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1 two protons that are later exchanged for one sodium ion.
2 a small cation-friendly cavity occupied by a sodium ion.
3 rfere with the interaction of SNAT2 with the sodium ion.
4 is stabilized by two disulphide bonds and a sodium ion.
5 eraction between the peroxide groups and the sodium ion.
6 prevent it from collapsing onto the smaller sodium ion.
7 e binding pocket requires the removal of the sodium ion.
8 hydrogen bonding network that is mediated by sodium ion.
9 r ~800 water molecules and for magnesium and sodium ions.
10 3 in the presence of potassium ions but not sodium ions.
11 yte battery, which involves the insertion of sodium ions.
12 the synapse, assisted by the co-transport of sodium ions.
13 rom singly charged precursor ions with bound sodium ions.
14 esting the disruption of hydrogen-bonding by sodium ions.
15 phate group, together with flanking zinc and sodium ions.
16 ysiology, including the balance of water and sodium ions.
17 us phase was correlated with the mobility of sodium ions.
18 o the binding sites for one chloride and two sodium ions.
19 D-glucose or D-galactose in the presence of sodium ions.
20 action with mediating water molecules and/or sodium ions.
21 the sulfate ions are coordinated directly to sodium ions.
22 considerably slowing down the permeation of sodium ions.
23 ter EAAC1 is coupled to cotransport of three sodium ions.
24 tage and are less attracted to potassium and sodium ions.
25 s results in a reduced capacity to transport sodium ions.
26 brane and an increased capacity to transport sodium ions.
27 o solid-state electrochemical reactions with sodium ions.
28 nd had a more organised structure around the sodium ions.
29 , 0.34% vs. 0.31%; silicon, 0.36% vs. 0.37%; sodium ion, 0.21% vs. 0.18%; and sulfate, 0.35% vs. 0.38
30 in methanol is K(K+) = 229 +/- 25 M(-1)) and sodium ions (3, K(Na+) = 84.2 +/- 7.9 M(-1) in methanol)
31 ltaenhC mutants showed a hypersensitivity to sodium ion, a phenotype associated with dysfunction of t
36 ree distinct GA dimeric species, detected as sodium ion adduct ions [2GA + 2Na](2+), and these are as
37 opropanol lead to a significant reduction in sodium ion adduction but are not as effective as acetoni
38 he effectiveness of this method for reducing sodium ion adduction is related to the low proton affini
40 bF(6), can significantly lower the extent of sodium ion adduction to the molecular ions of proteins a
41 produced as potassium, proton, or sometimes sodium ion adducts, whereas proton loss was dominant in
42 ction remains wide enough for the passage of sodium ions, aided by a continuous bridge of approximate
43 , mesitylenic acid, and solvent molecules on sodium ion all are critical in identifying the most favo
44 through a dual mechanism of intercalation of sodium ions along the x axis of the phosphorene layers f
45 ith cardiovascular hospitalizations, whereas sodium ion, aluminum, and magnesium, components abundant
46 gs differ from the conventional thought that sodium ions always lead to more severe fractures in the
47 ase kinase alpha, Snf1 was activated by both sodium ion and alkaline stress, suggesting that stress s
48 d, the M3 receptor is bound by an allosteric sodium ion and confined mostly in the inactive state wit
50 increased some of the allosteric effects of sodium ions and amiloride, whereas orthosteric ligand bi
51 nist and antagonist affinity, allosterism by sodium ions and amilorides, and receptor functionality w
52 small but significant decrease in hemolymph sodium ions and an increase in calcium ions after 24 h p
53 ynamics occur in the absence and presence of sodium ions and aspartate, but stall in sodium alone, pr
54 amate from synapses are driven by symport of sodium ions and counter-transport of a potassium ion.
55 ansporter, defining sites for aspartate, two sodium ions and d,l-threo-beta-benzyloxyaspartate, an in
57 of one glutamate to the cotransport of three sodium ions and one proton and the countertransport of o
58 sists of cotransport of glutamate with three sodium ions and one proton, followed by countertransport
59 forms gated paracellular channels and allows sodium ions and other small positively charged ions to c
60 formation, the enzyme is able to capture two sodium ions and transport them to the external side of t
61 serve electroneutrality and osmotic balance, sodium ions and water also flow into the intestinal lume
63 otential role of structured water molecules, sodium ions, and lipids/cholesterol in GPCR stabilizatio
64 ed and reduced states, Na(+)-NQR binds three sodium ions, and that the affinity for sodium is the sam
65 A core domain of six helices harbours two sodium ions, and the remaining four helices pack in a ro
67 ibule about 11 A above the substrate and two sodium ions, apparently stabilizing the extracellular ga
70 he heptahydrate and decahydrate in which the sodium ions are coordinated exclusively by water molecul
73 as active electrode materials of lithium or sodium ion batteries, catalysts for water splitting, and
76 and development efforts on room-temperature sodium-ion batteries (NIBs) have been revitalized, as NI
77 u of 0.1 V in PIBs, slightly higher than for sodium-ion batteries (SIBs) (0.01 V), and well above the
81 to develop high-energy-density cathodes for sodium-ion batteries (SIBs), low-cost, high capacity Na(
83 formance when used in lithium-ion batteries, sodium-ion batteries and supercapacitors, respectively.
87 be a scalable, low-cost cathode material for sodium-ion batteries exhibiting high capacity, long cycl
88 The as-prepared sample used as an anode in sodium-ion batteries exhibits the best rate performance
89 ough recent reports on cathode materials for sodium-ion batteries have demonstrated performances comp
93 nd the manufacturing feasibility of low cost sodium-ion batteries with existing lithium-ion battery i
94 ural and chemical evolution of tin anodes in sodium-ion batteries with in situ synchrotron hard X-ray
96 articular emphasis on lithium-ion batteries, sodium-ion batteries, catalysis of hydrogen evolution, o
97 re proposed to fabricate superior anodes for sodium-ion batteries, featuring high-rate capabilities a
98 pplications including lithium-ion batteries, sodium-ion batteries, lithium-sulfur batteries, lithium-
111 form of CTF-HUST-4 as an anode material in a sodium-ion battery achieving an excellent discharge capa
114 n, for the first time, we report a family of sodium-ion battery electrodes obtained by replacing step
116 ility, and cycle life, providing a practical sodium-ion battery powering an electric vehicle in frigi
118 major scientific challenge for a competitive sodium-ion battery technology is to develop viable anode
119 tion enables the fabrication of a discharged sodium-ion battery with a non-sodium metal anode, and th
125 med to understand the mechanistic effects of sodium ion binding on dynamic activation of the M3 musca
128 ucted to have altered ion selectivities, the sodium ion binding site nearest the extracellular side i
129 e D2.50-protonated receptor does not exhibit sodium ion binding to the D2.50 allosteric site and samp
133 has four transmembrane alpha helices, with a sodium ion bound between helices 2 and 4 at a site burie
135 ve charge introduced at position 124 and the sodium ions bound at Na3' and Na1 underlies the protecti
136 utation results from the substitution of the sodium ions bound within the dimer interface by the intr
139 validated the concept of full-titanium-based sodium ion cells through the assembly of symmetric cells
140 ncreased expression of certain voltage-gated sodium ion channel (NaV) isoforms in peripheral sensory
143 HN-associated VZV isolates induce changes in sodium ion channel currents known to be associated with
144 lular geometry, gap junctional coupling, and sodium ion channel distribution on propagation velocity
148 luates the delivery of dsRNA targeted to the sodium ion channel paralytic A (TcNav) gene in Tribolium
150 iscern the role of the cardiac voltage-gated sodium ion channel SCN5A in the etiology of dilated card
151 analogs that inhibit NaV1.7, a voltage-gated sodium ion channel that is a compelling target for impro
154 insensitive to gap junctional coupling when sodium ion channels are located entirely on the cell end
155 ent with what is known of human aura in that sodium ion channels are those predominantly involved in
156 on of Nav 1.6 and Nav 1.7 genes all encoding sodium ion channels the dysregulation of which is associ
157 by mutations in skeletal muscle chloride and sodium ion channels with considerable phenotypic overlap
160 djustment for gaseous copollutants, nitrate, sodium ion, chloride ion, magnesium, and nickel remained
162 monapride or [(3)H]spiperone depended on the sodium ion concentration but was independent of the comp
163 arkedly attenuated, there was no decrease in sodium ion concentration in tissue from outer medulla or
164 fferences in sodium ion mobility and in free sodium ion concentration, leading to differences in in-m
165 a dehydrated iron hexacyanoferrate with high sodium-ion concentration enables the fabrication of a di
166 demonstrate that sustained low extracellular sodium ion concentrations ([Na(+)]) directly stimulate o
168 thesis and are found to irreversibly inhibit sodium ion conductance in recombinantly expressed wild-t
170 analysis, we find that the particles form a sodium-ion conductive film on the anode, which stabilize
171 establish the molecular basis for allosteric sodium ion control in opioid signalling, revealing that
172 Notably, norbuprenorphine interacted with sodium ion-coordinating residues W293(6.48) and N150(3.3
174 h two preferred binding sites identified for sodium ions, corresponding to strong binding with the ox
177 easure H(2)O and H(2)S fluxes, respectively, sodium ion dilution and buffer acidification by proton r
178 in nature, is approximately 52% faster than sodium ion (DNa+ = 1.33, DCl- = 2.03[10(-9)m(2)s(-1)]).
179 into the cell, driven by the co-transport of sodium ions down their transmembrane concentration gradi
180 trides, (3) an electrostatically stabilizing sodium ion during nitride installation, (4) selecting th
184 erization of the cocrystalline solid-organic sodium ion electrolyte NaClO4 (DMF)3 (DMF=dimethylformam
188 tion light-driven H(+)/Na(+) pumps, ejecting sodium ions from cells in the presence of sodium and pro
190 upled with an Sb-based anode, the fabricated sodium-ion full-cells also exhibit excellent rate and cy
195 rate, and Na1 and Na2 for two co-transported sodium ions) have been resolved, we still lack a mechani
197 aling the presence and fundamental role of a sodium ion in mediating allosteric control of receptor f
200 titative assessment of the metabolic role of sodium ions in cellular processes and their malfunctions
202 wo-dimensional conductivity, owing to mobile sodium ions in lattice planes, between which are insulat
203 was used to study the molecular mobility of sodium ions in model cheeses through measurements of the
204 M7 and Asp-405 on TM8 and support a role for sodium ions in stabilizing substrate-bound conformers.
205 ic, mucoadhesive thickener, the retention of sodium ions in the mouth is prolonged due to the mucoadh
206 for the first time the detailed locations of sodium ions in the selectivity filter of a sodium channe
208 ke potassium ions in potassium channels, the sodium ions in these channels appear to be hydrated and
210 while earlier NMR results in the presence of sodium ions indicated a unimolecular, antiparallel quadr
212 in some prokaryotes, complex I may transport sodium ions instead, and three subunits in the membrane
214 ride ions: Potassium ions, being larger than sodium ions, interact only weakly with phospholipid head
216 rocess that critically regulates the flow of sodium ions into excitable cells, is a common functional
219 reveals that the presence of binding-pocket sodium ions is necessary to stabilize the locked-occlude
223 pid/protein ratios, 0% and 1% added NaCl) on sodium ion mobility ((23)Na NMR), in-mouth sodium releas
224 se composition, thus inducing differences in sodium ion mobility and in free sodium ion concentration
226 rmness and perceived hardness, and increased sodium ion mobility, in vivo sodium release and both sal
227 e cheeses, perceived hardness, and decreased sodium ion mobility, in vivo sodium release, saltiness a
228 of a Saccharomyces cerevisiae strain lacking sodium ion (Na(+)) efflux transporters and increased sal
229 igma-1 receptors and inhibited voltage-gated sodium ion (Na+) channels in both native cardiac myocyte
233 f 2 conserved residues (S278 and N401) and a sodium ion (Na2); and the second, by the electrostatic i
239 s associated with counterion condensation of sodium ions onto this part of gp32, which compensates fo
243 e rotor ring from the vacuolar-type (V-type) sodium ion-pumping adenosine triphosphatase (Na+-ATPase)
245 Sodium is globally available, which makes a sodium-ion rechargeable battery preferable to a lithium-
246 nto this part of gp32, which compensates for sodium ion release from the nucleic acid upon its bindin
247 uptake of neurotransmitter with one or more sodium ions, removing neurotransmitter from the synaptic
248 ges that may illuminate the pathway by which sodium ions return to the endoneurial space after they h
249 the ion movements or to the pathway taken by sodium ions returning to their original endoneurial loca
251 tran, and altered apical side tight junction sodium ion selectivity, compared with wild-type mice.
253 that single-particle mass spectra with weak sodium ion signals can be produced by the desorption of
254 ne)amiloride 2 (HMA) supposedly bind in this sodium ion site and can influence orthosteric ligand bin
259 A receptor (hA2AAR), in which the allosteric sodium ion site was elucidated, makes it an appropriate
262 he seven-transmembrane bundle core, with the sodium ion stabilizing a reduced agonist affinity state,
266 The energy required to remove calcium and sodium ions subsequent to nerve excitation was estimated
267 fied new materials design rules for emerging sodium-ion systems that do not apply to lithium-ion syst
268 ions in raw montmorillonites are replaced by sodium ions, the resulting Na(+)-montmorillonite does no
271 om-centric Pt sites are formed by binding to sodium ions through -O ligands, the ensemble being equal
273 R must contain structures that (1) allow the sodium ion to pass through the hydrophobic core of the m
274 s reveal that the GLIC channel is open for a sodium ion to transport, but presents a approximately 11
276 al stimulation, the channel opens and allows sodium ions to enter the cell, inducing a fast upstroke
279 kidney and small intestine, transports three sodium ions together with one divalent anion substrate,
281 ria, a close model for the human enzyme, and sodium ion transport across the mitochondrial inner memb
283 show that the surface diffusion barrier for sodium ion transport is a sensitive function of the chem
286 functional theory calculations suggest that sodium ions undergo occupancy-dependent stepwise inserti
288 ence of binding-pocket leucine substrate and sodium ions, we have sampled plausible conformational st
289 ile range increases in OCM, EC, silicon, and sodium ion were associated with estimated increases in m
294 suggest specific localization sites for the sodium ions, which correspond with experimentally determ
295 D52A(2.50) directly affected the mobility of sodium ions, which readily migrated to another pocket fo
296 cations would strongly favor the passage of sodium ions while hindering translocation of chloride io
297 ns incorporated explicit water molecules and sodium ions, while NMR experiments utilized (15)N-enrich
298 consumption during the diffusion process of sodium ions, while the carbon-coated structure can incre
299 03Asn mutation facilitates coordination of a sodium ion with Lys101 O, Asn103 N and O(delta1), Tyr188
300 This is because of strong interactions of sodium ions with the carbonyl region of a phospholipid m
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