ライフサイエンスコーパス: smooth pursuit

1 are implicated in generating and maintaining smooth pursuit.

2 on is associated with sluggish initiation of smooth pursuit.

3 signals are implicated in the generation of smooth pursuit.

4 Changes in head position had no effect on smooth pursuit.

5 the role of basal ganglia in the control of smooth pursuit.

6 saccades (i.e., fast vergence) and conjugate smooth pursuit.

7 cteristics of visually guided and predictive smooth pursuit.

8 e ability to produce conjugate adaptation of smooth pursuit.

9 observed in the covered eye during vertical smooth pursuit.

10 ved in the nonfixating eye during horizontal smooth pursuit.

11 ia as they performed horizontal and vertical smooth pursuit (0.2 Hz, +/-10 degrees ) under monocular

12 Smooth pursuit abnormality in subjects with schizophreni

13 ugh mounting evidence supports the idea that smooth pursuit abnormality marks the genetic liability t

14 We tested smooth pursuit adaptation during monocular viewing in st

15 To induce smooth pursuit adaptation experimentally, we used a step

16 retina is known to be necessary for guiding smooth pursuit adaptation.

17 Ocular smooth pursuit and fixation are typically viewed as sepa

18 Smooth pursuit and fixation suppression of VOR were mild

19 bulo-ocular reflex, vestibulo-collic reflex, smooth pursuit and gaze holding.

20 (1) nearly completely abolished ipsilateral smooth pursuit and impaired contralateral pursuit, (2) a

21 slow eye movements such as fixational drift, smooth pursuit and low-amplitude mechanical vibrations o

22 mined the effects of the microstimulation on smooth pursuit and on the compensation for target veloci

23 the visual cortical pathways that drive both smooth pursuit and perception.

24 , we analyzed variability in visually driven smooth pursuit and perceptual reports of target directio

25 in 36 preterm and 33 full-term subjects and smooth pursuit and saccades in 21 preterm and 19 full-te

26 ral algorithms for how the motor systems for smooth pursuit and saccadic eye movements might extract

27 correlated both with initial acceleration of smooth pursuit and with peak gain, but was not significa

28 ntitatively examine the control of saccades, smooth pursuit, and antisaccades in children who were bo

29 ts, including microsaccades, small saccades, smooth pursuit, and fixation.

30 M subsystem superimposes saccadic turns upon smooth pursuit; and (5) the two systems in combination a

31 As expected from previous results, the smooth pursuit before the first saccade reflected a vect

32 rom premotor pathways mediating saccades and smooth pursuit, but not from secondary vestibulo-ocular

33 raretinal signals, such as efference copy of smooth pursuit commands, are required to compensate for

34 cal Alzheimer's disease showed lower gain in smooth pursuit compared to controls.

35 ies of 10, 20, and 30 deg/s in six patients; smooth pursuit could not be elicited in four patients.

36 Smooth pursuit deficits in the subjects with schizophren

37 Smooth pursuit deficits were assessed outside the fMRI a

38 Sinusoidal smooth pursuit did not differentiate between controls an

39 pia as they performed horizontal or vertical smooth pursuit during monocular viewing.

40 The model was tested on data from several smooth pursuit experiments and reproduced all major char

41 ily studies have shown that abnormalities of smooth pursuit eye movement are increased in the adult r

42 Smooth pursuit eye movement gain (equal to the ratio of

43 Qualitative smooth pursuit eye movement score was significantly wors

44 In the smooth pursuit eye movement system, neural integration i

45 compare brain hemodynamic response during a smooth pursuit eye movement task in patients with schizo

46 d 14 healthy comparison subjects performed a smooth pursuit eye movement task while undergoing 1.5-T

47 During smooth pursuit eye movement, observers often misperceive

48 MT into estimates of target motion to drive smooth pursuit eye movement.

49 Abnormal smooth pursuit eye movements (SPEMs) are some of the mos

50 ve to other patients and control subjects in smooth pursuit eye movements and on the antisaccade task

51 schizophrenia and has a potential to disrupt smooth pursuit eye movements and other visual functions

52 astriate visual cortex and are used to drive smooth pursuit eye movements and perceptual judgments of

53 cortical and sub-cortical systems mediating smooth pursuit eye movements and sensorimotor gating.

54 ited the temporal specificity of learning in smooth pursuit eye movements and the well-defined anatom

55 lity to perform visually guided saccades and smooth pursuit eye movements and to suppress visually gu

56 Smooth pursuit eye movements are abnormal in patients wi

57 Deficits in smooth pursuit eye movements are an established phenotyp

58 Smooth pursuit eye movements are continuous, slow rotati

59 Smooth pursuit eye movements are generated by a motor sy

60 Learning was induced in smooth pursuit eye movements by repeated presentation of

61 The present paper asks how primate smooth pursuit eye movements choose targets, by analysis

62 By means of infrared oculography, smooth pursuit eye movements during a 17 degrees /second

63 es for saccades and increasing responses for smooth pursuit eye movements from posterior/medial to an

64 Abnormal smooth pursuit eye movements have been found in many sch

65 ard systems alter motor behavior, we studied smooth pursuit eye movements in monkeys trained to assoc

66 ulus form and contrast for the initiation of smooth pursuit eye movements in monkeys, we show that vi

67 Abnormal smooth pursuit eye movements in patients with schizophre

68 of Caenorhabditis elegans to the control of smooth pursuit eye movements in primates, and from the c

69 Patients with schizophrenia have abnormal smooth pursuit eye movements in response to a step-ramp

70 Abnormal smooth pursuit eye movements in schizophrenia and relate

71 bance in a frontotemporal network subserving smooth pursuit eye movements in schizophrenia.

72 theory of mind in autism to abnormalities of smooth pursuit eye movements in schizophrenia.

73 oal was to test the hypothesis that abnormal smooth pursuit eye movements in schizophrenic patients a

74 We have used motor learning in smooth pursuit eye movements of monkeys to determine how

75 je cells during trial-over-trial learning in smooth pursuit eye movements of monkeys.

76 the neural code for sensory-motor latency in smooth pursuit eye movements reveals general principles

77 nnection between visual motion estimates and smooth pursuit eye movements to measure stimulus-respons

78 es instructive signals for motor learning in smooth pursuit eye movements under natural conditions, s

79 ation/target gap and overlap conditions) and smooth pursuit eye movements using an infrared pupil-tra

80 Smooth pursuit eye movements were assessed during both t

81 the stimulus, we assessed the initiation of smooth pursuit eye movements when two targets move in di

82 und only small idiosyncratic anisotropies in smooth pursuit eye movements, a motor action requiring a

83 leading (small anticipatory) saccades during smooth pursuit eye movements, and cancellation of reflex

84 consists of orienting saccades and tracking smooth pursuit eye movements, and found strong physiolog

85 During smooth pursuit eye movements, both tracking position and

86 activity in the frontal eye fields controls smooth pursuit eye movements, but the relationship betwe

87 ficient velocity discrimination and impaired smooth pursuit eye movements, inasmuch as the brain regi

88 round stimuli sweep across the retina during smooth pursuit eye movements, non-pursued targets are us

89 In smooth pursuit eye movements, the response to a brief pe

90 dic target trajectories and emit pre-emptive smooth pursuit eye movements--prior to the emergence of

91 TEMENT When an object moves, we view it with smooth pursuit eye movements.

92 time scale comparable with the initiation of smooth pursuit eye movements.

93 ppocampi and the right fusiform gyrus during smooth pursuit eye movements.

94 trong sequelae in the direction and speed of smooth pursuit eye movements.

95 e cerebral cortex is part of the circuit for smooth pursuit eye movements.

96 ides the visual inputs for behaviors such as smooth pursuit eye movements.

97 d the range of dynamics normally seen during smooth pursuit eye movements.

98 t is associated with a profound asymmetry in smooth pursuit eye movements.

99 l-by-trial variations in neural activity and smooth pursuit eye movements.

100 area (MT) are correlated with variability in smooth pursuit eye movements.

101 ects were larger while the animal was making smooth pursuit eye movements.

102 e relationship between motion perception and smooth pursuit eye movements.

103 precise motor timing by studying learning in smooth pursuit eye movements.

104 Because both smooth pursuit eye tracking dysfunction and obstetrical

105 otic (MZ) twins have suggested that abnormal smooth pursuit eye tracking is an indicator of genetic l

106 In this study, the authors compared smooth pursuit eye tracking, a biological trait marker a

107 ts and many of their relatives show impaired smooth pursuit eye tracking.

108 constant velocity (16.67 degrees per second) smooth pursuit eye tracking.

109 th pursuit gain measure, which is a ratio of smooth pursuit eye velocity in response to both retinal

110 e we show that electrical stimulation of the smooth-pursuit eye movement region in the arcuate sulcus

111 Gain control is also an integral part of the smooth-pursuit eye movement system.

112 signal-to-noise ratio for the initiation of smooth-pursuit eye movements as a function of time and c

113 half-angle rule of ocular kinematics during smooth-pursuit eye movements from eccentric positions.

114 Here we use visually guided smooth-pursuit eye movements in primates as a testing gr

115 ure-based attention on motion perception and smooth-pursuit eye movements in response to moving dicho

116 tracked target motion with normal, high-gain smooth-pursuit eye movements right up until the target w

117 Smooth-pursuit eye movements transform 100 ms of visual

118 Also, the schizophrenic patients' smooth-pursuit eye movements were tested in response to

119 gion is known to be involved in saccadic and smooth-pursuit eye movements, we propose that a nearby r

120 mensional derivative of eye position, during smooth-pursuit eye movements.

121 complications, and their relatives had worse smooth-pursuit eye movements.

122 intermediate stage in the neural circuit for smooth-pursuit eye movements.

123 sition dependence as seen in visually guided smooth-pursuit eye movements.

124 analysis to show that the initial changes in smooth-pursuit eye speed are driven by low-level motion

125 Smooth-pursuit eye velocity to a moving target is more a

126 the locus of this and other ketamine-induced smooth-pursuit eye-movement deficits involves NMDA recep

127 or placebo in double-blind fashion during a smooth-pursuit eye-movement task.

128 Eye movements were recorded during smooth pursuit, fixation stability, and free-viewing tas

129 Sinusoidal smooth pursuit function decreases modestly for horizonta

130 sis was performed with saccadic velocity and smooth pursuit gain as dependent variables and comparing

131 No major improvement in smooth pursuit gain could be attributed to drug treatmen

132 However, the traditional smooth pursuit gain in response to both retinal and extr

133 Normal smooth pursuit gain in response to both retinal and extr

134 The traditional smooth pursuit gain measure, which is a ratio of smooth

135 measures: peak saccadic velocity and average smooth pursuit gain.

136 impairment of saccadic velocity but not with smooth pursuit gain.

137 Smooth-pursuit gains were 0.28 to 1.19, 0.11 to 0.68, an

138 tion, newly developed measures of predictive smooth pursuit (ie, in the presence of only extraretinal

139 rence for saccades was found in SPL1 and for smooth pursuit in IPS5.

140 unction in the initiation and maintenance of smooth pursuit in schizophrenia.

141 beliefs) can account for several features of smooth pursuit in schizophrenia: namely, a reduction in

142 ration of disruptive leading saccades during smooth pursuit is thought to be mediated by frontal-thal

143 Smooth-pursuit latencies tended to be slightly longer in

144 may explain the influence of eye position on smooth pursuit maintenance.

145 ctory of oblique saccades, and initiation of smooth pursuit, may aid in diagnosing these different ty

146 both cases, results showed no alterations in smooth pursuit, meaning that its velocity was unaffected

147 uggests normal retinal motion processing and smooth pursuit motor output.

148 e dragonfly makes a head saccade followed by smooth pursuit movements to orient its direction-of-gaze

149 tic nystagmus (MOKN), monocular asymmetry of smooth pursuit (MSP), and perceived monocular speed bias

150 ces a model of oculomotor control during the smooth pursuit of occluded visual targets.

151 ries to accommodate the cardinal features of smooth pursuit of partially occluded targets that have b

152 lly explained by motor deficits in saccades, smooth pursuit, or fixation.

153 the movement was purposeful, as in vertical smooth pursuit, or whether it was inappropriate, as in a

154 Humans and monkeys are able to adapt their smooth pursuit output when challenged with consistent er

155 half syndrome (3), saccadic palsy (28), and smooth pursuit palsy (46).

156 y--to provide Bayes optimal solutions to the smooth pursuit problem.

157 l motion signals delivered to one eye during smooth pursuit produce adaptation in the fellow eye.

158 rrelations were computed between measures of smooth pursuit (qualitative rating, peak gain, saccade f

159 SC neurons also exhibit fixation-related and smooth-pursuit-related activity.

160 ly, 100 to 200 trials were used to adapt the smooth pursuit response.

161 c eye movements, a specific component of the smooth-pursuit response shown to be abnormal in schizoph

162 ccades and the ratios of leading saccades to smooth-pursuit response time and to total saccadic eye-m

163 tion, but for some separations evoked larger smooth pursuit responses from both humans and monkeys th

164 cal and physiological mechanisms that govern smooth pursuit, saccades, and the vestibulo-ocular refle

165 onary stimuli and stimuli designed to elicit smooth pursuit, saccades, optokinetic nystagmus (OKN), v

166 performed visually guided saccade (VGS) and smooth-pursuit (SmP) tasks.

167 assive and active following of a predictable smooth pursuit stimulus in order to establish if predict

168 The smooth pursuit stimulus was presented in pairs that were

169 rimate sensorimotor systems, for example the smooth pursuit system and their ability to compensate fo

170 s in conjugate eye position as tested during smooth-pursuit, thereby verifying that the responses wer

171 s fail to show any disruption of eye motion, smooth pursuit velocity being unaffected.

172 phases of nystagmus were also affected, but smooth pursuit, vergence, and the vestibuloocular reflex

173 isually guided eye movements (e.g., saccades/smooth pursuit/vergence).

174 Abnormal vertical smooth pursuit was present in 17 (57%) of 30 subjects: n

175 nce of genetic factors on characteristics of smooth pursuit were evaluated in young adult monozygotic

176 Horizontal and vertical smooth pursuit were measured in different eye-in-orbit p

177 zheimer's disease, CBS and PSP, saccades and smooth pursuit were measured in three FTLD subtypes, inc