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1 ides, suggestive of a role for TM6SF2 in the small intestine.
2 then contrasted with what is known about the small intestine.
3 not previously demonstrated in the mammalian small intestine.
4 nce of segmented filamentous bacteria in the small intestine.
5 n spectra compared to those of the colon and small intestine.
6 olon cancer, being largely restricted to the small intestine.
7 and subsequent protein digestibility in the small intestine.
8 cause significant lipid accumulation in the small intestine.
9 inflammatory cytokines and Th17 cells in the small intestine.
10 e distribution of metformin in the liver and small intestine.
11 ntly more tumors in the colon and the entire small intestine.
12 nvironmental samples, mouse feces, and mouse small intestine.
13 nct gluten-degradation patterns in the mouse small intestine.
14 mas, while the bulk of the disease is in the small intestine.
15 he transport and absorption of lipids in the small intestine.
16 ver, and a putative target of metformin, the small intestine.
17 liver but did not affect distribution in the small intestine.
18 and reduced expression of granzyme B in the small intestine.
19 ainst microbes and food Ags derived from the small intestine.
20 ddition to increasing markers of AAMs in the small intestine.
21 an digestion processes in mouth, stomach and small intestine.
22 ters and promotes fructose absorption by the small intestine.
23 pancreas ducts where they join and enter the small intestine.
24 of NCR(-) and NCR(+) ILC3 subsets within the small intestine.
25 rofold cells (M-cells) in Peyer's patches of small intestine.
26 , and 7), to deleteLpcat3specifically in the small intestine.
27 nsumption induces expression of Txnip in the small intestine.
28 TG2 as its principal protein partner in the small intestine.
29 lial cells overlying lymphoid tissues in the small intestine.
30 rate-limiting for glucose absorption in the small intestine.
31 mbling the segmentation motor pattern of the small intestine.
32 lack of interference by radioactivity in the small intestine.
33 nsferase (GPAT3) was examined throughout the small intestine.
34 key discoveries regarding stem cells of the small intestine.
35 of hypoglycaemia following infusion into the small intestine.
36 homeostasis on the epithelial surface of the small intestine.
37 ort processes along the vertical axis of the small intestine.
38 the accumulation of genetic mutations in the small intestine.
39 susceptible to IR-induced tissue injury than small intestine.
40 ie many of the phenotypes seen in the mutant small intestine.
41 tty acids and lipid metabolites in the mouse small intestine.
42 immune system, facilitating colonization of small intestine.
43 l role in looping morphogenesis of the avian small intestine.
44 ntestinal tract (GIT) model: mouth; stomach; small intestine.
45 uniquely altered in the chronically inflamed small intestine.
46 d GLP-1 secretion from isolated perfused rat small intestine.
47 ystem, including the esophagus, stomach, and small intestine.
48 g reaching the surfaces between villi in the small intestine.
49 induces a massive secretion of 26RFa by the small intestine.
50 a and colonize the epithelial surface of the small intestine.
51 rpa1 as an L cell-enriched transcript in the small intestine.
52 en and mucosa and moderately with the distal small intestine.
53 s well as inflammation and remodeling of the small intestine.
54 enic Th1 and/or Th17 cell populations in the small intestine.
55 o express SLAMF4 only after migrating to the small intestine.
56 d increased expression of ABCG5 and ABCG8 in small intestine.
57 hich emptied into and progressed through the small intestine.
58 ate of delivery of nutrients to the proximal small intestine.
59 cally lacked FDC-containing GALT only in the small intestine.
60 he primary site of injury, namely the distal small intestine.
61 h1 ChIP-seq datasets from the cerebellum and small intestine.
62 ed in brush-border membranes from kidney and small intestine.
63 a propria phagocytes and inflammation in the small intestine.
64 xpression in organotypic cultures of Ano1 WT small intestine.
65 nt role in maintaining the physiology of the small intestine.
66 ed in loss of slow waves in smooth muscle of small intestine.
67 low levels in the colon as compared with the small intestine.
68 e mouse stomach and colon and from the human small intestine.
69 act with and modulate the environment in the small intestine.
70 y decrease their intracellular levels in the small intestine.
71 d unsaturated fatty acids found in the upper small intestine.
72 ltaneously deleted Chk1 and Apc in the mouse small intestine.
73 y intact anthocyanins were absorbed from the small intestine.
74 tes along the proximal-to-distal axis of the small intestine.
75 g glucose-induced GLP-1 secretion from human small intestine.
76 er cholesterol transporters, in the proximal small intestine.
77 laughter age, but reduces clearance from the small intestine.
78 ed information on survivin expression in the small intestine.
79 , and cholecystokinin (CCK), secreted by the small intestine.
80 irectly by modulation of BMP activity in the small intestine.
81 s immune-mediated inflammatory damage to the small intestine.
82 T cells (T(EM)) found only in the lungs and small intestine.
83 mportantly, no adenomas were observed in the small intestine.
84 in the apical surface of enterocytes in the small intestine.
85 YY (PYY), from enteroendocrine cells in the small intestine.
86 nd food crops was found to be highest in the small intestine.
87 eferentially enzymatically hydrolyzed in the small intestine.
88 postnatal formed lymphoid tissues within the small intestines.
89 sis, are also influenced by vitamin A in the small intestines.
90 ducing microbiota and SCFA production in the small intestines.
91 w lumen, with adaptability to both large and small intestines.
92 both colon (1.5-fold increase, P < 0.05) and small intestine (1.5-fold increase, P < 0.05) after 30 a
93 lso found in the subepithelial region of the small intestine 24h after pollens were gavaged to mice.
94 tly decreased (11)C-donepezil binding in the small intestine (-35%; P = 0.003) and pancreas (-22%; P
95 n was found to be up-regulated in the distal small intestine, a tissue involved in chylomicron synthe
97 ls developed exacerbated inflammation in the small intestine after challenge with anti-CD3 antibodies
98 We showed that upregulation of SGLT1 in the small intestine after glucose ingestion is promoted by g
99 (TG) are absorbed by the enterocytes of the small intestine after luminal hydrolysis into monacylgly
100 monstrated that microcapsules released Lf in small intestine allowing 6.5 times higher concentration
101 quitous, but poorly defined, disorder of the small intestine among people living in conditions of pov
102 The increasing pH as the yogurt enters the small intestine and a slower gastrointestinal transit ti
103 s taken up by dendritic cells throughout the small intestine and additionally by F4/80(+) cells in th
104 in primary cultures of enteroids from human small intestine and anion current in enteroid monolayers
109 ed by mesenteric ischemia/reperfusion in the small intestine and by dextran sulfate sodium in the col
110 2(IEKO)) led to malignant tumors in both the small intestine and colon in 86% of the mice and to meta
111 on intestinal epithelial cells (IECs) in the small intestine and colon is required for enteric IFN-la
113 fferentiation into Th2 and Th17 cells in the small intestine and colon, and inhibits T-cell different
114 known to be a stem cell marker in the murine small intestine and colon, however the localization of L
115 ial alimentary tract, including the stomach, small intestine and colon, was analyzed in normal human
119 the intestinal epithelial cells in the host small intestine and has evolved strategies to antagonize
120 th the deep muscular plexus (ICC-DMP) in the small intestine and ICC associated with the submucosal p
122 er investigate the role of Chk1 in the mouse small intestine and its potential role as a therapy for
123 was used to determine the gastrointestinal (small intestine and liver) retention, and presumably met
125 , although is involved in clearance from the small intestine and longer-term clearance from the ceca.
126 nal accessibility during passage through the small intestine and modulated the formation of the degra
127 53,193 individual epithelial cells from the small intestine and organoids of mice, which enabled the
129 n reduced numbers of parasitic adults in the small intestine and reduced larval output throughout inf
130 in specialized enteroendocrine cells of the small intestine and released by fat ingestion, facilitat
131 the induction of the IL-1beta pathway in the small intestine and reversed by treatment with an exogen
135 visceral fat and decreased the length of the small intestine and the circumference of the ileum.
138 s are one of 4 secretory cell linages in the small intestine and the source of IL-25, a critical init
139 enterocyte surface in the mid-region of the small intestine and translocate through the epithelial c
140 on-like peptide-1 (GLP-1) is produced in the small intestines and in nucleus tractus solitarius (NTS)
141 ve tract (mouth, throat, esophagus, stomach, small intestine, and colorectum) and digestive accessory
142 e of homeostasis and proper functions in the small intestine, and dysregulated miRNAs and proteins al
145 lling associated with type 2 immunity in the small intestine, and perhaps at other mucosal barriers p
146 luding the kidney, liver, lung, lymph nodes, small intestine, and spleen, confirmed that reciprocal d
147 r suppressor in the mouse colon, but not the small intestine, and that invasive ARID1A-deficient aden
150 oth the mRNA and protein levels revealed the small intestine as the major site of protein expression
152 ice had increased expression of Txnip in the small intestine as well as enhanced fructose uptake and
153 ls caused significant mucosal atrophy in the small intestine, as indicated by decreased cell prolifer
154 develop remarkable dilation of the stomach, small intestine, bladder, and ureters attributable to th
156 he lung, airways, lamina propria (LP) of the small intestine, brain, visceral adipose tissue, bone ma
158 provides a noninvasive option to assess the small intestine, but its use with respect to endoscopic
159 e III secretion system (T3SS2) to infect the small intestine, but knowledge of additional factors tha
160 s study, we studied PC dynamics in the human small intestine by cell-turnover analysis in organ trans
161 s as models for pathogenic infections in the small intestine by examining whether HIEs from different
164 e number and tightness of loops in the chick small intestine can be increased or decreased directly b
166 sease (CD) is an inflammatory disease of the small intestine caused by an immunologic hypersensitivit
167 iscovered that Giardia's colonization of the small intestine causes a systemic dysbiosis of aerobic a
168 e salts glycocholate and taurocholate in the small intestine causes dimerization of the transmembrane
169 genome-wide mutation patterns in ASCs of the small intestine, colon and liver of human donors with ag
170 or primordial germ cell-like cells and E18.5 small intestine, combined with functional annotation ana
171 reased expression of interferon gamma in the small intestine compared to wild-type mice exposed to sm
174 the derived COs represent colon rather than small intestine, containing stem cells, transit-amplifyi
176 e liver, upper large intestine contents, and small intestine contents between rest and stress imaging
181 duction of this key cytokine by ILC3s in the small intestine during development and under basal condi
184 at the slightly alkaline pHs simulating the small intestine environment, suggesting a potential of t
185 FAM7A transcript found in normal human fetal small intestine epithelial (FHs) cells to prove its iden
187 ence of dendritic cells and TRM cells in the small intestine epithelium also required alphavbeta6.
190 suggest that the absorptive capacity of the small intestine for fructose is limited, though the mole
191 g whether HIEs from different regions of the small intestine from different patients are susceptible
193 absorbed doses to the upper large intestine, small intestine, gallbladder wall, and lower large intes
194 of the deep muscular plexus (ICC-DMP) in the small intestine generate spontaneous Ca(2+) transients t
195 with the OA patients, more AYA patients had small-intestine GISTs (139 [35.5%] vs 1465 [27.3%], P =
198 of Cajal in the deep muscular plexus of the small intestine (ICC-DMP) are closely associated with va
199 y data from the Cftr-deficient colon and the small intestine identified dysregulated genes that belon
201 ls in stool as a means to surveil the distal small intestine in a murine model of NSAID enteropathy.
204 d on T lymphocytes mediates migration to the small intestine in response to a gradient of CCL25.
205 ne surveillance is distinct from that of the small intestine in terms of draining LNs, and identify p
206 an velocity of the magnetic pill through the small intestine in the fasting state was not significant
207 etary lipids are efficiently absorbed by the small intestine, incorporated into triglyceride-rich lip
209 changes in the myenteric innervation of the small intestine, intestinal wall and mucosal cells respo
213 ic cells (FDC) in the Peyer's patches in the small intestine is essential for the efficient spread of
217 riphery, particularly to Peyer's patches and small intestine lamina propria, where they upregulate LA
218 l dysbiosis alters immune homeostasis in the small intestine, leading to an increase in regulatory T
221 h tumors of the stomach and small intestine, small-intestine location was associated with improved su
222 images with thickened walls adjacent to the small intestine loops, and increase in the echogenicity
226 ems TM6SF2 deficiency resulted in defects in small intestine metabolism in response to dietary lipids
227 t but rather inhibiting gastric emptying and small intestine motility and strongly suggest the involv
228 stasis, and basic functions performed by the small intestine need to be tightly preserved to maintain
230 conjugates of insulin were absorbed from the small intestine of anaesthetised rats by means of the bi
231 observed an enlarged Th17 population in the small intestine of C57BL/6.IgA(-/-) mice compared with w
232 esent study, primary IECs were isolated from small intestine of C57BL/6J mice fed a low-fat diet (LFD
233 C57BL/6 mice with bacteria isolated from the small intestine of CD patients or healthy controls, sele
234 at an HFD promotes tumour progression in the small intestine of genetically susceptible, K-ras(G12Din
236 f both NCR(-) and NCR(+) ILC3 subsets in the small intestine of mice raised on a vitamin A-deficient
237 sion levels were determined in the liver and small intestine of neonatal hUGT1 mice that were orally
238 NS progenitors from tunica muscularis of the small intestine of newborn (postnatal day 0) wild-type C
239 We analyzed human proteins present in the small intestine of patients infected with V. cholerae O1
240 transglutaminase 2 (TG2) are abundant in the small intestine of patients with active celiac disease (
241 iardia colonization and proliferation in the small intestine of the host may, however, disrupt the ec
242 tion, bleeding, and oedema in the stomach or small intestine of wild-type (WT) mice; however, such se
243 Anti-CD3 aggravated inflammation in the small intestines of Slamf4(-/-) mice and Eat2a(-/-)Eat2b
247 helial and lamina propria lymphocytes of the small intestine, Peyer's patches, and mesenteric lymph n
248 mall droplets were fully digested within the small intestine phase, and led to a higher bioaccessibil
249 ethylated cytosine residues in the colon and small intestine, probably reflecting their high ASC divi
250 nstration of OCT-mediated transport into the small intestine provides evidence of direct effects of m
256 g of dietary triacylglycerol in the proximal small intestine results in physiological, hormonal and b
260 nt DCs had reduced numbers of T cells in the small intestine (SI), but not large intestine (LI), incl
262 6SF2 is expressed predominantly in liver and small intestine, sites for triglyceride-rich lipoprotein
263 AYA patients with tumors of the stomach and small intestine, small-intestine location was associated
264 e expression in ILCs and NK cells from mouse small intestine, spleen and liver, as part of the Immuno
265 term expansion of adult primary tissues from small intestine, stomach, liver and pancreas into self-a
266 ecovery of viable virus from mouse feces and small intestine suggest that these pests may play a role
267 f TM6SF2 protein being 10-fold higher in the small intestine than in the liver, dietary lipid absorpt
268 Celiac disease (CD) is a disease of the small intestine that occurs in genetically susceptible s
271 ts pro-apoptotic activity in mouse thymi and small intestines, the chromosomal instability caused by
272 lood via inhibition of its absorption in the small intestine, thus protecting against heart attack an
273 3 to -607 of the proximal Npc1l1 promoter in small intestine tissues from 24-week-old offspring fed e
274 cell confocal imaging or by cytofluorometry; small intestine tissues were analyzed by histology and i
276 gh the delivery of nutrients into the distal small intestine to promote satiety and suppress food int
278 used stem cell-derived enteroids from human small intestines to study enterovirus infections of the
279 ptosis in the myenteric ganglia of the adult small intestine, total myenteric neuronal numbers remain
280 We achieved full aPKC down-regulation in small intestine villi and colon surface epithelium using
281 tagenicity in site-of-contact tissues (e.g., small intestine) was generally more than dose-additive a
284 e of the lysophosphatidic acids (LPA) in the small intestine were also increased in response to UFP e
285 on, animals were euthanized and stomachs and small intestines were processed as whole mounts (submuco
286 intricate organs (esophagus, lung, liver and small intestine) were imaged with x-ray phase contrast c
287 s expressed at the crypt base throughout the small intestine, where it is enriched in crypt base colu
288 rtening, and inflammatory responses in mouse small intestine, whereas administration of D-4F attenuat
289 b(+)CD103(+) DCs induce Th2 responses in the small intestine, whereas CD11b(+)CD103(-) DCs perform th
290 wever, little is known about its role in the small intestine, which absorbs and senses nutrients.
291 for downregulation of glucose absorption in small intestine, which has been proposed as strategy for
293 HRH in T1D may improve GLP-1 function in the small intestine, which, in turn, diminishes TRL and redu
294 Streptococcus) were found in the stomach and small intestine, while anaerobic Lachnospiraceae and Rum
295 genesis, is expressed by Paneth cells in the small intestine, while group X sPLA2 is expressed by Pan
296 mulated cholecystokinin (CCK) hormone in the small intestine, while ILDR1 in EpH4 mouse mammary epith
297 to increased nutrient delivery to the lower small intestine with release of satiety-promoting gut ho
298 ocrine cells in both crypts and villi of the small intestine, with no changes observed in goblet or P
299 , permitting proper placement of the lengthy small intestine within the confines of the body cavity.
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