ライフサイエンスコーパス: sodium depletion

1 seeking out salt or consuming salt following sodium depletion.

2 are typically attractive, particularly after sodium depletion.

3 r FoxP2 and become c-Fos activated following sodium depletion.

4 on the consumption of salt itself following sodium depletion.

5 downstream targets that are activated during sodium depletion.

6 ange mRNA abundance was increased by dietary sodium depletion.

7 by potassium depletion but only minimally by sodium depletion.

8 ve similar but lower efficacies and increase sodium depletion.

9 During sodium depletion, 7-NI (partially) and -NAME (completely

10 During sodium depletion, 7-NI decreased RIF cGMP, but the reduc

11 Except in instances of severe, prolonged sodium depletion, a sodium-specific appetite has not bee

12 ation after furosemide diuresis and 22 hr of sodium depletion affects the amount of water or 0.3 M Na

13 During sodium depletion and angiotensin II coinfusion, (Pyr(1))

14 re indicate that a strong natural motivator, sodium depletion and associated salt appetite, also lead

15 Sodium depletion and captopril led to activation and dif

16 nal abnormalities are also common, including sodium depletion and hypovolemia, hypophosphatemia and h

17 sults indicate that repeated experience with sodium depletion and ingestion affects both behavioral a

18 ant (albeit reduced) salt appetite following sodium depletion, and a normal conditioned taste aversio

19 anscription level in SONs and PVNs following sodium depletion, and as a consequence of diurnal rhythm

20 neurons, which are selectively activated by sodium depletion, and by the A2 noradrenergic neurons, w

21 Following sodium depletion, and in the absence of either sucrose o

22 ent as WT mice, even during profound dietary sodium depletion, as a result of the upregulation of tra

23 During sodium depletion, both PD and -NAME caused a similar dec

24 DNA and protein content were not altered by sodium depletion but were significantly lowered by losar

25 ading, volume contraction, and potassium and sodium depletion completely corrects alkalosis by a rena

26 that provoke a robust sodium appetite (e.g., sodium depletion, deoxycorticosterone acetate) decrease

27 Experience with sodium depletion enhances its ingestion and may have a d

28 cleus accumbens of rats that had experienced sodium depletions had significantly more dendritic branc

29 ced by various experimental procedures (e.g. sodium depletion; hypovolaemia).

30 Animals receiving repeated sodium depletions increased sodium ingestion across init

31 tion of the renin- angiotensin system during sodium depletion increases renal nitric oxide production

32 food intake, decreases salt ingestion after sodium depletion, induces pica, and produces robust cond

33 Because dietary sodium depletion markedly increases ouabain-insensitive

34 he upregulation of renal AT1 mRNA induced by sodium depletion occurs conjointly with an elevation of

35 ance index (P<0.001 for all) irrespective of sodium depletion or angiotensin II (0.5 ng/kg per minute

36 of renin-angiotensin system activation with sodium depletion or angiotensin II coinfusion.

37 s in sodium appetite induced by either acute sodium depletion or mineralocorticoid administration but

38 T1 receptor binding in the rats subjected to sodium depletion plus losartan did not differ from that

39 Also, sodium depletion produced increased heart rate and reduc

40 Thus, mild sodium depletion produces hypohedonia and cardiovascular

41 Potassium depletion but not sodium depletion resulted in an increase in KCC1 protein

42 Sodium depletion significantly increased RIF cGMP.

43 Yet, upon first reencounter in a novel sodium-depletion state to promote mesocorticolimbic reac

44 Sodium depletion substantially increased ouabain-insensi

45 t intakes after their second experience with sodium depletion than their first, and microstructural a

46 tes transmit information related to systemic sodium depletion to forebrain sites and are part of the

47 Here, we used sodium depletion to reversibly manipulate the appetitive

48 We have recently reported that sodium depletion up-regulates H,K-ATPase alpha-subunit m

49 Dopamine responses to the NaCl US following sodium depletion updated independent of prior experience

50 In addition, a history of sodium depletions was found to have cross-sensitization

51 ctivation of the renin-angiotensin system by sodium depletion will enhance the growth of cysts in ani

52 Following 48 hr of sodium depletion with a diuretic (furosemide) and a sodi