ライフサイエンスコーパス: sobrinus

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1 d dental caries (Streptococcus mutans and S. sobrinus).

2 ) for Streptococcus mutans and Streptococcus sobrinus.

3 d by infection with cariogenic Streptococcus sobrinus.

4 e construct to include both S. mutans and S. sobrinus.

5 replace the P1 protein, the spaA gene of S. sobrinus 6715 was isolated from plasmid pX1303 and inser

6 (GBP-2, GBP-3, and GBP-5) from Streptococcus sobrinus 6715 were compared structurally by mass spectro

7 tinomyces viscosus and Streptococcus mutans (sobrinus) 6715, and fed a cariogenic diet.

8 hildren under 18 mos were: S. mutans 70%, S. sobrinus 72%, P. gingivalis 23%, B. forsythus 11%, and A

9 vaginalis, and S. mutans with Streptococcus sobrinus (all p < 0.05) were positively associated with

10 Multivariate modeling employing HOT, S. sobrinus and S. mutans (PCR/qPCR), and sugar snacks sepa

11 S. mutans (sobrinus) and A. viscosus established readily in all ani

12 e strain each of S. gordonii, S. sanguis, S. sobrinus, and S. vestibularis.

13 nel of streptococci, including S. mutans, S. sobrinus, and Streptococcus australis, and Alloprevotell

14 tination of Streptococcus mutans 3209 and S. sobrinus B13.

15 hat SpaA plays a role in the virulence of S. sobrinus by specifically interacting with fluid-phase sa

16 educed dental caries after infection with S. sobrinus compared with sham-immunized animals.

17 In conclusion, S. mutans and S. sobrinus correlated with Romanian adolescents with carie

18 Thus, S. sobrinus GBP-2 and GBP-3 appear to be distinct proteins

19 S. sobrinus GBP-5 may be a proteolytic fragment of GBP-3, o

20 None of these S. sobrinus GBPs appeared to have a strong antigenic relati

21 Streptococcus sobrinus glucosyltransferase (GTF), an enzyme involved i

22 ere immunized as above or with Streptococcus sobrinus GTF and then infected with S. sobrinus to explo

23 also inhibited the ability of Streptococcus sobrinus GTF to synthesize insoluble glucan.

24 at 7- to 14-day intervals with peptides, S. sobrinus GTF, or phosphate-buffered saline.

25 ibody from the communized group inhibited S. sobrinus GTF-mediated insoluble glucan synthesis in vitr

26 soluble glucan synthesis by S. mutans and S. sobrinus GTFs (P < 0.0001 and P < 0.05, respectively).

27 Two mutants of Streptococcus sobrinus incapable of aggregation by high-molecular-weig

28 eptococcus mutans (p < 0.005), Streptococcus sobrinus (p < 0.005), and Bifidobacteria (p < 0.0001) we

29 The results show that even though S. sobrinus produces several proteins capable of binding al

30 s, failed to reveal cross-reactivity with S. sobrinus proteins.

31 ectrophoretic mobility similar to that of S. sobrinus SpaA protein and a minor 210-kDa protein and a

32 The Streptococcus sobrinus SpaA protein and the Streptococcus mutans P1 pr

33 was inhibited specifically by antisera to S. sobrinus SpaA protein.

34 iment, rats were infected with Streptococcus sobrinus strain 6715 under an identical protocol.

35 ted were Streptococcus mutans, Streptococcus sobrinus, Streptococcus sanguinus, Lactobacillus acidoph

36 occus sobrinus GTF and then infected with S. sobrinus to explore the effects of immunization on immun

37 S. pyogenes, S. gordonii, S. mutans, and S. sobrinus, using PCR amplification, and sequenced.

38 associated with S-ECC, and S. mutans with S. sobrinus was associated with lesion recurrence (p < 0.05

39 s from culture supernatants of Streptococcus sobrinus were able to bind avidly to Sephadex G-75.

40 ess to dental care, whereas S. mutans and S. sobrinus were detected infrequently in Swedish adolescen

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