ライフサイエンスコーパス: smoking habit

1 d by the bone substratum, periodontitis, and smoking habit.

2 grafted vs. pristine bone, and for a heavier smoking habit.

3 patients with periodontitis that reported a smoking habit.

4 168) was composed according to age, sex, and smoking habits.

5 tion practices in relation to their personal smoking habits.

6 ression was not associated with the maternal smoking habits.

7 t role in nicotine metabolism and consequent smoking habits.

8 possible determinants of disease, including smoking habits.

9 osures to cigarette smoke than self-reported smoking habits.

10 ting abruption was increased irrespective of smoking habits.

11 f this research was to examine self-reported smoking habits according to measures of socioeconomic st

12 e optical density of MP (iris color, gender, smoking habits, age, and lens density).

13 physical activity patterns, medication use, smoking habits, alcohol consumption, and other lifestyle

14 hanges in LC biochemistry may strengthen the smoking habit among subjects with major depression.

15 ty patients with a greater than one pack/day smoking habit and generalized moderate to severe chronic

16 t why some adolescents progress to a regular smoking habit and others do not.

17 After adjusting for smoking habits and a wide range of established and poten

18 be the key substance responsible for tobacco-smoking habits and appears to trigger reinforcement via

19 , as well as other medical conditions (i.e., smoking habits and body mass index), were considered in

20 II Nutrition Cohort collected information on smoking habits and exposure to ETS during childhood and

21 nces between groups in terms of gender, age, smoking habits and indications for treatment.

22 mpleted a self-administered questionnaire on smoking habits and lifestyle factors.

23 ng Illumina 450K array) were integrated with smoking habits and ultrasound-measured carotid plaque sc

24 ting for age, education level, osteoporosis, smoking habit, and body mass index, the ORadjusted was 4

25 old density, alcoholic beverage consumption, smoking habit, and cardiovascular disease (odds ratio OR

26 ndent of age, sex, weight, height, cigarette smoking habit, and past history of fracture.

27 and data collection, last visit to dentist, smoking habit, and present occupation, the association m

28 index by 5 kg/m2, quitting a 10 cigarette/d smoking habit, and reducing dietary cholesterol intake b

29 after adjusting for age, menopausal status, smoking habit, and sexual exposure history.

30 However, age, smoking habits, and especially DM2 were significantly as

31 city, maternal age, parental occupation, and smoking habits, and they differed only slightly by pater

32 isease severity, as well as its extent and a smoking habit, appear to be factors that influence the c

33 the coronary atheromatous plaque burden and smoking habit are associated with a reduced acute local

34 because previous studies used self-reported smoking habits as surrogates for children's true exposur

35 ion measurements and complete information on smoking habits available.

36 nce of venous thromboembolism in relation to smoking habits, both in the absence of surgery and in th

37 We also investigated whether a history of smoking habits cessation may affect the risk of periodon

38 , family history (chi2 = 6.26; P = .01), and smoking habit (chi2 = 10.06; P = .007) as independent ri

39 ost fit 40%) fitness categories by strata of smoking habit, cholesterol level, blood pressure, and he

40 blood pressure, blood cholesterol, cigarette smoking habit, diabetes, and supplement use.

41 that differed between patients and controls (smoking habits, diabetes mellitus, years of education, a

42 eas other variables (i.e., TT, sex, age, and smoking habit) do influence alveolar BT.

43 ntervals) adjusted for maternal age, height, smoking habits, education, and time period (5-year group

44 ernal height, maternal age, parity, mother's smoking habits, education, country of birth, and year of

45 models, adjusting for age, body mass index, smoking habits, ethnicity, and reproductive factors.

46 se findings were independent of age, gender, smoking habits, ethnicity, and standard lipids differenc

47 d's parents provided information about their smoking habits for each year from age 15 years to the ch

48 Data regarding smoking habits, general health, and medications were col

49 were gathered on age, sex, bone substratum, smoking habit, history of periodontitis, and prosthetic

50 There was no heterogeneity across sex, smoking habit, histotype, and epidermal growth factor re

51 for age, sex, physical activity status, BMI, smoking habits, intake of nuts, and other confounders.

52 ence of risk modification by factors such as smoking habit, known to be associated with stroke.

53 Patients should be advised that their smoking habit may result in poorer bone regeneration aft

54 dies confirm these findings, modification of smoking habits may prevent or delay age-related declines

55 ied into 3 categories based on self-reported smoking habits: never (43.2%), former (50.5%), and curre

56 haled from cigarettes is nicotine, hence the smoking habit of SZP may represent an attempt to use nic

57 th the residential construction date and the smoking habits of residents.

58 After positing a distribution for the smoking habits of workers and referents, a distribution

59 nce of proximal coronary artery atheroma and smoking habit on the stimulated release of tissue plasmi

60 narcotics were more likely to have a current smoking habit (P < .001) with perianal disease (P = .046

61 tic aneurysm diameter (P<0.0001) and current smoking habit (P=0.0446) also predicted the primary outc

62 although this was not independent of current smoking habit (P=0.1993).

63 65 years or older and younger than 65 years, smoking habit, past history of fracture, and hip and non

64 f PRAL, independent of age, body mass index, smoking habit, physical activity, diagnosed osteoporosis

65 ormation about age, gender, medical history, smoking habit, physical examination and results of imagi

66 tantially increased regardless of changes in smoking habits suggests that factors other than smoking

67 ls have in reducing tobacco use, many have a smoking habit themselves.

68 e matched by age, time of randomization, and smoking habit to an equal number of controls (who had re

69 other risk factors, and related drinking and smoking habits to the cumulative probability of dying be

70 okers even after adjusting for self-reported smoking habits, urinary cotinine, and well-known cardiov

71 ge, body weight, dietary and alcohol intake, smoking habits, use of medications, and occurrence of di

72 Smoking habit was ascertained at baseline and yearly by

73 Information on anthropometry, diet, and smoking habits was obtained through a questionnaire.

74 w-up (1980 to 1992), in which information on smoking habits was updated every 2 years.

75 ong biotypes, whereas: 1) TT, 2) age, and 3) smoking habit were often predictors of reduction in BT i

76 on-attendance to periodontal maintenance and smoking habits were also associated with less favorable

77 ometry, occupational endotoxin exposure, and smoking habits were assessed at 5-year intervals.

78 ons as well as ethnic and marital status and smoking habits were considered.

79 rol subjects matched on the basis of age and smoking habit who remained free of vascular disease duri

80 001), independent of age of the individuals, smoking habits, whole-blood storage time, and various in