ライフサイエンスコーパス: slit

1 tered subcutaneously (SCIT) or sublingually (SLIT).

2 le with nonlocal interactions with the other slit.

3 -based diffuser and postarray-based dialysis slit.

4 y rescue drugs, accounted for 26.3% of total SLIT.

5 % compared to the enhancement in an isolated slit.

6 challenge (DBPCFC) after 12 months of peanut SLIT.

7 ed for subsequent axonal repulsion away from Slit.

8 ctive SLIT/placebo OIT or active OIT/placebo SLIT.

9 a placebo-controlled trial of peanut OIT and SLIT.

10 cts could be a factor affecting dropout from SLIT.

11 moved behind a narrow vertical or horizontal slit.

12 on, when an object is moving behind a narrow slit.

13 slits, with absorptive sidewalls between the slits.

14 ce modes and the resonance of the individual slits.

15 a conventional characterization using double slits.

16 ted both pro- and anti-angiogenic effects of Slits.

17 n axon guidance protein, either by targeting slit-1 mRNA or, potentially, by modulating the canonical

18 Additionally, miR-124 regulates Smed-slit-1, which encodes an axon guidance protein, either b

19 Following 12 months of SLIT, 10 subjects (30%) passed the DBPCFC without sympto

20 ilopodia form and elongate toward sources of Slit, a response that we find is required for subsequent

21 Continuous rate of SLIT achieved about 90%, suggesting relatively high leve

22 is in the Drosophila ventral nerve cord of a slit allele (slit-UC) that cannot be cleaved revealed th

23 ion over time and, in case of the horizontal slit, also across visual hemifields.

24 After applying all selection criteria, 2851 SLIT and 71 275 control patients were selected for the s

25 ine the component-specific effects of peanut SLIT and determine whether peanut component testing coul

26 The axonal wiring molecule Slit and its Round-About (Robo) receptors are conserved

27 Robo-Slit and Plexin-Semaphorin signaling participate in vari

28 nditional knockout mice deficient in various Slit and Robo proteins and found that Slit2 potently and

29 We investigated the role of Slit and Robo receptors in wiring Drosophila higher-orde

30 d receptor knockout mice shows that PlexinA1-Slit and Robo-Slit signaling have complementary roles du

31 s and meta-analyses have confirmed that both SLIT and SCIT are effective in patients with seasonal AR

32 odocyte, accompanied by a tighter filtration slit and the appearance of TJ-like structures between th

33 Strong dispersion is produced in the slits and slow sound conditions are induced.

34 nce suggested that sublingual immunotherapy (SLIT) and subcutaneous immunotherapy (SCIT) would be con

35 Rhinitis symptoms were lower in the SCIT, SLIT, and SLIT-TOL groups (P < .001) compared with those

36 and in genetic mouse models have identified Slit- and NTRK-like family (Slitrks) as candidate genes

37 ovement over transmission without a metallic slit array.

38 Subwavelength metallic slit arrays have been shown to exhibit extraordinary opt

39 sensitive schemes for THz spectroscopy with slit arrays manufactured by standard UV photolithography

40 along with robust support of pharyngeal gill slits as a shared deuterostome character, provide the fo

41 We started SLIT at our clinic in October-December, 2014.

42 We started SLIT at our clinic in October-December, 2014.

43 Here we appraise evidence for SCIT versus SLIT based on indirect evidence from Cochrane reviews an

44 Cells were also counted in magnified slit beam photographs masked to molecular diagnosis when

45 are rigidly backed slotted panels, with each slit being loaded by an array of Helmholtz resonators.

46 We recruited 38 patients who began SLIT between November 2014 and September 2015.

47 Two orthogonal sets of slits (bigrating) allows this narrow-band effect to be b

48 am of the podocyte glycocalyx that spans the slit, but none are observed upstream of the slit diaphra

49 s study is to clear the clinical efficacy of SLIT by comparing with other therapies, such as subcutan

50 ture responsiveness to the midline repellant Slit by expressing the endosomal sorting receptor Commis

51 o and dropout from sublingual immunotherapy (SLIT) by verifying patient backgrounds 1 year after star

52 Fabry-Perot resonance supported inside each slit can be spectrally shifted across the working wavele

53 e plasmonic ribbons situated inside metallic slits can efficiently block the coupling channel for res

54 After SLIT cessation, asthma medication use fell by an additio

55 dult trials can be used to improve pediatric SLIT clinical development.

56 second low affinity binding site in the Robo-Slit complex as well as suggesting the role of the Ig2 d

57 Dscam1 appears to modify the output of Robo/Slit complexes so that signaling is no longer repulsive.

58 were selectively impaired in the horizontal slit condition, indicating specific difficulties in long

59 domain and inhibits its activity; therefore, SLIT-dependent activation of RhoA is mediated by ROBO in

60 Moreover, when the same slit-dependent shape slivers were shuffled, thereby prev

61 is likely to mediate temporal integration of slit-dependent shape views, generating a slit-invariant

62 ver, there is no definitive evidence to show slit diaphragm (SD) to TJ transition in vivo Here, we re

63 e direct and systems-level evidence that the slit diaphragm and podocyte cytoskeleton are regulated t

64 preserved nephrin surface expression on the slit diaphragm and reduced proteinuria in diabetic mice,

65 n proteins nephrin and neph1 localize to the slit diaphragm and transduce signals in a Src family kin

66 cs, leading to aberrant nephrin turnover and slit diaphragm disassembly.

67 phrin and regulate podocyte cytoskeleton and slit diaphragm dynamics, MAGI2 mutations have not been d

68 ulates podocyte shape, structure, stability, slit diaphragm insertion, adhesion, plasticity, and dyna

69 gene Coq2, the silencing of which disrupted slit diaphragm morphology.

70 Expression of several key slit diaphragm protein was down regulated in pGR KO mice

71 g protein, essential for the assembly of the slit diaphragm protein-lipid supercomplex.

72 in intracellular movements of this critical slit diaphragm protein.

73 ces Ca(2+) influx, oxidative stress, loss of slit diaphragm proteins, and apoptosis.

74 Here, we discovered that two Drosophila slit diaphragm proteins, orthologs of the human genes en

75 d PHB2 at mitochondrial membranes and at the slit diaphragm, a specialized cell junction at the filtr

76 s a key structural component of the podocyte slit diaphragm, and proper expression of nephrin on the

77 luding effacement and disorganization of the slit diaphragm, followed by foot process disappearance,

78 a specialized cell junction at the podocyte slit diaphragm, MEC-2 is found in neurons required for t

79 t determinant of the structural integrity of slit diaphragm, which is a critical component of kidney'

80 tions is a unique cell junction known as the slit diaphragm, which is physically connected to the act

81 ation apparatus in mice lacking the critical slit diaphragm-associated protein CD2AP, highlighting th

82 treatment led to decreased expression of the slit diaphragm-associated proteins podocin, nephrin, and

83 ecific protein, is the main component of the slit diaphragm.

84 nd decreased expression of components of the slit diaphragm.

85 and the podocyte intercellular junction, or slit diaphragm.

86 need to invoke direct size selection by the slit diaphragm.

87 slit, but none are observed upstream of the slit diaphragm.

88 of the Pals1 ortholog caused alterations in slit-diaphragm-like structures.

89 re previously shown to result in loss of the slit diaphragms of the podocytes, leading to the hypothe

90 acunar channels and effacement of nephrocyte slit diaphragms.

91 to an interference effect analogous to multi-slit diffraction.

92 nd integral field spectroscopy with numerous slit dispersive paths, has no moving parts and provides

93 SLIT dose-finding studies for other pollens might start

94 Second, we detect no midline localization of Slit during brain development.

95 e SLIT plays a role in tumor suppression, as SLIT-encoding genes are inactivated in several types of

96 Mechanistically, Slit evokes changes in filopodium dynamics by increasing

97 The atomically flat angstrom-scale slits exhibit little surface charge, allowing elucidatio

98 Feynman stated that the double-slit experiment "...has in it the heart of quantum mecha

99 pproach by analyzing a version of the double-slit experiment augmented with postselection, showing th

100 that the intensity pattern formed in a three-slit experiment is seemingly in contradiction with the m

101 e demonstration of a two-dimensional Young's Slits experiment.

102 ges between children and adults treated with SLIT for allergic rhinoconjunctivitis.

103 OIT and SLIT for peanut allergy induce rapid suppression of baso

104 In this cohort of subjects undergoing SLIT for peanut allergy, lower baseline levels of IgE ag

105 OIT appeared far more effective than SLIT for the treatment of PA but was also associated wit

106 otherapy (OIT) and sublingual immunotherapy (SLIT) for food allergy hold promise; however, the immuno

107 se of the drug for sublingual immunotherapy (SLIT) for Japanese Cedar pollinosis (JCP).

108 se of the drug for sublingual immunotherapy (SLIT) for Japanese Cedar pollinosis (JCP).

109 eptor and found that it binds the C-terminal Slit fragment specifically and transduces a SlitC signal

110 he cellular and molecular mechanisms of Robo/Slit function.

111 ng lung cancer; however, it is not clear how SLIT functions in lung cancer.

112 Slit glycoproteins signaling through Roundabout (Robo) r

113 ture such as a one-dimensional (1D) metallic slit grating, these modes all exist and can potentially

114 es, FK506 and Elafin, was related to reduced slit guidance ligand 3 (SLIT3), an antimigratory factor.

115 standard, whereas sublingual immunotherapy (SLIT) has emerged as an effective and safe alternative.

116 be necessary to guarantee the quality of the SLIT-HDM products and to demonstrate their effectiveness

117 n-specific structure of the interendothelial slit (IES).

118 We reported the clinical efficacy of SLIT in the first and the second treated year.

119 y rescue drugs, accounted for 16.8% of total SLIT in the first follow-up year.

120 s study is to clear the clinical efficacy of SLIT in the second treated year by comparing with other

121 SLIT in the second treated year showed good clinical eff

122 s study is to clear the clinical efficacy of SLIT in the third treated year by comparing with other t

123 SLIT in the third treated year showed good clinical effi

124 mpared the clinical efficacy in 2017, of 112 SLIT in the third treated year with 38 SCIT, 364 primary

125 ever, genetic evidence supporting a role for Slits in ocular neovascularization is lacking.

126 ro-ellipse filters consisting of microfuidic slits in series gradually narrowed.

127 PRKL-1/Prickle containing PCP pathway and a Slit-independent SAX-3/Robo pathway cooperate to regulat

128 69D (RPTP69D) and loss of midline-localized Slit inhibit formation of specific axon collaterals thro

129 The slit-invariant representation of the various shapes also

130 We show that slit-invariant shape information is most accurate in the

131 preventing their spatiotemporal integration, slit-invariant shape information was reduced dramaticall

132 Importantly, the slit-invariant shape representations matched the convent

133 of slit-dependent shape views, generating a slit-invariant whole-shape percept.

134 Instead, Slit is enriched in the mushroom body, a neuronal struct

135 In vivo, Slit is proteolytically cleaved into N- and C-terminal f

136 Sublingual immunotherapy (SLIT) is a potential efficacious and safe treatment opti

137 al acuity, cycloplegic objective refraction, slit lamp as well as fundus examinations.

138 Visual outcomes, slit lamp biomicroscopy, intraocular pressure (IOP), and

139 Slit lamp examination revealed the presence of bilateral

140 ch visit includes (1) Clinical evaluation: a slit lamp examination, fundoscopy, intraocular pressure

141 CASE PRESENTATION: We evaluated slit lamp examination, fundoscopy, optical coherence tom

142 ternal ocular infections were examined under slit lamp microscope.

143 om the angiographic images and marked at the slit lamp using a needle to make a cut to the depth of t

144 sts were visual acuity, clinical evaluation (slit lamp), Amsler chart, color fundus photographs, infr

145 or and posterior segments examination with a slit-lamp and a direct ophthalmoscope respectively.

146 ereoacuity, refraction, clinical findings of slit-lamp and dilated fundus examinations.

147 e (IOP) measurement, and corneal pachymetry; slit-lamp and optic nerve examination; automated visual

148 e posterior hyaloid membrane observed during slit-lamp biomicroscopy after posterior vitreous detachm

149 e posterior hyaloid membrane observed during slit-lamp biomicroscopy in patients with posterior vitre

150 All corneas were examined by using slit-lamp biomicroscopy to determine the severity of FEC

151 f treatment, cumulative dose, Orlando stage (slit-lamp biomicroscopy), and serum concentrations of am

152 cted visual acuity recorded in LogMAR units, slit-lamp biomicroscopy, and optical coherence tomograph

153 After maximal mydriasis, slit-lamp biomicroscopy, and photography, imaging of the

154 ents had their ocular surface evaluated with slit-lamp biomicroscopy, and tear production quantified

155 Best-corrected visual acuity, slit-lamp biomicroscopy, dilated fundus examination, wid

156 al ocular findings, including visual acuity, slit-lamp biomicroscopy, spectral-domain optical coheren

157 CA was assessed by ophthalmologists using slit-lamp biomicroscopy.

158 Best-corrected vision, IOP, comprehensive slit-lamp evaluation, and anterior segment (AS) optical

159 Slit-lamp exam revealed a corneal ulcer with feathery ma

160 Age, visual acuity (VA), slit-lamp examination of anterior vitreous (SLAV), and c

161 punctum diameter (not readily measurable by slit-lamp examination), rather than the surface diameter

162 rected visual acuity, applanation tonometry, slit-lamp examination, indirect ophthalmoscopy, digital

163 nations, including visual acuity, perimetry, slit-lamp examination, intraocular pressure, and fundus

164 changes in corneal opacity were detected by slit-lamp examination, the corneas of homozygous mutant

165 he D-Eye device, followed by dilated retinal slit-lamp examination, to grade DR according to a 5-step

166 edge was noted in postoperative visits under slit-lamp examination.

167 CVA) visual acuity in 4 m, 80 cm, and 40 cm; slit-lamp examination; and tomography.

168 each visit, graft survival was determined by slit-lamp examination; best spectacle-corrected visual a

169 cuity (DCVA) in 4 m, 80 cm, 60 cm, and 40 cm slit-lamp examination; defocus testing; contrast sensiti

170 Slit-lamp examinations at 3 days and 1, 2, and 4 weeks a

171 -corrected visual acuity (BCVA) assessments, slit-lamp examinations, and stereoscopic fundus photogra

172 Retrospective review of charts and slit-lamp images of 564 consecutive patients from the pr

173 the sizes of stromal infiltrates measured on slit-lamp photographs 30 days after treatment.

174 were transient, and most interventions were slit-lamp procedures.

175 Surgical, postoperative slit-lamp, and histopathologic assessments of PPC were p

176 s included measurement of best-corrected VA, slit-lamp, examination, indirect ophthalmoscopy, and ult

177 Thus, Slit locally stimulates directional filopodial extension

178 ental characterization of two distant double-slit masks illuminated by chaotic light, in the absence

179 cs pharmacologically or genetically disrupts Slit-mediated repulsion and produces severe axon guidanc

180 tion framework based on shift-excitation and slit-modulation, followed by mathematical post-processin

181 Dscam1 and Robo1 form a complex dependent on Slit-N.

182 full-length Slit, whereas Dscam1 only binds Slit-N.

183 cal pinhole before being focused through the slit of a spectrometer.

184 red light from both beams is imaged onto the slit of an imaging spectrograph as two spatially separat

185 specialized cell junction at the filtration slit of glomerular podocytes.

186 l simulations of RBC flow through the venous slits of the human spleen.

187 ssion resonance of an array of square-shaped slits on a silicon substrate at ~0.3 THz, we were able t

188 ficant reduction of IL-5 was observed in the SLIT or untreated group.

189 oach involves designing an array of periodic slits or grating apertures that enables coupling of the

190 The shape representation is invariant to slit orientation and is similar to that evoked by a full

191 shape should be invariant to changes in the slit orientation.

192 s, but no patient was discontinued by AEs in SLIT patients.

193 en with PA were randomized to receive active SLIT/placebo OIT or active OIT/placebo SLIT.

194 ndicates that the neuronal guidance molecule SLIT plays a role in tumor suppression, as SLIT-encoding

195 The number and size of the filtration slit pores decreased.

196 (SNM), designed with approximately 7 nm-wide slit-pores to provide middle molecule selectivity by lim

197 n mirror placed in front of the spectrometer slit positions the Raman signals onto different pixel ro

198 ates both Semaphorin and Slit signaling, and Slit processing generates two active fragments, each exe

199 argely by the local availability of SCIT and SLIT products of proved value and personal (patient) pre

200 The Slit protein is a major midline repellent for central ne

201 of the Robo family and the secreted protein Slit provides important signals in the development of th

202 We identified PlexinA1 as a Slit receptor and found that it binds the C-terminal Sli

203 ess, which reduces surface expression of the Slit receptor Roundabout1 (Robo1).

204 Double mutants for the Slit receptors Dscam1 and robo1 strongly resemble the sl

205 vity to the conserved Netrin attractants and Slit repellents is insufficient to explain the guidance

206 taneously mediates NELL2 repulsion, inhibits Slit repulsion, and facilitates Netrin attraction to ach

207 scue drugs, accounted for 9 and 24% of total SLIT, respectively.

208 By using the Slit-Robo axon guidance pathway to target neuronal midli

209 rivers of osteosarcoma metastasis, including Slit-Robo GTPase-Activating Protein 2 (Srgap2).

210 Additionally, SRGAP2 and other genes in the Slit-Robo pathway have altered transcript levels in a su

211 o further evaluate the potential role of the Slit-Robo pathway in osteosarcoma.

212 axons in Robo1/2 mutant embryos showed that Slit-Robo repulsive signaling was not required for post-

213 at Netrin1-DCC attractive signaling, but not Slit-Robo repulsive signaling, remains active in hindbra

214 ways, serves as an essential co-receptor in Slit-Robo signaling.

215 viral infection in the host by targeting the Slit/Robo pathway with modulation of cytoskeletal elemen

216 o the best of our knowledge, a newly defined SLIT/ROBO/Myo9b/RhoA signaling pathway that restricts lu

217 least two independent mechanisms to overcome Slit-Robo1 repulsion in pre-crossing commissural axons h

218 n of commissureless (comm), an antagonist of Slit-Roundabout midline repulsion, through an unknown me

219 Single-growth-cone imaging reveals that Slit/RPTP69D are not required for general branch initiat

220 partially to entirely blocked by introducing slits, s = 95, 195, 245, 295 and 395 nm.

221 Both SCIT and SLIT showed good clinical efficacy without significant d

222 Both SCIT and SLIT showed good clinical efficacy without significant d

223 mer--present as an ultra-thin coating on the slit sidewalls.

224 ckout mice shows that PlexinA1-Slit and Robo-Slit signaling have complementary roles during commissur

225 Thus, PlexinA1 mediates both Semaphorin and Slit signaling, and Slit processing generates two active

226 0 pathways: KRAS, TGF-beta, WNT, NOTCH, ROBO/SLIT signalling, G1/S transition, SWI-SNF, chromatin mod

227 ions with hydrated diameters larger than the slit size can still permeate through, albeit with reduce

228 del, we find that the repulsive guidance cue Slit stimulates the formation and elongation of actin-ba

229 aily treatment with placebo (n = 277) or HDM SLIT tablet (dosage groups: 6 SQ-HDM [n = 275] or 12 SQ-

230 Asthma onset was less frequent in SLIT tablet group than in non-AIT group (odds ratio: 0.6

231 and relative to the pretreatment period) in SLIT tablet group than in the non-AIT group (P<.001).

232 (relative to the pretreatment period) in the SLIT tablet group vs the non-AIT group (P=.004).

233 se dust mite (HDM) sublingual immunotherapy (SLIT) tablet (MK-8237; Merck & Co, Kenilworth, NJ/ALK-Ab

234 patients received sublingual immunotherapy (SLIT) tablet (Oralair, Stallergenes(c)) and symptomatic

235 ite (HDM) sublingual allergen immunotherapy (SLIT) tablet is a potential novel treatment option for H

236 out asthma were randomized to a daily SQ HDM SLIT-tablet (12 SQ-HDM dose) or placebo for up to approx

237 The SQ HDM SLIT-tablet (ALK) has been developed for treatment of ho

238 mild asthma included in these studies, grass SLIT-tablet did not increase TEAE frequency, severe loca

239 HDM SLIT-tablet effects were numerically greater than all ph

240 n the first North American trial of use of a SLIT-tablet for HDM allergy, 12 SQ-HDM was well tolerate

241 igated the efficacy and safety of the SQ HDM SLIT-tablet in adults with moderate-to-severe HDM-induce

242 Using data from eight trials of grass SLIT-tablet in subjects with allergic rhinitis with/with

243 eeks of treatment in 2 house dust mite (HDM) SLIT-tablet trials (n = 1768).

244 ing entire pollen seasons in 6 timothy grass SLIT-tablet trials (n = 3094) and 2 ragweed SLIT-tablet

245 SLIT-tablet trials (n = 3094) and 2 ragweed SLIT-tablet trials (n = 658) and during the last 8 weeks

246 SLIT-tablet trials allowed rescue medication use, wherea

247 terogeneity and use of rescue medications in SLIT-tablet trials, effects on nasal symptoms with timot

248 In grass and ragweed SLIT-tablet trials, overall improvement in TNSSs relativ

249 In HDM SLIT-tablet trials, TNSS overall improvement relative to

250 o groups receiving either SCIT injections or SLIT tablets or neither.

251 d treatment of AR patients with grass pollen SLIT tablets was associated with slower AR progression,

252 abase, AR patients treated with grass pollen SLIT tablets were compared with a control group not havi

253 cy of grass pollen sublingual immunotherapy (SLIT) tablets in AR and their impact on asthma onset and

254 reatment effect of sublingual immunotherapy (SLIT)-tablets with pharmacotherapy for seasonal allergic

255 t trial to assess the efficacy/safety of HDM SLIT-tablets in North American subjects with HDM-induced

256 SLIT-tablets offer the additional benefit of long-term e

257 The treatment effect of timothy grass SLIT-tablets was considered similar between pediatric (n

258 asal symptoms with timothy grass and ragweed SLIT-tablets were nearly as great as with MFNS and numer

259 ng the clinical development of timothy grass SLIT-tablets.

260 ildren and adults treated with timothy grass SLIT-tablets.

261 port ion transport through ultimately narrow slits that are fabricated by effectively removing a sing

262 ed with the development of pharyngeal 'gill' slits, the foremost morphological innovation of early de

263 Our preliminary data showed that SLIT therapy in viro-immunological controlled HAART trea

264 omagnetic near-fields in the vicinity of the slits through the excitation of surface plasmons.

265 well controlled by ICS, the addition of HDM SLIT to maintenance medications improved time to first m

266 ermal paths (i.e. labyrinths) by introducing slits to control the impact of the unobstructed "line-of

267 h grass pollen sublingual immunotherapy (the SLIT-TOL group; n = 6), patients with untreated seasonal

268 s symptoms were lower in the SCIT, SLIT, and SLIT-TOL groups (P < .001) compared with those in the SA

269 patients (n = 14), sublingual immunotherapy (SLIT)-treated patients (n = 12), participants who comple

270 acy data showed a significant improvement in SLIT-treated patients compared to controls (TCS: P = 0.0

271 ical and immunologic outcomes for our peanut SLIT trial.

272 Obtaining large sample sizes for pediatric SLIT trials is challenging, but a Bayesian approach usin

273 zes are needed for sublingual immunotherapy (SLIT) trials because of inherent data variability second

274 ptors Dscam1 and robo1 strongly resemble the slit-UC phenotype, suggesting they cooperate in longitud

275 sophila ventral nerve cord of a slit allele (slit-UC) that cannot be cleaved revealed that midline re

276 educed for horizontal compared with vertical slit-viewing in ASD.

277 Adherence of SLIT was 89+/-12%.

278 At 3 years, SLIT was discontinued for 8 weeks, followed by another 1

279 SLIT was significantly effective compaired with other ph

280 SLIT was significantly effective compared with other pha

281 SLIT was significantly effective than other pharmacother

282 stead of a quantum wave passing through both slits, we have a localized particle with nonlocal intera

283 from baseline and after 12 months of peanut SLIT were assayed using ImmunoCAP for IgE and IgG4 again

284 Both SCIT and SLIT were significantly better than other pharmacotherap

285 Both SCIT and SLIT were significantly better than other pharmacotherap

286 odological limitations; these suggested that SLIT, when used in patients with both asthma and allergi

287 ing alone shows a preference for full-length Slit, whereas Dscam1 only binds Slit-N.

288 ltration barrier is known as a 'size cutoff' slit, which retains nanoparticles or proteins larger tha

289 lar basement membrane is thickened, podocyte slit width is increased and sub-podocyte space coverage

290 ) for straight beam to 31 W m(-1) K(-1) for slit width of 395 nm.

291 in glomerular basement membrane and podocyte slit width, as well as the decrease in sub-podocyte spac

292 mpared the clinical efficacy in 2016, of 133 SLIT with 46 SCIT, 351 primary pharmacotherapy that star

293 We compared the clinical efficacy of 191 SLIT with 48 SCIT, 191 primary pharmacotherapy that star

294 We included 33 subjects who underwent peanut SLIT with a DBPCFC of 2500 mg of peanut protein performe

295 SLIT with rBet v 1 neither improved the clinical reactiv

296 ith the placebo and rBet v 1-treated groups, SLIT with rMal d 1 reduced rMal d 1-induced oral symptom

297 ld enhancement in a two-dimensional array of slits with micrometer dimensions in a metallic film can

298 ty and efficacy of sublingual immunotherapy (SLIT) with 2 formulations containing either rMal d 1 or

299 Sublingual immunotherapy (SLIT) with peanut changes clinical and immune responses

300 eams in graphene based on collinear pairs of slits, with absorptive sidewalls between the slits.