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1 have also been implicated, including tobacco smoke.
2 xposures to secondhand and thirdhand tobacco smoke.
3 g relating to characteristics of parents who smoke.
4 eutic targets for breast cancer patients who smoke.
5 ression with adjustment for age, gender, and smoking.
6 nvolved in the reinforcing effect of tobacco smoking.
7 ysical inactivity, and 4.8 years for current smoking.
8 smoking initiation and past 30-day cigarette smoking.
9  offer a lower risk alternative to cigarette smoking.
10  previously been found to be associated with smoking.
11 e, Barrett's esophagus, obesity, and tobacco smoking.
12 e due to correlation between EA and maternal smoking.
13 the disease are differentially methylated by smoking.
14 d after overnight abstinence and again after smoking.
15 phageal reflux disease, central obesity, and smoking.
16 d rs1079597, with an adjustment for habitual smoking.
17 d educate patients regarding ocular risks of smoking.
18 te its efficacy during acute abstinence from smoking.
19  for > 60 pack-years of smoking versus never smoking.
20 -a disorder that can be accompanied by heavy smoking.
21  modified by parity, latitude, fetal sex, or smoking.
22  current nicotine dependence (DSM criteria), smoking 10 or more cigarettes per day, and a willingness
23  women was 1.3% (95% CI 0.8-1.9) for tobacco smoking, 2.1% (1.1-3.4) for smokeless tobacco use, and 3
24 h severe COPD than in controls who had never smoked (3166+/-402 vs. 1515+/-152 mug per milliliter) an
25                            The prevalence of smoking (42.6% to 36.5%; RR = 0.86) and sedentary lifest
26 likely men (97% vs 92%), more likely to have smoked (57% vs 45%), and less likely to be overweight/ob
27                       Furthermore, cigarette smoke, a leading risk factor for lung cancer, was also i
28 otine infusion study that followed overnight smoking abstinence.
29 IOG were positively correlated with years of smoking across the smokers.
30 i (15q25.1 and 19q13.2) were associated with smoking adjusted lung function, and 15q25.1 reached geno
31 iations, with the exception of daily tobacco smoking (adjusted OR 1.74, 95% CI 1.08-2.81), any illici
32 s (adjusted OR, 7.3; P = .007), and parental smoking (adjusted OR, 3.8; P = .028).
33 ckly, following the addition of ozone to the smoke aerosol, and ClNO2(g) production promptly followed
34                We aimed at investigating how smoking affects blood DNA methylation in MS patients, by
35 inner than in children whose mothers had not smoked after correction for age, sex, birth weight, heig
36 for age, height, weight, pack-years, current smoking, age at completed full-time education, spiromete
37  to estimate hypothetical effects of habits (smoking, alcohol consumption, and fat and carbohydrates
38                      Excluding the factor of smoking, an exogenous source of glyoxal, levels of these
39 n and tissue remodeling induced by cigarette smoke and highlight another potential target to modulate
40 This paper provides an overview of cigarette smoke and urban air pollution, considering how their com
41 adjustment for lifetime number of cigarettes smoked and secondhand smoke exposure.
42 married, living in the rural area, cigarette smoking and alcohol drinking were associated with insomn
43      Several lifestyle factors, most notably smoking and alcohol, are associated with SP risk.
44 s significant relationships between maternal smoking and ASD risk.
45 s examining the association between maternal smoking and autism spectrum disorder (ASD) in offspring
46 n on the risks of nicotine dependence, heavy smoking and consequent lung cancer.
47 , this "risk paradox" is partly explained by smoking and disease-associated weight loss.
48           Frequency of combustible cigarette smoking and e-cigarette use within the past 30 days (0 d
49 ion of rs660895-G with PD, and suggests that smoking and HLA-DRB1 are involved in common pathways, po
50 actors alcohol consumption, body mass index, smoking and maternal smoking during pregnancy.
51 es examined the association between prenatal smoking and NEC-associated infant mortality rates with a
52 ortance of distinct contributing factors for smoking and non-smoking lung cancer.
53                                         Both smoking and non-smoking patients were studied, to determ
54 h lower total mortality after adjustment for smoking and other potential confounders (1 cup per day:
55 cts or from confounding influences shared by smoking and severe mental illness.
56 oms as children were more likely to initiate smoking and to start smoking younger.
57 ected men ages 40-60, independent of tobacco smoking and traditional risk factors.
58    Sociodemographic, mental/physical health, smoking and treatment characteristics (nicotine replacem
59       CVRF freedom was defined as no current smoking and untreated blood pressure <140/90 mm Hg, fast
60 >/=3 days [frequent]) and daily intensity of smoking and vaping (number of cigarettes smoked per day,
61 s in the United States, with inhalation (ie, smoking and vaping) being one of the most common routes
62 crease subsequent frequency and intensity of smoking and vaping.
63 uency and intensity of combustible cigarette smoking and vaping.
64 ions with seropositivity were similar across smoking and/or drinking exposure groups: HRfor low expos
65 faces (including sofas), exposure to tobacco smoke, and prematurity; protective factors include breas
66 nd validate a novel approach to the study of smoking, and addiction more generally.
67 nicity, modified Charlson comorbidity index, smoking, and alcohol use.
68 , educate patients regarding ocular risks of smoking, and discuss with patients smoking cessation opt
69 omoting oxidative stress: diabetes, obesity, smoking, and excessive pollution.
70 ere observed with poor oral hygiene, tobacco smoking, and oral cancer.
71 .58) after adjustment for baseline LTL, age, smoking, and percentage of body fat.
72 levated blood pressure, physical inactivity, smoking, and poor glucose control) are associated with i
73 1.35, 0.09), controlling for sex, age, site, smoking, and race/ethnicity.
74 rted information on diet, physical activity, smoking, and stress during early pregnancy.
75 stinctive sensory and behavioural aspects of smoking, and understanding the neural effects of such pr
76 t, sedentary lifestyle, alcohol consumption, smoking, and urinary sodium excretion levels measured at
77 cal activity, diet, alcohol consumption, and smoking-and cognition, adjusting for demographic factors
78 e UK Biobank (N=48,931) using heavy vs never smoking as a proxy phenotype (P=3.6 x 10(-4), OR=1.05, a
79 ienced greater alleviation of their urges to smoke, as assessed by the Brief Questionnaire on Smoking
80 tus for lung adenocarcinoma risk among never-smoking Asian women, we conducted a meta-analysis of 11
81 ood pressure (BP), adverse serum lipids, and smoking associate with cognitive deficits.
82 ears) of systolic BP, total-cholesterol, and smoking associated inversely with midlife visual and epi
83                                              Smoking-associated DNA hypomethylation has been observed
84                                        Thus, smoking-associated hypomethylation may be a consequence
85 ess onset, exposure to environmental tobacco smoke, bacterial colonization, and breastfeeding were as
86 data identifies the same cortical region for smoking behavior as found with fMRI data.
87       We also studied 5 loci associated with smoking behavior.
88 n ages 43 and 60-64 years were influenced by smoking behavior.
89            We conclude that varenicline as a smoking cessation agent differs from nicotine through tr
90  risks of smoking, and discuss with patients smoking cessation options.
91 udies were included if they reported nurses' smoking cessation practices in relation to their persona
92  status (slow/normal metabolism) may improve smoking cessation rates.
93 slation (n=35), tobacco taxation (n=11), and smoking cessation services (n=3).
94 ettes per day, pack-years smoked, time since smoking cessation) as risk predictors.
95  nAChR PAM compound for possible efficacy in smoking cessation, in smokers with or without schizophre
96 ) of alpha7 receptors, JNJ-39393406, may aid smoking cessation.
97 her be studied in a larger clinical trial of smoking cessation.
98 reduction, increasing physical activity, and smoking cessation.
99 rettes per day, and a willingness to attempt smoking cessation.
100  5 days a week, for 8 weeks, in a ventilated smoking chamber, or ambient air (controls).
101 a decreased frequency of SSI in patients who smoke cigarettes and to confirm that smoking is signific
102                          Mild asthmatics who smoke cigarettes may develop unstable disease and neutro
103 signatures derived from the smoke of machine-smoked cigarettes.
104        In 17 patients and 10 age-matched non-smoking control subjects we examined brachial artery flo
105 in cholesterol), and clinical variables (C) (smoking, diabetes mellitus, and peripheral arterial dise
106                                     Obesity, smoking, diabetes, hypertension, and cardiac and metabol
107  factors were measured at baseline (obesity, smoking, diabetes, prehypertension, hypertension, and hy
108 arital status, material deprivation history, smoking, drinking and socioeconomic status, working-age
109 aspects of smoking exposure (smoking status, smoking duration, cigarettes per day, pack-years smoked,
110 eports suggest that offspring of mothers who smoke during pregnancy have greater risk of developing d
111            In 227 children whose mothers had smoked during pregnancy, the peripapillary RNFL was 5.7
112                                Both maternal smoking during pregnancy and low birth weight have been
113           To examine the association between smoking during pregnancy and severe mental illness in of
114 methylation changes associated with maternal smoking during pregnancy have been described in placenta
115 strong support for a causal role of maternal smoking during pregnancy in offspring depression, rather
116 ailed to find support for a causal effect of smoking during pregnancy on risk of severe mental illnes
117                                     Maternal smoking during pregnancy was associated with risk of TD/
118 nown to be strongly associated with maternal smoking during pregnancy, and thus their association wit
119 ption, body mass index, smoking and maternal smoking during pregnancy.
120                         Exposure to maternal smoking during uterine life and low birth weight were in
121              A significant CYP2A6 genotype x smoking effect was found in the dorsal anterior cingulat
122                                      Besides smoking, epidemiological studies have linked female sex
123 evalent hypertension, diabetes mellitus, and smoking exist within states and within counties in the c
124 found altered macrophage polarization in the smoke-exposed Mmp28(-/-) lung consistent with other publ
125                                     Maternal smoke exposure decreases mesenchymal proliferation and m
126 d impact on gene expression compared to 3R4F smoke exposure in vitro.
127 ess about the detrimental effects of tobacco smoke exposure on children's respiratory health.
128 evidence for positive association of tobacco smoke exposure with rhinitis, while the effect is mainly
129                                      Tobacco smoke exposure, lung function at 6 weeks, infant growth,
130 e number of cigarettes smoked and secondhand smoke exposure.
131 models considered age and various aspects of smoking exposure (smoking status, smoking duration, ciga
132 alth effects from air pollution and wildfire smoke exposures.
133 tudied the effects of TNFalpha and cigarette smoke extract on human coronary artery endothelial cells
134 ular proteins but was inhibited by cigarette smoke extract via oxidative disruption of actin polymeri
135 ith gas-diffusion separation of phenols from smoked food sample and analytes absorption into a NaOH s
136 effective point-of-need screening phenols in smoked food samples.
137  the brain processes involved in (simulated) smoking for the first time, and validate a novel approac
138 mbinations of the following MPOWER policies: smoke-free legislation (n=35), tobacco taxation (n=11),
139 wo studies assessing the association between smoke-free legislation and perinatal mortality, one show
140                            Implementation of smoke-free legislation was associated with reductions in
141 s the effect of implementing a comprehensive smoke-free policy on rates of physical assaults in a lar
142 ial cultures (HBECs) to air or whole tobacco smoke from cigarettes vs. LCs.
143                                              Smoke from the burning of biomass fuels has been linked
144 although this was not independent of current smoking habit (P=0.1993).
145 tion practices in relation to their personal smoking habits.
146 168) was composed according to age, sex, and smoking habits.
147                                              Smoking had a significant effect on this association (Pi
148                          Exposure to tobacco smoke has been associated with harmful effects on child
149 lic risk factors with increased risk of HCC; smoking has a significant effect on this association.
150 unfavorable lifestyle factors were included: smoking, heavy alcohol use, unhealthy diet and physical
151 ogate of human papillomavirus) and cigarette smoking history (pack-years) randomly assigned to clinic
152 nts with intracranial aneurysm (53.8%) had a smoking history vs 163 of 564 patients without intracran
153 ing for clinical site, patient age, reported smoking history, body mass index (BMI), diabetes, HIV, a
154 ssion analyses adjusted for body mass index, smoking, hypertension, diabetes, and systemic steroid us
155 tors included body mass index >/=30, current smoking, hypertension, diabetes, and total cholesterol >
156 e 10), mental health problems, daily tobacco smoking, illicit drug use, and dependence were all more
157 radrenergic alpha2a agonist, reduced tobacco smoking in a 4-week trial and in animal models has been
158 cotine pharmacokinetics resembling cigarette smoking in humans.
159 at perceived stress is associated with daily smoking in most countries.
160                        Paternal grandmothers smoking in pregnancy showed no associations.
161 nteraction of a variant in NOD2 with current smoking in relation to the risk for CD (frameshift varia
162 ctical difficulties associated with subjects smoking in the modern neuroscientific laboratory environ
163  effective interventions to prevent paternal smoking in the presence of children would reduce LRTI-re
164 ly rising and in utero exposure to cigarette smoke increases the risk of AA and bronchopulmonary dysp
165 ltivariate analysis, only PV replication and smoking independently increased the risk of bladder canc
166                             Paternal tobacco smoking independently increased the risk of LRTI 1.76-fo
167 ma and wheezing, including airborne viruses, smoke, indoor dampness, cockroaches, and poor access to
168                                          The smoke-induced chromatin changes include initial repressi
169 a protective role of miR-218-5p in cigarette smoke-induced inflammation and COPD.
170 ermore, genome expression studies revealed a smoke-induced up-regulation of Rho-GTPase-dependent acti
171  group also had more diabetes, dyslipidemia, smoking, infarcts from small-vessel disease, and "other
172                                              Smoking initiation and escalation are affected different
173 ronment and genetic factors are critical for smoking initiation and nicotine addiction.
174 d with greater risk for subsequent cigarette smoking initiation and past 30-day cigarette smoking.
175 tle evidence of a causal association between smoking initiation and schizophrenia, in either directio
176  evidence consistent with a causal effect of smoking initiation on schizophrenia risk (OR 1.73, 95% C
177  and 9 loci with convincing evidence of gene-smoking interaction (GxSMK) on obesity-related traits.
178                          Study-specific gene-smoking interaction effects were calculated and pooled u
179                                    Cigarette smoking is a leading cause of preventable mortality worl
180                                    Cigarette smoking is a risk factor for many perioperative complica
181                                              Smoking is a well-documented risk factor in various canc
182                                    Cigarette smoking is an established environmental risk factor for
183                                              Smoking is associated with a relatively homogenous distr
184 nts who smoke cigarettes and to confirm that smoking is significantly independently associated with S
185 arrett's Esophagus score) based on male sex, smoking, length of BE, and baseline low-grade dysplasia
186                                    Male sex, smoking, length of BE, and baseline-confirmed low-grade
187  However, the adverse events associated with smoking limit its clinical significance.
188 nct contributing factors for smoking and non-smoking lung cancer.
189 a use (great or moderate risk to health from smoking marijuana occasionally) and marijuana use (past
190 lving 807 incident lung cancer cases and 807 smoking-matched controls.
191             Our results suggest that tobacco smoking may alter the genetic susceptibility to overall
192 l approach of investigating population-level smoking metrics as moderators.
193  Our study shows that using population-level smoking metrics uncovers significant relationships betwe
194 ays, and shared seventeen genes with a mouse smoking model and twenty genes with previous emphysema s
195  binding studies, RNA-interference, a murine smoking model, and expression quantitative trait locus a
196 : Our study provides the first evidence that smoking modifies the previously reported inverse associa
197 greatest among black participants, those who smoked more than 20 cigarettes daily, current marijuana
198                                    Cigarette smoking more than doubled HSIL risk.
199                                      Neither smoking nor early-life exposures were associated with FV
200 ithelial cells (HBEC) confirm that cigarette smoke not only downregulates CFTR activity but also inhi
201                                              Smoking, not meeting physical activity guidelines, and b
202 d implant-related factors (sex, patient age, smoking, number of remaining teeth, percentage of teeth
203 le risk factors include sedentary lifestyle, smoking, obesity, diabetes mellitus, obstructive sleep a
204                            Other hotspots of smoke occurrence are found over state and national parks
205 OS was associated negatively with cumulative smoking (odds ratio [OR]: 0.992; 95% CI 0.984-1.000 per
206                     Mice were exposed to the smoke of 5 cigarettes per day, 5 days a week, for 8 week
207 ,5-dimethylfuran signatures derived from the smoke of machine-smoked cigarettes.
208  in addition to inferring a causal effect of smoking on mLOY.
209 nt smokers can lessen the impact of maternal smoking on offspring pulmonary function and decrease the
210 e of this study was to examine the impact of smoking on respiratory diseases, hypertension and myocar
211 mice and humans revealed that the effects of smoking on risk for IBD depend on genetic variants.
212               To evaluate if abstinence from smoking on the day of surgery is associated with a decre
213                     They were less likely to smoke or to be left-handed.
214                            In the absence of smoking or hypertension, 0.09% (95% CI, 0.02% to 0.35%)
215     Analogous to the development model, ever smoking (OR 6.70; 95%CI 6.41-6.99), prior asthma (OR 6.4
216 ed-stress resulted in a 5% increased odds of smoking (OR = 1.05; 95%CI = 1.03-1.06).
217  percent uninsured occurred in patients with smoking- or infection-related cancers.
218 non-smokers and that constituents of tobacco smoke other than nicotine affect inflammatory processes.
219 x, race, state (Iowa or North Carolina), and smoking (pack years), to estimate associations between e
220 d Ovarian Cancer Screening Trial (PLCO) ever-smoking participants (1,463 lung cancer cases and 915 lu
221 metric measures from a subset of 6,425 never-smoking participants without respiratory symptoms or dis
222                         Both smoking and non-smoking patients were studied, to determine whether effe
223  in patients with short duration of disease, smoking patients, and young patients.
224               The mean numbers of cigarettes smoked per day at 2 weeks and 1-month were significantly
225  of smoking and vaping (number of cigarettes smoked per day, number of vaping episodes per day, and n
226 er cross-ancestry GWAS meta-analysis for any smoking phenotype, we reconfirmed the well-known CHRNA5-
227 g for covariates (age, sex, education, race, smoking, physical activity, and obesity), people with vi
228 f the five major HRBs-alcohol use; cigarette smoking, physical inactivity, unhealthy diet, and illici
229           Other contributing factors include smoking, pollution and work-related exposures.
230 t when we matched pooled ORs with adult male smoking prevalence (z = 2.55, p = 0.01) in each country,
231                                              Smoking prevalence reduced significantly across the thre
232 sures is significantly associated with lower smoking prevalence, with anticipated future reductions i
233    Although we acknowledge the importance of smoking prevention and cessation, this is a large topic
234                                    Cigarette smoke promotes cell death and features of pancreatitis i
235 al was observed for increasing pack-years of smoking ( Ptrend = .008), with HR for death of 1.49 (95%
236  over 25 kg/m(2), diabetes, past and current smoking, red meat consumption, saturated fat and cholest
237 erosols were generated with standard intense smoking regimens with careful consideration for dose by
238                  In models adjusted for age, smoking, region, and population density, we did not obse
239                                  In study 1, smoking relapse was associated with less gray-matter vol
240 re associated with smoking sooner during the smoking relapse-analog task.
241 l sensitivity analyses: after excluding most smoking-related cancer cases (OR, 1.10 per procedure; 95
242 l therapeutic regimen to help improve MCC in smoking-related chronic bronchitis.
243 amining whether R-E training could attenuate smoking-related craving and behavior.
244 suggest that the effects of TNP on mood- and smoking-related outcomes may vary depending on the ovari
245  and Sept 30, 2015; 225 (4.9%) of which were smoking-related.
246 tudy design, methodological factors, age and smoking significantly affected the relationship.
247 ients, and in mice with bleomycin, cigarette smoke, silica, or sepsis-induced lung injury.
248 idence interval [CI]: 11.0, 64.4; P = .006), smoking (smokers vs nonsmokers, 45.2 mm(3); 95% CI: 7.1,
249 x, chronic disease, socioeconomic status and smoking social integration was inversely related to ten-
250  beta = 0.79; P < .001) were associated with smoking sooner during the smoking relapse-analog task.
251 for baseline Gender-Age-Physiology stage and smoking status (hazard ratio per 10% visual GGR increase
252 Patients and Methods We analyzed survival by smoking status among 1,037 patients from two large US pr
253     Most eye care providers assess patients' smoking status and educate patients regarding ocular ris
254 ) were not significantly associated with the smoking status of the nurse.
255  The association of airflow obstruction with smoking status was stronger in women (odds ratio for ex-
256 t as an independent variable identified age, smoking status, body mass index, haemoglobin, serum uric
257  sex, primary tumour type, age at diagnosis, smoking status, chemotherapy drug class, and duration of
258 n models fitted the association of age, sex, smoking status, diabetes mellitus, educational level, al
259 cluded baseline covariates: race, education, smoking status, diabetes, and cardiovascular disease.
260 n of eye care providers who assess patients' smoking status, educate patients regarding ocular risks
261 were adjusted for age, sex, body mass index, smoking status, education, energy intake, examination ye
262  age, body mass index, race, supplement use, smoking status, educational level, income, and aspirin u
263 tes diagnosis, systolic blood pressure, BMI, smoking status, estimated glomerular filtration rate, LD
264                            Neural effects of smoking status, nicotine, and varenicline were tested fo
265 ight was maintained across strata defined by smoking status, sex, and age, but the excess was greates
266 age and various aspects of smoking exposure (smoking status, smoking duration, cigarettes per day, pa
267  C-reactive protein, HbA1c, height, obesity, smoking status, triglycerides, type 2 diabetes, waist-hi
268 th, family history, alcohol consumption, and smoking status, which suggests that most risk factor ass
269  sex, employment grade, body mass index, and smoking status.
270 : 0.63, 0.91; P-trend < 0.01), regardless of smoking status.
271 risk factor such as body mass index, age, or smoking status.
272 nts with periodontitis, especially those who smoke, suffer from a high rate of tooth loss.
273 significant risk factor for AMD in LSOCA was smoking; the relative risk vs never-smokers was 3.4 for
274 ing duration, cigarettes per day, pack-years smoked, time since smoking cessation) as risk predictors
275 C in relatively young users who did not also smoke tobacco.
276 se issues, and provide a close simulation of smoking tobacco cigarettes.
277 e, as assessed by the Brief Questionnaire on Smoking Urges (BQSU).
278 95% CI, 1.05 to 2.10) for > 60 pack-years of smoking versus never smoking.
279 86) for any dependence; and the OR for daily smoking was 2.00 (1.89-2.12).
280                                     Maternal smoking was associated with prolonged mechanical ventila
281                               Information on smoking was collected biennially during follow-up.
282                                      Current smoking was most prevalent in the west Southeast and Mid
283 y participants aged between 36-55 and 56-80, smoking was positively associated with the risk of incid
284                                 A history of smoking was significantly associated with intracranial a
285  magnitude of association with maternal ever-smoking was stronger in male children compared with fema
286 model, adjusting for pack-years of cigarette smoking, was used to calculate hazard ratios and 95% con
287       BP, serum lipids, body mass index, and smoking were assessed in all follow-ups.
288 is, higher comorbidity scores and history of smoking were associated with a higher odds of the primar
289 ge, sex, and duration and daily frequency of smoking were gathered using a questionnaire.
290                         Higher odds of daily smoking were observed in both cohorts for those adolesce
291 ion, high blood sugar, and regular cigarette smoking were rare.
292  body-mass index (BMI) z-score and household smoking were strong predictors of higher BDE-153 levels.
293 ), self-reported physical inactivity, and/or smoking) were included.
294 el health impacts and improve adjustment for smoking when studying other risk factors with more subtl
295                                     Maternal smoking with obligatory nicotine inhalation is associate
296 e prospectively evaluated the association of smoking with patient survival.
297 related behavior and brain activation during smoking withdrawal among smokers with ADHD.
298                                              Smoking withdrawal negatively impacts inhibitory control
299 er, height, early pregnancy body mass index, smoking, year of delivery, maternal pregestational diabe
300 more likely to initiate smoking and to start smoking younger.

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