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1 eep across nights and a shorter baseline REM sleep latency.
2 receptor, Rdl(A302S), specifically decreases sleep latency.
3 netics of GABA(A) receptor signaling dictate sleep latency.
4 leep apnea was demonstrated only for reduced sleep latency.
5 tonin synthesis, pupillary light reflex, and sleep latency.
6 ion was seen between Epworth scores and mean sleep latency.
7 uration and intensity of NREMS and prolonged sleep latency.
8 egions, increased sleep times, and shortened sleep latencies.
9 normal sleep latency (>10 minutes), moderate sleep latency (5 to 10 minutes), or severe sleep latency
10 mokers, current smokers had a longer initial sleep latency (5.4 minutes, 95% confidence interval (CI)
14 In the phase II study, tasimelteon reduced sleep latency and increased sleep efficiency compared wi
16 with placebo, modafinil decreased nighttime sleep latency and increased slow-wave sleep time in coca
18 s disrupted by methamphetamine by decreasing sleep latency and increasing sleep efficiency compared w
19 flies are resistant to the effects of CBZ on sleep latency and that mutant RDL(A302S) channels are re
20 zed by decreased total sleep time, increased sleep latency, and decreased sleep efficiency, without d
21 ariables-male sex, sleepiness, nocturnal REM sleep latency, and extent of oxygen desaturation-could r
22 period significantly reduced REM, shortened sleep latency, and increased EEG delta power in rats.
23 duction in plasma tryptophan concentrations, sleep latency, and REM latency, as well as increased REM
24 thanol microinjections significantly reduced sleep latency, and tended (P<0.06) to increase total sle
25 imination, delayed gastric emptying, altered sleep latency, anxiety-like behavior, and age-dependent
26 piness Scale), objective sleepiness (reduced sleep latency as determined by the Multiple Sleep Latenc
27 groups of patients who were defined by mean sleep latency as having normal sleep latency (>10 minute
29 afinil was associated with increased daytime sleep latency, as measured by the Multiple Sleep Latency
30 ystemic zolpidem administration also reduced sleep latency, but less so than for histamine neurons.
31 phically assessed (upper or lower quartiles) sleep latency, continuity, and duration (RRs = 2.2-4.7;
32 stress-induced insomnia in humans: increased sleep latency, decreased non-REM (nREM) and REM sleep, i
33 These experiments uncouple the regulation of sleep latency from that of sleep duration and suggest th
34 fined by mean sleep latency as having normal sleep latency (>10 minutes), moderate sleep latency (5 t
35 slow-wave sleep time, total sleep time, and sleep latency in cocaine-dependent and healthy participa
40 ore and after sleep onset, such as prolonged sleep latency, loss of stage 3-4 sleep, reduced rapid ey
42 e sleep latency (5 to 10 minutes), or severe sleep latency (<5 minutes) (analysis of variance, P = 0.
44 cy, sleep onset and rapid eye movement [REM] sleep latencies, non-REM and REM sleep stages, and wakef
45 hted effect sizes for subjective measures of sleep latency, number of awakenings, wake time after sle
47 ), a 5-min decrease in the MSLT-derived mean sleep latency (OR = 1.9, 95% CI = 1.3 to 2.8), a 90-min
48 s with Parkinson disease exhibited increased sleep latency (P = .04), reduced sleep efficiency (P = .
50 ion was seen between Epworth scores and mean sleep latency (Pearson correlation, -0.17 [95% CI, -0.35
51 In many patients, a short rapid eye movement sleep latency (REML) during the NPSG is also observed bu
52 n ICU patients is characterized by prolonged sleep latencies, sleep fragmentation, decreased sleep ef
53 In the phase III study, tasimelteon improved sleep latency, sleep efficiency, and wake after sleep on
54 rsomnolence was quantified with the multiple sleep latency test (MSLT) and survival analysis was used
55 deficiency, is diagnosed using the Multiple Sleep Latency Test (MSLT) following nocturnal polysomnog
56 ent (REM) periods (2omSOREMPs) on a Multiple Sleep Latency Test (MSLT) raise the possibility of narco
58 ts with cataplexy but with a normal multiple sleep latency test (MSLT) result, or if MSLT is not inte
60 tive sleepiness was assessed by the Multiple Sleep Latency Test in all subjects, problem sleepiness b
61 Sleepiness Scale and the objective multiple sleep latency test may evaluate different, complementary
63 sleep latency as determined by the Multiple Sleep Latency Test), and neurobehavioral functioning (la
64 rements included polysomnography, a multiple sleep latency test, an oral glucose tolerance test, dete
65 e sleep latency, as measured by the Multiple Sleep Latency Test, and a nearly significant decrease in
66 tory tests such as polysomnography, multiple sleep latency test, and actigraphy, along with referral
68 ormed with the use of the nighttime Multiple Sleep Latency Test, the Clinical Global Impression of Ch
74 me, 2 nights of polysomnography and multiple sleep latency tests in the laboratory, and body composit
75 age change group showed significantly longer sleep latencies than the gentle handling group, indicati
77 ent from baseline in mean (+/-SEM) nighttime sleep latency (the interval between the time a person at
78 oring and sleep logs to measure time in bed, sleep latency (time required to fall asleep), sleep dura
80 sleep, and with increases in latency to REM sleep, latency to persistent sleep, and percent Stage 3/
83 5 (standard deviation (SD), 1.2) hours, mean sleep latency was 21.9 (SD, 29.0) minutes, mean sleep du
87 e the onset of melatonin secretion and short sleep latencies were observed close to the temperature n
88 eye movement (REM) sleep and shortened NREM sleep latency with a simultaneous decrease in core tempe
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