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1 antineutrophil cytoplasm antibody-associated small vessel vasculitis.
2 he PR3 ANCA immune response in patients with small vessel vasculitis.
3 Strauss Syndrome is an eosinophil-associated small vessel vasculitis.
4 ealing, and open lung biopsy revealed active small vessel vasculitis.
5  ischemic infarctions, presumed secondary to small vessel vasculitis.
6 arthritis, systemic lupus erythematosus, and small vessel vasculitis.
7 ic antibodies (ANCAs) can result in systemic small vessel vasculitis.
8  (ANCA) causes vascular injury that leads to small-vessel vasculitis.
9         A large number of entities can mimic small-vessel vasculitis.
10 ophil cytoplasmic antibody (ANCA)-associated small-vessel vasculitis.
11 ten than other patients with ANCA-associated small-vessel vasculitis.
12 th polyarteritis nodosa and one patient with small-vessel vasculitis.
13 patients with clinical manifestations due to small-vessel vasculitis.
14 rysms of renal and hepatic arteries, whereas small vessel vasculitis affected the skin and glomeruli,
15            NOTCH activation did not occur in small-vessel vasculitis affecting branches of the vasa v
16 ophil cytoplasmic antibody (ANCA)-associated small-vessel vasculitis (ANCA-SVV) and to gather evidenc
17 nase 3 are detected in sera of patients with small vessel vasculitis and participate in the pathogene
18 ic or soft plaques of the coronary arteries, small vessel vasculitis and small aneurysm.
19 een consistently detected in ANCA-associated small-vessel vasculitis, and this association prompted u
20 angiitis (Churg-Strauss, EGPA) is a systemic small-vessel vasculitis associated with asthma and eosin
21 neutrophil cytoplasm autoantibody-associated small vessel vasculitis based on antineutrophil cytoplas
22 strointestinal disease, febrile attacks, and small-vessel vasculitis characteristic of Behcet disease
23 entic glomerulonephritis (NCGN) and systemic small vessel vasculitis in humans.
24  vascular lesions associated with autoimmune small-vessel vasculitis may arise from activation of cir
25 e IgG (ANCA IgG), derived from patients with small-vessel vasculitis, on neutrophil adhesion.
26 omerular lesions with crescents, mimicking a small vessel vasculitis such as ANCA-associated GN, are
27 d crescentic glomerulonephritis and systemic small vessel vasculitis, such as microscopic polyangiiti
28  microdissected glomeruli from patients with small vessel vasculitis (SVV) had markedly higher levels
29 l cytoplasmic autoantibody (ANCA)-associated small-vessel vasculitis (SVV) and systemic lupus erythem
30    Microscopic polyangiitis is an autoimmune small-vessel vasculitis that often manifests as focal an
31  three patients with polyarteritis nodosa or small-vessel vasculitis were homozygous for the p.Gly47A

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