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1 me closely mirrored the transcriptome of the small intestinal mucosa.
2 mediated inhibition of TLR4 signaling in the small intestinal mucosa.
3 elial or crypt cell regions of healthy human small intestinal mucosa.
4 in human intestinal Caco-2 cells and rodent small intestinal mucosa.
5 s in splenocytes and epithelial cells of the small intestinal mucosa.
6 orizin hydrolase (LPH) was purified from rat small intestinal mucosa.
7 the fractalkine receptor CX3CR1 in the human small intestinal mucosa.
8 hesion within the healthy and diseased human small intestinal mucosa.
9 ance of normal rates of protein synthesis in small intestinal mucosa.
10 ions are composed of heterotopic stomach and small intestinal mucosa.
11 ogically mediated inflammatory damage to the small-intestinal mucosa.
12 compared these data to RNA-Seq from both the small intestinal mucosa and colonic mucosa of healthy co
13 impact on the structure and function of the small intestinal mucosa and suggest signaling through th
14 orption, morphology and DNA proliferation of small intestinal mucosa, and gastrointestinal transit we
15 homeobox genes are expressed in human adult small intestinal mucosa, and some are found predominantl
16 n the luminal aspect of the bacteria-exposed small-intestinal mucosa, and was followed by a decrease
19 is a syndrome characterized by damage of the small intestinal mucosa caused by the gliadin fraction o
20 is a syndrome characterized by damage of the small intestinal mucosa caused by the gluten fraction of
21 tissue, the decline in the synthesis rate of small intestinal mucosa during insulin deprivation may b
23 29b-reduced endogenous miR-29b levels in the small intestinal mucosa increases cyclin-dependent kinas
26 studies reveal that growth inhibition of the small intestinal mucosa is associated with increased exp
27 earch into the structure and function of the small intestinal mucosa is becoming increasingly focused
30 of the molecular and cellular biology of the small-intestinal mucosa is providing insights into the r
33 of DQ2-mediated antigen presentation in the small intestinal mucosa of Celiac Sprue patients therefo
34 G2 can be activated by dietary gluten in the small intestinal mucosa of celiac sprue patients, our fi
35 n (GFP)-tagged gammadelta T cells within the small intestinal mucosa of mice infected with DsRed-labe
36 the gammadelta TCR are more abundant in the small intestinal mucosa of patients with celiac disease
38 messenger RNA targeting relationships in the small intestinal mucosa provides insight into the molecu
39 an villous height/crypt depth (Vh/Cd) in the small-intestinal mucosa was significantly lower and the
40 , dramatic, and reversible remodeling of the small intestinal mucosa with significant villus shorteni
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