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1 thmatics with moderate to severe obstructive sleep apnea syndrome.
2 tients, 697 (68 percent) had the obstructive sleep apnea syndrome.
3 s and sleep in patients with the obstructive sleep apnea syndrome.
4 important in the pathogenesis of obstructive sleep apnea syndrome.
5 ic cardiovascular abnormality in obstructive sleep apnea syndrome.
6 l occlusion which results in the obstructive sleep apnea syndrome.
7 been tested as pharmacologic treatments for sleep apnea syndrome.
8 he ratio of %EFV to %VAF loss decreased with sleep apnea syndrome (1.34+/-0.3 vs. 0.52+/-0.08, p<0.05
9 ously regarding the evolution of obstructive sleep apnea syndrome and persistence of abnormal pharyng
10 d disorders of ventilatory control including sleep apnea syndromes and obesity hypoventilation syndro
11 s included obesity hypoventilation syndrome, sleep apnea syndrome, hypertension, gastroesophageal ref
13 rved a significant prevalence of obstructive sleep apnea syndrome in patients in waiting list for LT,
14 ting, surgical treatment for the obstructive sleep apnea syndrome in school-age children did not sign
19 gs confirm previous reports that obstructive sleep apnea syndrome is associated with reduced parasymp
22 studies have suggested that the obstructive sleep apnea syndrome may be an important risk factor for
23 ions of these findings for the management of sleep apnea syndrome must be verified by appropriate cli
24 ne the independent effect of the obstructive sleep apnea syndrome on the composite outcome of stroke
25 ity (body mass index [BMI] >35), obstructive sleep apnea syndrome, or other causes of respiratory fai
27 ARS) (23%) than in subjects with obstructive sleep apnea syndrome (OSAS) (0.06%), parasomnia (0.7%),
28 ight is modestly associated with obstructive sleep apnea syndrome (OSAS) among young children, but st
30 ittent hypoxia (CIH) occurs with obstructive sleep apnea syndrome (OSAS) and provokes systemic endoth
31 ative treatment in children with obstructive sleep apnea syndrome (OSAS) associated with significant
33 he upper airway in children with obstructive sleep apnea syndrome (OSAS) have not been established.
37 the pathophysiology of pediatric obstructive sleep apnea syndrome (OSAS) is suggested by the observat
40 few studies suggesting that the obstructive sleep apnea syndrome (OSAS) may compromise optic nerve h
41 estigate the correlation between obstructive sleep apnea syndrome (OSAS) risk with periodontal diseas
42 asthma patients with concomitant obstructive sleep apnea syndrome (OSAS) seems to have a favorable im
44 s been reported in patients with obstructive sleep apnea syndrome (OSAS), and these two chronic condi
49 al breathing in 10 children with obstructive sleep apnea syndrome (OSAS; age, 4.3 +/- 2.3 years) and
50 ent (Mallampati score III or IV, obstructive sleep apnea syndrome, reduced mobility of cervical spine
51 rillation, and hypertension, the obstructive sleep apnea syndrome retained a statistically significan
52 syndrome (FES) and body mass index (BMI) in sleep apnea syndrome (SAS) patients compared to normal s
53 eart disease, morbid obesity associated with sleep apnea syndrome, sickle cell disease, and polycythe
55 n, 5 to 9 years of age, with the obstructive sleep apnea syndrome to early adenotonsillectomy or a st
57 In an unadjusted analysis, the obstructive sleep apnea syndrome was associated with stroke or death
59 sized that, in children with the obstructive sleep apnea syndrome without prolonged oxyhemoglobin des
60 y performed in children with the obstructive sleep apnea syndrome, yet its usefulness in reducing sym
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