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1 , and associates with EEG differences during sleep loss.
2 siological sleepiness in response to chronic sleep loss.
3 out the cellular adaptations that occur with sleep loss.
4 eep and learning impairments associated with sleep loss.
5 campus-related cognitive deficits induced by sleep loss.
6 may be involved in regulating sensitivity to sleep loss.
7 prevented cognitive deficits associated with sleep loss.
8 ial feature of performance impairment due to sleep loss.
9 on of psychomotor vigilance as a function of sleep loss.
10 ndicative of a homeostatic response to acute sleep loss.
11 n the brain and body of animals experiencing sleep loss.
12 generalize to conditions of chronic partial sleep loss.
13 dicate that the hippocampus is vulnerable to sleep loss.
14 reduced expression of heat-shock genes after sleep loss.
15 es compensatory REM sleep in response to REM sleep loss.
16 is associated with intermittent hypoxia and sleep loss.
17 ntain daytime sleep in the face of nighttime sleep loss.
18 to maintain normal blood sugar levels during sleep loss.
19 ic dysfunction, and potential biomarkers for sleep loss.
20 occurs regardless of the approach to achieve sleep loss.
21 uscle Bmal1 reduced the recovery response to sleep loss.
22 The hippocampus is particularly sensitive to sleep loss.
23 etes risk and inflammation, independently of sleep loss.
24 reased, indicating a homeostatic response to sleep loss.
25 in sleep timing and homeostatic responses to sleep loss.
26 nd memory deficits comparable to those after sleep loss.
27 might be causal for individual responses to sleep loss.
28 higher level of arousal despite the similar sleep loss.
29 d differently in males and females following sleep loss.
30 rom it may require more time than from acute sleep loss.
31 multiplicative effect on performance during sleep loss.
33 c adverse metabolic effects independently of sleep loss, a parallel group design was used to study 26
35 ast, chronic sleep restriction but not acute sleep loss activates microglia, promotes their phagocyti
37 addition, experimental studies suggest that sleep loss alters cerebrospinal fluid Abeta dynamics, de
47 r to promptly curtail the chronic effects of sleep loss and effectively screen for underlying, potent
48 on on which to consider interactions between sleep loss and emotional reactivity in a variety of clin
51 on of this circuit in young flies results in sleep loss and lasting deficits in adult courtship behav
53 mework for a positive feedback loop, whereby sleep loss and neuronal excitation accelerate the accumu
55 ation (PSD), a condition distinct from total sleep loss and one experienced by millions on a daily an
56 es in cognitive performance and sleep during sleep loss and recovery, as well as a new approach for p
57 omnography was performed to measure baseline sleep loss and responses to isoflurane anesthesia at 1%
58 nt with a body of literature suggesting that sleep loss and sleep fragmentation are associated with b
59 miological studies have shown a link between sleep loss and the obesity 'epidemic,' and several obser
61 exposed to the shifting schedule revealed no sleep loss, and stress measures were not altered in shif
64 multifaceted view on cerebral correlates of sleep loss at night and propose that genetic predisposit
67 nd recovery differ between acute and chronic sleep loss, but the physiological basis for these time c
68 tal animal and human studies have found that sleep loss can impair metabolic control and body weight
73 al variability exists in the degree to which sleep loss compromises learning, the mechanistic reasons
75 different lights as a countermeasure against sleep-loss decrements in alertness, melatonin and cortis
76 ow-frequency brain electrical activity after sleep loss demonstrate that sleep need is homeostaticall
78 more complete understanding of the issues of sleep loss during residency training can inform innovati
79 in A1AR availability were more resilient to sleep-loss effects than those with a subtle increase.
82 le of a single species with a convergence on sleep loss exhibited by several independently evolved po
83 ictors of individual responses to subsequent sleep loss exposures chronically or intermittently, acro
85 To date it is not known whether sleep or sleep loss has any effect on proliferation of cells in t
88 die from one form of sleep deprivation, but sleep loss has not been shown to cause death in well-con
91 However, society-specific consequences of sleep loss have rarely been explored, and no function of
92 ive and executive functioning-resulting from sleep loss in a healthy, racially-diverse adult populati
95 studies are warranted to better characterize sleep loss in eczema and develop strategies for treatmen
96 ep regulation to quantify performance during sleep loss in the absence of caffeine and a dose-depende
97 tudies in humans and rodents also found that sleep loss increases peripheral markers of inflammation,
99 d the degree of such neural vulnerability to sleep loss: individuals with highest trait anxiety showe
100 mechanisms for the observed changes comprise sleep loss-induced changes in appetite-signaling hormone
101 myo-inositol and glycine levels, suggesting sleep loss-induced modifications downstream of mGluR5 si
102 estigated potential underlying mechanisms of sleep-loss-induced changes in behavior by high-density e
104 PER3 predicts individual differences in the sleep-loss-induced decrement in performance and that thi
113 Even though the overall consensus is that sleep loss leads to metabolic perturbations promoting th
115 ed on-call workload was associated with more sleep loss, longer shift duration, and a lower likelihoo
117 These findings demonstrate that sleep and sleep loss modify experience-dependent cortical plastici
118 It is not yet clear how acute and chronic sleep loss modify neuronal activities and lead to adapti
119 ty, longer episode duration, less subjective sleep loss, more guilt, and more work/activity impairmen
122 ruption and anxiety disorders, the impact of sleep loss on affective anticipatory brain mechanisms, a
123 way may underlie both the adverse effects of sleep loss on cognition and the subsequent changes in co
124 e reviewed studies addressing the effects of sleep loss on cognition, performance, and health in surg
125 he negative effects of shiftwork and chronic sleep loss on health and productivity are now being appr
126 may be involved with the negative effects of sleep loss on health, and highlight the interrelatedness
128 omeostatic sleep response and the effects of sleep loss on memory, previous studies have not determin
131 leep and affective regulation, the impact of sleep loss on the discrimination of complex social emoti
133 conclude that circadian disruption, but not sleep loss or stress, are associated with jet lag-relate
134 ble from the energy-balance perturbations of sleep loss or the potentially stressful effects of the f
135 However, during sleep restriction (partial sleep loss) performance predictions based on such models
137 lesion size were associated with cumulative sleep loss (r=0.77 and r=0.62, respectively), and cumula
142 ults may also provide insight for predicting sleep loss responses in patients with schizophrenia and
144 use and effect association are still scarce, sleep loss seems to be an appealing target for the preve
146 understand the association between workload, sleep loss, shift duration, and the educational time of
147 correlations in neurobehavioral responses to sleep loss suggest that these trait-like differences are
148 sed to a similar extent after short and long sleep loss, suggesting that astrocytic phagocytosis may
149 nificant increase after prolonged and severe sleep loss, suggesting that it may promote the housekeep
150 beta' GABAA and GABABR3 receptors results in sleep loss, suggesting these receptors are the sleep-rel
151 tes these EB neurons are highly sensitive to sleep loss, switching from spiking to burst-firing modes
153 ay be more susceptible to weight gain during sleep loss than women due to a larger increase in daily
154 y insult, these results suggest that chronic sleep loss, through microglia priming, may predispose th
155 ind that increased mGluR5 availability after sleep loss tightly correlates with behavioral and electr
157 mechanisms linking circadian dysfunction and sleep loss to neurodegenerative diseases, with a focus o
158 ment of cognitive performance in response to sleep loss was significantly greater in the PER3(5/5) in
159 77 and r=0.62, respectively), and cumulative sleep loss was the strongest predictor of high sensitivi
160 e indeed costs associated with resiliency to sleep loss, we challenged natural allelic variants of th
161 ve CSR that mimics a common pattern of human sleep loss, we quantified a new procedure of sleep disru
162 nd sufficient to suppress starvation-induced sleep loss when animals encounter nutrient-poor food sou
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