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1 ty of the SET9 promoter in coordination with Snail.
2 was specifically upregulated by Slug but not Snail.
3 ies of sequence-specific repressors, such as Snail.
4 ssociated with symmetry breaking in the pond snail.
5 dent degradation of the E-cadherin repressor SNAIL.
6 -34a and its direct targets Notch, Slug, and Snail.
7 directly facilitate capture by the predatory snail.
8 , and the epithelial-to-mesenchymal mediator Snail.
9 ion of HDAC1, DNMT3b, DNMT1, and mSin3A with SNAIL.
10 a known EMT regulator Slug, but not TWIST or Snail.
11 EndMT master regulatory transcription factor SNAIL.
12 cy, characterized by increased expression of Snail.
13 most obviously chiral animals, the gastropod snails.
14 rough contact with water containing infected snails.
15 a gondii was not detected in field-collected snails.
16 e and average life span of latently infected snails.
17 l T. gondii contamination in field-collected snails.
18 interactions with animals such as isopods or snails.
20 control of stem cell migration and show that snail-1, snail-2 and zeb-1 EMT transcription factor homo
21 f stem cell migration and show that snail-1, snail-2 and zeb-1 EMT transcription factor homologs are
23 T cells, when stably transfected with SNAIL (SNAIL/293T), displayed suppressed transcription and tran
29 iates that explain empirical observations of snail abundance under the actual climatic forcings exper
30 ng protein (AChBP), a humanized chimera of a snail AChBP, which has 71% sequence similarity with the
31 Methylmercury concentrations in clams and snails also declined with productivity, and consumer con
33 xpression changes, including upregulation of Snail and beta-catenin, and increased cell migration and
36 RNA) mediates a physical interaction between Snail and enhancer of zeste homolog 2 (EZH2), an enzymat
37 e often governed by the transcription factor Snail and entail the loss of apical junctions from epith
38 hosphorylation to promote the translation of Snail and Mmp-3 mRNAs, and the induction of epithelial-t
39 induced expression of cyclin-D1, cyclin-E1, snail and MMP2 and inhibited the expression of P53 and P
40 cells express conserved factors such as Msx, Snail and Pax3/7 and generate melanin-containing pigment
42 n MDA-MB-231 cells impaired the induction of Snail and Slug expression by EGF, and this effect was as
43 ty of E-cadherin transcriptional repressors, snail and slug, induced by transforming growth factor-be
46 ation of Cat L, decreased binding of CUX1 to Snail and the E-cadherin promoter, reversed EMT, and dec
47 d repertoire of EMT-related events including Snail and Twist expression, tumor cell motility, and ano
48 lated hypoxia-induced transcription factors, Snail and Twist1, leading to decreased transactivation o
50 that miR-31 directly represses Pmar1, Alx1, Snail and VegfR7 within the PMC gene regulatory network
58 field experiment that tracked parasitism in snails and people at two matched villages after prawns w
59 ortion of large, warm-adapted species (i.e., snails and predatory dipterans) relative to small-bodied
62 prostate (ARCaP-M and ARCaP-E overexpressing Snail) and breast (MDA-MB-468, MDA-MB-231, and MCF-7 ove
64 find suitable intermediate hosts (freshwater snails) and mature larvae, definitive hosts (ungulates).
65 vated the expression of mRNA for N-cadherin, Snail, and GHRH GHRH antagonist reduced the average volu
66 kton/filter feeding clam, periphyton/grazing snail, and leaves/shredding amphipod (Hyalella azteca).
68 ically in the venom glands of predatory cone snails, animals that synthesize a remarkably diverse set
71 ed and spread into novel areas where Bulinus snails are endemic, and how hybridisation could increase
72 aeological shellmounds, many species of land snails are found abundantly distributed throughout the o
77 GF-beta target genes such as PAI1, MMP9, and Snail as well as augmented TGF-beta1 secretion in mesenc
78 phalaria glabrata granulin (BgGRN)] from the snail B. glabrata, a natural host for the human blood fl
84 etical studies on the ecology of the aquatic snails (Bulinus spp. and Biomphalaria pfeifferi) that se
85 eem likely that malleodectids specialised on snails but probably also consumed a wider range of prey
86 we demonstrate that susceptible B. glabrata snails can be made resistant to infection with S. manson
87 ch patterns, like the Levy walks made by mud snails, can have their mechanistic origins in chaotic ne
88 MB-468, MDA-MB-231, and MCF-7 overexpressing Snail) cancer cells expressed lower E-cadherin activity,
90 rces for aquatic consumers like tadpoles and snails, causing bottom-up effects on wetland ecosystems.
94 present ideas for modernizing and scaling up snail control, including spatiotemporal targeting, envir
97 results suggests the use of ad hoc models of snail demography depending on habitat type (e.g., natura
99 mean of estimated thresholds of Oncomelania snail density below which the schistosomiasis transmissi
100 sed to estimate the threshold of Oncomelania snail density for interrupting schistosomiasis transmiss
101 m the periphyton to the water column in high-snail density/high-infection ponds were up to 50% higher
102 factor-beta and transglutaminase 2 stimulate Snail-dependent invadosome formation in rat and human sy
103 ctility can strengthen junctions to override Snail-dependent junctional disassembly and postpone EMT
104 s direct evidence of gain of resistance in a snail-digenean infection model using a defined factor to
105 g/kg) and BAF values (0.05) obtained for the snail directly exposed to contaminated soil were lower t
106 e without microplastics, indicating that the snails do not recognize solid nonfood particles in the s
110 anti-formin drug treatment converts dextral snail embryos to a sinistral phenocopy, and in frogs, dr
112 microbial ecology of microbes grazed by tree snails enables effective management when conservation re
114 (Zn) associated with natural particles using snails enriched with a less common Zn stable isotope.
115 tions in aquatic snails with measurements of snail excretion and tissue stoichiometry to show that pa
116 mal Snail expression in mesoderm, or ectopic Snail expression in ectoderm, is sufficient to drive ear
117 hen contractile myosin is absent, the normal Snail expression in mesoderm, or ectopic Snail expressio
121 ncoding snail family zinc finger 1, known as Snail) expression is sufficient to promote prolonged TGF
122 to Snail2/P-cadherin in the chick, but both Snail factors and Zeb2 fulfil a similar role in chick an
123 To address these questions, we collected 102 snail faecal samples as a proxy for diet, and 102 matche
124 structure was significantly distinct between snail faeces and leaf samples, but the same microbes occ
126 ater, oocysts were detected by microscopy in snail faeces and tissues for 10 and 3 days respectively.
127 etalloproteinases (Mmp3 and -9; P < 0.01), a snail family member (Snai3; P < 0.001), and osteopontin
131 factor 1, known as Twist) or Snai1 (encoding snail family zinc finger 1, known as Snail) expression i
132 how that the reactivation of Snai1 (encoding snail family zinc finger 1, known as Snail1) in mouse re
133 anscription factor (EMT-TF), a member of the Snail family, serves as a master regulator of the gland-
134 transition-like mechanism that required the Snail-family member Escargot and, in subdomains, Decapen
138 y, genetic and pharmacological inhibition of Snail function restores 4E-BP1 expression and sensitizes
140 of MMPs as well as the presence of twist and snail genes in TS Huh-7 cells were reversed by LDE225, a
142 d by studies of predator-induced shell form, snail growth rates and life history parameters of a few
147 nvestigating schistosomes in their human and snail hosts and the development of therapeutics and vacc
148 s is often very specific, such that suitable snail hosts define the geographical ranges of diseases c
149 nd T. brevior L3s to infect new, susceptible snail hosts following their release from experimentally
151 As expected from a reduction in infected snails, human schistosomiasis prevalence was 18 +/- 5% l
152 tic dynamics in the movement patterns of mud snails (Hydrobia ulvae) moving in controlled experimenta
155 A rats and RA patients, whereas knockdown of Snail in CIA joints prevents cartilage invasion and join
159 temic molluscicide for controlling slugs and snails in a wide range of agricultural and horticultural
160 tration and retention of T. gondii by marine snails in laboratory aquaria, and to test for natural T.
161 ers, as the majority ( approximately 75%) of snails in oiled habitats never reached standing unoiled
162 as dissected from non-endangered native tree snails in the same family as Achatinella to confirm that
164 s increased chemoresistance-inducing factor, Snail, in recipient epithelial cells and promote prolife
166 g expression of the EMT transcription factor Snail increased TF, coagulant properties, and early meta
172 nd also increased cercaria production by the snail intermediate hosts, causing opposing effects on ta
173 y elements of the biology and ecology of the snail intermediate hosts, together with an improved unde
175 increased in more southeastern wetlands, and snail (intermediate host) community composition had stro
182 cal mechanism of action within the recipient snail is to close off the entrance to the sperm digestio
183 ions: movements of an endangered raptor, the snail kite (Rostrhamus sociabilis plumbeus), and human m
186 ignificant modularity in annual dispersal of snail kites (all adults, males only, females only, and j
189 4E-BP1 expression inversely correlates with Snail level in cancer cell lines and clinical specimens.
191 quencies in isolated populations of a marine snail (Littorina saxatilis), re-established with perturb
194 erin, and decreased those of vimentin, Slug, Snail, matrix metalloproteinase (MMP)-2, -9, and activit
195 erin protein levels; increased expression of SNAIL, matrix metalloproteinase 2, and integrin beta1; a
196 By concentrating oocysts in faecal pellets, snails may facilitate entry of T. gondii into the nearsh
197 these results suggest that marsh periwinkle snails may have been adversely affected following exposu
202 In a second movement assay, there was no snail movement standing unoiled structure in chambers wi
207 uggest that a positive feedback loop between Snail-nuclear Cat L-CUX1 drives EMT, which can be antago
212 , together with an improved understanding of snail-parasite interactions, will aid to identify, plan,
215 uce human schistosomiasis have evolved from 'snail picking' campaigns, a century ago, to modern wide-
216 sease transmission, and thus the dynamics of snail populations are critically important for schistoso
217 k of it) of ecological growth rates of local snail populations by contrasting novel ecological and en
218 sing gene silencing it was demonstrated that Snail positively regulated the expression of EndMT marke
219 verished asexual populations of a freshwater snail, Potamopyrgus antipodarum, from distinct habitats
220 ession of Snail in cancer cell lines lacking Snail profoundly represses 4E-BP1 expression, promotes c
226 E treatment resulted in nuclear retention of snail regulator FBXL5 that was concurrent with suppressi
227 enhance ADM development, the contribution of Snail-related protein Slug (Snai2) to ADM development is
231 f HIV on expression of molecules involved in SNAIL repressor complex formation and demonstrated that
232 employ live-imaging methods to visualize the Snail repressor, which establishes the boundary between
233 derm due to transcriptional quenching by the Snail repressor, which precludes recruitment of CBP and
235 ayer exploit the paralogous EMT-TFs Slug and Snail, respectively, which induce distinct EMT programs.
236 rm hunting to fish hunting among marine cone snails, resulting in the adaptive radiation of fish-hunt
237 fish, making them easier to engulf with the snail's distended false mouth, which functions as a net.
238 ations of W compartmentalization were in the snail's hepatopancreas based on wet chemistry and synchr
243 ell motility and expression of beta-catenin, Snail, Slug, Zeb1 and N-cadherin, and upregulated E-cadh
244 overexpression of key transcription factors (Snail, Slug, Zeb1) or by acquiring drug resistance produ
246 in is downregulated during tumorigenesis via Snail/Slug-mediated E-cadherin transcriptional reduction
247 ependence 1 (GFI1) is comprised of conserved Snail/Slug/Gfi1 (SNAG) and zinc finger motifs separated
248 . elegans include migration vectors (such as snails, slugs and isopods) and pathogens (such as micros
250 epithelial-mesenchymal transition regulator Snail (Snai1) can cooperate with Kras in acinar cells to
251 find that the gene for EMT master regulator Snail (SNAI1), but not Slug (SNAI2), shows evidence of P
252 293T cells, when stably transfected with SNAIL (SNAIL/293T), displayed suppressed transcription a
255 n extrapolation from a random sample of land snail species via two independent approaches, that we ma
257 channels in the venom of a worm-hunting cone snail suggests that a closely related ancestral toxin en
261 la mustelina is a critically endangered tree snail that subsists entirely by grazing microbes from le
264 explanation for the capacity of this asexual snail to spread by adaptive evolution or plasticity to d
266 oxicity assay was then conducted by exposing snails to oil coated Spartina stems in chambers for peri
267 rasite compatibility experiments by exposing snails to schistosome larvae recovered from the urine of
268 nia sea otters, the ability of kelp-dwelling snails to transmit terrestrially derived pathogens has n
271 n and decreased expression of N-cadherin and snail transcription factor -2 ( SNAI2) (also called SLUG
272 BR2 and the EMT inducers ZEB1, ZEB2, and the snail transcriptional repressor SNAI2, each crucial fact
273 INE) compounds results in reversal of EMT in snail-transduced primary human mammary epithelial cells
275 ted by various signaling pathways, including Snail, Twist and transforming growth factor-beta1 as wel
276 CoCl2-induced EMT most likely via HIF1-alpha-Snail/Twist signaling pathway, and copper depletion may
278 Vc1.1, a peptide from predatory marine cone snail venom, inhibits Cav2.2 channels by activating pert
279 no acid peptides present in predatory marine snail venoms that function as allosteric antagonists of
281 bpopulation expresses higher levels of SLUG, SNAIL, VIMENTIN and N-CADHERIN while show a lack of expr
282 expressed lower E-cadherin activity, higher Snail, vimentin, and Cat L activity, and a p110/p90 acti
285 d with flow-altering cuffs demonstrated that Snail was expressed preferentially at low shear stress s
287 i exposure and prey specialization on marine snails was identified in threatened California sea otter
293 kingly, some early-recovery mRNAs, including Snail, were elevated first during apoptosis, implying th
294 y sub-additive behavior is also observed for snail, whereby removal of the proximal enhancer causes a
295 in, and decreased expression of vimentin and snail, which is partially attributed to inhibiting Akt/G
296 s of the Cavu River harbour many B truncatus snails, which are capable of transmitting S haematobium-
297 itual is the "love" dart shooting of helicid snails, which has courted many theories regarding its pr
298 ist assessment of 632 species of marine cone snails with human impacts and projected ocean thermal st
299 of trematode parasite infections in aquatic snails with measurements of snail excretion and tissue s
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