ライフサイエンスコーパス: sodium chlorate

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1 cient cells were generated by treatment with sodium chlorate.

2 Treatment of HeLa cells with sodium chlorate, a potent inhibitor of proteoglycan sulf

3 Pretreatment of HEp-2 cells with 50 mM sodium chlorate, a selective inhibitor of sulfation, for

4 Surprisingly, we found that sodium chlorate, a sulfation inhibitor, did not inhibit

5 face HSPGs with, respectively, heparinase or sodium chlorate abrogated HSC adhesion to CCN2(4).

6 ls is inhibited if cells are pretreated with sodium chlorate, an inhibitor of sulfate incorporation,

7 E fragment include heparin, heparan sulfate, sodium chlorate and heparinase, the low-density lipoprot

8 Chlorine gas and sodium chlorate are two base chemicals produced through

9 t study, inhibition of tyrosine sulfation by sodium chlorate decreased the secretion of processed CCK

10 lumbelliferyl-beta-D-xyloside) or sulfation (sodium chlorate) enhanced the release of apoE from cells

11 eased glycosaminoglycan chain sulfation with sodium chlorate enhances BMP2 morpho-genetic bioactivity

12 xylopyranoside or by the sulfation inhibitor sodium chlorate, indicating that SR-BI-mediated LDL-CE u

13 m with heparinase III, or growth of cells in sodium chlorate, indicating that sulfated heparan sulfat

14 in embryonic stem cells with heparinases or sodium chlorate inhibited differentiation of embryonic s

15 tion of the endothelial cell's glycocalyx or sodium chlorate inhibition of endothelial cell sulfated

16 The crystallization of sodium chlorate (NaClO3) is a classic example of spontan

17 antibody, suramin, or treatment with either sodium chlorate or heparitinase, demonstrating an autocr

18 Inhibition of proteoglycan sulfation by sodium chlorate or incubation of cells with labeled lipo

19 rior organ cultures treated with 20 or 50 mM sodium chlorate, or 1 mM beta-xyloside.

20 er treatment of MEF cell lines with heparin, sodium chlorate, or heparinase II, demonstrating that he

21 rowth of expressing cells in the presence of sodium chlorate, significantly reduced the potency for M

22 were treated with various concentrations of sodium chlorate, so as to express a range of endogenous

23 alb/c3T3 fibroblasts were treated with 50 mM sodium chlorate to completely inhibit (99%) sulfation of

24 face glycosaminoglycans or by treatment with sodium chlorate to decrease cellular sulfation.

25 Treatment of whole kidneys with sodium chlorate to disrupt proteoglycan synthesis result

26 Growth of N18 and BHK cells in sodium chlorate to eliminate all sulfation decreased vir

27 t VSMC were generated by treating cells with sodium chlorate to inhibit the sulfation of HSPG.

28 cholerae neuraminidase, tunicamycin, or 5 mM sodium chlorate, which blocks sulfation of surface prote

29 de, which limits host cell surface GAGs, and sodium chlorate, which decreases surface sulfation.

30 Sodium chlorate, which inhibits cell proteoglycan synthe

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