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1 voxamine for the treatment of social phobia (social anxiety disorder).
2 nical and clinical neurobiological models of social anxiety disorder.
3 .9% of subjects with noncomorbid generalized social anxiety disorder.
4 to examine a gaze-contingent intervention in social anxiety disorder.
5 account for the known neurobiology of human social anxiety disorder.
6 ularly those with a social component such as social anxiety disorder.
7 ng children are at marked risk of developing social anxiety disorder.
8 performance and social anxiety in women with social anxiety disorder.
9 ed the long-term outcome of psychotherapy in social anxiety disorder.
10 role in the pathophysiology and treatment of social anxiety disorder.
11 st intervention for the initial treatment of social anxiety disorder.
12 ive benefits for sertraline nonresponders in social anxiety disorder.
13 the neurodevelopmental mechanisms underlying social anxiety disorder.
14 ion with approved treatments for generalized social anxiety disorder.
15 for the acute management of the symptoms of social anxiety disorder.
16 ral therapy (CBT) in adults with generalized social anxiety disorder.
17 py (CBT) have been shown to be effective for social anxiety disorder.
18 gment a full course of comprehensive CBT for social anxiety disorder.
19 ht serve as biomarkers for interventions for social anxiety disorder.
20 s in developing preventive interventions for social anxiety disorder.
21 of behavioral inhibition are a precursor of social anxiety disorder.
22 on was associated with a lifetime history of social anxiety disorder.
23 disorder, and 6.2% (CI, 4.7% to 7.9%) had a social anxiety disorder.
24 rally well-tolerated treatment for pediatric social anxiety disorder.
25 ion that overlap with features of autism and social anxiety disorder.
26 rs appear to be responsible for the onset of social anxiety disorder.
30 ssociation between behavioral inhibition and social anxiety disorder and an inverse relationship betw
32 re, of interest, more frequently observed in social anxiety disorder and specific phobia than in PTSD
34 sults support a conditioning contribution to social anxiety disorder and underscore the importance of
35 th introversion (a core personality trait in social anxiety disorder) and with increased limbic activ
36 vity in amygdala and insula in patients with social anxiety disorder, and genetic studies are increas
37 n in healthy participants, participants with social anxiety disorder, and participants with acute str
38 pressive disorder (MDD), panic disorder, and social anxiety disorder are among the most prevalent and
40 e) and adolescents (12-17 years of age) with social anxiety disorder as their predominant psychiatric
41 disorder, posttraumatic stress disorder, and social anxiety disorder, as well as autism and schizophr
42 n, posttraumatic stress disorder, addiction, social anxiety disorder, bipolar disorder, schizophrenia
43 linical severity associated with generalized social anxiety disorder, both alone and with comorbid ps
44 ic therapy were both efficacious in treating social anxiety disorder, but there were significant diff
47 vel gaze-contingent music reward therapy for social anxiety disorder designed to reduce attention dwe
50 Adult outpatients with DSM-IV generalized social anxiety disorder for 6 months or longer were rand
53 year, only 0.5% of subjects with generalized social anxiety disorder had been accurately diagnosed.
54 Persons with average-severity generalized social anxiety disorder had probabilities of graduating
55 ral inhibition, a temperamental precursor of social anxiety disorder (haplotype P = 3 x 10(-5); odds
56 ta-analysis of interventions for adults with social anxiety disorder, identified from published and u
58 efficacy of psychodynamic therapy and CBT in social anxiety disorder in a multicenter randomized cont
61 dynamic therapy were efficacious in treating social anxiety disorder, in both the short- and long-ter
62 ncerning clinical neurobiological aspects of social anxiety disorder, including the developmental neu
69 cial phobia (gSP), also known as generalized social anxiety disorder, is characterized by excessive f
70 chizophrenia, and borderline personality and social anxiety disorders, may be reduced by oxytocin adm
72 medicated participants (N=22 generalized and social anxiety disorder/N=23 controls) recruited from Wa
73 169 medication-free adults with generalized social anxiety disorder, of whom 144 completed the 12-we
74 ugmentation strategy for exposure therapy in social anxiety disorder, one that may be especially effe
75 humans diagnosed as having specific phobia, social anxiety disorder, panic disorder with or without
76 isorder, selective mutism, specific phobias, social anxiety disorder, panic disorder, agoraphobia, an
77 edged as an important etiologic mechanism in social anxiety disorder, past psychophysiological experi
78 the treatment of depression, panic disorder, social anxiety disorder, posttraumatic stress disorder,
79 ne women 19-60 years of age with generalized social anxiety disorder received placebo intranasal spra
87 nnectivity were measured in 38 patients with social anxiety disorder (SAD) to predict subsequent trea
88 fect, but whether anxiety syndromes, such as social anxiety disorder (SAD), are characterized by an o
89 abnormalities underlie pathological anxiety, social anxiety disorder (SAD)-although among the most co
94 rrelations) that discriminated patients with social anxiety disorder (SAD, N=16) from controls (N=19)
95 d better outcomes on self-report measures of social anxiety disorder severity (Liebowitz Social Anxie
98 ies of posttraumatic stress disorder (PTSD), social anxiety disorder, specific phobia, and fear condi
99 ety disorders: generalized anxiety disorder, social anxiety disorder, specific phobia, and panic diso
100 with confirmed diagnoses of panic disorder, social anxiety disorder, specific phobias, generalized a
101 disorder, generalized anxiety disorder, and social anxiety disorder, suggest that CRF1 receptor anta
102 py yielded greater reductions of symptoms of social anxiety disorder than the control condition on bo
105 community cohort of HMO members, generalized social anxiety disorder was rarely diagnosed or treated
106 Adults (n = 40) with a DSM-IV diagnosis of social anxiety disorder were randomized to placebo or yo
107 disorder, obsessive-compulsive disorder, or social anxiety disorder were randomly assigned by princi
109 In an outpatient setting, 495 patients with social anxiety disorder were randomly assigned to manual
110 ve outcomes for individuals with generalized social anxiety disorder who remain symptomatic after ini
112 utilization of health services; generalized social anxiety disorder with comorbid psychopathology wa
113 e in the short-term treatment of generalized social anxiety disorder, with efficacy and tolerability
114 ort to assess the conditioning correlates of social anxiety disorder within an ecologically enhanced
115 and anxiety (n = 59), panic disorder and/or social anxiety disorder without comorbid MDD (n = 56), a
116 ment are the best-established treatments for social anxiety disorder, yet many individuals remain sym
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