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1  ratio = 1.43, 95% CI: 1.33, 1.5 for regular snoring).
2 sons say that they "don't know" whether they snore.
3 refreshing sleep; and 33% reported that they snored.
4 al congestion is a risk factor for apnea and snoring.
5  the basis of either sleep apnea or habitual snoring.
6 ren had obstructive apnea and 21 had primary snoring.
7 ess and standardized questionnaires assessed snoring.
8 nd 23% of the variability in self-reports of snoring.
9 l index or self-reported history of habitual snoring.
10 ng and 1.55 (95% CI: 1.42, 1.70) for regular snoring.
11 e back were directly associated with regular snoring.
12 sus 75 +/- 16 kg) and prevalence of habitual snoring (78% versus 28%).
13  obstructive sleep apnea, and also that even snoring alone affects neurocognitive function.
14 ere 1.29 (95% CI: 1.22, 1.37) for occasional snoring and 1.55 (95% CI: 1.42, 1.70) for regular snorin
15 scriminating between 13 subjects with no EDS/snoring and 21 patients with EDS and snoring were identi
16 cant genetic correlation between obesity and snoring and between obesity and excessive daytime sleepi
17                           Patients with mild snoring and body mass index lower than 26 are unlikely t
18 covariates, the positive association between snoring and CVD was attenuated but remained statisticall
19 om these data that self-reported symptoms of snoring and daytime sleepiness in older men have a genet
20 he variation and covariation of obesity with snoring and daytime sleepiness.
21 etes showed a consistent association between snoring and diabetes within the categories of these vari
22 opulation were similar to those for frequent snoring and included: male sex, higher Body Mass Index,
23                             A total of 1,010 snoring and nonsnoring children ages 5-7 years were pros
24    Position therapy plays a role in treating snoring and obstructive sleep apnea syndrome (OSAS).
25 nce interval (CI): 1.16, 1.27 for occasional snoring and odds ratio = 1.43, 95% CI: 1.33, 1.5 for reg
26                        We conclude that both snoring and RDI are independently associated with excess
27                               The effects of snoring and RDI on sleepiness were little affected by ad
28           To examine the association between snoring and risk of developing type II diabetes mellitus
29 The authors examined the association between snoring and risk of hypertension in a cohort of 73,231 U
30 ty (U3P) has been advocated for treatment of snoring and sleep apnea/hypopnea syndrome (SAHS), but of
31 gh for the most part the genetic variance in snoring and sleepiness was nonoverlapping with the genet
32  subjects may disavow knowledge of their own snoring and suggest that future studies consider the "do
33 tion was very strong in persons who reported snoring and weak in those who did not.
34 I motoneurons is common during sleep causing snoring and, in serious cases, airway obstruction that i
35 sed surrogate information to define SDB (eg, snoring) and were based on small clinic populations, or
36 g and maintaining sleep, unrefreshing sleep, snoring, and the presence of physician-diagnosed sleep d
37 time below 90% oxygen saturation; history of snoring; and presence of hypertension, defined as restin
38 the "don't know" response to questions about snoring as a response of potential interest.
39 tems addressed the presence and frequency of snoring behavior, waketime sleepiness or fatigue, and hi
40 useful clinical adjunct to the evaluation of snoring children, with more accurate identification of t
41 inical evaluation and referral of habitually snoring children.
42 try may contribute to diagnostic accuracy in snoring children.
43 en and in a subsequent post hoc sample of 94 snoring children.
44 osed on the basis of characteristic history (snoring, daytime sleepiness) and physical examination (i
45 rity for menopausal women with complaints of snoring, daytime sleepiness, or unsatisfactory sleep.
46  sleep apnea or >/= 2 hallmarks of OSA: loud snoring, daytime sleepiness, witnessed apnea, and hypert
47 noring index (SI, expressed as the number of snoring events per hour measured on an acoustic analytic
48 roarousal index (P = 0.008); and symptoms of snoring, fatigue, and sleepiness (P < 0.001).
49 core was also seen across five categories of snoring frequency, from 6.4 (4.2) in current nonsnorers
50                                              Snoring history was obtained via a self-completion quest
51  in the generation of apneas, hypopneas, and snoring in healthy subjects.
52                                      Whether snoring increases risk of CVD remains unclear; most prev
53                                      Whether snoring increases the risk of hypertension remains uncle
54 cale ranging from 0 to 10) and the objective snoring index (SI, expressed as the number of snoring ev
55                                              Snoring is also associated with excess sleepiness, altho
56                    These data suggested that snoring is associated with a modest but significantly in
57                                              Snoring is common in sleep apnea patients but is not use
58                   These results suggest that snoring is independently associated with elevated risk o
59 ndicator of obstructive sleep apnea, whereas snoring is not very specific.
60  an independent effect of snoring or whether snoring is simply a marker for obstructive sleep apnea.
61 gical mechanism underlying this association, snoring may help clinicians identify individuals at high
62                      These data suggest that snoring may increase risk of hypertension in women, inde
63 ning 103 subjects (14 nonsnoring non-EDS, 21 snoring non-EDS, 68 snoring with EDS).
64 ether this reflects an independent effect of snoring or whether snoring is simply a marker for obstru
65                                              Snoring patterns were ascertained by questionnaire.
66 e waves in the UA, similar to those found in snoring, produce reflex inhibition of inspiratory motor
67 h OSAS, compared to 26 children with primary snoring (PS).
68 ildren with OSA and 19 children with primary snoring (PS).
69 slightly attenuated the risk (for occasional snoring, RR = 1.41 (95% CI: 1.22, 1.63); for regular sno
70  RR = 1.41 (95% CI: 1.22, 1.63); for regular snoring, RR = 2.03 (95% CI: 1.71, 2.40); p for trend < 0
71 ary outcome measures included the subjective snoring severity (SS, measured on a visual analogue scal
72 rent nonsnorers to 9.3 (4.8) in subjects who snored six to seven nights per week (p < 0.001).
73 rom 15% in never-snorers to 39% in those who snored six to seven nights per week.
74 ng and support the large-scale evaluation of snoring sound characters as markers of surveillance and
75                    Our findings suggest that snoring sound detection may be helpful in determining ob
76 p, CCA-IMT was significantly correlated with snoring sound energies of 0-20 Hz (r = 0.608, p = 0.036)
77  obstruction detected by either DISE or mean snoring sound intensity (301-850 Hz), and AHI could sign
78 ignificantly, inversely correlated with mean snoring sound intensity (301-850 Hz; OR, 0.84, 95% CI 0.
79 ficantly, positively associated with maximal snoring sound intensity (40-300 Hz; odds ratio [OR], 1.2
80 study recruited 36 OSAHS patients for 6-hour snoring sound recordings during in-lab full-night polyso
81                                              Snoring sounds generated by different vibrators of the u
82           Good-quality signals of full-night snoring sounds in an ordinary sleep condition obtained f
83 artery intima-media thickness (CCA-IMT) with snoring sounds in OSA patients.
84 a head-positioning pillow (HPP) could reduce snoring sounds in patients with mild-to-moderate positio
85         Our findings suggest that underlying snoring sounds may cause carotid wall thickening and sup
86 dy aimed to investigate associations between snoring sounds, obstruction sites, and surgical response
87 hysical activity, other sleep disorders, and snoring status.
88 ith no significant change in the relation of snoring to ESS score after adjustment for RDI in multiva
89                              The relation of snoring to sleepiness was seen at all levels of RDI, wit
90           To further explore the relation of snoring to sleepiness, we conducted a cross-sectional co
91                    Ten symptomatic patients (snoring, upper airway resistance syndrome [UARS], or OSA
92 ciated with risk of diabetes (for occasional snoring vs. nonsnoring, relative risk (RR) = 1.48 (95% c
93 ence interval (CI): 1.29, 1.70); for regular snoring vs. nonsnoring, RR = 2.25 (95% CI: 1.91, 2.66);
94                                 Frequency of snoring was assessed using mailed questionnaires at base
95 , waist circumference, and other covariates, snoring was associated with a significantly higher preva
96 alyses adjusted for age and body mass index, snoring was associated with risk of diabetes (for occasi
97                                 In addition, snoring was associated with significantly higher systoli
98  no EDS/snoring and 21 patients with EDS and snoring were identified by receiver operator curve analy
99  was accompanied by a virtual elimination of snoring, which fell from 16.5 +/- 3.0% of total sleep ti
100 d 44% of the cohort answered questions about snoring with a "don't know" response.
101 4 nonsnoring non-EDS, 21 snoring non-EDS, 68 snoring with EDS).
102 vere sleep apnea, and self-reported habitual snoring without PDSA (HS), a surrogate for mild sleep ap
103                             Features include snoring, witnessed apnoeas, and sleepiness.

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