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1 olving disruptions of social behavior (e.g., social withdrawal).
2 avioral changes and counteracted PCP-induced social withdrawal.
3 imal model for some aspects of schizophrenic social withdrawal.
4 tment resulted in significant improvement in social withdrawal.
5 rlocomotion, stereotype behavior, ataxia and social withdrawal.
6 motor retardation, and passive or apathetic social withdrawal.
7 responses, including anorexia, lethargy, and social withdrawal.
8 expression of anxiety-related behaviors and social withdrawal.
9 eractivity (15 [19%] vs 86 [14%], p<0.0001), social withdrawal (142 [18%] vs 75 [12%], p=0.002), and
10 iatric disorders associated with symptoms of social withdrawal and anhedonia, both of which are stron
11 an improve the abnormal behaviors (including social withdrawal and immobility) that are commonly asso
14 and understanding speech, which can lead to social withdrawal and isolation, have been linked to cha
15 T, 70%); elevated anxiety and/or depression, social withdrawal, and attention problems (internalizing
17 (-/-) mice exhibit impulsive aggressiveness, social withdrawal, and decreased sexual motivation, toge
18 ecklist subscales for irritability, lethargy/social withdrawal, and hyperactivity; the Behavior Probl
19 d consequences of child and early-adolescent social withdrawal; and (c) present a developmental frame
20 headstrong, attention deficit, peer conflict/social withdrawal, antisocial behaviors, and social comp
21 hought disorder), negative symptoms (such as social withdrawal, apathy and emotional blunting) and ot
22 h negativity amplitude and passive-apathetic social withdrawal at left and right frontal electrodes.
23 nstructs previously employed in the study of social withdrawal; (b) examine the predictors, correlate
24 t prenatal stress preparation to investigate social withdrawal behaviors that may have relevance to t
25 LY225910 blocked both PCP- and AM251-induced social withdrawal, but not URB597 effect in control rats
26 indicated fewer attention problems and less social withdrawal compared with the placebo group (N=26)
27 cal well-being; loss of psychosocial assets; social withdrawal; family disruption; and lower levels o
28 ination (rotarod), and depressive behaviors (social withdrawal, immobility in the tail suspension tes
29 ent CB agonist CP55,940 reversed PCP-induced social withdrawal in a CB(1)-dependent manner, whereas p
31 tranasal OT failed to reverse stress-induced social withdrawal in female mice and reduced social inte
32 tor of endocannabinoid degradation, reversed social withdrawal in PCP-treated rats via stimulation of
33 role played by the endocannabinoid system in social withdrawal induced by sub-chronic administration
35 while "negative" psychotic symptoms, such as social withdrawal, lowered the risk of serious violence.
38 ipulation of each population mediates either social withdrawal or behavioral despair, but not both.
40 K), we also examined whether the PCP-induced social withdrawal resulted from deficient CB(1)-mediated
41 for social interaction, and that PCP-induced social withdrawal results from deficient endocannabinoid
42 ificant improvement was found on the CGI and social withdrawal subscale of the Aberrant Behavior Chec
43 cipitates psychopathologies characterized by social withdrawal such as post-traumatic stress disorder
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