ライフサイエンスコーパス: slow-wave sleep

1 ction, P < 0.02), urinary catecholamines, or slow wave sleep.

2 ather tended to increase waking and decrease slow wave sleep.

3 ave ripples observed in quiet wakefulness or slow wave sleep.

4 al decrease in effective interactions during slow-wave sleep.

5 during wakefulness, to a stable focus during slow-wave sleep.

6 EEGs is the K-complex (KC), which occurs in slow-wave sleep.

7 ating cellular and network plasticity during slow-wave sleep.

8 uent in waking and paradoxical sleep than in slow-wave sleep.

9 oscillatory activity (spindle waves) during slow-wave sleep.

10 ay severe sleep loss as a result of unstable slow-wave sleep.

11 ll brain nodes to best match wakefulness and slow-wave sleep.

12 role in organizing cortical activity during slow-wave sleep.

13 r are reactivated during rest and subsequent slow-wave sleep.

14 ge II and REM but is preempted by arousal in slow-wave sleep.

15 preempted development of EUCR and 2P during slow-wave sleep.

16 ring behaviours such as alert immobility and slow-wave sleep.

17 ponse to auditory stimuli is greatest during slow-wave sleep.

18 as the slow (0.1-0.5 Hz) oscillation during slow-wave sleep.

19 ncreased wakefulness at the expense of deep, slow-wave sleep.

20 timescale during single sharp-wave bursts of slow-wave sleep.

21 ay dilator activity in sleep and/or enhanced slow-wave sleep.

22 al cell spikes in the rat hippocampus during slow-wave sleep.

23 play of cortical cell spike sequences during slow-wave sleep.

24 s deviations from perfect balance, mostly in slow-wave sleep.

25 ef periods of desynchronization prevalent in slow-wave sleep.

26 is important for this transformation during slow-wave sleep.

27 during waking and accelerated tenfold during slow-wave sleep.

28 rousal but to rapidly and selectively induce slow-wave sleep.

29 onists induce wakefulness and reduce REM and slow-wave sleep.

30 th one learned sequence were replayed during slow-wave sleep.

31 lective memory generalization during REM and slow-wave sleep.

32 , that it also does not arise during natural slow-wave sleep.

33 cting total sleep time, sleep efficiency, or slow-wave sleep.

34 ot occur with MECIII input inhibition during slow-wave sleep.

35 delta oscillations have been associated with slow-wave sleep.

36 d fast-sigma oscillations, especially during slow-wave sleep.

37 ory gating, thalamocortical rhythmicity, and slow-wave sleep.

38 ity in healthy humans during wakefulness and slow-wave sleep.

39 ith performance improvement were specific to slow-wave sleep.

40 ivation process for specific memories during slow-wave sleep.

41 s both REM sleep and wakefulness and reduces slow-wave sleep 2.

42 ists during subsequent quiet wakefulness and slow-wave sleep, a process that may facilitate the conso

43 in extended sleep paralleling changes in EEG slow-wave sleep activity.

44 These results suggest that enhancing slow-wave sleep acutely after trauma may have a benefici

45 g, i.p.) was found to significantly increase slow wave sleep and decrease REM sleep in rats implanted

46 s thalamic network activity occurring during slow wave sleep and paroxysmal discharges critically dep

47 In men, age-related changes in slow wave sleep and REM sleep occur with markedly differ

48 Hcrt cells are silent in slow wave sleep and tonic periods of REM sleep, with occ

49 Slow wave sleep and total sleep time are indistinguishab

50 Ebselen decreased slow-wave sleep and affected emotional processing by inc

51 spent in rapid eye movement (REM) sleep and slow-wave sleep and an increase in muscle tone during RE

52 ("sharp wave-ripples") occur during rest and slow-wave sleep and are thought to be important for memo

53 During slow-wave sleep and deep anesthesia, the rat hippocampus

54 sleep spindles (6-14 Hz) occur mostly during slow-wave sleep and have been hypothesized to involve th

55 disability and autism which may impact both slow-wave sleep and information processing during waking

56 robability of an awakening event during both slow-wave sleep and rapid eye movement sleep.

57 xtinguished conditioned fear, increased both slow-wave sleep and rapid-eye movement (REM) sleep.

58 6% more time in a transitional state between slow-wave sleep and REM sleep (tS-R) compared with that

59 natomical substrate for behaviors, including slow-wave sleep and seizure suppression evoked by stimul

60 s showed differentially smaller increases in slow-wave sleep and smaller reductions in stage 2 sleep

61 ane potential oscillations are slower during slow-wave sleep and under anesthesia.

62 neural network oscillations associated with slow-wave sleep and various epilepsies.

63 during quiescent states such as anesthesia, slow-wave sleep, and awake immobility.

64 ty and beta-cell function, for time spent in slow-wave sleep, and for EEG spectral power in the delta

65 patterns of activity associated with waking, slow-wave sleep, and generalized seizures.

66 aneous BOLD fluctuations across wakefulness, slow-wave sleep, and rapid-eye-movement sleep.

67 assessed for presence of HEP within stage 2, slow-wave sleep, and REM sleep in 40 children with prima

68 ivity and temperature patterns, increases in slow-wave sleep, and shifts in EEG spectral power, sever

69 e rat brain ventricles specifically enhances slow wave sleep, apparently by antagonizing the effects

70 and systemic disease and injury (e.g. fever, slow-wave sleep, appetite suppression and neuroendocrine

71 Slow-wave sleep as well as generalized absence seizures

72 nized network oscillations representative of slow-wave sleep, as well as absence seizures, were demon

73 henomenon, which is also termed asymmetrical slow wave sleep (ASWS).

74 water, however, fur seals exhibit asymmetric slow-wave sleep (ASWS), resembling the unihemispheric sl

75 t patterns of sleep: bilaterally symmetrical slow-wave sleep (BSWS) as seen in terrestrial mammals an

76 e unique in that they display both bilateral slow-wave sleep (BSWS), as seen in all terrestrial mamma

77 involved no further significant decrease in slow wave sleep but an increase in time awake of 28 minu

78 scillation which occurs predominantly during slow-wave sleep, but may also play a role during awake s

79 ng of HA neurons during wakefulness promotes slow-wave sleep, but not rapid eye movement sleep, durin

80 Slow-wave sleep consists in slowly recurring waves that

81 vity of cholinergic brainstem neurons during slow-wave sleep continues to have a functional impact up

82 We propose that rapid eye movement (REM) and slow-wave sleep contribute differently to the formation

83 During slow-wave sleep, cortical neurons display synchronous fl

84 The mean (SEM) percentage of deep slow wave sleep decreased from 18.9% (1.3%) during early

85 Short REM latency and slow wave sleep deficits are familial.

86 Short REM latency was also associated with slow wave sleep deficits.

87 al oscillations that underlie drowsiness and slow-wave sleep depend on rhythmic inhibition of relay c

88 rst half of the night, which is dominated by slow-wave sleep, did not improve recall.

89 ogic markers of sleep homeostasis, including slow-wave sleep, electroencephalographic slow-wave activ

90 g spontaneous wakefulness as contrasted with slow wave sleep; exhibited progressive increases during

91 ced a transient decrease in REM sleep and in slow-wave sleep followed by a slight improvement of slee

92 ng speech understanding, sensory gating, and slow-wave sleep for a subset of elderly individuals.

93 and is subsequently replayed during rest or slow-wave sleep for consolidation of the encoded experie

94 The decline in slow wave sleep from early adulthood to midlife was para

95 .5 Hz) cerebral cortical oscillations during slow-wave sleep has recently lead to the suggestion that

96 urons that are specifically activated during slow-wave sleep have not previously been described in th

97 During slow-wave sleep, HVC neurons responded preferentially to

98 ry inputs, as in development in utero, or in slow-wave sleep (i.e., throughout the entire lifespan),

99 ce and decreases paradoxical (REM) sleep and slow wave sleep in rats.

100 s probably correlated with the appearance of slow-wave sleep in postnatal animals.

101 oth the visual cortex and hippocampus during slow-wave sleep in rats.

102 The signature of slow-wave sleep in the electroencephalogram (EEG) is lar

103 , decreased delta sleep ratio, and decreased slow wave sleep [in percentage]) were stable, as predict

104 n chronically sleep restricted subjects, low slow-wave sleep intensity over the right prefrontal cort

105 indicate that the control of unihemispheric slow-wave sleep is likely to be a function of interponti

106 In contrast, slow-wave sleep is more directly involved in the consoli

107 synchronization of cortical activity during slow-wave sleep is still controversial, with some studie

108 Slow-wave sleep is thought to be important for retuning

109 sleepwalking, i.e. the partial arousal from slow-wave sleep, is today well-documented, the detailed

110 s bursts occurring in the hippocampus during slow-wave sleep, leading to the selective erasure of inf

111 rospinal fluid Abeta dynamics, decrements in slow-wave sleep may decrease the clearance of Abeta from

112 first time, a mechanistic explanation of how slow-wave sleep may promote consolidation of recent memo

113 eversible state abnormalities, while reduced slow-wave sleep may represent a more persistent trait ab

114 empirical functional changes observed during slow-wave sleep, namely a global shift of the brain's dy

115 sleep (ASWS), resembling the unihemispheric slow-wave sleep of odontocetes (toothed whales).

116 During subsequent slow-wave sleep, one sound was unobtrusively presented t

117 No slow wave sleep or rapid eye movement sleep stages could

118 ity of transition to wakefulness from either slow wave sleep or rapid eye movement sleep.

119 (active) and Down (quiescent) states during slow-wave sleep or anesthesia.

120 These oscillations occur during slow-wave sleep or at rest.

121 can provide novel insights into neocortical slow-wave sleep oscillations and their relationship to r

122 secretion was significantly associated with slow wave sleep (P<.001).

123 reased sleep efficiency, p = 0.005, enhanced slow-wave sleep, p = 0.0004, and minimized sleep-related

124 SDB during non-REM sleep (stage 2: P = 0.03; slow-wave sleep: P = 0.001).

125 o the brain ventricles specifically enhances slow-wave sleep, presumably by antagonizing the effects

126 ring eating, drinking, awake immobility, and slow-wave sleep, produce a large field excitatory postsy

127 oning, re-exposure to the odorant context in slow-wave sleep promoted stimulus-specific fear extincti

128 ation (TMR) of specific memory traces during slow-wave sleep promotes the emergence of explicit knowl

129 ppocampus resemble those found in nonprimate slow wave sleep, quantitative studies of these oscillati

130 -cell recordings manifesting Up/Down states (slow-wave sleep, quiet wakefulness), probably as a resul

131 s administered to the LH, rats had increased slow-wave sleep, rapid-eye movement (REM) sleep, and sle

132 portion = 1.24, 95% CI: 1.14, 1.33) and less slow wave sleep (relative proportion = 0.86, 95% CI: 0.7

133 ced REM sleep, while leaving active wake and slow wave sleep relatively intact.

134 nimal studies support the hypothesis that in slow-wave sleep, replay of waking neocortical activity u

135 tions between the different phases of sleep: Slow-wave sleep requires low ACh concentrations in the b

136 During slow-wave sleep, rhythmic burst firing is prominent, whe

137 ty that underlie rhythmic motor patterns and slow-wave sleep rhythms.

138 sufficient to rapidly and selectively induce slow-wave sleep.SIGNIFICANCE STATEMENT The function of m

139 occur during both behavior (awake SWRs) and slow-wave sleep (sleep SWRs).

140 Given the reported function of slow-wave sleep states in neocortical and hippocampal me

141 In particular, during episodes of slow wave sleep (SWS) and rapid eye movement (REM) sleep

142 Cortical activity during slow wave sleep (SWS) differs from that during REM sleep

143 equences involving four or more cells during slow wave sleep (SWS) immediately following, but not pre

144 p as a reference, an increase in stage 2 and slow wave sleep (SWS) were protective from hypersomnolen

145 cally exhibit reduced discharge rates during slow wave sleep (SWS), a subpopulation of GABAergic inte

146 tuned for hippocampal ensemble spike data in slow wave sleep (SWS), even in the absence of prior beha

147 that result in reduced interference include slow wave sleep (SWS), NMDA receptor antagonists, benzod

148 In sleeping zebra finches, we observed slow wave sleep (SWS), rapid eye movement (REM) sleep, a

149 Slow wave sleep (SWS), the deepest sleep stage hallmarke

150 During slow wave sleep (SWS), traces of neuronal activity patte

151 and humans has suggested the existence of a slow wave sleep (SWS)-promoting/electroencephalogram (EE

152 nists of 5-HT(2A) have been shown to enhance slow wave sleep (SWS).

153 cant increase in wakefulness and decrease in slow wave sleep (SWS).

154 ly observed in electrophysiogical studies of slow wave sleep (SWS).

155 eep onset; (ii) during Stage 3-4 sleep, i.e. slow wave sleep (SWS); (iii) during rapid eye movement (

156 ally sleeping common marmosets, we show that slow-wave sleep (SWS) alters neural responses in the pri

157 ippocampal sharp-wave/ripple (SWR) bursts in slow-wave sleep (SWS) and are sharply reduced during REM

158 luding several markers of sleep homeostasis: slow-wave sleep (SWS) and electroencephalogram (EEG) slo

159 GABA neuron firing rate decreased 53% during slow-wave sleep (SWS) and increased 79% during REM, rela

160 z EEG slow oscillation (SO) is a hallmark of slow-wave sleep (SWS) and is critically involved in slee

161 s by brief (60- 90 min) naps containing both slow-wave sleep (SWS) and rapid eye movement (REM) sleep

162 s the vigilance states of quiet waking (QW), slow-wave sleep (SWS) and rapid eye movement (REM) sleep

163 erns of EEG activity as a function of awake, slow-wave sleep (SWS) and rapid-eye movement (REM) sleep

164 two well-characterized physiological states, slow-wave sleep (SWS) and rapid-eye-movement sleep (REM)

165 However, only in slow-wave sleep (SWS) beta- and gamma-oscillations are a

166 respond to 6-h sleep deprivation (SD) with a slow-wave sleep (SWS) EEG delta (1.0 to 4.0 Hz) power re

167 During natural slow-wave sleep (SWS) in nonanesthetized cats, silent (d

168 Thus, slow-wave sleep (SWS) is characterized by EEG spindles a

169 Slow-wave sleep (SWS) is characterized by synchronized n

170 In the hippocampus, slow-wave sleep (SWS) is marked by high-frequency networ

171 Slow oscillations during slow-wave sleep (SWS) may facilitate memory consolidatio

172 ery similar; all three produced increases in slow-wave sleep (SWS) only in the dark period with no ch

173 ep (BSWS) as seen in terrestrial mammals and slow-wave sleep (SWS) with a striking interhemispheric E

174 thening of associated neural circuits during slow-wave sleep (SWS), a process known as "cellular cons

175 as they cycled normally between waking (W), slow-wave sleep (SWS), and rapid eye movement (REM) slee

176 to determine wake after sleep onset (WASO), slow-wave sleep (SWS), and rapid eye movement (REM) slee

177 points, including paradoxical sleep (PS) and slow-wave sleep (SWS), as well as the circadian rhythmic

178 pid eye movement (NREM) sleep, also known as slow-wave sleep (SWS), is thought to be the most "restor

179 e for 48 h in a respiration chamber, whereas slow-wave sleep (SWS), rapid eye movement (REM)-sleep, t

180 of freely moving rats, and the effects on W, slow-wave sleep (SWS), REM sleep, and levels of phosphor

181 d electroencephalogram (EEG) activity during slow-wave sleep (SWS), similar to that observed in all t

182 s, memory consolidation occurs partially via slow-wave sleep (SWS)-dependent replay of activity patte

183 lectrophysiological activity observed during slow-wave sleep (SWS).

184 higher during active waking (AW) than during slow-wave sleep (SWS).

185 fulness, but in the reverse direction during slow-wave sleep (SWS).

186 motor memory after targeted reactivation in slow-wave sleep (SWS).

187 ial role in the induction and maintenance of slow-wave sleep (SWS).

188 aking or rapid eye movement (REM) sleep from slow-wave sleep (SWS).

189 paradoxical sleep (PS; aka REM) than during slow-wave sleep (SWS).

190 (50%) in daily wakefulness at the expense of slow-wave sleep (SWS).

191 as positively correlated with the amounts of slow-wave sleep (SWS).

192 d 5-HT declined progressively from waking to slow-wave-sleep (SWS) and then to rapid-eye-movement (RE

193 (PZ(Vgat)) neurons in behaving mice produces slow-wave-sleep (SWS), even in the absence of sleep defi

194 During slow-wave sleep, temporal coordination of hippocampal sh

195 ere positively correlated with the amount of slow-wave sleep that patients obtained between training

196 We propose that during slow-wave sleep the tight functional coupling between GA

197 In fact, during slow-wave sleep, the patterns of CA1 pyramidal cell ense

198 and spontaneous ventilation were observed in slow wave sleep time (45 min vs 28 min), rapid eye movem

199 reased nighttime sleep latency and increased slow-wave sleep time in cocaine-dependent participants.

200 ttern A human IgGs on rapid eye movement and slow-wave sleep time parameters in the inactive phase an

201 s bilaterally placed in the preoptic region, slow-wave sleep time was significantly decreased, but RE

202 Comparison of slow-wave sleep time, total sleep time, and sleep latenc

203 hypothesis is that neurons fire less during slow-wave sleep to recover from the "fatigue" accrued du

204 o sleep states, rapid eye movement sleep and slow-wave sleep, to offline memory processing.

205 reactivation of these representations during slow-wave sleep transforms episodic representations into

206 nd suppressed REM sleep time while increased slow wave sleep typifies the inactive phase, findings th

207 ep in an unusual manner, with unihemispheric slow wave sleep (USWS) and suppressed REM sleep, it is u

208 en RR neurons and SPW-R events in subsequent slow wave sleep was diminished.

209 t bursting occurs only during states such as slow-wave sleep, when little or no information is relaye

210 the first half of the night is dominated by slow-wave sleep, whereas during the second half, rapid e

211 by behavioural state, and was maximal during slow-wave sleep, which may explain the propensity for ne

212 d sounds were re-presented during subsequent slow-wave sleep while participants underwent functional

213 WS), as seen in all terrestrial mammals, and slow-wave sleep with interhemispheric electroencephalogr

214 Slow-wave sleep would thus begin with spindle oscillatio