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1 ounterpart Na-exposed rats (pair-fed 216 ppm sodium acetate).
2 cetonitrile, followed by a cleanup step with sodium acetate.
3 thanebis(diazene-N-oxide-N'-hydroxylate) and sodium acetate.
4 degrees C to produce methane by pyrolysis of sodium acetate.
5 thanebis(diazene-N-oxide-N'-hydroxylate) and sodium acetate.
6 es from 9:1 DMF/water mixtures buffered with sodium acetate.
7 ses for both Whatman 1PS paper and 30% TlNO3/sodium acetate.
8 s concentrations of potassium, rubidium, and sodium acetate.
9 ot to a normal meal or one supplemented with sodium acetate.
10 in calli supplemented with [U-(13)C]glucose, sodium acetate (1,2-(13)C2) and sodium octanoate (1,2,3,
12 ere administered lead acetate, 100 mg/kg, or sodium acetate, 36 mg/kg (control), by i.p. for 7 days.
13 urbit[6]uril analogue (1) in aqueous buffer (sodium acetate, 50 mM, pH 4.74, 25 degrees C) toward a v
16 the literature conditions (acetic anhydride/sodium acetate) affords both the indole 6 and the indoli
17 phaE328A was sensitive to chemical rescue by sodium acetate, an effect that was not reproduced by pho
18 product at 100 degrees C in the presence of sodium acetate and 1% sodium dodecyl sulfate generates f
21 tified via intravenous infusion of [1-(13)C] sodium acetate and mass isotopomer distribution analysis
22 creased levels of select components, notably sodium acetate and sodium bicarbonate, restored the leve
24 r of salts (sodium sulfate, sodium fluoride, sodium acetate, and sodium chloride) on the thermodynami
27 in was inhibited by acetylsalicylic acid and sodium acetate as well as by N-acetylated glucosamine an
28 situ from partial transformation of aqueous sodium acetate at 300 degrees C and 2.4-3.5 GPa and that
29 , potassium phosphate, potassium acetate and sodium acetate) at varying concentrations (0.8 to 1.6 M)
35 A solution of AlCl(3).6H(2)O in a pH 4.0 sodium-acetate buffer was mixed with an aqueous solution
36 acidified 5 M NaCl solution directly into a sodium acetate-buffered solution containing a DOTA (1,4,
37 experiments, different acids or acetic acid/sodium acetate buffers at different pHs were used to evo
39 on the observations of different patterns of sodium acetate clusters that are characteristic for each
40 ensity, are quantified as functions of salt (sodium acetate) concentration for the interactions of th
46 matography using sodium hydroxide (NaOH) and sodium acetate gradients, coupled with integrated pulsed
47 e phase modifiers (i.e., ammonium formate or sodium acetate); however, these ESI adduct signals are l
49 deprotected and cyclized in the presence of sodium acetate in refluxing ethanol to afford (+/-)-abys
52 is is associated with changes of glucose and sodium acetate metabolism, demonstrating HRMAS NMR as a
54 ve to 500 mM concentrations of NaCl, Na2SO4, sodium acetate (NaAc), KCl, K2SO4, potassium acetate (KA
55 In contrast, the replacement of NaCl with sodium acetate, or pretreatment of cells with the Cl- ch
57 ution conditions used (3.8 mM protein, 50 mM sodium acetate, pH 4.5, 20 degreesC), and that it tumble
59 solution containing guanidinium thiocyanate, sodium acetate, phenol and chloroform, followed by centr
60 m-proton antiporter genes also contribute to sodium acetate, potassium acetate, and ammonium acetate
61 s, replacement of 300 mosmol/kgH2O NaCl with sodium acetate resulted in down-regulation of the gamma
63 its for sodium chloride, potassium chloride, sodium acetate, sodium fluoride, sodium dodecyl sulfate
64 ure for up to 1 month in the ethylene glycol-sodium acetate solvent system without the requirement fo
66 Zymomonas mobilis mutant (AcR) demonstrating sodium acetate tolerance that has potential importance i
67 itter was incubated with 100 mM of [2-(13)C] sodium acetate under thermophilic anaerobic conditions.
68 ocks, including chromium, iron, nitrate, and sodium acetate, were selected to represent the shocks of
69 romatic C-H activation was first promoted by sodium acetate with [Cp*MCl2]2 (M = Rh, Ir) at room temp
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