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1 ratio = 1.43, 95% CI: 1.33, 1.5 for regular snoring).
2 sons say that they "don't know" whether they snore.
3 refreshing sleep; and 33% reported that they snored.
4 al congestion is a risk factor for apnea and snoring.
5 the basis of either sleep apnea or habitual snoring.
6 ren had obstructive apnea and 21 had primary snoring.
7 ess and standardized questionnaires assessed snoring.
8 nd 23% of the variability in self-reports of snoring.
9 l index or self-reported history of habitual snoring.
10 ng and 1.55 (95% CI: 1.42, 1.70) for regular snoring.
11 e back were directly associated with regular snoring.
14 ere 1.29 (95% CI: 1.22, 1.37) for occasional snoring and 1.55 (95% CI: 1.42, 1.70) for regular snorin
15 scriminating between 13 subjects with no EDS/snoring and 21 patients with EDS and snoring were identi
16 cant genetic correlation between obesity and snoring and between obesity and excessive daytime sleepi
18 covariates, the positive association between snoring and CVD was attenuated but remained statisticall
19 om these data that self-reported symptoms of snoring and daytime sleepiness in older men have a genet
21 etes showed a consistent association between snoring and diabetes within the categories of these vari
22 opulation were similar to those for frequent snoring and included: male sex, higher Body Mass Index,
25 nce interval (CI): 1.16, 1.27 for occasional snoring and odds ratio = 1.43, 95% CI: 1.33, 1.5 for reg
29 The authors examined the association between snoring and risk of hypertension in a cohort of 73,231 U
30 ty (U3P) has been advocated for treatment of snoring and sleep apnea/hypopnea syndrome (SAHS), but of
31 gh for the most part the genetic variance in snoring and sleepiness was nonoverlapping with the genet
32 subjects may disavow knowledge of their own snoring and suggest that future studies consider the "do
34 I motoneurons is common during sleep causing snoring and, in serious cases, airway obstruction that i
35 sed surrogate information to define SDB (eg, snoring) and were based on small clinic populations, or
36 g and maintaining sleep, unrefreshing sleep, snoring, and the presence of physician-diagnosed sleep d
37 time below 90% oxygen saturation; history of snoring; and presence of hypertension, defined as restin
39 tems addressed the presence and frequency of snoring behavior, waketime sleepiness or fatigue, and hi
40 useful clinical adjunct to the evaluation of snoring children, with more accurate identification of t
44 osed on the basis of characteristic history (snoring, daytime sleepiness) and physical examination (i
45 rity for menopausal women with complaints of snoring, daytime sleepiness, or unsatisfactory sleep.
46 sleep apnea or >/= 2 hallmarks of OSA: loud snoring, daytime sleepiness, witnessed apnea, and hypert
47 noring index (SI, expressed as the number of snoring events per hour measured on an acoustic analytic
49 core was also seen across five categories of snoring frequency, from 6.4 (4.2) in current nonsnorers
54 cale ranging from 0 to 10) and the objective snoring index (SI, expressed as the number of snoring ev
60 an independent effect of snoring or whether snoring is simply a marker for obstructive sleep apnea.
61 gical mechanism underlying this association, snoring may help clinicians identify individuals at high
64 ether this reflects an independent effect of snoring or whether snoring is simply a marker for obstru
66 e waves in the UA, similar to those found in snoring, produce reflex inhibition of inspiratory motor
69 slightly attenuated the risk (for occasional snoring, RR = 1.41 (95% CI: 1.22, 1.63); for regular sno
70 RR = 1.41 (95% CI: 1.22, 1.63); for regular snoring, RR = 2.03 (95% CI: 1.71, 2.40); p for trend < 0
71 ary outcome measures included the subjective snoring severity (SS, measured on a visual analogue scal
74 ng and support the large-scale evaluation of snoring sound characters as markers of surveillance and
76 p, CCA-IMT was significantly correlated with snoring sound energies of 0-20 Hz (r = 0.608, p = 0.036)
77 obstruction detected by either DISE or mean snoring sound intensity (301-850 Hz), and AHI could sign
78 ignificantly, inversely correlated with mean snoring sound intensity (301-850 Hz; OR, 0.84, 95% CI 0.
79 ficantly, positively associated with maximal snoring sound intensity (40-300 Hz; odds ratio [OR], 1.2
80 study recruited 36 OSAHS patients for 6-hour snoring sound recordings during in-lab full-night polyso
84 a head-positioning pillow (HPP) could reduce snoring sounds in patients with mild-to-moderate positio
86 dy aimed to investigate associations between snoring sounds, obstruction sites, and surgical response
88 ith no significant change in the relation of snoring to ESS score after adjustment for RDI in multiva
92 ciated with risk of diabetes (for occasional snoring vs. nonsnoring, relative risk (RR) = 1.48 (95% c
93 ence interval (CI): 1.29, 1.70); for regular snoring vs. nonsnoring, RR = 2.25 (95% CI: 1.91, 2.66);
95 , waist circumference, and other covariates, snoring was associated with a significantly higher preva
96 alyses adjusted for age and body mass index, snoring was associated with risk of diabetes (for occasi
98 no EDS/snoring and 21 patients with EDS and snoring were identified by receiver operator curve analy
99 was accompanied by a virtual elimination of snoring, which fell from 16.5 +/- 3.0% of total sleep ti
102 vere sleep apnea, and self-reported habitual snoring without PDSA (HS), a surrogate for mild sleep ap
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