ライフサイエンスコーパス: sleep-disordered breathing

1 CIH-induced neuropathology in patients with sleep disordered breathing.

2 le lung disease, pulmonary hypertension, and sleep disordered breathing.

3 n the predominant abnormality leading to the sleep-disordered breathing.

4 lt patients evaluated by polysomnography for sleep-disordered breathing.

5 c respiratory cycles in adults evaluated for sleep-disordered breathing.

6 al impairments observed in a rodent model of sleep-disordered breathing.

7 uld have a role in preventing or alleviating sleep-disordered breathing.

8 desaturation (DeltaSa(O(2))) associated with sleep-disordered breathing.

9 ic obstructive pulmonary disease, asthma and sleep-disordered breathing.

10 diovascular risk factor levels in those with sleep-disordered breathing.

11 ace, we studied the effect of spaceflight on sleep-disordered breathing.

12 uality during spaceflight is not degraded by sleep-disordered breathing.

13 nography is invaluable for the evaluation of sleep-disordered breathing.

14 specific sleep-related parameters, including sleep-disordered breathing.

15 is hypothesis in a group of subjects without sleep-disordered breathing.

16 resence or absence of insulin resistance and sleep-disordered breathing.

17 who have clinically significant REM-specific sleep-disordered breathing.

18 s that insulin resistance is associated with sleep-disordered breathing.

19 me individuals may partially protect against sleep-disordered breathing.

20 When abnormal, these interactions lead to sleep-disordered breathing.

21 effects of oral antihistamines on asthma and sleep-disordered breathing.

22 omnography studies to assess the presence of sleep-disordered breathing.

23 malities in children with SCD are related to sleep-disordered breathing.

24 al importance to the patients suffering from sleep-disordered breathing.

25 sitive pressure ventilation in children with sleep-disordered breathing.

26 s is higher among subjects with than without sleep-disordered breathing.

27 eful second-line treatment for children with sleep-disordered breathing.

28 rdered breathing compared with those without sleep-disordered breathing: 4.8 versus 0.9% (p=0.003) fo

29 entral apnea, Cheyne-Stokes respiration, and sleep-disordered breathing-age interaction terms were si

30 ongest in older participants in whom overall sleep-disordered breathing also increased atrial fibrill

31 implicated, including apnoea of prematurity, sleep disordered breathing and congestive heart failure.

32 urbations during sleep (e.g. those caused by sleep disordered breathing and periodic leg movements) m

33 ined the cross-sectional association between sleep- disordered breathing and self-reported CVD in 6,4

34 Compared with those without sleep-disordered breathing and adjusting for age, sex, b

35 events, we aimed to assess the prevalence of sleep-disordered breathing and associated clinical featu

36 Although research supports a sleep-disordered breathing and atrial fibrillation assoc

37 Associations between sleep-disordered breathing and cardiovascular disease (C

38 ights the complex interrelationships between sleep-disordered breathing and cardiovascular disease, p

39 Obese females are less predisposed to sleep-disordered breathing and have higher serum leptin

40 e directionality of the relationship between sleep-disordered breathing and heart failure is controve

41 the association between objectively measured sleep-disordered breathing and hypertension (defined as

42 born preterm exhibit increased incidence of sleep-disordered breathing and hypertension, suggesting

43 ontinuous positive airway pressure (CPAP) on sleep-disordered breathing and its consequences in heart

44 more in-depth discussion of indications for sleep-disordered breathing and recurrent throat infectio

45 gnificant relation was also observed between sleep-disordered breathing and ventricular ectopic beats

46 Obesity is a strong causal factor for sleep-disordered breathing, and because of the ongoing o

47 trategies for management of hypoventilation, sleep-disordered breathing, and cough insufficiency are

48 d glycation endpoints, autonomic neuropathy, sleep-disordered breathing, and genetic susceptibility t

49 icity and alcohol's contribution to obesity, sleep-disordered breathing, and hypertension.

50 re, specific exercise, opioids, treatment of sleep-disordered breathing, and interventions to address

51 The probability of moderate-to-severe sleep-disordered breathing (apnea-hypopnea index >/=15%)

52 The prevalence of sleep-disordered breathing (apnea-hypopnea index of 15 o

53 isk factors for excessive sleepiness: severe sleep-disordered breathing (apnea-hypopnea index, >30 ep

54 t prevalence estimates of moderate to severe sleep-disordered breathing (apnea-hypopnea index, measur

55 overnight by 18-channel polysomnography for sleep-disordered breathing, as defined by the apnea-hypo

56 The primary outcome was prevalence of sleep-disordered breathing, assessed by the apnoea-hypop

57 We found a dose-response association between sleep-disordered breathing at base line and the presence

58 ible predisposition of the pregnant woman to sleep-disordered breathing because of these changes, and

59 We analyzed data on sleep-disordered breathing, blood pressure, habitus, and

60 ent in HFpEF patients, but renal disease and sleep-disordered breathing burdens are similar.

61 opause is considered to be a risk factor for sleep-disordered breathing, but this hypothesis has not

62 Obstructive sleep disordered breathing can cause death and significa

63 Central sleep-disordered breathing can occur with associated car

64 on, a standard-of-care management option for sleep-disordered breathing, can itself trigger specific

65 s, dyslipidemia, obstructive sleep apnea and sleep-disordered breathing, certain cancers, and major c

66 Sleep-disordered breathing (characterized by recurrent a

67 Among older women, those with sleep-disordered breathing compared with those without s

68 drigeminy) were more common in subjects with sleep-disordered breathing compared with those without s

69 Sleep-disordered breathing contributes to cardiac chambe

70 The prevalence of sleep-disordered breathing, depending on the apnea-hypop

71 articipants who had objective assessments of sleep-disordered breathing during pregnancy were asked t

72 Prospective data suggest that sleep-disordered breathing enhances risk for incident an

73 The frequency of sleep-disordered breathing episodes remained high at 0 a

74 ds of complex arrhythmias than those without sleep-disordered breathing even after adjustment for pot

75 dered breathing, can itself trigger specific sleep-disordered breathing events including air leaks, p

76 airway resistance can increase the risk for sleep-disordered breathing events.

77 relationship between insulin resistance and sleep-disordered breathing for potential confounding var

78 ry artery disease, congestive heart failure, sleep-disordered breathing, gastro-oesophageal reflux di

79 The prevalence of moderate-to-severe sleep-disordered breathing (>/=15 events per h) was 23.4

80 rdered breathing compared with those without sleep-disordered breathing had an increased risk of deve

81 ent coronary heart disease, individuals with sleep-disordered breathing had four times the odds of at

82 Sleep-disordered breathing has also been linked to these

83 thway whereby incident CVD causes or worsens sleep-disordered breathing has not been studied.

84 The prevalence of sleep-disordered breathing has not been well studied in

85 Common polysomnographic measures of sleep-disordered breathing have shown a disappointing ab

86 Individuals with severe sleep-disordered breathing have two- to fourfold higher

87 have also highlighted the manifestations of sleep disordered breathing in children with sickle cell

88 compensation suggesting that exacerbation of sleep disordered breathing in REM (compared to NREM) sle

89 between the use of replacement hormones and sleep-disordered breathing in a sample of 2,852 noninsti

90 lic consequences and community prevalence of sleep-disordered breathing in mildly obese, but otherwis

91 Sleep-disordered breathing in neuromuscular diseases is

92 e in addressing pitfalls in the diagnosis of sleep-disordered breathing in neuromuscular diseases, id

93 changes, and results of published studies of sleep-disordered breathing in pregnancy are discussed.

94 We estimated the prevalence of sleep-disordered breathing in the United States for the

95 for sleep duration, sleep fragmentation, and sleep-disordered breathing) in the development of cognit

96 m the brain, and hypoxemia characteristic of sleep-disordered breathing increases Abeta production.

97 iated with an increased likelihood of having sleep-disordered breathing, independent of known confoun

98 In addition, sleep disordered breathing, inflammation, left ventricul

99 Sleep-disordered breathing is a common disorder with a r

100 Sleep-disordered breathing is a prevalent condition asso

101 Thus, sleep-disordered breathing is a prevalent condition in m

102 Sleep-disordered breathing is also more common in patien

103 Sleep-disordered breathing is associated with an increas

104 A significant portion of sleep-disordered breathing is associated with ApoE epsil

105 Sleep-disordered breathing is associated with major morb

106 Sleep-disordered breathing is correlated with RMS and th

107 Sleep-disordered breathing is highly prevalent in elderl

108 These results suggest that sleep-disordered breathing is highly prevalent, with imp

109 The findings suggest that sleep-disordered breathing is likely to be a risk factor

110 Nevertheless, sleep-disordered breathing is often unrecognized in chil

111 Sleep-disordered breathing is prevalent in the general p

112 If sleep-disordered breathing is shown in future follow-up

113 intermittent hypoxia (IH), such as occurs in sleep-disordered breathing, is associated with neurobeha

114 reathing, the current evidence suggests that sleep-disordered breathing may function as a risk factor

115 sis was limited to those 27 patients who had sleep-disordered breathing (more than 5 apneas or hypopn

116 Sleep-disordered breathing occurs after surgery even in

117 Sleep-disordered breathing occurs in Alzheimer disease p

118 ompatible with modest to moderate effects of sleep-disordered breathing on heterogeneous manifestatio

119 lectively studied in populations at risk for sleep-disordered breathing or cardiovascular diseases.

120 None had symptoms of sleep-disordered breathing or conditions that impacted o

121 ent studies show either absence of change in sleep-disordered breathing or improved sleep cardiovascu

122 , incident CVD was associated with worsening sleep-disordered breathing over 5 years.

123 Sleep-disordered breathing, particularly the obstructive

124 gnition; however, it remains unclear whether sleep-disordered breathing precedes cognitive impairment

125 tion association, prospective data examining sleep-disordered breathing predicting incident atrial fi

126 oing obesity epidemic, previous estimates of sleep-disordered breathing prevalence require updating.

127 The high prevalence of sleep-disordered breathing recorded in our population-ba

128 owever, its indication for all patients with sleep-disordered breathing, regardless of daytime sympto

129 , and body mass index: (1) 228 subjects with sleep-disordered breathing (respiratory disturbance inde

130 ce index>or=30) and (2) 338 subjects without sleep-disordered breathing (respiratory disturbance inde

131 Although most children with sleep-disordered breathing respond to surgical treatment

132 We propose that sleep-disordered breathing results from loss of preBotC

133 tent hypoxia during sleep (IH), as occurs in sleep disordered breathing (SDB), induces spatial learni

134 ing evidence suggests an association between sleep-disordered breathing (SDB) and cognitive decline i

135 of the insertion/deletion polymorphism with sleep-disordered breathing (SDB) and hypertension in 1,1

136 tional association has been reported between sleep-disordered breathing (SDB) and insulin resistance,

137 Increasing recognition of sleep-disordered breathing (SDB) and its morbidity have

138 Sleep-disordered breathing (SDB) and sleep apnea have be

139 Sleep-disordered breathing (SDB) has been associated wit

140 Sleep-disordered breathing (SDB) has been noted commonly

141 Trazodone with L-tryptophan can treat sleep-disordered breathing (SDB) in an animal model of O

142 This study examined risk factors for sleep-disordered breathing (SDB) in children and adolesc

143 Sleep-disordered breathing (SDB) in children is associat

144 haracterization of anatomic risk factors for sleep-disordered breathing (SDB) in children.

145 Sleep-disordered breathing (SDB) is a common disorder in

146 Whether sleep-disordered breathing (SDB) is a risk factor for le

147 There is increasing evidence that sleep-disordered breathing (SDB) is an independent risk

148 Sleep-disordered breathing (SDB) is associated with dayt

149 Clinic-based studies suggest that sleep-disordered breathing (SDB) is associated with gluc

150 Sleep-disordered breathing (SDB) is associated with hype

151 Sleep-disordered breathing (SDB) is associated with path

152 Sleep-disordered breathing (SDB) is both prevalent and a

153 Sleep-disordered breathing (SDB) is common in patients w

154 ng of intrinsic information in children with sleep-disordered breathing (SDB) is different from healt

155 Yet, sleep-disordered breathing (SDB) is highly prevalent in

156 xcessive daytime sleepiness in patients with sleep-disordered breathing (SDB) is not well defined.

157 Prevalence of sleep-disordered breathing (SDB) is reported to increase

158 Most polysomnograms are performed because sleep-disordered breathing (SDB) is suspected, but perio

159 We assessed the extent to which sleep-disordered breathing (SDB) may explain association

160 The effect of sleep-disordered breathing (SDB) on right heart structur

161 ol abnormalities in predisposing to familial sleep-disordered breathing (SDB) was assessed in 31 subj

162 Genetic determinants of sleep-disordered breathing (SDB), a common set of disord

163 ss body weight is positively associated with sleep-disordered breathing (SDB), a prevalent condition

164 sibility (UAC) is increased in children with sleep-disordered breathing (SDB), but during wakefulness

165 In this case-control family study of sleep-disordered breathing (SDB), we describe the distri

166 however, the relation of sleepiness to mild sleep-disordered breathing (SDB), which affects as much

167 Children with mild sleep-disordered breathing (SDB), who may not be recomme

168 ronic sleepwalkers frequently presented with sleep-disordered breathing (SDB).

169 d to discrepant estimates of the severity of sleep-disordered breathing (SDB).

170 and functional status in subjects with mild sleep-disordered breathing (SDB).

171 en linked to disrupted sleep associated with sleep-disordered breathing (SDB).

172 ial triggers for arrhythmia in patients with sleep-disordered breathing (SDB).

173 Evaluation for sleep-disordered breathing should be a priority for meno

174 The relationship of sleep-disordered breathing (SOB) to neuropsychological d

175 Compared with the 193 women without sleep-disordered breathing, the 105 women (35.2%) with s

176 though stroke can lead to the development of sleep-disordered breathing, the current evidence suggest

177 y cross-sectional area in an animal model of sleep-disordered breathing, the English bulldog.

178 Cross-sectional studies have linked sleep-disordered breathing to poor cognition; however, i

179 thing in neuromuscular diseases, identifying sleep-disordered breathing triggered by noninvasive vent

180 OSA, and 16 healthy control subjects in whom sleep disordered breathing was excluded by complete over

181 Sleep-disordered breathing was ascertained by apnea-hypo

182 Sleep-disordered breathing was associated more strongly

183 Sleep-disordered breathing was defined as an apnea-hypop

184 Sleep-disordered breathing was defined by an AHI of at l

185 inverse association between hormone use and sleep-disordered breathing was evident in various subgro

186 Sleep-disordered breathing was indicated by the frequenc

187 pause, perimenopause, and postmenopause with sleep-disordered breathing was investigated with a popul

188 The prevalence of sleep-disordered breathing was modeled as a function of

189 Sleep-disordered breathing was not associated with globa

190 Determination of sleep-disordered breathing was performed through clinica

191 Sleep-disordered breathing was quantified by the apnea-h

192 Sleep-disordered breathing was quantified by the respira

193 time of the sleep study, moderate levels of sleep-disordered breathing were common, with a median Re

194 rdered breathing, the 105 women (35.2%) with sleep-disordered breathing were more likely to develop m

195 To reduce the confounding influence of sleep-disordered breathing, which is related to both inc

196 ted with cardiorespiratory diseases, such as sleep-disordered breathing with apnoea, congestive heart

197 to determine the independent association of sleep-disordered breathing with risk of mild cognitive i