ライフサイエンスコーパス: slowness

1 beneath the same region also have anomalous slowness.

2 This slowness allows infection to become well established; th

3 inally, we investigate the trade-off between slowness and sparseness when combined in one objective f

4 In contrast to this claim, we here show that slowness and sparsity drive the representations towards

5 partial inactivation might produce weakness, slowness, and loss of independence of one or two adjacen

6 tion might produce dissociation of weakness, slowness, and relative independence of movement, and whi

7 teria: shrinking, exhaustion, sedentariness, slowness, and weakness.

8 The slowness anomalies are not accompanied by significant tr

9 eria (weight loss, exhaustion, low activity, slowness) at >/=2 visits, or at 1 visit with >/=1 criter

10 indings; other possible explanations include slowness due to comorbidities misinterpreted as bradykin

11 like phenotypes with dystonia, vertical gaze slowness, dysexecutive syndrome, predominant axial rigid

12 Parkinson's disease is associated with slowness, especially of sequential movements, and is cha

13 by resting tremor, rigidity, bradykinesia or slowness, gait disturbance, and postural instability.

14 Heritability was high for slowness in eating (84%; 95% CI: 83%, 86%) and satiety r

15 ite [enjoyment of food, food responsiveness, slowness in eating (SE), satiety responsiveness (SR), an

16 "enjoyment of food," "food responsiveness," "slowness in eating," and "satiety responsiveness." Herit

17 ing of lysozyme arises not from any inherent slowness in the formation of the native structure but ra

18 here, exhibiting extremes of "fastness" and "slowness" in their life histories.

19 with the only difference that SSA maximizes slowness instead of sparsity.

20 This unsupervised temporal slowness learning (UTL) was substantial, increased with

21 la, and the Mid-Atlantic Ridge have observed slownesses more than 64 percent greater than predicted b

22 otection is also poor because of the extreme slowness of an intramolecular Galpha refolding step (iso

23 ontribute significantly to the rate-limiting slowness of cellulose hydrolysis.

24 The slowness of concerted transfer of H* between Os(IV)NHPh

25 The mechanism of this dynamic slowness of contracted airways probably involves intrins

26 It started with slowness of gait and mood dysfunction.

27 Bradykinesia as defined by slowness of initiation of movement and a progressive red

28 Parkinson's disease defines bradykinesia as 'slowness of initiation with progressive reduction in spe

29 lymph node infiltration assay to address the slowness of metastasis of tumor xenografts.

30 se: an age-dependent and levodopa-responsive slowness of movement associated with diminished dopamine

31 n understanding the exact mechanisms driving slowness of movement has been impeded due to the heterog

32 The characteristic slowness of movement in Parkinson's disease relates to a

33 cardinal motor symptoms of tremor, rigidity, slowness of movement, and impairments of posture, gait,

34 disease causes symptoms including stiffness, slowness of movement, and tremor.

35 afflicts patients later in life with tremor, slowness of movement, gait instability, and rigidity.

36 of progressive mild left-sided weakness and slowness of movements.

37 bined effect of the conduction delay and the slowness of muscle contraction.

38 The slowness of nucleation can be reconciled with the simila

39 mechanistic insights on the counterintuitive slowness of PT at water-hydrophobe boundaries and its re

40 selectivity filter are at the origin of the slowness of recovery in K(+) channels.

41 Finally, the slowness of the lockdown process is dictated by the need

42 n intersystem barrier and is responsible for slowness of the spin-forbidden deprotonation of (1)HNO b

43 signal-to-noise phase, the arrival time and slowness of which agree with theoretical predictions for

44 PAs are an emergent phenomenon in which the "slowness" of the network is due to interactions between

45 ic latencies that obviate the impact of limb slowness on executing response decisions.

46 , known PD motor deficits characterized by a slowness or inability to switch between motor programs.

47 ve the conflict it emerges from (despite its slowness), or does it operate on future conflicts that i

48 ame properties may be equally derived from a slowness principle.

49 zophrenia is a consequence of depression and slowness, rather than a primary feature of the disease.

50 eurologic condition characterized by tremor, slowness, stiffness, and unstable posture.

51 quently showed signs of movement poverty and slowness, tremor and subtle cognitive deficits.

52 also could produce dissociated weakness and slowness versus loss of independence in a given finger m

53 teria-defined bradykinesia, characterized by slowness with progressive reduction in amplitude and spe