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1 lular TG storage was induced in the cells by sodium lactate.
2 es after intravenous infusions of hypertonic sodium lactate.
3 racellular solutions that contained 20-80 mm sodium lactate.
6 erents to myocardial ischaemia, lactic acid, sodium lactate, acidic phosphate buffer and hypercapnia
9 xytocin-induced contractility in response to sodium lactate and other weak acids (1-20 mM) were studi
14 f 10-100 microg ml-1 of lactic acid, but not sodium lactate, concentration-dependently stimulated 18
15 enous administration of the anxiogenic agent sodium lactate elicited robust increases in blood pressu
18 ole of plasma cortisol levels in determining sodium lactate-induced panic by reporting psychological,
19 the hypothalamus as putative substrates for sodium lactate-induced panic-like responses in vulnerabl
22 Asn administered either alone or prior to a sodium lactate infusion had no effect on the primed rat'
25 s: a 20-minute baseline, 20-minute 0.5-mol/L sodium lactate infusion, and 15-minute postinfusion peri
26 r experience panic attacks after intravenous sodium lactate infusions by an as yet unexplained mechan
29 acid decarboxylase inhibitor L-allylglycine, sodium lactate infusions or yohimbine elicits panic-like
30 is 'priming' effect induces a sensitivity to sodium lactate infusions that results in a panic-like re
31 ial interaction (SI) test, after intravenous sodium lactate infusions, a phenomenon similar to patien
32 ther show both in vitro and in vivo that the sodium lactate-mediated inhibition of CD4+ T cell motili
33 dy investigates the effect of treatment with sodium lactate (NaLac) on the changes in brain energy st
35 ice were given intraperitoneal injections of sodium lactate or small interfering RNA against Gpr81.
36 late transporters (MCT) or replacing LA with sodium lactate revealed that LA effects are MCT-1- and p
38 and cortisol responses to infusion of 0.5 M sodium lactate were compared among seven subjects with P
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