ライフサイエンスコーパス: smooth

1 e analyzed with generalized additive models (smoothing).

2 as peri-stimulus time histograms and kernel smoothing.

3 ues, as we demonstrate by preparing a dense, smooth, 5.3-mum-thick PbSe QD film via doctor-blading.

4 In this paper, we propose an iterative smoothing algorithm with structure sparsity (ISSS) metho

5 trongly charged NPs (>2 e/nm(2)), both types smooth and DNA-functionalized NPs, show an attractive po

6 anning electron microscopy both revealed the smooth and flat nature of the nanosheets.

7 dicated the archaeal community structures of smooth and pustular mats were significantly different (g

8 f key biogeochemical properties of two mats (smooth and pustular) from Shark Bay, Australia.

9 imals, including humans, flies generate both smooth and rapid saccadic movements to stabilize their g

10 The resulting smooth and uniform C-coatings sheathing the inner core m

11 Here, we present a novel Bayesian smoothing approach (called ABBA) to detect differentiall

12 spatial resolution, the neural code must be smooth at the voxel and functional level such that simil

13 atients were included in the series, 19 with smooth-bordered lesions and 26 with irregularly bordered

14 The domains are large, have smooth boundaries, and can merge quickly, consistent wit

15 ed DCX and classical lissencephaly including smooth brain and abnormal cortical morphology.

16 Accordingly, human primary smooth cells secrete IL-26 in response to proinflammator

17 Classical lissencephaly is characterized by smooth cerebral surface and cortical thickening that res

18 We then turn to the theory of smooth Chebyshev nets to address the inverse design of h

19 Mycobacterium canettii, which has a smooth colony morphology, is the tuberculous organism re

20 The process takes place under smooth conditions, nonanionic conditions, and with a hig

21 Two proteins, FlgK and FlgL, assure a smooth connectivity between the hook and the filament.

22 With silicate addition, a smooth, continuous, coherent and apparently amorphous ir

23 and HVB series, strong and weak types form a smooth continuum with no sharp break in properties.

24 onospecific stands of Spartina alterniflora (smooth cordgrass) or Avicennia germinans (black mangrove

25 Locally weighted scatterplot smoothing curves were used to display visual acuity (VA)

26 nterpreting the size-selectivity in terms of smooth cylindrical pores using the centerline approximat

27 ate that an arcsin transformation of Laplace-smoothed data is at least as good as state-of-the-art mo

28 show that myosin II contractility drives the smooth dermal mesenchyme into a pattern of surface bumps

29 strategies of material design are needed to smooth each pivotal step.

30 MOmega; 10.8 Omega cm(2)) at 1 kHz and very smooth electrode surfaces.

31 Applications for smooth electron waveguides in 2D Dirac-Weyl systems are

32 direction of eye movement, and we show that smooth eye movements modulate MT responses in a systemat

33 We used visually driven smooth eye movements to find the 3D space-time function

34 anticline with bends on both limbs, while a smooth fault plane will develop a roll-over anticline wi

35 brain stimulation (DBS) is modulated by the smooth feedback signals.

36 Circular elliptical microstructures produce smooth flow minimally reducing any damage.

37 , which corresponds to the commonly observed smooth generation spectra.

38 Smooth glass was found to provide the upper and lower bo

39 n fingers gives rise to stable contacts with smooth, hard objects is surprisingly slow.

40 ter the interaction, indicating a relatively smooth hot electron distribution at the rear-side of the

41 kDa) were obtained by the ultrafiltration of smooth hound viscera protein hydrolysates, produced by N

42 o hydrodynamical simulations, where pressure smoothing is determined by the integrated thermal histor

43 onent of Rs, but global-scale models tend to smooth its spatial heterogeneity by aggregating factors

44 rystal cross-sections possessed a symmetric, smooth lattice misorientation with respect to the c-axis

45 Smooth M. canettii colonies grew from 68% of lungs and 3

46 Mycobacterium bovis strains was greater than smooth "M. canettii" and M. kansasii.

47 Smooth mats possessed higher archaeal diversity, dominat

48 The methanogenic community in smooth mats was dominated by hydrogenotrophic Methanomic

49 How rapid body saccades and smooth movement interact for simultaneous object pursuit

50 following the bar, with surprisingly little smooth movement.

51 stent airflow obstruction had greater airway smooth muscle (Asm) area with decreased periostin and tr

52 mechanism along the cholinergic nerve-airway smooth muscle (ASM) axis that underlies prolonged airway

53 on, mucus hypersecretion and abnormal airway smooth muscle (ASM) contraction.

54 is commonly associated with increased airway smooth muscle (ASM) mass.

55 f human LTCCs during SN DA-like and arterial smooth muscle (aSM)-like activity patterns using whole-c

56 KEY POINTS: Non-muscle (NM) and smooth muscle (SM) myosin II are both expressed in smoot

57 of non-muscle (NM) isoforms of myosin II in smooth muscle (SM) tissues and their possible role in co

58 crease phosphorylation of myosin in vascular smooth muscle (VSM) cells, causing persistent constricti

59 oth muscle actin, myosin heavy chain 11, and smooth muscle 22 alpha.

60 Interestingly, LPS down-modulated alpha-smooth muscle actin (activated HSC marker) and collagen

61 The expression of alpha-smooth muscle actin (alpha-SMA) and Collagen I were redu

62 ted the induction of activation marker alpha-smooth muscle actin (alpha-SMA) in rat and mouse HSCs.

63 Alpha-smooth muscle actin (alpha-SMA) is a marker of activated

64 n of EMT resulted in the generation of alpha-smooth muscle actin (alpha-SMA)-positive myofibroblasts

65 ells were enriched in proximity to the alpha-smooth muscle actin (alpha-SMA+) area within mild fibros

66 population with elevated expression of alpha-smooth muscle actin (alphaSMA) located immediately adjac

67 aracterized by increased expression of alpha-smooth muscle actin and collagen 1.

68 oxaban deactivated HSC, with decreased alpha-smooth muscle actin and mRNA expression of other HSC act

69 In contrast, alpha-smooth muscle actin expression by infarct myofibroblasts

70 Exposure to mtDNA augments alpha-smooth muscle actin expression in NHLFs.

71 ed with cholangiocyte supernatants and alpha-smooth muscle actin levels were measured.

72 en fluorescent protein costaining with alpha-smooth muscle actin or collagen 1alpha in left ventricul

73 otoxic, and a more potent inhibitor of alpha-smooth muscle actin protein expression than CCG-203971.

74 pha 1, matrix metalloproteinase 2, and alpha-smooth muscle actin) markers.

75 er cells expressed increased levels of alpha-smooth muscle actin, a marker of CAF, compared with MSC

76 c flow contained reduced myosin heavy chain, smooth muscle actin, and desmin, and increased markers o

77 on of the fibroblast markers vimentin, alpha-smooth muscle actin, and S100A4.

78 n levels of fibronectin-1, collagen I, alpha-smooth muscle actin, CTGF, and PAI-1, but decreased Smad

79 ion of selective VSMCs markers such as alpha smooth muscle actin, myosin heavy chain 11, and smooth m

80 fibroblasts, shown by up-regulation of alpha-smooth muscle actin, pro-collagen 1, and F-actin express

81 sis (IPF) involves the accumulation of alpha-smooth muscle actin-expressing myofibroblasts arising fr

82 wth factors and cytokines, decrease of alpha-smooth muscle actin-positive ASFs, and finally in a sign

83 tor receptor-alpha-positive cells, and alpha-smooth muscle actin-positive blood vessels were assayed

84 ctor receptor-alpha-positive cells, 4) alpha-smooth muscle actin-positive blood vessels, and 5) of ke

85 a 1.35-fold increase in proliferative alpha-smooth muscle actin-positive cells in the lungs of ITSN-

86 l effects of infarct scar maturation, causes smooth muscle alpha-actin fiber formation, up-regulation

87 These findings reveal that disruption of smooth muscle alpha-actin filaments in smooth muscle cel

88 Furthermore, disruption of smooth muscle alpha-actin filaments in wild-type smooth

89 ed blood vessels of all caliber and putative smooth muscle and astroglial basement membrane compartme

90 nced atherosclerotic plaques with diminished smooth muscle and collagen content.

91 t this locus in primary cultures of vascular smooth muscle and endothelial cells.

92 MP) as well as adjacent urethra comprised of smooth muscle and peri-urethral mesenchyme.

93 reases STOC activity in contractile vascular smooth muscle and show that a critical step is the activ

94 A role for smooth muscle ARHGEF1 in asthmatic airway hyper-responsi

95 Perivascular access to smooth muscle basement membrane compartments also exhibi

96 characterized by excessive pulmonary artery smooth muscle cell (PASMC) proliferation, migration, and

97 restingly, ADAMTS-4 was directly involved in smooth muscle cell (SMC) apoptosis.

98 Smooth muscle cell (SMC) death contributes to plaque des

99 Smooth muscle cell (SMC) differentiation is essential fo

100 Vascular smooth muscle cell (SMC) proliferation and endothelial c

101 elial cell (BmxCreER(T2)-driven)-specific or smooth muscle cell (SMC, SmmhcCreER(T2)- or TaglnCre-dri

102 Vascular smooth muscle cell (VSMC) apoptosis precipitates AAA for

103 in pathophysiologic stimulation of vascular smooth muscle cell (VSMC) migration and proliferation.

104 articles were safe to rat pulmonary arterial smooth muscle cell and to the lungs, as evidenced by the

105 , DRP1 inhibition attenuated mouse and human smooth muscle cell calcification.

106 kinase inhibition directly attenuates airway smooth muscle cell contraction independent of its protec

107 Smooth muscle cell contraction significantly increased t

108 , SRF alone is not sufficient for regulating smooth muscle cell development.

109 (PROCR, rs867186 (p.Ser219Gly)) and vascular smooth muscle cell differentiation (LMOD1, rs2820315).

110 GAS5) suppresses TGF-beta/Smad3 signaling in smooth muscle cell differentiation of mesenchymal progen

111 wered blood pressure, which was dependent on smooth muscle cell expression of Panx1 and independent o

112 capacity and beating rate and suppressed the smooth muscle cell formation.

113 ar cell functions, including endothelial and smooth muscle cell growth, proliferation, and migration;

114 Histopathologic findings of smooth muscle cell hypertrophy and stroma-like cells, co

115 Loss of smooth muscle cell hypoxia inducible factor-1alpha under

116 and evidence of higher biological activity (smooth muscle cell loss and fibrin deposition) in the FP

117 llular phenotypes was analyzed with vascular smooth muscle cell migration assays and platelet aggrega

118 d integration site) signaling and regulating smooth muscle cell survival, as well as differentiation

119 In vivo, vascular smooth muscle cell/mesangial cell-specific overexpressio

120 Activated CD4 T cells connect to airway smooth muscle cells (ASMCs) in vitro via lymphocyte-deri

121 portant in regulating healthy primary airway smooth muscle cells (ASMCs), whereas changed expression

122 equency of Ca(2+) oscillations within airway smooth muscle cells (ASMCs).

123 o, knockdown of T-cadherin from human aortic smooth muscle cells (HASMCs) with synthetic phenotype si

124 tractile markers in co-cultured human aortic smooth muscle cells (HASMCs).

125 ABSTRACT: Smooth muscle cells (myocytes) of resistance-size arteri

126 pha and auxiliary beta1 subunits in arterial smooth muscle cells (myocytes).

127 stressful conditions, pulmonary artery (PA) smooth muscle cells (PASMCs) exhibit a "cancer-like" pro

128 In parallel, pulmonary arterial smooth muscle cells (PASMCs) from Cox4i2(-/-) mice showe

129 , the role of HIF-1alpha in pulmonary artery smooth muscle cells (PASMCs) remains controversial.

130 ute to the proliferation of pulmonary artery smooth muscle cells (PASMCs), and inhibition of phosphod

131 dases, ADAM10 and ADAM17 in pulmonary artery smooth muscle cells (PASMCs).

132 1.5 protein expression were decreased in PAH smooth muscle cells (primary culture).

133 because voltage-dependent Ca(2+) channels in smooth muscle cells (SMC) provide the Ca(2+) that trigge

134 BB)-stimulated proliferation of human venous smooth muscle cells (SMC) was measured by a DNA-binding

135 Kir2 channels was observed in ICC but not in smooth muscle cells (SMC).

136 es, inhibited proliferation and migration of smooth muscle cells (SMCs) and promoted the tube formati

137 Vascular smooth muscle cells (SMCs) can resist and repair artery

138 expression could be resolved in ICC but not smooth muscle cells (SMCs) in the IAS and rectum.

139 Loss and dysfunction of smooth muscle cells (SMCs) in the vasculature may cause

140 n vitro differentiation into fibroblasts and smooth muscle cells (SMCs) is also described.

141 expression of 5-HTT induced proliferation of smooth muscle cells (SMCs); however, this phenotype coul

142 r calcium ([Ca(2+)]cyt) dynamics in vascular smooth muscle cells (VSMC).

143 OCs) in proliferative and migratory vascular smooth muscle cells (VSMCs) are quite intricate with man

144 The importance of TSPANs in vascular smooth muscle cells (VSMCs) is unexplored.

145 Specific ablation of Plk1 in vascular smooth muscle cells (VSMCs) led to reduced arterial elas

146 MicroRNAs are key regulators of vascular smooth muscle cells (VSMCs) phenotypic switch, one of th

147 ABSTRACT: In vascular smooth muscle cells (VSMCs), stimulation of canonical tr

148 In vascular smooth muscle cells (VSMCs), stimulation of SOCs compose

149 apurinic/apyrimidinic endonuclease I protect smooth muscle cells against oxidant-induced cell death.

150 ts due to this ACTA2 mutation in both aortic smooth muscle cells and adventitial fibroblasts may cont

151 II (30 nm) also increased TRPM4 currents in smooth muscle cells and constricted cerebral arteries fr

152 the major KV1 channel expressed in vascular smooth muscle cells and is abundantly localized on the p

153 Mural cells (vascular smooth muscle cells and pericytes) play an essential rol

154 steady-state pHi persisted only in vascular smooth muscle cells but not endothelial cells.

155 th muscle alpha-actin filaments in wild-type smooth muscle cells by various mechanisms activates nucl

156 revealed that the origin of aortic vascular smooth muscle cells can be traced back to progenitor cel

157 real-time imaging was performed in vascular smooth muscle cells expressing a FRET-biosensor comprisi

158 A discrete population of smooth muscle cells forms in the embryo and is postnatal

159 teins and cocaine was confirmed in pulmonary smooth muscle cells from cocaine injected HIV-transgenic

160 Airway smooth muscle cells generated pro-MMP-1, which was prote

161 this study was to determine whether vascular smooth muscle cells in cultured microvascular networks m

162 e of the ischemic cascade: selective loss of smooth muscle cells in juveniles but not adults shortly

163 x18 selectively marks pericytes and vascular smooth muscle cells in multiple organs of adult mouse.

164 localized on endothelial cells and synthetic smooth muscle cells in the aortic intima.

165 Extensive proliferation of immature smooth muscle cells in the primitive embryonic dorsal ao

166 on of smooth muscle alpha-actin filaments in smooth muscle cells increases reactive oxygen species le

167 s likely that in its absence, contraction of smooth muscle cells is impaired.

168 c increase in the proliferation of pulmonary smooth muscle cells on exposure to HIV-proteins and/or c

169 orylated SMAD2/3 in human pulmonary arterial smooth muscle cells on treatment with cocaine and Tat.

170 n and pharmacological inhibition in vascular smooth muscle cells reveal that cytochrome b5 reductase

171 aorta establishes the long-lived lineages of smooth muscle cells that make up the wall of the adult a

172 nted Sox10(+) stem cells differentiated into smooth muscle cells to stabilize functional microvessels

173 DRP1 inhibition in human smooth muscle cells undergoing osteogenic differentiatio

174 hallenge, and bronchoscopy, and their airway smooth muscle cells were grown in culture.

175 scovered globin expressed in fibroblasts and smooth muscle cells with unknown function.

176 nerve terminals and electrically coupled to smooth muscle cells within the gastric musculature.

177 hus potentiating AngII signaling in vascular smooth muscle cells without an increase in the exogenous

178 luripotent stem cell-derived cardiomyocytes, smooth muscle cells, and endothelial cells (in a 2:1:1 r

179 hen seeded the scaffold with cardiomyocytes, smooth muscle cells, and endothelial cells that had been

180 odest reduction of proliferation in vascular smooth muscle cells, but given low proliferative capacit

181 ted Ca(2+) channels in the adjacent vascular smooth muscle cells, causing vasoconstriction.

182 In cardiomyocytes and smooth muscle cells, cyclic AMP (cAMP) and subsequent ca

183 ells, which encompass pericytes and vascular smooth muscle cells, is a hallmark of CADASIL and other

184 om different origins, including endothelial, smooth muscle cells, macrophages, hepatocytes, adipocyte

185 In airway smooth muscle cells, these Ca(2+) oscillations are cause

186 hat neoarterioles were aberrantly covered by smooth muscle cells, with increased interprocess spacing

187 ression was found in human and murine airway smooth muscle cells.

188 xylase domain activity were increased in PAH smooth muscle cells.

189 and an enhanced ability to differentiate to smooth muscle cells.

190 rentially expressed in vascular and visceral smooth muscle cells.

191 ein secretion by lipid-loaded human vascular smooth muscle cells.

192 crostructure including elastin, collagen and smooth muscle cells.

193 mmation, and proliferation in fibroblast and smooth muscle cells.

194 y 45-fold higher in isolated cerebral artery smooth muscle cells.

195 1.3% of vessels with recruitment of vascular smooth muscle cells; VSMCs) in the presence of enhanced

196 pithelial ion transport and fluid secretion, smooth muscle constriction, neuronal excitability, and c

197 Tracheal smooth muscle contains significant amounts of myosin bin

198 ss, but how they interact to regulate airway smooth muscle contractility is not fully understood.

199 enzyme expression, endothelial dysfunction, smooth muscle contractility, and vascular remodeling.

200 eletal dense bodies, and impaired intestinal smooth muscle contractility.

201 matrix and, to our knowledge, novel model of smooth muscle contractility.

202 a vital mechanism for the control of airway smooth muscle contraction and thus are a critical factor

203 congenital disorder characterized by loss of smooth muscle contraction in the bladder and intestine.

204 oded by TMEM16A control neuronal signalling, smooth muscle contraction, airway and exocrine gland sec

205 n, transforming growth factor-beta, vascular smooth muscle contraction, and the hedgehog and Wnt sign

206 ching morphogenesis, the frequency of airway smooth muscle contraction, and the rate of developmental

207 , and identified distinct pathways linked to smooth muscle contraction, inflammatory cytokines, immun

208 re preceded by long-duration waves of airway smooth muscle contraction.

209 trix was prepared from decellularized airway smooth muscle cultures.

210 and suggest its role in establishing normal smooth muscle cytoskeletal-contractile coupling.

211 fic demethylase and promote adipogenesis and smooth muscle development.

212 t CD146 is transiently expressed in vascular smooth muscle development.

213 by promoting monocyte firm adhesion, whereas smooth muscle EphA2 expression may regulate the progress

214 a) channels are key determinants of vascular smooth muscle excitability.

215 nic hedgehog expression, leading to aberrant smooth muscle formation and defective contraction of the

216 airment in BKCa channel function in vascular smooth muscle from diabetic patients through unique mech

217 similar alterations occur in native vascular smooth muscle from humans with type 2 diabetes is unclea

218 tudy, we evaluated BKCa function in vascular smooth muscle from small resistance adipose arteries of

219 Endothelial and smooth muscle function were diminished in uterine (and t

220 nscription co-factor CRP2 was a regulator of smooth muscle gene expression.

221 a histone acetyltransferase and a driver of smooth muscle gene expression.

222 istically with SRF and myocardin to regulate smooth muscle gene expression.

223 , GATA6 and CRP2 required CSRP2BP for robust smooth muscle gene promoter activity.

224 CSRP2BP synergistically activated smooth muscle gene promoters in an SRF-dependent manner.

225 lar matrix, which enhanced subsequent airway smooth muscle growth by 1.5-fold (P < 0.05), which was d

226 ransiently increasing MMP activation, airway smooth muscle growth, and airway responsiveness.

227 sthma, mast cells are associated with airway smooth muscle growth, MMP-1 levels are associated with b

228 KATP channels in vascular smooth muscle have a well-defined role in regulating vas

229 e thickening, subepithelial fibrosis, airway smooth muscle hyperplasia and increased angiogenesis.

230 creased thickness, p = 0.005) and within the smooth muscle layer (p = 0.004).

231 ion of extracellular matrix (ECM) and larger smooth muscle mass are correlated with increased airway

232 selective genetic deletion of melanopsin in smooth muscle mostly removed the light-induced, but not

233 showed that the interaction between CBFbeta-smooth muscle myosin heavy chain (SMMHC; encoded by CBFB

234 Smooth muscle myosin light chain kinase (smMLCK) is a me

235 Vascular smooth muscle NOX4, the common denominator of ischemia w

236 and provide a functional innervation of the smooth muscle of the bowel wall.

237 compartment that began to express markers of smooth muscle precursors and adventitial fibrocytes, res

238 A distinct population of CD146(+) smooth muscle progenitor cells emerges during embryonic

239 on to advanced atherosclerosis by regulating smooth muscle proliferation and extracellular matrix dep

240 regulator of O2-dependent NO degradation in smooth muscle remains elusive.

241 ignalling pathway, thereby regulating airway smooth muscle remodelling in asthma.

242 sing calpain knockout mice attenuated airway smooth muscle remodelling in mouse asthma models.

243 proliferation of ASMCs and attenuated airway smooth muscle remodelling in mouse asthma models.

244 However, the role of calpain in airway smooth muscle remodelling remains unknown.

245 mmalian cells, including epithelia, vascular smooth muscle tissue, electrically excitable cells, and

246 Contractile stimulation of tracheal smooth muscle tissues stimulates phosphorylation of the

247 muscle (SM) myosin II are both expressed in smooth muscle tissues, however the role of NM myosin in

248 Airway smooth muscle treated with activated mast cell supernata

249 antly influencing the phenotypic tonicity in smooth muscle via ROCK2: a lack of tone in ASM may be as

250 ay inflammation, mucus, fibrosis, and airway smooth muscle were no different in Ormdl3(Delta2-3/Delta

251 and subsequent differentiation into coronary smooth muscle, and restores Wt1 activity upon MI.

252 eosinophils, lung Il13 levels, collagen, and smooth muscle, as well as a significant depletion of gob

253 LAM is characterized by neoplastic growth of smooth muscle-alpha-actin-positive cells that destroy lu

254 lso found that TSPAN2 is highly expressed in smooth muscle-enriched tissues and down-regulated in in

255 ation of RLC phosphorylation in tonic airway smooth muscle.

256 gy-dynamic but not fully differentiated from smooth muscle.

257 c field stimulation (EFS) in bovine tracheal smooth muscle.

258 Our findings suggest that airway smooth muscle/mast cell interactions contribute to asthm

259 Smooth-muscle actin expression by stellate cells and CD3

260 e myofibroblastic markers vimentin and alpha-smooth-muscle actin in developing kidneys.

261 d epithelioid cells (LAM cells) that express smooth-muscle and melanocyte-lineage markers, harbor mTO

262 rtner polypeptides in regulation of vascular smooth-muscle cell contractility.

263 Smooth-muscle cells from mouse tracheas were assayed in

264 pidly activated RhoA, ERK, and Akt in airway smooth-muscle cells, but only in the presence of TSG-6.

265 ization of freshly dispersed ICC and colonic smooth muscles, suggesting that this conductance is acti

266 apidity of pCPI-17 inactivation in mammalian smooth muscles.

267 f striated muscles and attachment plaques of smooth muscles.

268 9% of synapses were formed with dendrites of smooth neurons in S1.

269 tiple covariates to be modelled as linear or smooth non-linear continuous functions.

270 atterns such as undulating motion, efficient smoothing of irregularities, and the generation of cusps

271 of the expected abrupt transition we find a smooth one.

272 substrates were formulated (i.e. containing smooth or skeletal muscle ECM) and used to culture MPCs

273 This instability can be smoothed out by increasing the bending rigidity of the c

274 platelets unlike planar control titania and smooth PDMS surfaces.

275 This temporal smoothing process helps perceptual continuity by preserv

276 nnection between visual motion estimates and smooth pursuit eye movements to measure stimulus-respons

277 TEMENT When an object moves, we view it with smooth pursuit eye movements.

278 We measured this pressure-smoothing scale by quantifying absorption correlations i

279 expression is altered, loss of PGX3 prevents smooth stomatal closure, and overexpression of PGX3 acce

280 To this end, a smooth substrate and two different high aspect ratio top

281 The device can be realized on any smooth substrate surface and operates with low energy co

282 beta-(phenylsulfonyl)vinyl AA-esters undergo smooth sulfone-stannane interchange to stereoselectively

283 niform encapsulates with good sphericity and smooth surface for SOE, compared to FOE powder.

284 The microelectrode array sites showed a very smooth surface mainly composed of thin-film polycrystall

285 imple benchtop polishing procedures render a smooth surface that supports propagation of surface plas

286 in the dry free-flowing form; however, less smooth surface with wrinkles was observed when the initi

287 s of the crystal and refers to an atomically smooth surface.

288 d that wheat bran flakes have a 'rough' and 'smooth' surface with substantially different affinity to

289 ndreds of micrometers in diameter with ultra-smooth surfaces and sub-micrometer wall thicknesses have

290 the other hand, moderately charged NPs with smooth surfaces as well as DNA-functionalized NPs with n

291 rs units correspond to a model consisting of smooth surfaces within these cavities.

292 ond-order critical phase transition, i.e., a smooth switch to huddling when the environment gets cold

293 the sampling design and probability, applied smoothing techniques to produce stable trends, fitted li

294 s present results from Vesta suggesting that smooth terrains with heightened hydrogen concentrations

295 ic layers and was up to seven-fold higher in smooth than pustular mats.

296 ion can bypass the instability by inducing a smooth transition from an open to a closed bud.

297 calculated by the presented method reveal a smooth transition from low to high LETs which is an adva

298 ological materials from damage by creating a smooth transition from strong to weak that dissipates la

299 s covered by hollow spinose sclerites, and a smooth, ventral girdle flanks an extensive mantle cavity

300 ver time, yet, for dynamic stimuli, temporal smoothing would blur dynamics and reduce sensitivity to