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1 Four children died, of whom only one had a skull fracture.
2 racranial injury can occur in the absence of skull fracture.
3 was done on 156 children, of whom 107 had a skull fracture.
4 whom had severe head injuries with multiple skull fractures.
6 iocervical junction, and tend also to have a skull fracture, a thin film of subdural haemorrhage, but
7 geminal nerve anesthesia-one following basal skull fracture and another following large posterior fos
8 tabase of all patients with TBI (findings of skull fracture and/or intracranial hemorrhage on an init
9 uding (a) healed rib fractures, (b) hairline skull fractures and a compression deformity of the thora
10 s, non-severe injury mechanism, no palpable skull fracture, and acting normally according to the par
11 Lower GCS score, midline shift, depressed skull fracture, and epidural hematoma are key risk facto
12 tracranial hemorrhage (other than epidural), skull fracture, and higher head/neck injury severity.
13 severe injury mechanism, no signs of basilar skull fracture, and no severe headache) had a negative p
14 DVST was seen in only those patients with skull fractures extending to a dural sinus or jugular bu
15 bulbs in 57 (40.7%) of the 140 patients with skull fractures extending to a dural sinus or jugular bu
18 anatomically exquisite portrayals of growing skull fractures for neurosurgical planning, but its rout
20 the acutely ill patient or identification of skull fractures in the assessment of a patient with head
22 of consciousness, post-traumatic amnesia, or skull fracture) in Olmsted County, Minnesota, during the
23 was diagnosed (eg, cervical spine fracture, skull fracture, intracranial bleeding, liver and spleen
24 venograms were reviewed for the presence of skull fractures, intracranial hemorrhage, and traumatic
25 ospective database on all patients with TBI (skull fracture/intracranial hemorrhage on head computed
26 physical examination findings suggestive of skull fracture (likelihood ratio [LR], 16; 95% CI, 3.1-5
27 were brain contusion with subdural hematoma, skull fracture, loss of consciousness or amnesia for mor
28 ldren who were admitted to hospital or had a skull fracture (n = 883) were included in the study.
30 ed tomography (CT) was done in children with skull fractures on radiography and in those without frac
31 that mTBI model did not produce brain edema, skull fracture or sensorimotor coordination dysfunctions
32 shift (OR, 6.8; 95% CI, 3.4-13.8), depressed skull fracture (OR, 6.5; 95% CI, 3.7-11.4), and epidural
33 onsciousness for 30 minutes to 24 hours or a skull fracture), or severe (loss of consciousness or amn
34 , ejected from vehicle, fall >1 m, suspected skull fracture, or GCS score <15 at 2 hours) had an LR o
35 for atraumatic brain abnormalities, isolated skull fractures, or chronic intracranial hemorrhage.
36 er age, traumatic brain injury severity, and skull fractures predict anterior pituitary disorders, wh
37 traumatic brain injury; seven studies), and skull fractures (risk ratio, 1.73; 95% CI, 1.03-2.91; si
39 2 through 1995, 87 consecutive children with skull fractures visible at plain radiography were referr
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