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1 lular TG storage was induced in the cells by sodium lactate.
2 es after intravenous infusions of hypertonic sodium lactate.
3 racellular solutions that contained 20-80 mm sodium lactate.
4      Continuous intraventricular infusion of sodium lactate (1.0 or 10.0 microM/h) or vehicle was ini
5 ptake and this inhibition was potentiated by sodium lactate (25 mM).
6 erents to myocardial ischaemia, lactic acid, sodium lactate, acidic phosphate buffer and hypercapnia
7                      In CD4+ T helper cells, sodium lactate also induces a switch towards the Th17 su
8                                Extracellular sodium lactate and lactic acid inhibit the motility of C
9 xytocin-induced contractility in response to sodium lactate and other weak acids (1-20 mM) were studi
10                     A detailed comparison of sodium, lactate and cortisol from saliva is reported, de
11 e metabolites: 1-(13)C-pyruvic acid, 1-(13)C-sodium lactate, and 1-(13)C-acetic acid.
12 ces exposed to 0 mM glucose were given 20 mM sodium lactate as a metabolic substrate.
13  density by >80% (0.95-1.76 muA/cm(2)) using sodium lactate as the sole carbon source.
14 f 10-100 microg ml-1 of lactic acid, but not sodium lactate, concentration-dependently stimulated 18
15 enous administration of the anxiogenic agent sodium lactate elicited robust increases in blood pressu
16 nt activities in pulmonary C fibres, whereas sodium lactate had no effect.
17 y-like states and a similar vulnerability to sodium lactate-induced cardioexcitatory responses.
18 ole of plasma cortisol levels in determining sodium lactate-induced panic by reporting psychological,
19  the hypothalamus as putative substrates for sodium lactate-induced panic-like responses in vulnerabl
20                                              Sodium lactate induces flashbacks in persons with PTSD w
21                 This study addressed whether sodium lactate induces flashbacks or other intrusive PTS
22  Asn administered either alone or prior to a sodium lactate infusion had no effect on the primed rat'
23                                              Sodium lactate infusion has induced flashbacks accompani
24 roduced during 'priming' as well as during a sodium lactate infusion into 'primed' rats.
25 s: a 20-minute baseline, 20-minute 0.5-mol/L sodium lactate infusion, and 15-minute postinfusion peri
26 r experience panic attacks after intravenous sodium lactate infusions by an as yet unexplained mechan
27 has no effect on anxiogenic responses due to sodium lactate infusions in 'primed' rats
28                                  Intravenous sodium lactate infusions or the noradrenergic agent yohi
29 acid decarboxylase inhibitor L-allylglycine, sodium lactate infusions or yohimbine elicits panic-like
30 is 'priming' effect induces a sensitivity to sodium lactate infusions that results in a panic-like re
31 ial interaction (SI) test, after intravenous sodium lactate infusions, a phenomenon similar to patien
32 ther show both in vitro and in vivo that the sodium lactate-mediated inhibition of CD4+ T cell motili
33 dy investigates the effect of treatment with sodium lactate (NaLac) on the changes in brain energy st
34 between saline or Ringer's lactate (compound sodium lactate or Hartmann's solution).
35 ice were given intraperitoneal injections of sodium lactate or small interfering RNA against Gpr81.
36 late transporters (MCT) or replacing LA with sodium lactate revealed that LA effects are MCT-1- and p
37                                              Sodium lactate was substituted iso-osmotically for sodiu
38  and cortisol responses to infusion of 0.5 M sodium lactate were compared among seven subjects with P
39                            Administration of sodium lactate, which bypasses the block in the glycolyt

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