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1 Periodic bursting is driven by a delayed and slowly decaying afterhyperpolarization (sAHP) current as
2                          These neurons had a slowly decaying afterhyperpolarization at light offset.
3 ong-range temporal correlations indicated by slowly decaying autocorrelation functions in neuronal ac
4  Ca(2+) transient, with the largest and most slowly decaying Ca(2+) transients in small-diameter, IB(
5 SR) and the Na+/Ca2+ exchanger in the small, slowly decaying Ca2+ transients of failing human ventric
6 z), however, resulted in an additional, more slowly decaying component (latency > 50 ms, duration > 2
7 e activated) that appeared to contain only a slowly decaying component (tau = 12 msec).
8 e difference may arise from integration of a slowly decaying component of the spike-mediated inhibiti
9 rent during repolarization developed a large slowly decaying component within 10 min.
10   This was due to a relative increase in the slowly decaying component, despite similar initial ampli
11 The pulse scheme is designed to preserve the slowly decaying components of both 1H-15N and methyl 13C
12 ons of image intensity consisted of fast and slowly decaying components.
13                                         Such slowly decaying currents could be evoked in PCs by actio
14                                              Slowly decaying currents were repeatedly evoked in ADP-
15        In some cells, choline activated also slowly decaying currents, confirming previous reports th
16                 Depending on the animal, the slowly-decaying excitation could be elicited by as littl
17                       In 14 of 16 animals, a slowly-decaying excitation could be evoked by bending.
18     In five of the nine animals tested, this slowly-decaying excitation could be evoked with bending
19                                            A slowly-decaying excitation was said to be present if aft
20  We report here that bending can also have a slowly-decaying excitatory effect on the CPG's frequency
21 slices, stimulation of mitral cells elicited slowly decaying, GABAA receptor-mediated reciprocal IPSC
22 (rapidly decaying) tunneling regime and the (slowly decaying) hopping regime in the general theory of
23  stimulus-induced dispersal, suggestive of a slowly decaying internal defensive state.
24 l- current in the ON segment and generated a slowly decaying inward OFF current in charge movement tr
25 icular nucleus (RTN) display a long-lasting, slowly decaying IPSC.
26 etagamma1 receptors, was responsible for the slowly decaying IPSCs.
27  early [Formula: see text] K-like state, the slowly decaying late intermediate [Formula: see text], a
28 zed and undergo Neel relaxation, producing a slowly decaying magnetic flux, which is detected by the
29  waves to neighboring astrocytes and trigger slowly decaying NMDA receptor-mediated inward currents i
30        During a long (2000 ms) TEST pulse, a slowly decaying ON current was also observed.
31 twi)betadeltagamma receptor isoform produces slowly decaying synaptic currents typical of SCS that tr
32 ytic release of glutamate, which activates a slowly decaying transient inward current (SIC) in CA1 py
33 ged from short, strongly damped responses to slowly decaying, weakly damped oscillations.
34                      In contrast, ACh evoked slowly decaying whole-cell currents that, being sensitiv

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