  
  
    62 e cisplatin toxicity but was associated with skin rash in a majority of patients and occasional serio
    63 treatment was interrupted because of grade 3 skin rash in four patients in the placebo arm, and none 
  
    65 ost common toxicities were anemia, acne-like skin rash, leukopenia, fatigue and malaise, and nausea a
  
    67 hematologic toxicities of docetaxel included skin rashes, mucositis, and mild elevations of serum tra
    68 st abnormalities, nausea/vomiting, diarrhea, skin rashes, mucositis, and palmoplantar erythrodysesthe
  
  
  
    72   Anemia, gastrointestinal side effects, and skin rashes occurred at a higher incidence among patient
    73 re supported by histologic findings from the skin rash of a human subject who received an attenuated 
  
  
    76 y more frequent at contrast-enhanced CT were skin rash (P = .0311), skin redness (P = .0055), skin sw
  
    78 elationship to study drug, were an acne-like skin rash, predominantly on the face and upper torso (86
    79 so 2 apparent treatment failures in cases of skin rash, raising questions about the efficacy or suita
    80 associated vasculitis manifested by purpuric skin rashes, renal abnormalities, and elevated cryoglobu
    81 ade 3 adverse event in the AZD8931 group was skin rash (three [20%] of 15 patients with available dat
  
    83  four [4%] vs two [2%] postoperatively), and skin rash (two [1%] vs 21 [15%] preoperatively; 0 vs eig
  
  
  
  
  
  
    90 (> 5 days) and/or associated with fever, and skin rashes were consistently experienced by heavily (HP
  
    92 toimmune events with the early appearance of skin rashes were observed in patients with stable diseas
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