ライフサイエンスコーパス: sleep disruption

1 e potential to cause per se narcoleptic-like sleep disruption.

2 acterized by recurrent nocturnal hypoxia and sleep disruption.

3 leep associated with oxygen desaturation and sleep disruption.

4 e, bowel/bladder and sexual dysfunction, and sleep disruption.

5 termine the effect of environmental noise on sleep disruption.

6 hey generally suggest modest and nonspecific sleep disruptions.

7 g neural mechanisms that explain age-related sleep disruption?

8 anisms linking non-rapid-eye-movement (NREM) sleep disruption, Abeta, and AD; (ii) a role for NREM sl

9 Although a clinical association between sleep disruption and AD has long been appreciated, emerg

10 C1 cell activation likely contributes to the sleep disruption and adverse autonomic consequences of s

11 Despite the co-occurrence of sleep disruption and anxiety disorders, the impact of sl

12 e and central types of sleep apnea result in sleep disruption and arterial oxyhemoglobin desaturation

13 Sleep disruption and arterial oxyhemoglobin desaturation

14 echanisms underlying the interaction between sleep disruption and behavior remain poorly understood.

15 sis of a direct pathological role of PLMS in sleep disruption and can be important for the discussion

16 onfused about the time of day and experience sleep disruption and fatigue.

17 Comorbid medical problems, such as sleep disruption and growth suppression, continue to be

18 logy with both non-rapid eye movement (NREM) sleep disruption and memory impairment in older adults.

19 latonin supplementation for the treatment of sleep disruption and other neurological diseases such as

20 topic dermatitis (AD) experience significant sleep disruption, and clinically, the disease is noted t

21 arly-onset patients had more energy, minimal sleep disruption, and greater suicidality, while typical

22 Inability to communicate, sleep disruption, and limitations on visiting were parti

23 han 40 million Americans suffer from chronic sleep disruption, and the development of effective treat

24 han 40 million Americans suffer from chronic sleep disruption, and the development of effective treat

25 Sleep disruption appears to be a core component of Alzhe

26 with impaired NREM SWA, these data implicate sleep disruption as a mechanistic pathway through which

27 ii) the potential diagnostic utility of NREM sleep disruption as a new biomarker of AD; and (iv) the

28 ruption, Abeta, and AD; (ii) a role for NREM sleep disruption as a novel factor linking cortical Abet

29 ed in many atopic diseases that can underlie sleep disruptions as a consequence of the presence of no

30 Clinicians need to consider that the chronic sleep disruption associated with nightmares may affect t

31 adequate interventions to prevent and treat sleep disruption because of their high relevance to our

32 survivors suffering from moderate or greater sleep disruption between 2 and 24 months after surgery,

33 mproved with sleeping drugs, suggesting that sleep disruption contributes to their neurological decli

34 Nocturnal sleep disruption develops in Alzheimer disease (AD) owin

35 quality of life caused by intense pruritus, sleep disruption, dietary and nutritional concerns, and

36 tion and those with more negative affect and sleep disruption during marijuana withdrawal were more l

37 r the functional consequences of age-related sleep disruption, focusing on memory impairment as an ex

38 the MICU appeared to be more susceptible to sleep disruptions from environmental factors than patien

39 Sleep disruption has deleterious effects, even in normal

40 rat model is associated with large long-term sleep disruption, however, the vehicle, DMSO/PEG had as

41 with PTCHD1 deletion show symptoms of ADHD, sleep disruption, hypotonia, aggression, ASD, and ID.

42 urst; however, the effects of other forms of sleep disruption (i.e. spontaneous arousals and apnoea-i

43 ent factors associated with sound levels and sleep disruption in a range of representative ICUs.

44 ynchrony, a characteristic of shift work and sleep disruption in humans, also leads to metabolic path

45 sleep loss, we quantified a new procedure of sleep disruption in mice by a week of consecutive sleep

46 The etiology of sleep disruption in patients in intensive care units (IC

47 nderstanding the neural mechanism underlying sleep disruption in response to environmental perturbati

48 Such findings suggest that sleep disruption in the elderly, mediated by structural

49 nts; and (2) the environmental etiologies of sleep disruption in the ICU are multifactorial.

50 zure activity, rapid sleep onset and reduced sleep disruption in the other recording.

51 r understanding of the mechanisms underlying sleep disruptions in AS.

52 exposed to high sound levels and substantial sleep disruption irrespective of factors including previ

53 Sleep disruption is a core feature of PTSD supporting th

54 ENT In the light of increasing evidence that sleep disruption is crucially involved in the progressio

55 Sleep disruption is prevalent in patients with cancer an

56 er emerging studies suggest that age-related sleep disruption may be one key factor that renders the

57 a support a neuropathological model in which sleep disruption may contribute to the maintenance and/o

58 ing that like many other stressors, extended sleep disruption may lead to a state of sustained microg

59 s in circadian rhythms may contribute to the sleep disruption observed in older adults.

60 Circadian rhythm and sleep disruptions occur frequently in individuals with a

61 rimental studies of these different forms of sleep disruption show deranged physiology from subcellul

62 dation is more susceptible to the effects of sleep disruption than is the acquisition (learning) of s

63 may affect the degree of hypoxic stress and sleep disruption that occurs in response to upper airway

64 Though stress causes complex sleep disruptions that are different in females and male