ライフサイエンスコーパス: smoke inhalation

1 in comparison with animals not subjected to smoke inhalation.

2 nt of the pulmonary inflammatory response to smoke inhalation.

3 ury who underwent bronchoscopy for suspected smoke inhalation.

4 elease ventilation (n = 12) for 48 hrs after smoke inhalation.

5 espiratory distress syndrome induced by wood smoke inhalation.

6 stablished ovine model of cutaneous burn and smoke inhalation.

7 eneity and strength of the acute response to smoke inhalation.

8 be of great value for studies of sepsis with smoke inhalation.

9 of MnSOD/kg (n = 6) intravenously 1 hr after smoke inhalation.

10 tion of early adverse respiratory effects of smoke inhalation.

11 ted alveolar epithelial barrier injury after smoke inhalation.

12 p receiving 3000 units of MnSOD 24 hrs after smoke inhalation (263 +/- 44 mL/min vs. 182 +/- 36 mL/mi

13 Adult sheep (n = 61) received smoke inhalation (48 breaths) and a 40% third-degree bur

14 were more severe than in animals exposed to smoke inhalation alone.

15 man activated protein C attenuated ALI after smoke inhalation and bacterial challenge in sheep, witho

16 In humans and in an ovine model of combined smoke inhalation and burn injury, bronchospasm and acute

17 airway obstruction that occur after combined smoke inhalation and burn injury.

18 del of severe respiratory failure created by smoke inhalation and cutaneous flame bum injury.

19 ffective in treating acute lung injury after smoke inhalation and pneumonia in sheep.

20 oles were obstructed by cast formation after smoke inhalation and pneumonia.

21 spiratory irritation, menthol may facilitate smoke inhalation and promote nicotine addiction and smok

22 , to conscious sheep subjected to a combined smoke inhalation and third-degree burn injury to 40% of

23 to validate its use in patients with severe smoke inhalation-associated acute lung injury requiring

24 The high mortality associated with smoke inhalation-associated acute lung injury results fr

25 gimens are increasingly being used to manage smoke inhalation-associated acute lung injury.

26 Smoke inhalation caused a significant increase in the al

27 The results of the study show that smoke inhalation causes injury to both the alveolar epit

28 Although prior studies have shown that smoke inhalation causes lung endothelial injury and form

29 iodontitis (EP) in the presence of cigarette smoke inhalation (CSI).

30 responsible for a substantial percentage of smoke inhalation deaths.

31 ke and cutaneous flame bum injury depends on smoke inhalation dose.

32 in which 1) animals or subjects experienced smoke inhalation exposure, 2) they were treated with neb

33 nodosum simultaneously, several weeks after smoke inhalation in a house fire.

34 roducible model of hyperdynamic sepsis after smoke inhalation in sheep.

35 spiratory distress syndrome caused by severe smoke inhalation in swine, airway pressure release venti

36 here was a significantly higher incidence of smoke inhalation in the cultured epithelial autograft gr

37 Following combined burn and smoke inhalation injury (40% of total body surface area,

38 ody surface area third-degree flame burn and smoke inhalation injury after tracheostomy.

39 ation would mitigate acute lung injury after smoke inhalation injury and burn.

40 anisms underlying lung inflammation in toxic smoke inhalation injury are unknown.

41 ction in sheep with severe combined burn and smoke inhalation injury by preventing the formation of a

42 he sheep in the sepsis group received cotton smoke inhalation injury followed by instillation of Pseu

43 Combined burn and smoke inhalation injury frequently results in acute lung

44 lease ventilation in the management of early smoke inhalation injury has not been studied.

45 The severity of smoke inhalation injury has systemically reaching effect

46 Smoke inhalation injury is a serious medical problem tha

47 and decreased fluid flux in a combined burn/smoke inhalation injury model.

48 Patients with severe burn and/or smoke inhalation injury suffer both systemic and pulmona

49 erial catheters and underwent an LD50 cotton smoke inhalation injury via a tracheostomy under halotha

50 Twenty-four hours after smoke inhalation injury, the animals were reanesthetized

51 cuss understanding of the pathophysiology of smoke inhalation injury, the best evidence-based treatme

52 ary dysfunction in sheep subjected to severe smoke inhalation injury.

53 clinical studies of patients with burns and smoke inhalation injury.

54 e lung injury secondary to combined burn and smoke inhalation injury.

55 for pulmonary pathology in burn victims with smoke inhalation injury.

56 mplication of burn patients with concomitant smoke inhalation injury.

57 for pulmonary pathology in burn victims with smoke inhalation injury.

58 vasoconstriction following combined burn and smoke inhalation injury.

59 p with ARDS resulting from combined burn and smoke inhalation injury.

60 ARDS) in sheep with severe combined burn and smoke inhalation injury.

61 esis that neutrophils play a pivotal role in smoke inhalation injury.

62 e in vivo scenario is not straightforward as smoke inhalation involves a number of other components.

63 Acute lung injury secondary to smoke inhalation is a major source of morbidity and mort

64 Patients with acute lung injury after smoke inhalation often develop pneumonia subsequently co

65 there is no information about the effect of smoke inhalation on the function of the alveolar epithel

66 Treatment with MnSOD given after smoke inhalation seems to be less effective then pretrea

67 onary microvascular permeability in combined smoke inhalation/third-degree burn injury, but does not

68 n flame burn and 48 breaths of cooled cotton smoke inhalation under deep anesthesia and analgesia.

69 Combined burn and smoke inhalation was associated with increased expressio

70 dmitted to the burn intensive care unit when smoke inhalation was suspected.

71 After smoke inhalation with 48 breaths of cotton smoke, the an