ライフサイエンスコーパス: slit lamp

1 the angiogenic response is measured using a slit lamp.

2 Lens clarity was assessed by slit lamp.

3 ained by using a digital camera mounted on a slit lamp.

4 mal rabbits and observed periodically with a slit lamp.

5 ted by clinical score and photographs with a slit lamp.

6 ea was marked in the sitting position at the slit lamp.

7 ted from the retro-optic portion seen at the slit lamp.

8 The injected eyes were monitored by slit lamp, a handheld tonometer, indirect ophthalmoscopy

9 sts were visual acuity, clinical evaluation (slit lamp), Amsler chart, color fundus photographs, infr

10 examined for the presence of hyphema using a slit lamp and gonioscopy.

11 corneal restoration was studied "in vivo" by slit lamp and in tissue sections by means of both light

12 Examination of mouse strains with a slit lamp and indirect ophthalmoscopy revealed that stra

13 scale, 0 to 10) assessed subjectively at the slit lamp and objectively using automated image analysis

14 Five of the 16 cases had slit lamp and/or topographic features consistent with ke

15 or and posterior segments examination with a slit-lamp and a direct ophthalmoscope respectively.

16 ereoacuity, refraction, clinical findings of slit-lamp and dilated fundus examinations.

17 e (IOP) measurement, and corneal pachymetry; slit-lamp and optic nerve examination; automated visual

18 Three of these recurrences were missed by slit-lamp, and 6 of these were missed by IVCM.

19 Surgical, postoperative slit-lamp, and histopathologic assessments of PPC were p

20 al acuity, cycloplegic objective refraction, slit lamp as well as fundus examinations.

21 isual acuity, autorefraction, visual fields, slit lamp assessment of the anterior and posterior segme

22 t detailed ophthalmic examinations including slit-lamp assessment and dilated retinal photographs.

23 , including best-corrected visual acuity and slit-lamp assessment of lens opacities using the Lens Op

24 , including best-corrected visual acuity and slit-lamp assessment of lens opacities using the Lens Op

25 Each mouse was examined by slit lamp at 18 and 24 months of age and scored for degr

26 All eyes were examined by slit lamp at baseline and 3, 6, 9, 24, 48, and 72 hours

27 Disease was monitored by clinical score, slit lamp, bacterial plate count, a myeloperoxidase (MPO

28 Cortical cataract was assessed using the slit-lamp-based Oxford Clinical Cataract Classification

29 neovascular response was quantified using a slit lamp biomicroscope.

30 e treatments with a diode laser adapted to a slit lamp biomicroscope.

31 ication and better optical sectioning than a slit-lamp biomicroscope, confocal microscopy is ideally

32 etter, no history of eye disease, and normal slit lamp biomicroscopic and ophthalmoscopic examination

33 Corneal topography, visual acuity, and slit lamp biomicroscopic examination were performed in a

34 Comparison was made between slit lamp biomicroscopy (SLB) and photographic grading.

35 Slit lamp biomicroscopy and corneal pachymetry were perf

36 t II), epithelial closure was monitored with slit lamp biomicroscopy and fluorescein staining, and co

37 ular pressure, best corrected visual acuity, slit lamp biomicroscopy and medical history were obtaine

38 with cataractous stages visually observed by slit lamp biomicroscopy and retroillumination photograph

39 of optic disk hemorrhage was evaluated with slit lamp biomicroscopy at each clinic visit prior to an

40 Slit lamp biomicroscopy disclosed the clinical features

41 ssure was normal in all of the study groups; slit lamp biomicroscopy examinations revealed that no ce

42 fts were evaluated for signs of rejection by slit lamp biomicroscopy over 8 weeks.

43 Slit lamp biomicroscopy was performed throughout the per

44 Eyes were examined by slit lamp biomicroscopy with fluorescein solution to ass

45 rosis were determined with stereomicroscopy, slit lamp biomicroscopy, alpha-smooth muscle actin (alph

46 nockout mice were screened for cataract with slit lamp biomicroscopy, and dissected lenses were exami

47 In vivo lens changes were monitored by slit lamp biomicroscopy, and enucleated lenses were exam

48 ssed at various times following injection by slit lamp biomicroscopy, electroretinography (ERG), bact

49 (HBL-) was assessed bacteriologically and by slit lamp biomicroscopy, electroretinography, histology,

50 Visual outcomes, slit lamp biomicroscopy, intraocular pressure (IOP), and

51 rabbit corneas (3 +/- 0.4) quantified using slit lamp biomicroscopy.

52 ty of haze in the rabbit eye was graded with slit lamp biomicroscopy.

53 dividuals in a large family were examined by slit lamp biomicroscopy.

54 All grafts were evaluated clinically by slit lamp biomicroscopy.

55 Graft survival was evaluated by slit lamp biomicroscopy.

56 r UVB exposure and for as long as 10 days by slit lamp biomicroscopy.

57 At 24 and 72 hours, slit-lamp biomicroscopy (and additionally indirect ophth

58 e posterior hyaloid membrane observed during slit-lamp biomicroscopy after posterior vitreous detachm

59 Corneal grafts were evaluated by ophthalmic slit-lamp biomicroscopy and analyzed by Kaplan-Meier sur

60 24, 48, and 72 hours and were re-examined by slit-lamp biomicroscopy and by indirect ophthalmoscopy.

61 ic nerve damage was assessed by stereoscopic slit-lamp biomicroscopy and fundus photography and by co

62 is study, the AC cell response, evaluated by slit-lamp biomicroscopy and graded using a standard grad

63 , 24, 48, and 72 hours after injection using slit-lamp biomicroscopy and laser flare photometry.

64 , both eyes of each patient were examined by slit-lamp biomicroscopy and white-light IVCM (Confoscan

65 All the eyes in the study were examined by slit-lamp biomicroscopy at baseline and 6, 9, 24, 48, an

66 All eyes were examined by slit-lamp biomicroscopy at baseline, 3, 6, 9, 24, 48, an

67 Demographic and clinical characteristics, slit-lamp biomicroscopy findings, and dilated ophthalmos

68 er flare photometry were consistent with the slit-lamp biomicroscopy flare findings up to grade 3+.

69 All eyes were evaluated by slit-lamp biomicroscopy for inflammatory response at 3,

70 e posterior hyaloid membrane observed during slit-lamp biomicroscopy in patients with posterior vitre

71 Slit-lamp biomicroscopy showed refractile, polychromatic

72 All corneas were examined by using slit-lamp biomicroscopy to determine the severity of FEC

73 A uveitis specialist used slit-lamp biomicroscopy to grade the AC cells on a scale

74 arts), Orbscan, retinoscopy, refraction, and slit-lamp biomicroscopy were performed.

75 f treatment, cumulative dose, Orlando stage (slit-lamp biomicroscopy), and serum concentrations of am

76 raocular defects by indirect ophthalmoscopy, slit-lamp biomicroscopy, and ERG to discover new spontan

77 pressure measurement, ultrasound pachymetry, slit-lamp biomicroscopy, and laser scanning in vivo conf

78 cted visual acuity recorded in LogMAR units, slit-lamp biomicroscopy, and optical coherence tomograph

79 After maximal mydriasis, slit-lamp biomicroscopy, and photography, imaging of the

80 ents had their ocular surface evaluated with slit-lamp biomicroscopy, and tear production quantified

81 Best-corrected visual acuity, slit-lamp biomicroscopy, dilated fundus examination, wid

82 e were compared with untreated animals using slit-lamp biomicroscopy, flow cytometry, and ELISA.

83 ncorrected and best-corrected visual acuity, slit-lamp biomicroscopy, Goldmann applanation tonometry,

84 History and ocular examination, including slit-lamp biomicroscopy, gonioscopy, specular microscopy

85 assessment of best-corrected visual acuity, slit-lamp biomicroscopy, intraocular pressure measuremen

86 stionnaire; ophthalmic examination including slit-lamp biomicroscopy, noncontact tonometry, fundus ph

87 Different approaches, including slit-lamp biomicroscopy, ophthalmoscopic examination, ul

88 y by a masked grader, applanation tonometry, slit-lamp biomicroscopy, optic nerve evaluation, and A-s

89 al ocular findings, including visual acuity, slit-lamp biomicroscopy, spectral-domain optical coheren

90 All corneas were examined using slit-lamp biomicroscopy, ultrasonic pachymetry, and conf

91 es used for imaging the anterior segment are slit-lamp biomicroscopy, ultrasound biomicroscopy, schei

92 underwent anterior segment examination with slit-lamp biomicroscopy.

93 Eyes were examined weekly by slit-lamp biomicroscopy.

94 CA was assessed by ophthalmologists using slit-lamp biomicroscopy.

95 kly for cataract development by conventional slit-lamp biomicroscopy.

96 Progression of cataracts was monitored by slit-lamp biomicroscopy.

97 , 6, 24, 48, and 72 hours after treatment by slit-lamp biomicroscopy.

98 NOP was graded at the slit lamp by an ophthalmologist using the Lens Opacity C

99 The slit-lamp characteristics of the corneal ulceration, cor

100 The visual clinical eye examination, slit lamp clinical studies, TUNEL, CD11b, and F4/80 assa

101 Slit lamp, clinical score, RT-PCR, ELISA, myeloperoxidas

102 A slit-lamp digital camera acquired images of the neovascu

103 roscopy and were photographed once a week by slit-lamp digital camera and scored for opacity.

104 morphology and histology were evaluated with slit lamp, digital confocal microscopy and hematoxylin a

105 he severity of endophthalmitis was graded by slit lamp, electroretinography, histological examination

106 Testing was performed with a photo slit lamp equipped with two 2x teleconverters and a digi

107 Careful slit lamp evaluation and ancillary testing can not only

108 ure (IOP) and pattern ERG (PERG), performing slit lamp evaluation of the anterior chamber, analyzing

109 the corneal phenotype was mild with a normal slit lamp evaluation.

110 Best-corrected vision, IOP, comprehensive slit-lamp evaluation, and anterior segment (AS) optical

111 corrected distance visual acuity (CDVA), and slit lamp evidence of corneal complications.

112 nograms [ERGs]), and cataract formation with slit lamp exam (biweekly).

113 Slit-lamp exam revealed a corneal ulcer with feathery ma

114 s, and pathologic changes were determined by slit lamp examination (SLE) and histopathologic analysis

115 Slit lamp examination (SLE) and histopathology were perf

116 Pathologic changes were monitored by slit lamp examination (SLE) and histopathology.

117 Slit lamp examination (SLE) and measurement of erosions

118 Mice underwent slit lamp examination (SLE) at 1, 3, 5, 7, and 9 days af

119 Eyes were graded for disease by slit lamp examination (SLE) every 6 hours until 24 hours

120 Slit lamp examination (SLE) of rabbit eyes was performed

121 Eyes were scored by slit lamp examination (SLE), and bacterial colony-formin

122 ated in rabbit and mouse keratitis models by slit lamp examination (SLE), bacterial enumeration, and/

123 Slit lamp examination (SLE), electroretinography, and my

124 e recovered from the corneas after the final slit lamp examination (SLE).

125 Corneal virulence was evaluated by slit lamp examination and bacterial cultures in both a r

126 Ocular pathology was evaluated by slit lamp examination and myeloperoxidase activity of in

127 orrelated well with clinical grading both at slit lamp examination and when looking at the images the

128 animals, cataract formation was followed by slit lamp examination at regular intervals.

129 rehensive eye examination, including dilated slit lamp examination by an ophthalmologist and digital

130 erity of stromal keratitis were monitored by slit lamp examination in a masked fashion.

131 al function and potential acuity, and to the slit lamp examination of the lens, posterior capsule, an

132 showed no obvious loss of lens clarity, but slit lamp examination revealed the emergence of opacific

133 Slit lamp examination revealed the presence of bilateral

134 In the rabbit model, ocular damage (slit lamp examination score) mediated by the parent stra

135 tivity, mediated infections characterized by slit lamp examination scores significantly lower than th

136 Slit lamp examination showed that control mice, C3(-/-)

137 Slit lamp examination showed that, from an early age, ne

138 ms of clinical signs (slit lamp examination, slit lamp examination), and viable bacteria.

139 Mean clinical scores, slit lamp examination, adenosine diphosphatase (ADPase),

140 Development of keratitis was assessed by slit lamp examination, and inflammatory cells in the cor

141 was evaluated by indirect ophthalmoscopy and slit lamp examination, and retinal changes were evaluate

142 Slit lamp examination, dilated fundus examination, and e

143 ch visit includes (1) Clinical evaluation: a slit lamp examination, fundoscopy, intraocular pressure

144 CASE PRESENTATION: We evaluated slit lamp examination, fundoscopy, optical coherence tom

145 ced by UMCR1 were documented by photographs, slit lamp examination, histopathologic analysis, and qua

146 oplegic retinoscopy, A-scan ultrasonography, slit lamp examination, indirect ophthalmoscopy, and kera

147 y was assessed over this same time period by slit lamp examination, indirect ophthalmoscopy, electror

148 e comparatively analyzed at multiple ages by slit lamp examination, intraocular pressure recording, a

149 ity, refractive error, intraocular pressure, slit lamp examination, pachymetry measurements and endot

150 rastromal models in terms of clinical signs (slit lamp examination, slit lamp examination), and viabl

151 mouse mutations by phenotypic screening with slit lamp examination.

152 Eyes were evaluated by slit lamp examination.

153 treated with spantide, and after infection, slit lamp examination; clinical score; bacterial counts;

154 were determined and pathology was scored by slit-lamp examination (SLE).

155 Cataract was diagnosed using a slit-lamp examination and defined as any lens opacity in

156 Cataracts were assessed on slit-lamp examination and were graded according to the L

157 es given before presentation, visual acuity, slit-lamp examination findings, corneal sensation, dose

158 ed demonstrate that IVCM is complementary to slit-lamp examination in the follow-up of HSK, particula

159 backscatter measurement by combined IVCM and slit-lamp examination may improve the outcome of HSK.

160 Slit-lamp examination of 10 affected individuals was con

161 Age, visual acuity (VA), slit-lamp examination of anterior vitreous (SLAV), and c

162 Ocular viral cultures were obtained after slit-lamp examination on days 1, 3, 5, 7, 9, 11, and 14.

163 keratitis was graded in a masked fashion by slit-lamp examination on days 2, 3, 5, 7, 9, 11, and 14.

164 Slit-lamp examination revealed progressive pathology, an

165 Slit-lamp examination showed chemosis, ciliary injection

166 Slit-lamp examination showed that Lyst mutant mice unifo

167 Patients were followed up clinically using slit-lamp examination to determine evidence of tumor dis

168 icles in the inferior AC that were missed by slit-lamp examination were detected by OCT.

169 to oval epithelial lesions in the cornea in slit-lamp examination with mild to moderate conjunctival

170 punctum diameter (not readily measurable by slit-lamp examination), rather than the surface diameter

171 njunctival impression cytology, 44% and 48%; slit-lamp examination, 20% and 66%; tear film break-up t

172 eal features of FECD and cataract density at slit-lamp examination, and corneal backscatter using in

173 rected visual acuity, applanation tonometry, slit-lamp examination, indirect ophthalmoscopy, digital

174 nations, including visual acuity, perimetry, slit-lamp examination, intraocular pressure, and fundus

175 Glaucoma diagnosis based on slit-lamp examination, measurement of intraocular pressu

176 al discomfort, despite minor lens opacity on slit-lamp examination, minor loss of best-corrected visu

177 changes in corneal opacity were detected by slit-lamp examination, the corneas of homozygous mutant

178 rameters were indiscernible or overlooked at slit-lamp examination, they proved to be excellent indic

179 he D-Eye device, followed by dilated retinal slit-lamp examination, to grade DR according to a 5-step

180 Changes in ocular disease, determined by slit-lamp examination, were measured at 3, 16, 22, and 2

181 edge was noted in postoperative visits under slit-lamp examination.

182 cataract was classified into seven stages by slit-lamp examination.

183 acuity and refraction tests, funduscopy, and slit-lamp examination.

184 CVA) visual acuity in 4 m, 80 cm, and 40 cm; slit-lamp examination; and tomography.

185 each visit, graft survival was determined by slit-lamp examination; best spectacle-corrected visual a

186 cuity (DCVA) in 4 m, 80 cm, 60 cm, and 40 cm slit-lamp examination; defocus testing; contrast sensiti

187 s included measurement of best-corrected VA, slit-lamp, examination, indirect ophthalmoscopy, and ult

188 evaluate the severity of herpetic keratitis, slit lamp examinations (SLE) were performed every other

189 Slit lamp examinations (SLEs) were performed at 24, 36,

190 Lesions were evaluated by slit lamp examinations over a 2-week period after infect

191 Slit-lamp examinations at 3 days and 1, 2, and 4 weeks a

192 Slit-lamp examinations performed in childhood or adultho

193 Standardized slit-lamp examinations were performed by trained study o

194 -corrected visual acuity (BCVA) assessments, slit-lamp examinations, and stereoscopic fundus photogra

195 imaging system is composed of a photographic slit lamp for biomicroscopic examination of the fundus,

196 3 individual examination sections (general, slit lamp, fundus) for the 3 diseases, 5 of the 9 possib

197 es, the OCT cell counts correlated well with slit-lamp grades in all three regions (Spearman's rho co

198 mian gland atrophy and acini appearance, and slit-lamp grading of lid debris and telangiectasias were

199 alpebral aperture (PA) were measured using a slit lamp graticule.

200 Doorknobs, slit-lamp headrests and chinrests, and computer keyboard

201 Clinical score, slit lamp, histopathology, bacterial counts, and polymor

202 er and were observed and graded for haze via slit lamp, imaged, and graded by macrophotography.

203 Slit lamp images showed an increase in nuclear light sca

204 Film-based slit lamp images taken at baseline and at 5- and 10-year

205 alpebral aperture were analyzed from digital slit lamp images.

206 Retrospective review of charts and slit-lamp images of 564 consecutive patients from the pr

207 By using slit-lamp in conjunction with IVCM, we detected 17 recur

208 Slit-lamp laser photocoagulation was sufficient in half

209 Fifteen eyes were treated with slit-lamp laser.

210 ternal ocular infections were examined under slit lamp microscope.

211 5 hours after infection and were examined by slit lamp microscopy 24, 36, and 48 hours after infectio

212 mutagenesis was characterized using RT-PCR, slit lamp microscopy and histologic methods.

213 (-/-), and C57BL/6 WT mice were screened via slit lamp microscopy or ex vivo analysis.

214 wed the absence of beaded filaments, whereas slit lamp microscopy showed a slowly emerging and progre

215 analyses, light and electron microscopy, and slit lamp microscopy.

216 d are transparent as determined by light and slit lamp microscopy.

217 in situ by direct ophthalmic examination and slit lamp microscopy.

218 Slit-lamp microscopy and histology were used to examine

219 ts were examined twice a week for 8 weeks by slit-lamp microscopy and were photographed once a week b

220 abbit eyes were swabbed and then examined by slit-lamp microscopy at 0, 5, 10, 15, 20, and 25 hours a

221 xamined by means of indirect ophthalmoscopy, slit-lamp microscopy, and fundus photography to establis

222 was not visible clearly through the graft by slit-lamp microscopy.

223 the corneal polarization axis (CPA), with a slit-lamp-mounted corneal polarimeter.

224 he same experimental period, consistent with slit lamp observations.

225 y measures included change in visual acuity, slit-lamp observations, and adverse events.

226 ivo before and after dilation by time-domain slit lamp optical coherence tomography (SL-OCT).

227 The patients were examined with the slit-lamp, optical pachymetry, and CM before undergoing

228 Laser photocoagulation was performed at the slit lamp or during pars plana vitrectomy for telangiect

229 Slit lamp photographs taken during a population-based pr

230 In addition, slit lamp photographs were graded for nuclear cataracts,

231 the sizes of stromal infiltrates measured on slit-lamp photographs 30 days after treatment.

232 locally resident siblings underwent digital slit lamp photography and were administered a questionna

233 Scheimpflug slit-lamp photography, as well as a variety of standard

234 Meibography and slit-lamp photos were captured digitally and saved for a

235 Standardized digital slit-lamp pictures were quantitatively and objectively e

236 Disease was graded by clinical score, slit lamp, plate count, real-time RT-PCR, and ELISA assa

237 sease was determined by mean clinical score, slit lamp, plate counts, and histopathology, and antigen

238 were transient, and most interventions were slit-lamp procedures.

239 were transient, and most interventions were slit-lamp procedures.

240 A modified slit lamp ("scatterometer"), with a video camera and syn

241 Using mouse genetic approaches and slit lamp screening, we identified two mouse cataractous

242 Dissected lenses were analysed by slit-lamp stereophotomicroscopy, western blot, tryptic-d

243 om the angiographic images and marked at the slit lamp using a needle to make a cut to the depth of t

244 Lens grading at the slit lamp, using standardized photographs and a grading

245 The slit beam of a photographic slit lamp was monitored with a video camera through one