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1 sn-1-Diacylglycerol lipase alpha (DAGL-alpha) is the mai

2 Oleic acid occupied typically the sn-1/sn-3 positions but when together with FAs 20:1, 20:2, 18

3 1-Oleoyl-2acetyl-sn-glycerol (OAG), a TRPC agonist, elicited responses in

4 lycerols rich in CLA, with a ratio of sn-1,3/sn-1,2 regioisomers of 21.8, compared to 2.3 for Novozym

5 mpared to their interesterified mix, using a sn-1,3 stereospecific lipase, to determine if there was

6 muscle revealed that HSL KO mice accumulated sn-1,3 DAG and not sn-1,2 DAG.

7 he diacylglycerol analogue 1-oleoyl-2-acetyl-sn-glycerol (OAG) reversed the inhibitory effect of cand

8 the diacylglycerol analog 1-oleoyl-2-acetyl-sn-glycerol (OAG), an agent that causes activation of Ca

9 the diacylglycerol analog 1-oleoyl-2-acetyl-sn-glycerol and CsA blocked cell death and arachidonic a

10 se were blocked by CsA and 1-oleoyl-2-acetyl-sn-glycerol but not by pyrrophenone or EGTA.

11 DTT-insensitive CDP-choline 1-alkyl-2-acetyl-sn-glycerol cholinephosphotransferase (PAF-CPT), and its

12 t both angiotensin II- and 1-oleoyl-2-acetyl-sn-glycerol-induced Ca(2+) entry in these cells, which w

13 nds 1-(4-hydroxy-3-methoxy) cinnamoyl-2-acyl-sn-glycero-3-phosphocholine and 1-(4-hydroxy-3,5-dimetho

14 1-(4-hydroxy-3,5-dimethoxy) cinnamoyl-2-acyl-sn-glycero-3-phosphocholine exhibited excellent antioxid

15 pound 1-(4-hydroxy-3-methoxy) benzoyl-2-acyl-sn-glycero-3-phosphocholine exhibited good antifungal ac

16 ycerophospholipids as 1-O-alk-1'-enyl-2-acyl-sn-glycero-3-phosphoethanolamine or plasmenylethanolamin

17 vity at the level of acyl-CoA:1-acylglycerol-sn-3-phosphate acyltransferase.

18 oxyacetone phosphate transport system and an sn-glycerol-3-phosphate dehydrogenase.

19 ngly unsaturated PC bilayers (sn-1: 16:0 and sn-2: 18:1...22:6; or sn-1 and sn-2: 18:1...22:6).

20 differences in the structure of the sn-1 and sn-2 acyl-binding sites of the protein.

21 s a cycle that enriches CPA at both sn-1 and sn-2 positions of PC and results in increased accumulati

22 on from fragment ions unique to the sn-1 and sn-2 positions, and the positions of carbon-carbon doubl

23 tidylcholine (PC) esterified at the sn-1 and sn-2 positions, with alpha-eleostearic acid (9Z, 11E, 13

24 ing unsaturated acyl chains in both sn-1 and sn-2.

25 n-1: 16:0 and sn-2: 18:1...22:6; or sn-1 and sn-2: 18:1...22:6).

26 of medium-chain fatty acids in the sn-2 and sn-3 positions of seed triacylglycerols (TAGs).

27 r species, total fatty acids, and sn-1+3 and sn-2 positions in the two lipid pools are similar, excep

28 tants, the esterification of both sn-1,3 and sn-2 positions of glycerol was impacted, and their cutin

29 tivity, with hydrolysis from both sn-1,3 and sn-2 sites being equally favoured.

30 G) molecular species, total fatty acids, and sn-1+3 and sn-2 positions in the two lipid pools are sim

31 through controlling both mRNA elongation and sn/snoRNA synthesis, the 7SK snRNP is a key regulator of

32 etail, their triacylglycerols identified and sn-2 positional arrangement of fatty acid constituents a

33 regioselective preparation of sn-1 mono and sn-1,3 diacylglycerols rich in CLA, with a ratio of sn-1

34 Oxidized 1-palmitoyl-2-arachidonoyl-sn-glycero-3-phosphocholine (OxPAPC) and its derivatives

35 , the products of 1-palmitoyl-2-arachidonoyl-sn-glycero-3-phosphocholine (PAPC) oxidation that contai

36 lipids (oxidized 1-palmitoyl-2-arachidonoyl-sn-glycero-3-phosphocholine [Ox-PAPC]) and proinflammato

37 olipids (oxidized 1-palmitoyl-2-arachidonoyl-sn-glycero-3-phosphocholine [OxPAPC]) promote endothelia

38 ALE: Oxidation of 1-palmitoyl-2-arachidonoyl-sn-glycero-3-phosphorylcholine (OxPAPC) generates a grou

39 ated phospholipid 1-palmitoyl-2-arachidonoyl-sn-glycero-phosphocholine.

40 produce the endocannabinoid, 2-arachidonoyl-sn-glycerol (2-AG) upon antigen activation.

41 1, increased endocannabinoid (2-arachidonoyl-sn-glycerol (2-AG)) levels in the taste organ, and enhan

42 ation of the endocannabinoid, 2-arachidonoyl-sn-glycerol (2-AG), in the amygdala.

43 vation of the endocannabinoid 2-arachidonoyl-sn-glycerol (2AG), is tightly controlled by the cell's r

44 process was not completely regiospecific at sn-1,3 positions, due to the spontaneous acyl migration

45 ses in increasingly unsaturated PC bilayers (sn-1: 16:0 and sn-2: 18:1...22:6; or sn-1 and sn-2: 18:1

46 is enables a cycle that enriches CPA at both sn-1 and sn-2 positions of PC and results in increased a

47 regioselectivity, with hydrolysis from both sn-1,3 and sn-2 sites being equally favoured.

48 o PCs having unsaturated acyl chains in both sn-1 and sn-2.

49 lyzed the acylation and de-acylation of both sn positions of PC, with a preference for the sn-2 posit

50 in these mutants, the esterification of both sn-1,3 and sn-2 positions of glycerol was impacted, and

51 t altering the bioavailability determined by sn-2 stereochemistry, could delay lipid absorption at th

52 NAs), small nuclear, nucleolar, cytoplasmic (sn-, sno-, scRNAs, respectively), transfer (tRNAs), and

53 f diacylglycerol to form 3-acetyl-1,2-diacyl-sn-glycerol (acetyl-TAG).

54 ted the exchange of gA between 1,2-dierucoyl-sn-glycero-3-phosphocholine (DC22:1PC) or 1,2-dioleoyl-s

55 is approach is demonstrated to differentiate sn-positional and double-bond-positional isomers, such a

56 (Arr(Tr)) with rhodopsin in 1,2-diheptanoyl-sn-glycero-3-phosphocholine (DHPC) micelles is investiga

57 om Escherichia coli in mixed 1,2-diheptanoyl-sn-glycerol-3-phosphocholine/1-myristoyl-2-hydroxy-sn-gl

58 o-3-phosphocholine (DOPC)/1,2-dihexadecanoyl-sn-glycero-3-phosphocholine (DPPC) membranes.

59 ethiol) and phospholipid [1,2-dihexadecanoyl-sn-glycero-3-phosphocholine, 1,2-dipalmitoyl-sn-glycero-

60 o-3-phosphocholine (DMPC) and 1,2-dihexanoyl-sn-glycero-3-phosphocholine (DHPC) self-assemble to form

61 LPS, endotoxin) in water with 1,2-dihexanoyl-sn-glycero-3-phosphocholine as detergent.

62 ged 80:20 diC12:0PC:diC12:0PG [1,2-dilauroyl-sn-glycero-3-phospho-(1'-rac-glycerol)] liposomes were i

63 o both zwitterionic diC12:0PC (1,2-dilauroyl-sn-glycero-3-phosphocholine) liposomes and negatively ch

64 Bilayers prepared using 1,2-dilauroyl-sn-glycero-3-phosphocholine, a lipid with 12 carbon acyl

65 t on the membrane surface in 1,2-dimyristoyl-sn-glycero-3-phosphatidylcholine (DMPC)/1,2-dimyristoyl-

66 3-phosphatidylcholine (DMPC)/1,2-dimyristoyl-sn-glycero-3-phosphatidylglycerol (DMPG) and 1-palmitoyl

67 -sn-glycero-3-phosphocholine/1,2-dimyristoyl-sn-glycero-3-phospho- (1'-rac-glycerol)/cholesterol lipi

68 1,2-Dimyristoyl-sn-glycero-3-phosphocholine (DMPC) and its mixtures with

69 show that liquid crystalline 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) and POPC/POPS 3:1 lip

70 s and the zwitterionic lipid 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) are investigated as c

71 ct tOmpA folding kinetics in 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) liposomes, suggesting

72 ree different phospholipids (1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC), 1,2-dipalmitoyl-sn-g

73 itions in single bilayers of 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC)/1,2-dipalmitoyl-sn-gl

74 Conversely, in the 1,2-dimyristoyl-sn-glycero-3-phosphocholine bilayer, the overall hydroph

75 Using model membranes of 1,2-dimyristoyl-sn-glycero-3-phosphocholine lipids at pH > pHagg, we fou

76 ) to EmrE reconstituted into 1,2-dimyristoyl-sn-glycero-3-phosphocholine liposomes by (31)P MAS NMR.

77 melting of lipid domains in 1,2-dimyristoyl-sn-glycero-3-phosphocholine vesicles is observed to occu

78 obular actin-supported DMPC (1,2-dimyristoyl-sn-glycero-3-phosphocholine) bilayers, deposited via the

79 the phase behaviour of DMPC (1,2-dimyristoyl-sn-glycero-3-phosphocholine) multilamellar vesicles.

80 g HA FP to TMD-reconstituted 1,2-dimyristoyl-sn-glycero-3-phosphocholine/1,2-dimyristoyl-sn-glycero-3

81 s of Abeta(1-40) interacting 1,2-dimyristoyl-sn-glycero-3-phosphoglycerol (DMPG) bilayers.

82 ueous solution the phospholipids dimyristoyl-sn-glycero-3-phosphocholine (DMPC) and 1,2-dihexanoyl-sn

83 Repeated addition of 1,2-dioctanoyl-sn-glycerol (DiC8) resulted in sustained plasma membrane

84 gated and the results show that 1,2-dioleoyl-sn-glycero-3-[phospho-L-serine] (DOPS) small unilamellar

85 -organic frameworks (MOFs) with 1,2-dioleoyl-sn-glycero-3-phosphate (DOPA) is presented.

86 osphatidic acid, whereby 300 nM 1,2-dioleoyl-sn-glycero-3-phosphate (DOPA), but not the control 1,2-d

87 rganic solvent by coating with 1, 2-dioleoyl-sn-glycero-3-phosphate (DOPA).

88 Using 1,2-dioleoyl-sn-glycero-3-phosphatidylcholine (DOPC) nanoliposomes, w

89 timulated 2-fold by liver PC or 1,2-dioleoyl-sn-glycero-3-phosphatidylethanolamine lipids.

90 oyl-sn-glycero-3-phosphocholine/1,2-dioleoyl-sn-glycero-3-phosphochol ine with varying concentrations

91 -3-phosphocholine (DC22:1PC) or 1,2-dioleoyl-sn-glycero-3-phosphocholine (DC18:1PC) lipid vesicles us

92 nm diameter) composed of either 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC) (fluid at room temper

93 ptors that can efficiently bind 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC) in nonpolar solvents.

94 B subunit (CTB) to a GM1-doped 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC) lipid bilayer were in

95 es, composed of an FDA-approved 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC) lipid monomer.

96 ation of nanoscale, fluid-phase 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC) liposomes contacting

97 ncer, delivery of miR-630 using 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC) nanoliposomes resulte

98 ere prepared by spreading giant 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC) vesicles on porous an

99 ate (DOPA), but not the control 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC), binds directly to S6

100 ry lipid mixture SM/cholesterol/1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC), focusing on the impo

101 he various lipids investigated, 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC)-based proteoliposomes

102 ero-3-phosphocholine (POPC) and 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC).

103 model peptides in a bilayer of 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC).

104 ro-3-phosphocholine (DPhPC) and 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC)/1,2-dihexadecanoyl-sn

105 ylcholine unilamellar vesicles [1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC)/1,2-dipalmitoyl-sn-gl

106 ave alamethicin (alm) pore in a 1,2-dioleoyl-sn-glycero-3-phosphocholine bilayer at 313 K indicates t

107 rce for each compound through a 1,2-dioleoyl-sn-glycero-3-phosphocholine bilayer is determined by mol

108 it is embedded show that in the 1,2-dioleoyl-sn-glycero-3-phosphocholine bilayer, charged residues of

109 Here we show, using supported 1,2-dioleoyl-sn-glycero-3-phosphocholine lipid bilayers in different

110 act voltammetry with the aid of 1,2-dioleoyl-sn-glycero-3-phosphocholine liposomes.

111 apparent mismatch produced by a 1,2-dioleoyl-sn-glycero-3-phosphocholine thicker bilayer could be a s

112 tion of ternary lipid mixtures (1,2-dioleoyl-sn-glycero-3-phosphocholine/sphingomyelin/cholesterol) i

113 apthol SBN intercalation into a 1,2-dioleoyl-sn-glycero-3-phosphocoline (DOPC) bilayer.

114 ntly complexed with two lipids, 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine (DOPE) and 1,2-dioleoyl

115 with fusogenic properties such as 2-dioleoyl-sn-glycero-3-phosphoethanolamine (DOPE) are integrated i

116 ) and the zwitterionic liposome 1,2-Dioleoyl-sn-Glycero-3-Phosphoethanolamine (DOPE) were tethered on

117 enic lipids (10-30mol% DOTAP or 1,2-dioleoyl-sn-glycero-3-phosphoethanolamine, 1-20mol% DOPE or 1,2-d

118 cently labeled lipid, NBD-DOPE [1,2-dioleoyl-sn-glycero-3-phosphoethanolamine-N-(7-nitro-2-1,3-benzox

119 lamellar vesicles membranes made of dioleoyl-sn-glycero-3-phosphocholine (DOPC) or 1-palmitoyl-2-oleo

120 thyleneimine (PEI)(1.8 kDa), and 1,2-dioleyl-sn-glycero-3-phosphoethanolamine (DOPE) units (the nanoc

121 icomponent lipid bilayers of 1,2-dipalmitoyl-sn-glycero-3-phosphatidylcholine (DPPC), 1,2-distearoyl-

122 are less permeable than pure 1,2-dipalmitoyl-sn-glycero-3-phosphatidylcholine or DSPC bilayers.

123 sn-glycero-3-phosphocholine, 1,2-dipalmitoyl-sn-glycero-3-phospho-(1'-rac-glycerol)] units.

124 luid at room temperature) or 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC) (gel at room temperat

125 layers of binary mixtures of 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC) and asialo-(GA1), dis

126 ify solute partitioning into 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC) lipid vesicles as a f

127 ents with stiffer, gel-phase 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC) liposomes verified th

128 pid vesicles having either a 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC) or mixed-DPPC/cardiol

129 cero-3-phosphocholine (DMPC)/1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC) phospholipid mixtures

130 ero-3-phosphocholine (DMPC), 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC), and 1-palmitoyl-2-ol

131 (l(d)) bilayers derived from 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC).

132 cero-3-phosphocholine (DOPC)/1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC)], we report a dramati

133 -plane phonon excitations in 1,2-dipalmitoyl-sn-glycero-3-phosphocholine above and below the main tra

134 glycero-3-phosphocholine and 1,2-dipalmitoyl-sn-glycero-3-phosphocholine in the liquid-ordered (lo) a

135 nt molecular ratios of DPPC (1,2-dipalmitoyl-sn-glycero-3-phosphocholine) and POPC (1-palmitoyl-2-ole

136 iant unilamellar vesicles of 1,2-dipalmitoyl-sn-glycero-3-phosphocholine.

137 ultilayers consisting of 1:1 1,2-dipalmitoyl-sn-glycero-3-phosphocholine/1,2-dioleoyl-sn-glycero-3-ph

138 -3-phosphocholine (DSPC) and 1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine (DPPE), were used to st

139 in antibodies for biotin-cap-1,2-dipalmitoyl-sn-glycero-3-phosphoethanolamine-N-(7-nitro-2-1,3-benz o

140 Chol (cholesterol) and Phos (1,2-dipalmitoyl-sn-glycerol-3-phospho-(1'rac-glycerol)) via disulfide bo

141 -sn-glycero-3-phosphocholine 1,2-diphytanoyl-sn-glycero-3-phosphocholine (DPhPC) and 1,2-dioleoyl-sn-

142 the self-assembly of stable 1,2-diphytanoyl-sn-glycero-3-phosphocholine 1,2-diphytanoyl-sn-glycero-3

143 The protein was dispersed in diphytanoyl-sn-glycero-3-phosphocholine lipid bilayers, and the spec

144 ted with a high proportion of 1,2 distearoyl-sn-glycero-3-phosphoethanolamine (DSPE) released up to 3

145 3-phosphatidylcholine (DPPC), 1,2-distearoyl-sn-glycero-3-phosphatidylcholine (DSPC), isostearyl isos

146 Two phospholipids, 1,2-distearoyl-sn-glycero-3-phosphocholine (DSPC) and 1,2-dipalmitoyl-s

147 nd the saturated phospholipid 1,2-distearoyl-sn-glycero-3-phosphocholine (DSPC), mixed with varying a

148 well as incubating samples of 1,2-distearoyl-sn-glycero-3-phosphocholine at 60 degrees C for 24-72 h

149 preferences of GA dimers from 1,2-distearoyl-sn-glycero-3-phosphocholine bilayers were significantly

150 olar ratio of cholesterol and 1,2-distearoyl-sn-glycero-3-phosphocholine, incorporating K[nido-7-CH3(

151 phatidylcholine and DSPE-PEG [1,2-distearoyl-sn-glycero-3-phosphoethanolamine-N-[amino(polythylene gl

152 Gal-(1 --> 4)-beta-D-GlcNAc-1,2-di-O-dodecyl-sn-glycero (B2NGL) served as model protein-GL complexes

153 reveal both the acyl chain assignment (i.e., sn-position) and the site-specific location of double bo

154 t of 1-palmitoyl-2-(5,6-epoxyisoprostane E2)-sn-glycero-3-phosphocholine, PEIPC, a proinflammatory mo

155 Both LPEATs could acylate either sn position of ether analogs of LPC The data show that t

156 ids attached via ester bonds to enantiomeric sn-glycerol 3-phosphate.

157 a female-sterile allele of the singed gene (sn(X2)) on FM7c with a sequence from balanced chromosome

158 ocholine (POVPC) and 1-palmitoyl-2-glutaroyl-sn-glycero-3-phosphocholine (PGPC), are two major oxidat

159 hocholine (POVPC) or 1-palmitoyl-2-glutaroyl-sn-glycero-3-phosphocholine, two oxidized phospholipids

160 phosphocholine], and 1-palmitoyl-2-glutaroyl-sn-glycero-3-phosphocholine, were identified in the extr

161 nsferase and mitochondrial acyl-CoA:glycerol-sn-3-phosphate acyltransferase and an increase in serine

162 amics of human tBid in 1-palmitoyl-2-hydroxy-sn-glycero-3-[phospho-RAC-(1-glycerol)] micelles.

163 urified Vo sector with 1-palmitoyl-2-hydroxy-sn-glycero-3-[phospho-rac-(1-glycerol)] resulted in sele

164 cerol-3-phosphocholine/1-myristoyl-2-hydroxy-sn-glycero-3-p hospho-(1'-rac-glycerol) micelles is pres

165 ore deeply inserted in 1-myristoyl-2-hydroxy-sn-glycero-3-phospho-1'-rac-glycerol (LMPG, anionic) tha

166 (LMPG, anionic) than in 1-lauroyl-2-hydroxy-sn-glycero-3-phosphocholine (LLPC, zwitterionic) micelle

167 Radiolabeling identified sn-2 monoacylglycerol as an initial glycerolipid interme

168 n length, as well as the presence of 16:0 in sn-1 of the unsaturated PCs and the total number of cis

169 ss ordered in unsaturated PCs having 16:0 in sn-1, as compared to PCs having unsaturated acyl chains

170 fatty acids are preferentially esterified in sn-2 position in hazelnut oil, while no significant pref

171 bisphosphate and the large, late increase in sn 1,2-diacylglycerol in fertilization.

172 rring loss-of-function mutations among known sn cultivars.

173 loomington Drosophila Stock Center have lost sn(X2) by this mechanism on a historical timescale.

174 26 cells, the LPA3 agonist 1-oleoyl-2-methyl-sn-glycero-3-phosphothionate (2S-OMPT) promoted erythrop

175 t HSL KO mice accumulated sn-1,3 DAG and not sn-1,2 DAG.

176 The U1 small nuclear (sn)RNA (U1) is a multifunctional ncRNA, known for its pi

177 tion of small nucleolar (sno)/small nuclear (sn)RNA genes is terminated by the RNA-binding proteins N

178 The U1 small nuclear (sn)RNA participates in splicing of pre-mRNAs by recogniz

179 nt CPS prefers the stereospecific numbering (sn)-1 position whereas E. coli CPS acts on sn-2 of phosp

180 d backbone, 1-hexadecyl-2-(11Z-octadecenoyl)-sn-glycero-3-phospho-(1'-myo-inositol), in which the sn-

181 hree regioisomers of 1,2-di(9Z-octadecenoyl)-sn-glycero-3-[phosphoinositol-x,y-bisphosphate] (PI(3,4)

182 en POPE (1-hexadecanoyl-2-(9-Z-octadecenoyl)-sn-glycero-3-phosphoethanolamine) was added.

183 was identified as 1-hexadecyl-2-octadecenoyl-sn-glycero-3-phosphocholine [PC(16:0e/18:1)] using tande

184 One interesting by-product (18% of sn-2 monoacylglyceride of DHA) remained at the end of th

185 skeletal muscle and that the accumulation of sn-1,3 DAG originating from lipolysis does not inhibit i

186 (TAG) by the acyl-CoA-dependent acylation of sn-1,2-diacylglycerol catalyzed by diacylglycerol acyltr

187 tidic acid that begins with the acylation of sn-glycerol-3-phosphate by PlsY using an acyl-phosphate

188 nt was attributed to decreased expression of sn-1,2 diacylglycerol acyltransferase and mitochondrial

189 ransmembrane helices observed as increase of sn-1 chain order, while thicker bilayers were compressed

190 We further showed that after incubation of sn-2-[(14)C]acyl-PC, formation of [(14)C]TAG was only po

191 ribosomes) and RNA modification (as part of sn/oRNPs), has also been identified as a subunit of arch

192 on of a phospholipid to the sn-3-position of sn-1,2-diacylglyerol, thus forming triacylglycerol and a

193 rth an optimal regioselective preparation of sn-1 mono and sn-1,3 diacylglycerols rich in CLA, with a

194 The proportion of sn-2-arachidonoyl-phosphatidylcholine (20:4-PC) inversel

195 diacylglycerols rich in CLA, with a ratio of sn-1,3/sn-1,2 regioisomers of 21.8, compared to 2.3 for

196 2 (PLA2) catalyze the hydrolysis reaction of sn-2 fatty acids of membrane phospholipids and are also

197 lycero-3-phosphocholine/1-palmitoyl-2-oleoyl-sn-glycero- 3-phosphoglycerol bilayer.

198 gatively charged lipid, 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphatidic acid acid (POPA), in supported

199 idylglycerol (DMPG) and 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphatidylcholine (POPC)/1-palmitoyl-2-ol

200 sphatidylcholine (POPC)/1-palmitoyl-2-oleoyl-sn-glycero-3-phosphatidylglycerol (POPG), as expected, w

201 membrane lipids, POPG (1-palmitoyl-2-oleoyl-sn-glycero-3-phospho-(1'-rac-glycerol) (sodium salt)), P

202 nding of TAT to anionic 1-palmitoyl-2-oleoyl-sn-glycero-3-phospho-1'-rac-glycerol (POPG) and neutral

203 osphocholine (POPC) and 1-palmitoyl-2-oleoyl-sn-glycero-3-phospho-L-serine (POPS) 3:1 mol/mole and at

204 pp), between Cu(2+) and 1-palmitoyl-2-oleoyl-sn-glycero-3-phospho-l-serine (POPS), a negatively charg

205 ) (sodium salt)), POPS (1-palmitoyl-2-oleoyl-sn-glycero-3-phospho-l-serine (sodium salt)), and gangli

206 enzymatic hydrolysis of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (PC), a zwitterionic lipid.

207 saturated phospholipids 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC) and 1,2-dioleoyl-sn-g

208 n membranes composed of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC) and 1-palmitoyl-2-ole

209 erol (POPG) and neutral 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC) liposomes.

210 ns specifically bind to 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC) membranes, whereas Cl

211 nce on the structure of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC) membranes.

212 nsaturated phospholipid 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC) the two-state model w

213 sphocholine (DPPC), and 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC)), and the average GM1

214 hosphocholine (DOPC) or 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC), by irradiating methy

215 we show that the ApoA1-1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC)-based particles are d

216 size and composition of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine (POPC)-containing PDs at neu

217 exchangeable mimics of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine and 1,2-dipalmitoyl-sn-glyce

218 In a 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine lipid bilayer and a plasma m

219 sins were inserted into 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine lipid nanodiscs and the kine

220 ased on solid-supported 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine membranes doped with differe

221 branes containing POPC (1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine) yielded an equilibrium diss

222 osphocholine) and POPC (1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine).

223 an its association with 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine, in bilayers with equal acyl

224 res in a fully hydrated 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphocholine/1-palmitoyl-2-oleoyl-sn-glyc

225 e lipid polymorphism of 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoethanolamine (POPE), using different

226 nfection-derived lipids 1-palmitoyl-2-oleoyl-sn-glycero-3-phosphoglycerol (POPG) and 1-palmitoyl-2-ol

227 the common phospholipid 1-palmitoyl-2-oleoyl-sn-glycerol-3-phosphocholine (16:0/18:1-PC), previously

228 kinin fragment 1-8, and 1-palmitoyl-2-oleoyl-sn-glycerol.

229 (sn)-1 position whereas E. coli CPS acts on sn-2 of phospholipids prompted us to investigate the pre

230 layers (sn-1: 16:0 and sn-2: 18:1...22:6; or sn-1 and sn-2: 18:1...22:6).

231 e (POVPC) and 1-palmitoyl-2-(9'-oxononanoyl)-sn-glycero-3-phosphocholine (PONPC), is of major interes

232 PLs), such as 1-palmitoyl-2-(5'-oxovaleroyl)-sn-glycero-3-phosphocholine (POVPC) and 1-palmitoyl-2-(9

233 ds, including 1-palmitoyl-2-(5'-oxovaleroyl)-sn-glycero-3-phosphocholine, in the lungs.

234 phospholipids, 1-palmitoyl-2-(5-oxovaleroyl)-sn-glycero-3-phosphocholine (POVPC) and 1-palmitoyl-2-gl

235 on addition of 1-palmitoyl-2-(5-oxovaleroyl)-sn-glycero-3-phosphocholine (POVPC) or 1-palmitoyl-2-glu

236 aining CD3delta segment in LPPG (1-palmitoyl-sn-glycero-3-phospho-(1'-rac-glycerol) (sodium salt)) mi

237 -(4-hydroxy-3-methoxy) cinnamoyl-2-palmitoyl-sn-glycero-3-phosphocholine exhibited good antibacterial

238 hydroxy-3,5-dimethoxy) cinnamoyl-2-palmitoyl-sn-glycero-3-phosphocholine exhibited good antioxidant a

239 the spontaneous acyl migration from position sn-2 to sn-1,3.

240 to interesterify ePL utilizing Lipozyme(R): sn-1,3 specific lipase.

241 PCATs were measured in the reverse reaction, sn-2-bound oleoyl, linoleoyl, and linolenoyl groups from

242 Lines homozygous for recessive sn mutations are early flowering and photoperiod insensi

243 genes and U small nuclear or nucleolar RNA (sn/snoRNA) loci that form intra- and inter-chromosomal c

244 acid, saturated fatty acids beta-sitosterol, sn-1 and 3 diglyceride values.

245 hibits RNAPII recruitment to RNAPII-specific sn/snoRNA genes, and reduces nascent snRNA and snoRNA sy

246 mponents of the 7SK snRNP on RNAPII-specific sn/snoRNA genes.

247 10:0 fatty acids in the Camelina sativa TAG sn-2 position, indicating a 10:0 CoA specificity that ha

248 currence of saturated fatty acids in the TAG sn-2 position is infrequent in seed oils.

249 The sn-2 position of TAGs in hybrid palm oil was shown to be

250 ith FAs 10:0, 12:0, 14:0, 20:1 and 20:2, the sn-2 preference of 16:0 was less clear.

251 rophosphatidylcholine (PC) esterified at the sn-1 and sn-2 positions, with alpha-eleostearic acid (9Z

252 hospholipids containing palmitic acid at the sn-1 position that could be exploited for the design of

253 In particular, occurrence at the sn-2 position allows optimal intestinal absorption condi

254 ances the probability that DHA chains at the sn-2 position in SDPC rise up to the bilayer surface, wh

255 T results in the incorporation of CPA at the sn-2 position of lysophosphatidic acid.

256 in transferring acyl groups modified at the sn-2 position of PC to the sn-1 position of this molecul

257 B expression generated TAGs with 14:0 at the sn-2 position, but not 10:0.

258 f concentrated fatty acids esterified at the sn-2 position.

259 However, a ricinoleoyl group at the sn-2-position of PC was removed 4-6-fold faster than an

260 orated into the sn-2 position of PC, but the sn-1 position of de novo DAG and indicated similar rates

261 Herein we describe the sn-glycerol-3-phosphate and dihydroxyacetone phosphate t

262 SDPC with SA (stearic acid) for the sn-1 chain and DHA (docosahexaenoic acid) for the sn-2 c

263 chain and DHA (docosahexaenoic acid) for the sn-2 chain is representative of polyunsaturated phosphol

264 OPC with OA (oleic acid) substituted for the sn-2 chain serves as a monounsaturated control.

265 n positions of PC, with a preference for the sn-2 position.

266 s involves the loss of a fatty acid from the sn-1/3 position in the first step followed by the loss o

267 n alpha,beta-unsaturated fatty acid from the sn-2 position in the second.

268 the transfer of a fatty acyl moiety from the sn-2 position of a phospholipid to the sn-3-position of

269 or linolenoyl than oleoyl moieties from the sn-2 position of PC to TAG.

270 have either 18:0 or 18:1 fatty acids in the sn-1 position and either 22:6 or 20:2 fatty acids in the

271 th increasing fatty acid chain length in the sn-1(3) position.

272 acylation of medium-chain fatty acids in the sn-2 and sn-3 positions of seed triacylglycerols (TAGs).

273 n and either 22:6 or 20:2 fatty acids in the sn-2 position for MS1 and MS2, respectively.

274 trate, CrDGTT1 preferred C16 over C18 in the sn-2 position of the glycerol backbone, but CrDGTT2 and

275 se TAGs contained up to 40 mol % 10:0 in the sn-2 position, nearly double the amounts obtained from c

276 C18 FAs, palmitic acid was typically in the sn-2 position.

277 ds were preferentially incorporated into the sn-2 position of PC, but the sn-1 position of de novo DA

278 of nascent and elongated acyl-CoAs into the sn-3 position of TAG.

279 e to the differences in the structure of the sn-1 and sn-2 acyl-binding sites of the protein.

280 function in proteoliposomes composed of the sn-1 chain perdeuterated lipids 14:0d27-14:1-PC, 16:0d31

281 nsferase (Lnt) catalyzes the transfer of the sn-1-acyl chain of phosphatidylethanolamine to this N-te

282 ransferase (LPAT) catalyzes acylation of the sn-2 position on lysophosphatidic acid by an acyl CoA su

283 in planta, E. coli CPS acts primarily on the sn-1 position of PC; coexpression of SfLPAT results in t

284 -2-1,3-benzoxadiazol-4-yl (NBD) group on the sn-2 C6 position, and were presumed to include phosphati

285 composition from fragment ions unique to the sn-1 and sn-2 positions, and the positions of carbon-car

286 s modified at the sn-2 position of PC to the sn-1 position of this molecule in plant cells).

287 er of an acetyl group from acetyl-CoA to the sn-3 position of diacylglycerol to form 3-acetyl-1,2-dia

288 m the sn-2 position of a phospholipid to the sn-3-position of sn-1,2-diacylglyerol, thus forming tria

289 Oleic acid occupied typically the sn-1/sn-3 positions but when together with FAs 20:1, 20:

290 ro-3-phospho-(1'-myo-inositol), in which the sn-1 position contains an ether-linked C16:0 chain; they

291 taneous acyl migration from position sn-2 to sn-1,3.

292 isoprenoid chains linked via ether bonds to sn-glycerol 1-phosphate (G1P), whereas bacteria and euka

293 ypically hydrolyze glycerophosphodiesters to sn-glycerol 3-phosphate (Gro3P) and their corresponding

294 clusters and suppresses the expression of U sn/snoRNA and histone genes.

295 ains can be either linear or branched, using sn-1,2 dipalmitoyl, dihexadecyl, diphytanoyl, and diphyt

296 ue material, [1-palmitoyl-2-(5-oxo-valeroyl)-sn-glycero-3-phosphocholine], and 1-palmitoyl-2-glutaroy

297 The accumulated DAG was sn-1,3 DAG, which is known not to activate PKC, and insu

298 triose phosphate acquisition pathway whereby sn-glycerol-3-phosphate is directly transported and inco

299 on of triacylglycerol composition along with sn-2 positional identification of the fatty acids consti

300 differences in mother's weight and diet with sn-18:1-16:0-18:1 as the most prevalent regioisomer in t

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