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1 ET with [(18)F]fluorodeoxyglucose and [(18)F]sodium fluoride.
2 phatase 2A and sensitive to okadaic acid and sodium fluoride.
3 sphatase inhibitors sodium orthovanadate and sodium fluoride.
4 tested versus a positive control dentifrice (sodium fluoride/0.30% triclosan/copolymer) in 440 medica
5 ce of phosphatase inhibitors including 50 mM sodium fluoride, 1 mM okadaic acid, and 0.5 mM calyculin
7 plaques using the radioactive tracers (18)F-sodium fluoride ((18)F-NaF) and (18)F-fluorodeoxyglucose
8 T/CT) imaging of atherosclerosis using (18)F-sodium fluoride ((18)F-NaF) has the potential to identif
9 investigated coronary arterial uptake of 18F-sodium fluoride (18F-NaF) and 18F-fluorodeoxyglucose (18
10 ion in the aortic valve were assessed by 18F-sodium fluoride (18F-NaF) and 18F-fluorodeoxyglucose (18
12 nt groups included control, Spirulina alone, sodium fluoride (20 mg/kg) alone, and sodium fluoride al
13 toxin-insensitive and was not additive with sodium fluoride, a cell-permeant activator of heterotrim
14 alone, sodium fluoride (20 mg/kg) alone, and sodium fluoride along with Spirulina (250 and 500 mg/kg)
15 of (99m)Tc-labeled diphosphonates and (18)F-sodium fluoride and discusses and compares the performan
16 ontaining 8% strontium acetate and 1,040 ppm sodium fluoride; and 3) containing 30% microaggregation
20 ith the aid of repeated PET imaging (FMT and sodium fluoride for bone), realignment to subsequent com
21 yclase activity stimulated by isoproterenol, sodium fluoride, guanyl-5'-imidodiphosphate, and forskol
22 of the pyrophosphatase activity of NURF with sodium fluoride has no significant effect on chromatin r
24 the role of Spirulina platensis in reversing sodium fluoride-induced thyroid, neurodevelopment and ox
25 te 1 to a state 2 chemically locked by 0.1 M sodium fluoride leads to an almost complete functional r
26 e activity was inhibited by sodium vanadate, sodium fluoride, N-ethylmaleimide, and phenylglyoxal but
27 tively assessed the impact of PET with (18)F-sodium fluoride (NaF PET) on intended management of Medi
28 Evidence Development policy, PET using (18)F-sodium fluoride (NaF PET) to identify osseous metastasis
30 ation of degradation species: newly observed sodium fluoride (NaF) and the expected sodium carbonate
31 examine the potential genotoxic influence of sodium fluoride (NaF) on mammalian cells by means of a m
33 ussis toxin, dose response relationships for sodium fluoride (NaF; range, 0.1-4 mmol/L), a Gi protein
38 rs (5 mmol/l sodium orthovanadate, 50 mmol/l sodium fluoride, or 5 mmol/l EDTA, but not 100 nmol/l ok
39 y, we previously studied the impact of (18)F-sodium fluoride PET (NaF PET) on the intended management
40 ssess skeletal tumor burden with 18F-labeled sodium fluoride PET/CT (18F-fluoride PET/CT) and evaluat
41 c resonance imaging (n = 3), and fluorine-18 sodium fluoride positron emission tomography (PET) (n =
42 )Tc-labeled diphosphonates and (18)F-labeled sodium fluoride provides functional information sensitiv
43 luence of a number of salts (sodium sulfate, sodium fluoride, sodium acetate, and sodium chloride) on
44 hloride, potassium chloride, sodium acetate, sodium fluoride, sodium dodecyl sulfate (SDS), and manga
46 ing dentifrices containing strontium acetate/sodium fluoride (SrAc2F) and potassium chloride/sodium m
47 ion or glycolytic inhibitors iodoacetate and sodium fluoride synergistically cooperated with NDI, thu
48 in preventing CAL and root caries versus the sodium fluoride/triclosan/copolymer control in xerostomi
49 es with a secondary amine in the presence of sodium fluoride using hydroquinine as chiral catalyst wa
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