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1 p with cardiopulmonary instability caused by sleep apnoea.
2 LVEF </=45%) treated for predominant central sleep apnoea.
3 arely developed specifically for obstructive sleep apnoea.
4 M, hypertension, angina, MI, and obstructive sleep apnoea.
5 or abnormal sleep behaviours and obstructive sleep apnoea.
6 c syndrome, type 2 diabetes, and obstructive sleep apnoea.
7 r airway motor disorders such as obstructive sleep apnoea.
8 infant death syndrome (SIDS) and obstructive sleep apnoea.
9 ced respiratory chemosensitivity and central sleep apnoea.
10 ion is impaired in patients with obstructive sleep apnoea.
11 ng hypertension in patients with obstructive sleep apnoea.
12 jor clinical problem of cyclical obstructive sleep apnoea.
13 echanism linking hypertension to obstructive sleep apnoea.
14 al neurostimulation in patients with central sleep apnoea.
15 ascular disease in patients with obstructive sleep apnoea.
16 a promising therapeutic approach for central sleep apnoea.
17 es, baseline blood pressure, and severity of sleep apnoea.
18 s widely used as a treatment for obstructive sleep apnoea.
20 intermittent hypoxia (CIH) is a hallmark of sleep apnoea, a condition associated with diverse clinic
23 cal disorders, including cerebral ischaemia, sleep apnoea, Alzheimer's disease, multiple sclerosis, a
26 ittent hypoxia (CIH) occurs in patients with sleep apnoea and has adverse effects on multiple physiol
28 bsence of RTN neurons probably underlies the sleep apnoea and lack of chemoreflex that characterize c
29 ion, but the association between obstructive sleep apnoea and myocardial infarction, stroke, and cong
30 specific physiological sleep disorders--eg, sleep apnoea and periodic limb movement disorder--are es
31 focus on the association between obstructive sleep apnoea and stroke reviewing both the epidemiologic
34 arction (MI), stroke, fractures, obstructive sleep apnoea, and cancer; mortality; and resolution of h
36 leep-related disorders including obstructive sleep apnoea (apnea), REM sleep behaviour disorder (RBD)
41 insufficiency in diverse conditions, such as sleep apnoea, cervical spinal injury or amyotrophic late
42 l trial of therapeutic NCPAP for obstructive sleep apnoea compared with a control group on subtherape
43 ause brain hypoperfusion such as obstructive sleep apnoea, congestive heart failure, cardiac arrhythm
45 olic abnormalities in those with obstructive sleep apnoea could reduce cardiovascular disease risk an
46 will be discussed, management of obstructive sleep apnoea could soon transition from a so-called one
49 n blood pressure in 118 men with obstructive sleep apnoea (Epworth score > 9, and a > 4% oxygen desat
53 in 24.5% (13/53); five of these patients had sleep apnoea/hypopnoea syndrome, six had depression and
54 tivity observed in patients with obstructive sleep apnoea (i.e. reflex compensation for an anatomical
55 cause of perinatal mortality in infants and sleep apnoea in adults, but the mechanisms of respirator
59 ignificantly reduced the severity of central sleep apnoea, including improvements in sleep metrics, a
62 There is convincing evidence to believe that sleep apnoea is a modifiable risk factor for stroke; how
68 primary sleep disorders such as obstructive sleep apnoea may worsen epilepsy and treatment of these
70 h of more than 4% SaO2 caused by obstructive sleep apnoea on overnight sleep study, were randomly ass
72 roup of elderly individuals with obstructive sleep apnoea (OSA) for comparison (n = 3, age 68 +/- 1 y
73 ther PA and vLTF are enhanced in obstructive sleep apnoea (OSA) participants compared to matched heal
74 re (CPAP) for moderate to severe obstructive sleep apnoea (OSA) syndrome have been established in mid
75 od O2 saturation profiles during obstructive sleep apnoea (OSA), have been shown to exhibit a heighte
76 od O2 saturation profiles during obstructive sleep apnoea (OSA), have been shown to exhibit a heighte
77 ation (SF), a primary feature of obstructive sleep apnoea (OSA), impairs hippocampal long-term potent
79 loop gain (LG) in patients with obstructive sleep apnoea (OSA), yet its effects on the other traits
83 t that acetazolamide may improve obstructive sleep apnoea (OSA).However, how acetazolamide affects th
84 who demonstrated spontaneous apnoeas during sleep, apnoea per se did not alter burst latency relativ
85 rate progression of, obstructive and central sleep apnoea, possibly through the development of periph
86 rm 36 mental component summary (MCS) and the sleep apnoea quality-of-life index symptoms domain (sym)
88 ure, the treatment of choice for obstructive sleep apnoea, reduces sleepiness and improves hypertensi
89 lysomnography showed features of obstructive sleep apnoea, stridor, and abnormal sleep architecture (
92 ntribution of genetic factors to obstructive sleep apnoea syndrome (OSAS) has led to a better underst
94 onalcoholic fatty liver disease, obstructive sleep apnoea syndrome, erectile dysfunction, periodontit
96 efit was larger in patients with more severe sleep apnoea than those who had less severe apnoea, but
97 past two decades indicates that obstructive sleep apnoea, through the effects of intermittent hypoxa
98 usitis, gastroesophageal reflux, obstructive sleep apnoea, vocal cord dysfunction, obesity, dysfuncti
99 he pathogenesis and treatment of obstructive sleep apnoea, we have developed a novel application of m
101 upon such mechanisms (as in the patient with sleep apnoea) will be prone to collapse during sleep.
102 (SERVE-HF) showed that treatment of central sleep apnoea with adaptive servoventilation in patients
103 cent insights and discoveries in obstructive sleep apnoea, with a focus on diagnostics and therapeuti
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