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1 bition of CYB5R3 increases NO bioactivity in small arteries.
2 density within the endothelial cell layer of small arteries.
3 g, kidney, and vasculature, most notably the small arteries.
4 owed 21.7 +/- 10.1 glomeruli and 5.0 +/- 2.8 small arteries.
5 ion of inflammation and high-risk plaques in small arteries.
6 acterized by obstructive vascular lesions in small arteries.
7 d levels of Rgs5 in their vascular plexa and small arteries.
8 NE in isolated human coronary arterioles and small arteries.
9 ally, by a greater hyperemic WSS stimulus in small arteries.
10 tionship could explain why FMD is greater in small arteries.
11 , right ventricular hypertrophy, and loss of small arteries.
12  on the pathobiology of lung capillaries and small arteries; 3) developing standard methods for asses
13  coronary circulation averaged 10 dyn/cm2 in small arteries and 19 dyn/cm2 in arterioles.
14 rization and constriction (myogenic tone) of small arteries and arterioles, and this response is a ke
15 ptions that are not necessarily fulfilled in small arteries and arterioles.
16  beta-amyloid in the media and adventitia of small arteries and capillaries of the leptomeninges and
17  large arteries and veins and more compliant small arteries and veins.
18 by different pathogenic mechanisms, of which small artery and large artery stroke are the most common
19 (MT) in isolated, pressurized rat mesenteric small arteries, and Ca2+ signalling in primary cultured
20 late best with a given function in large and small arteries, and that the genes in pulmonary veins wh
21 ted in 31% of brain injury patients, whereas small arteries are inferred to be abnormal in up to 69%
22 de or important neuroreceptors; whether many small arteries are occluded (especially downstream of na
23 pressure and intimal thickening of pulmonary small artery are a little more serious in nose-only CS e
24 exerts an anticontractile effect on adjacent small arteries, but this is lost in obesity-associated c
25 erstand how the cross-talk between large and small artery changes interacts in pressure wave transmis
26 e a potential target for overcoming diabetic small artery complications.
27                                              Small artery contractile function was examined in vitro
28  EPHB4 plays an essential role in regulating small artery contractility and blood pressure.
29 osterone, play a critical role in regulating small artery contractility and BP.
30  that isolated human coronary arterioles and small arteries dilate to NE via beta2-receptors on smoot
31   Regulation of shear stress was observed in small arteries during adenosine-induced increases in cor
32 s but completely abolished its regulation in small arteries during vasodilation.
33 rkers like large artery elasticity (LAE) and small artery elasticity (SAE) may predict cardiovascular
34 sk score reduction was due to an increase in small artery elasticity and a decrease in BP, and after
35     A panel of 10 tests, including large and small artery elasticity, resting and treadmill exercise
36  marginally significant, by using large- and small-artery elasticity measurements.
37                                              Small arteries exhibit tone, a partially contracted stat
38                           We discovered that small arteries from castrated Ephb6 gene KO males showed
39 oprusside) of isolated, pressurized coronary small arteries from lean control and Zucker obese fatty
40  (10(-9)-10(-5)m) was impaired in mesenteric small arteries from male and female OHF compared with of
41                                              Small arteries from patients with EH demonstrated eutrop
42  results demonstrate vascular hypertrophy in small arteries from patients with type 2D.
43                        Arterioles (n=39) and small arteries from the left ventricle of explanted huma
44 estigate the effects of bariatric surgery on small artery function and the mechanisms underlying this
45 ls given to hypercholesterolemic patients on small artery function in vitro.
46  a diet based on soy protein, but influences small artery function.
47 ng neurally stimulated contraction of intact small arteries have not yet been recorded.
48  improved endothelial function in peripheral small arteries in hypercholesterolemia patients.
49 the increased media/lumen ratio of large and small arteries in hypertension and increase arterial com
50                           5-LO expression in small arteries in PPH was more intense than in control a
51 traluminal transmural pressure of mesenteric small arteries in vitro results in a myogenic response t
52 te and areas of intimal proliferation within small arteries indicative of early transplant arterioscl
53     Furthermore, the greater FMD response in small arteries is accounted for, at least partially, by
54 netic hypertension, the altered structure of small arteries is due to either cellular hyperplasia or
55                Involvement of both large and small arteries is more common than that of either alone.
56                               Contraction of small arteries is regulated by the sympathetic nervous s
57 s (thrombi in glomerular capillary loops and small arteries, mesangiolysis) and ultrastructural lesio
58 ular pH (pH1) were studied in rat mesenteric small arteries mounted on a wire myograph.
59 tor effects of troglitazone and BRL 49653 in small arteries (n = 44) from human subcutaneous fat.
60 iking reduction and impairment of the distal small artery network in the heart and kidney.
61 umber of core biopsy samples, glomeruli, and small arteries obtained during 294 consecutive US-guided
62    Here we study blood pressure and isolated small arteries of mice with reduced expression of Na(+)
63  factor in hypertension, including large and small artery remodeling and functional changes.
64 aled NO, particularly in conditions in which small arteries represent the site of resistance.
65  noradrenaline-preconstricted rat mesenteric small arteries (RMSAs, i.d. 200-300 microm) was studied
66 ntly greater decrease in gradient across the small artery segment after inhaled NO and zaprinast comp
67 ques that improve the visualization of PE in small arteries should be used.
68 e analysis of smooth muscle abundance around small arteries (SMart) and terminal bronchioles (SMtb).
69                         Aortic stiffness and small-artery structure and function share various risk f
70 4 fluorescence in pressurized rat mesenteric small arteries subjected to low-frequency electrical fie
71 rast, is less hypoxic as it is perfused with small arteries that are often positive for the marker ne
72               Furthermore, FMD is greater in small arteries, though the reasons for this phenomenon a
73  major stimulus for the myogenic response of small arteries to pressure.
74 e smooth muscle cells of intact, functioning small arteries, together with measurement of arterial fo
75 evoke spreading dilatation in rat resistance small arteries under physiological pressure and flow.
76                     Reactivity of mesenteric small arteries was assessed by myography, and responses
77 ulin-dependent) diabetes mellitus (type 2D), small arteries were examined in vitro from carefully def
78                                              Small arteries were mounted on a wire myograph for isome
79                     We loaded rat mesenteric small arteries with the fluorescent Ca2+ indicator fluo-

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