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1 two protons that are later exchanged for one sodium ion.
2 a small cation-friendly cavity occupied by a sodium ion.
3 rfere with the interaction of SNAT2 with the sodium ion.
4  is stabilized by two disulphide bonds and a sodium ion.
5 eraction between the peroxide groups and the sodium ion.
6  prevent it from collapsing onto the smaller sodium ion.
7 e binding pocket requires the removal of the sodium ion.
8 hydrogen bonding network that is mediated by sodium ion.
9 r ~800 water molecules and for magnesium and sodium ions.
10  3 in the presence of potassium ions but not sodium ions.
11 yte battery, which involves the insertion of sodium ions.
12 the synapse, assisted by the co-transport of sodium ions.
13 rom singly charged precursor ions with bound sodium ions.
14 esting the disruption of hydrogen-bonding by sodium ions.
15 phate group, together with flanking zinc and sodium ions.
16 ysiology, including the balance of water and sodium ions.
17 us phase was correlated with the mobility of sodium ions.
18 o the binding sites for one chloride and two sodium ions.
19  D-glucose or D-galactose in the presence of sodium ions.
20 action with mediating water molecules and/or sodium ions.
21 the sulfate ions are coordinated directly to sodium ions.
22  considerably slowing down the permeation of sodium ions.
23 ter EAAC1 is coupled to cotransport of three sodium ions.
24 tage and are less attracted to potassium and sodium ions.
25 s results in a reduced capacity to transport sodium ions.
26 brane and an increased capacity to transport sodium ions.
27 o solid-state electrochemical reactions with sodium ions.
28 nd had a more organised structure around the sodium ions.
29 , 0.34% vs. 0.31%; silicon, 0.36% vs. 0.37%; sodium ion, 0.21% vs. 0.18%; and sulfate, 0.35% vs. 0.38
30 in methanol is K(K+) = 229 +/- 25 M(-1)) and sodium ions (3, K(Na+) = 84.2 +/- 7.9 M(-1) in methanol)
31 ltaenhC mutants showed a hypersensitivity to sodium ion, a phenotype associated with dysfunction of t
32 e carbonate source, but this also introduces sodium ions--a potential catalyst poison.
33               As in previous MD simulations, sodium ions accumulate in an enhanced manner near the C-
34 ree energy released by this reaction to pump sodium ions across the cell membrane.
35 ng oxidation of NADH with ubiquinone to pump sodium ions across the cytoplasmic membrane.
36 ree distinct GA dimeric species, detected as sodium ion adduct ions [2GA + 2Na](2+), and these are as
37 opropanol lead to a significant reduction in sodium ion adduction but are not as effective as acetoni
38 he effectiveness of this method for reducing sodium ion adduction is related to the low proton affini
39                      This method of reducing sodium ion adduction to proteins is simple and requires
40 bF(6), can significantly lower the extent of sodium ion adduction to the molecular ions of proteins a
41  produced as potassium, proton, or sometimes sodium ion adducts, whereas proton loss was dominant in
42 ction remains wide enough for the passage of sodium ions, aided by a continuous bridge of approximate
43 , mesitylenic acid, and solvent molecules on sodium ion all are critical in identifying the most favo
44 through a dual mechanism of intercalation of sodium ions along the x axis of the phosphorene layers f
45 ith cardiovascular hospitalizations, whereas sodium ion, aluminum, and magnesium, components abundant
46 gs differ from the conventional thought that sodium ions always lead to more severe fractures in the
47 ase kinase alpha, Snf1 was activated by both sodium ion and alkaline stress, suggesting that stress s
48 d, the M3 receptor is bound by an allosteric sodium ion and confined mostly in the inactive state wit
49                               A calcium ion, sodium ion and glycerol molecule were identified within
50  increased some of the allosteric effects of sodium ions and amiloride, whereas orthosteric ligand bi
51 nist and antagonist affinity, allosterism by sodium ions and amilorides, and receptor functionality w
52  small but significant decrease in hemolymph sodium ions and an increase in calcium ions after 24 h p
53 ynamics occur in the absence and presence of sodium ions and aspartate, but stall in sodium alone, pr
54 amate from synapses are driven by symport of sodium ions and counter-transport of a potassium ion.
55 ansporter, defining sites for aspartate, two sodium ions and d,l-threo-beta-benzyloxyaspartate, an in
56                           In the presence of sodium ions and no potassium ions, LJM-3064 adopts an an
57 of one glutamate to the cotransport of three sodium ions and one proton and the countertransport of o
58 sists of cotransport of glutamate with three sodium ions and one proton, followed by countertransport
59 forms gated paracellular channels and allows sodium ions and other small positively charged ions to c
60 formation, the enzyme is able to capture two sodium ions and transport them to the external side of t
61 serve electroneutrality and osmotic balance, sodium ions and water also flow into the intestinal lume
62 on, elemental carbon, organic carbon matter, sodium ion, and ammonium.
63 otential role of structured water molecules, sodium ions, and lipids/cholesterol in GPCR stabilizatio
64 ed and reduced states, Na(+)-NQR binds three sodium ions, and that the affinity for sodium is the sam
65    A core domain of six helices harbours two sodium ions, and the remaining four helices pack in a ro
66 xcellent electrode material for lithium-ion, sodium-ion, and lithium-sulfur batteries.
67 ibule about 11 A above the substrate and two sodium ions, apparently stabilizing the extracellular ga
68                 One citrate molecule and one sodium ion are bound per protein, and their binding site
69                                              Sodium ions are actively pumped out of the lumen of the
70 he heptahydrate and decahydrate in which the sodium ions are coordinated exclusively by water molecul
71                                              Sodium ion batteries are being considered as an alternat
72                    It is used as an anode in sodium ion batteries to deliver a high initial reversibl
73  as active electrode materials of lithium or sodium ion batteries, catalysts for water splitting, and
74 NaMF3, the prospective cathode materials for sodium ion batteries.
75 and tested as a promising anode material for sodium ion batteries.
76  and development efforts on room-temperature sodium-ion batteries (NIBs) have been revitalized, as NI
77 u of 0.1 V in PIBs, slightly higher than for sodium-ion batteries (SIBs) (0.01 V), and well above the
78                                      Organic sodium-ion batteries (SIBs) are potential alternatives o
79                                              Sodium-ion batteries (SIBs) are still confronted with se
80                                              Sodium-ion batteries (SIBs) have attracted increasing at
81  to develop high-energy-density cathodes for sodium-ion batteries (SIBs), low-cost, high capacity Na(
82 -performance negative-electrode material for sodium-ion batteries (SIBs).
83 formance when used in lithium-ion batteries, sodium-ion batteries and supercapacitors, respectively.
84                                              Sodium-ion batteries are emerging as a highly promising
85                                              Sodium-ion batteries are emerging as candidates for larg
86                              All-solid-state sodium-ion batteries are promising candidates for large-
87 be a scalable, low-cost cathode material for sodium-ion batteries exhibiting high capacity, long cycl
88   The as-prepared sample used as an anode in sodium-ion batteries exhibits the best rate performance
89 ough recent reports on cathode materials for sodium-ion batteries have demonstrated performances comp
90                                              Sodium-ion batteries have recently attracted significant
91                    Potential applications of sodium-ion batteries in grid-scale energy storage, porta
92                                  This allows sodium-ion batteries to have comparable voltages to lith
93 nd the manufacturing feasibility of low cost sodium-ion batteries with existing lithium-ion battery i
94 ural and chemical evolution of tin anodes in sodium-ion batteries with in situ synchrotron hard X-ray
95 ing electrochemical capacitors, lithium- and sodium-ion batteries, and lithium-sulfur batteries.
96 articular emphasis on lithium-ion batteries, sodium-ion batteries, catalysis of hydrogen evolution, o
97 re proposed to fabricate superior anodes for sodium-ion batteries, featuring high-rate capabilities a
98 pplications including lithium-ion batteries, sodium-ion batteries, lithium-sulfur batteries, lithium-
99                               In the case of sodium-ion batteries, thermodynamically, the use of a so
100  state-of-the-art carbon anode materials for sodium-ion batteries.
101 um-ion systems suggest untapped potential in sodium-ion batteries.
102 present a step forward in the development of sodium-ion batteries.
103 he growing area of new cathode materials for sodium-ion batteries.
104 material, as a viable positive electrode for sodium-ion batteries.
105 chanism of high-capacity antimony anodes for sodium-ion batteries.
106 sing negative electrode material for aqueous sodium-ion batteries.
107 ould have impact in advancing development of sodium-ion batteries.
108 as a negative electrode material for aqueous sodium-ion batteries.
109 r sodium:transition-metal ratio for nonoxide sodium ion battery cathode materials are reported.
110                       To improve lithium and sodium ion battery technology, it is imperative to under
111 form of CTF-HUST-4 as an anode material in a sodium-ion battery achieving an excellent discharge capa
112 nSb) alloys are synthesized and applied as a sodium-ion battery anode.
113  highest capacity value reported for organic sodium-ion battery anodes until now.
114 n, for the first time, we report a family of sodium-ion battery electrodes obtained by replacing step
115                     Organic room-temperature sodium-ion battery electrodes with carboxylate and carbo
116 ility, and cycle life, providing a practical sodium-ion battery powering an electric vehicle in frigi
117                                  The aqueous sodium-ion battery system is a safe and low-cost solutio
118 major scientific challenge for a competitive sodium-ion battery technology is to develop viable anode
119 tion enables the fabrication of a discharged sodium-ion battery with a non-sodium metal anode, and th
120                                  A symmetric sodium-ion battery with an aqueous electrolyte is demons
121              The advent of aqueous symmetric sodium-ion battery with high safety and low cost may pro
122 igh-rate capability and long cycle life in a sodium-ion battery.
123                                 At least two sodium ions bind before aspartate.
124                                 At least two sodium ions bind in close proximity to the substrate and
125 med to understand the mechanistic effects of sodium ion binding on dynamic activation of the M3 musca
126                     Recently we identified a sodium ion binding pocket in a high-resolution structure
127                                  Because the sodium ion binding pocket is highly conserved in other c
128 ucted to have altered ion selectivities, the sodium ion binding site nearest the extracellular side i
129 e D2.50-protonated receptor does not exhibit sodium ion binding to the D2.50 allosteric site and samp
130 teric pockets is significantly weakened upon sodium ion binding.
131 e residue proposed to form part of the third sodium ion-binding site.
132 inactive state, and releases a proton when a sodium ion binds Asp163.
133 has four transmembrane alpha helices, with a sodium ion bound between helices 2 and 4 at a site burie
134       The central cluster harbors a putative sodium ion bound to the highly conserved aspartate resid
135 ve charge introduced at position 124 and the sodium ions bound at Na3' and Na1 underlies the protecti
136 utation results from the substitution of the sodium ions bound within the dimer interface by the intr
137                               Significantly, sodium ions can successfully move out and into without c
138                    Addition of an allosteric sodium ion caused the receptor and ligand to adopt an in
139 validated the concept of full-titanium-based sodium ion cells through the assembly of symmetric cells
140 ncreased expression of certain voltage-gated sodium ion channel (NaV) isoforms in peripheral sensory
141                            The voltage-gated sodium ion channel (VGSC) belongs to the largest superfa
142                                          The sodium ion channel blocker was able to inhibit rCBF chan
143 HN-associated VZV isolates induce changes in sodium ion channel currents known to be associated with
144 lular geometry, gap junctional coupling, and sodium ion channel distribution on propagation velocity
145       The effect of gap junctional coupling, sodium ion channel distribution, and extracellular condu
146                    NaV1.7 is a voltage-gated sodium ion channel implicated by human genetic evidence
147               For example, the voltage-gated sodium ion channel Nav1.7 is expressed selectively in se
148 luates the delivery of dsRNA targeted to the sodium ion channel paralytic A (TcNav) gene in Tribolium
149                                  Single cell sodium ion channel recording was performed after 72 hr b
150 iscern the role of the cardiac voltage-gated sodium ion channel SCN5A in the etiology of dilated card
151 analogs that inhibit NaV1.7, a voltage-gated sodium ion channel that is a compelling target for impro
152 e, and reversible inhibitor of voltage-gated sodium ion channels (NaVs).
153 nctions as a potent agonist of voltage-gated sodium ion channels (NaVs).
154  insensitive to gap junctional coupling when sodium ion channels are located entirely on the cell end
155 ent with what is known of human aura in that sodium ion channels are those predominantly involved in
156 on of Nav 1.6 and Nav 1.7 genes all encoding sodium ion channels the dysregulation of which is associ
157 by mutations in skeletal muscle chloride and sodium ion channels with considerable phenotypic overlap
158  mediated in part by the Nav 1.6 and Nav 1.7 sodium ion channels.
159                                      Aqueous sodium-ion charge storage devices combined with biocompa
160 djustment for gaseous copollutants, nitrate, sodium ion, chloride ion, magnesium, and nickel remained
161                                         Both sodium ion concentration and pH were measured by scannin
162 monapride or [(3)H]spiperone depended on the sodium ion concentration but was independent of the comp
163 arkedly attenuated, there was no decrease in sodium ion concentration in tissue from outer medulla or
164 fferences in sodium ion mobility and in free sodium ion concentration, leading to differences in in-m
165 a dehydrated iron hexacyanoferrate with high sodium-ion concentration enables the fabrication of a di
166 demonstrate that sustained low extracellular sodium ion concentrations ([Na(+)]) directly stimulate o
167 ath by increasing intracellular chloride and sodium ion concentrations.
168 thesis and are found to irreversibly inhibit sodium ion conductance in recombinantly expressed wild-t
169 nts of the transition are open and closed to sodium ion conduction, respectively.
170  analysis, we find that the particles form a sodium-ion conductive film on the anode, which stabilize
171 establish the molecular basis for allosteric sodium ion control in opioid signalling, revealing that
172    Notably, norbuprenorphine interacted with sodium ion-coordinating residues W293(6.48) and N150(3.3
173                                              Sodium ion coordination with the reactant diazo compound
174 h two preferred binding sites identified for sodium ions, corresponding to strong binding with the ox
175              Only two events are observed of sodium ions crossing through the pore.
176 ed by the cardiac sodium channel [persistent sodium ion current (INa)].
177 easure H(2)O and H(2)S fluxes, respectively, sodium ion dilution and buffer acidification by proton r
178  in nature, is approximately 52% faster than sodium ion (DNa+ = 1.33, DCl- = 2.03[10(-9)m(2)s(-1)]).
179 into the cell, driven by the co-transport of sodium ions down their transmembrane concentration gradi
180 trides, (3) an electrostatically stabilizing sodium ion during nitride installation, (4) selecting th
181 e by a rotary motor powered by a proton or a sodium ion electrochemical gradient.
182 tural chemistry and appreciable voltages for sodium-ion electrochemistry.
183                  However, the performance of sodium-ion electrode materials has not been competitive
184 erization of the cocrystalline solid-organic sodium ion electrolyte NaClO4 (DMF)3 (DMF=dimethylformam
185  can lower the surface diffusion barrier for sodium ions, enabling stable electrodeposition.
186 odium gradient to facilitate the exchange of sodium ions for ionic calcium.
187                           The release of one sodium ion from the crystallographically determined sodi
188 tion light-driven H(+)/Na(+) pumps, ejecting sodium ions from cells in the presence of sodium and pro
189           Here, an all-stretchable-component sodium-ion full battery based on graphene-modified poly(
190 upled with an Sb-based anode, the fabricated sodium-ion full-cells also exhibit excellent rate and cy
191 n of reduced ferredoxin with generation of a sodium ion gradient.
192 ts human orthologues, the transporter uses a sodium-ion gradient for nucleoside transport.
193                                Transmembrane sodium-ion gradients provide energy that can be harnesse
194                          For the first time, sodium ions have been imaged sitting inside the 7-member
195 rate, and Na1 and Na2 for two co-transported sodium ions) have been resolved, we still lack a mechani
196                             Dysregulation of sodium ion homeostasis has been implicated in mechanisms
197 aling the presence and fundamental role of a sodium ion in mediating allosteric control of receptor f
198          These new findings suggest that the sodium ion in the allosteric binding pocket not only imp
199               The bound anion and the nearby sodium ion in the Na1 site organize a connection between
200 titative assessment of the metabolic role of sodium ions in cellular processes and their malfunctions
201 ith a trained panel and in vivo retention of sodium ions in human volunteers.
202 wo-dimensional conductivity, owing to mobile sodium ions in lattice planes, between which are insulat
203  was used to study the molecular mobility of sodium ions in model cheeses through measurements of the
204 M7 and Asp-405 on TM8 and support a role for sodium ions in stabilizing substrate-bound conformers.
205 ic, mucoadhesive thickener, the retention of sodium ions in the mouth is prolonged due to the mucoadh
206 for the first time the detailed locations of sodium ions in the selectivity filter of a sodium channe
207 ntials of mean force (PMFs) for chloride and sodium ions in the two receptors.
208 ke potassium ions in potassium channels, the sodium ions in these channels appear to be hydrated and
209 ioinosine, dipyridamole, and dilazep and was sodium ion-independent.
210 while earlier NMR results in the presence of sodium ions indicated a unimolecular, antiparallel quadr
211 olving large anharmonic displacements of the sodium ions inside multi-vacancy clusters.
212 in some prokaryotes, complex I may transport sodium ions instead, and three subunits in the membrane
213                                              Sodium ions interact with chemical moieties of the polys
214 ride ions: Potassium ions, being larger than sodium ions, interact only weakly with phospholipid head
215          The free energies of transferring a sodium ion into a prehydrated gate in functionally close
216 rocess that critically regulates the flow of sodium ions into excitable cells, is a common functional
217 r from the synapse by cotransport with three sodium ions into the surrounding cells.
218 may also indicate the return pathway for the sodium ions involved in impulse formation.
219  reveals that the presence of binding-pocket sodium ions is necessary to stabilize the locked-occlude
220 the unexpected temperature dependence of the sodium ions is no longer observed.
221                 Intra-axonal accumulation of sodium ions is one of the key mechanisms of delayed neur
222 partate release invariably follows that of a sodium ion located near the HP2 gate entrance.
223 pid/protein ratios, 0% and 1% added NaCl) on sodium ion mobility ((23)Na NMR), in-mouth sodium releas
224 se composition, thus inducing differences in sodium ion mobility and in free sodium ion concentration
225  phase, but no significant difference in the sodium ion mobility were obtained.
226 rmness and perceived hardness, and increased sodium ion mobility, in vivo sodium release and both sal
227 e cheeses, perceived hardness, and decreased sodium ion mobility, in vivo sodium release, saltiness a
228 of a Saccharomyces cerevisiae strain lacking sodium ion (Na(+)) efflux transporters and increased sal
229 igma-1 receptors and inhibited voltage-gated sodium ion (Na+) channels in both native cardiac myocyte
230 asma membranes in exchange for extracellular sodium ions (Na+).
231 GltPh coordinates L-aspartate as well as the sodium ion Na1.
232 trostatic interactions with D405 and another sodium ion (Na1).
233 f 2 conserved residues (S278 and N401) and a sodium ion (Na2); and the second, by the electrostatic i
234 lular electric fields and depletion of local sodium ion nanodomains.
235           We believe that the segregation of sodium ions next to the negatively charged sapphire subs
236 atter (OCM), elemental carbon (EC), silicon, sodium ion, nitrate, ammonium, and sulfate.
237                      Partial condensation of sodium ions occurs around a chromonic stack, with two pr
238 80A(7.45)) the negative allosteric effect of sodium ions on agonist binding.
239 s associated with counterion condensation of sodium ions onto this part of gp32, which compensates fo
240                           Addition of either sodium ions or the substrate 5-benzyl-l-hydantoin (L-BH)
241        They exhibit strong selectivities for sodium ions over other cations, enabling the finely tune
242                                            A sodium ion present in the active site suggests that dyna
243 e rotor ring from the vacuolar-type (V-type) sodium ion-pumping adenosine triphosphatase (Na+-ATPase)
244  than in lithium analogues due to the larger sodium-ion radius.
245  Sodium is globally available, which makes a sodium-ion rechargeable battery preferable to a lithium-
246 nto this part of gp32, which compensates for sodium ion release from the nucleic acid upon its bindin
247  uptake of neurotransmitter with one or more sodium ions, removing neurotransmitter from the synaptic
248 ges that may illuminate the pathway by which sodium ions return to the endoneurial space after they h
249 the ion movements or to the pathway taken by sodium ions returning to their original endoneurial loca
250                         The pathway taken by sodium ions returning to their original location and the
251 tran, and altered apical side tight junction sodium ion selectivity, compared with wild-type mice.
252                               Potentiometric sodium ion sensors were developed using a polyvinyl chlo
253  that single-particle mass spectra with weak sodium ion signals can be produced by the desorption of
254 ne)amiloride 2 (HMA) supposedly bind in this sodium ion site and can influence orthosteric ligand bin
255                     The distinctive delta-OR sodium ion site architecture is centrally located in a p
256                                          The sodium ion site is an allosteric site conserved among ma
257 -158 located in a position equivalent to the sodium ion site Na2 of LeuT.
258 (3)H]ZM-241,385) from both the wild-type and sodium ion site W246A mutant hA2AAR.
259 A receptor (hA2AAR), in which the allosteric sodium ion site was elucidated, makes it an appropriate
260 receptor confirmed its likely binding to the sodium ion site.
261                  Here, we show that external sodium ions stabilize the TRPV1 channel in a closed stat
262 he seven-transmembrane bundle core, with the sodium ion stabilizing a reduced agonist affinity state,
263  are evaluated as anode materials in aqueous sodium-ion storage devices.
264                      When used as anodes for sodium-ion storage, these 3D MXene films exhibit much im
265                         Exposure of cells to sodium ion stress, alkaline pH, or oxidative stress caus
266    The energy required to remove calcium and sodium ions subsequent to nerve excitation was estimated
267 fied new materials design rules for emerging sodium-ion systems that do not apply to lithium-ion syst
268 ions in raw montmorillonites are replaced by sodium ions, the resulting Na(+)-montmorillonite does no
269         In contrast to monovalent lithium or sodium ions, the reversible insertion of multivalent ion
270                           In the presence of sodium ions, the sequences with two and three single T l
271 om-centric Pt sites are formed by binding to sodium ions through -O ligands, the ensemble being equal
272 of electron carriers and limited mobility of sodium ions through the aluminium oxide layers.
273 R must contain structures that (1) allow the sodium ion to pass through the hydrophobic core of the m
274 s reveal that the GLIC channel is open for a sodium ion to transport, but presents a approximately 11
275 ) values could be altered by the addition of sodium ions to assays.
276 al stimulation, the channel opens and allows sodium ions to enter the cell, inducing a fast upstroke
277 ate of sufficient diameter to allow hydrated sodium ions to pass through.
278        The titration does not add sufficient sodium ions to the interlayers of the montmorillonite pl
279 kidney and small intestine, transports three sodium ions together with one divalent anion substrate,
280                                          The sodium ion-translocating NADH:quinone oxidoreductase (Na
281 ria, a close model for the human enzyme, and sodium ion transport across the mitochondrial inner memb
282                 An imbalance of chloride and sodium ion transport in several epithelia is a feature o
283  show that the surface diffusion barrier for sodium ion transport is a sensitive function of the chem
284  driven predominantly by active chloride and sodium ion transport.
285             In particular, ENA21, encoding a sodium ion transporter, was strongly induced in C. albic
286  functional theory calculations suggest that sodium ions undergo occupancy-dependent stepwise inserti
287                                An allosteric sodium ion was found bound to a highly conserved D2.50 r
288 ence of binding-pocket leucine substrate and sodium ions, we have sampled plausible conformational st
289 ile range increases in OCM, EC, silicon, and sodium ion were associated with estimated increases in m
290                  The mobility and release of sodium ions were assessed in model cheeses with three di
291        Upon the binding of pVI to DNA, three sodium ions were displaced from the DNA.
292 rmation during dough mixing, compared to the sodium ion, were determined.
293        Thus, a turn-on fluorescent probe for sodium ion, which does not respond to many other metal s
294  suggest specific localization sites for the sodium ions, which correspond with experimentally determ
295 D52A(2.50) directly affected the mobility of sodium ions, which readily migrated to another pocket fo
296  cations would strongly favor the passage of sodium ions while hindering translocation of chloride io
297 ns incorporated explicit water molecules and sodium ions, while NMR experiments utilized (15)N-enrich
298  consumption during the diffusion process of sodium ions, while the carbon-coated structure can incre
299 03Asn mutation facilitates coordination of a sodium ion with Lys101 O, Asn103 N and O(delta1), Tyr188
300    This is because of strong interactions of sodium ions with the carbonyl region of a phospholipid m

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