ライフサイエンスコーパス: sodium phosphate

1 les were determined, protein, ash, chloride, sodium, phosphate.

2 tment groups: saline solution, dexamethasone sodium phosphate, a nonparticulate steroid, and the part

3 r the elution of CRP, which contained 0.05 M sodium phosphate and 2.0 M NaCl (pH 4.5).

4 er gave higher purification performance than sodium phosphate and citrate-phosphate buffers in IMAC s

5 GII is intrinsically unfolded in 10 mM sodium phosphate and gives weak chromatosome protection,

6 loop of GI, termed GII-L, is stable in 10 mM sodium phosphate and is as effective as GI in chromatoso

7 Both sodium phosphate and magnesium citrate provided excellen

8 dominis plane (TAP) block with dexamethasone sodium phosphate and preperitoneal instillation of local

9 A cathartic (sodium phosphate) and an emetic (ipecac) were given to a

10 s stably folded at low ionic strength (10 mM sodium phosphate) and gives strong protection of chromat

11 ow concentrations of impurities (e.g., 20 mM sodium phosphate), and reduces or eliminates the catastr

12 g 10mmoll(-1) sodium tetraborate, 4mmoll(-1) sodium phosphate, and 25mmoll(-1) SDS.

13 concentrations of SSC, SSPE, PBS, TRIS, MES, sodium phosphate, and potassium phosphate buffers, and f

14 ouble-stranded forms of the 28-mer in 0.01 M sodium phosphate at 25 degrees C, although long-chain (c

15 ty (P(50) = 28.2 mmHg, n(max) = 2.6 in 0.1 M sodium phosphate at pH 7.4) compared to those of Hb A (P

16 y after 120days of storage at 4 degrees C in sodium phosphate buffer (0.1M, pH 7.5), whereas the free

17 U of chondroitinase ABC in 50 microl of 1 mM sodium phosphate buffer (pH 7.0) at 37 degrees C for 6 h

18 heir corresponding DNA/DNA duplexes in 10 mM sodium phosphate buffer (pH 7.0) were determined from di

19 were incubated individually for 2 h in 10 mM sodium phosphate buffer (pH 7.2) containing 0.14 M NaCl,

20 In sodium phosphate buffer (pH 7.4) at 37 degrees C, an unu

21 monocytogenes, and Candida albicans in 10 mM sodium phosphate buffer (pH 7.4).

22 ended mixture consisting in acetonitrile and sodium phosphate buffer 10mmolL(-1) pH = 3.50 (30:70v/v)

23 dine side chains of the mutant rHbs in 0.1 M sodium phosphate buffer at pH 7.0 than for those of Hb A

24 ate that was first dissolved in a 0.1-mmol/L sodium phosphate buffer at pH 7.6 and administered to th

25 ular weight of the proteins extracted with a sodium phosphate buffer containing 2.0% sodium dodecyl s

26 thyl phenylacetyl glucuronide (MPG) in 0.1 M sodium phosphate buffer in D2O at pD 7.4 over 18 h to mo

27 pH pulse produced by freeze-thawing in mixed sodium phosphate buffer induces the oligomerization of D

28 stabilizing agent (i.e., methylene blue and sodium phosphate buffer mixture), the in vitro stability

29 mV shift from CNTs to 7.4 atom % N-CNTs in a sodium phosphate buffer solution (pH 7.0) with 2.0 mM NA

30 n of single-strand DNA, 5'-ATGCTGATGC-3', in sodium phosphate buffer solution at 10 degrees C tempera

31 sition of peroxynitrite (OONO(-)) in aqueous sodium phosphate buffer solution at neutral pH was inves

32 ents of glucose-isomerase concentration in a sodium phosphate buffer that is actively varied over the

33 um dodecyl sulfate from proteins in water or sodium phosphate buffer was achieved by column chromatog

34 Also, when sodium phosphate buffer was used, increasing the ionic s

35 oward neutral to slightly acidic pH in 10 mM sodium phosphate buffer, mitigating the concern of disas

36 ns of our assay (0.05 M sodium citrate/0.1 M sodium phosphate buffer, pH 5.25, 0.2 M NaCl, 0.1 mM EDT

37 el adopts a random-coil conformation in 2 mm sodium phosphate buffer, pH 5.5, and becomes an alpha he

38 With increasing hydration (50 mM sodium phosphate buffer, pH 7.0; 50 mM NaCl), T(M) decre

39 mbda em = 393 nm) in Tris buffer, pH 9.0, or sodium phosphate buffer, pH 7.2, but under the same cond

40 , 0.01-0.06 mM] but not in 0.14 M NaCl/10 mM sodium phosphate buffer, pH 7.2/0.5 mM CaCl2/0.15 mM MgC

41 of 1.0 mug muL(-1) stock solutions in 100 mM sodium phosphate buffers (pH 1-12) at room temperature.

42 genes encoding the kidney-specific type IIa sodium phosphate co-transporter (SLC34A1), the calcium-s

43 The sodium phosphate co-transporters Npt2a and Npt2c play im

44 reduction in the protein levels of the renal sodium-phosphate co-transporter NaPi-IIa in the proximal

45 layed higher renal protein expression of the sodium-phosphate co-transporter NaPi-IIa, lower renal Kl

46 was dependent on alkyl side chain length and sodium phosphate concentration, but the same effects wer

47 ues were then minced and extracted with 5 mM sodium phosphate containing 1% Triton X-100.

48 Parathyroid hormone inhibits sodium-phosphate cotransport in proximal renal tubule ce

49 yroid hormone inhibits proximal renal tubule sodium-phosphate cotransport through a signaling complex

50 ogic regulator of proximal renal tubule cell sodium-phosphate cotransport, stimulates several signal

51 The sodium/phosphate cotransport inhibitor phosphonoformic a

52 Na-H exchanger 3, Na-K ATPase, and type IIa sodium phosphate cotransporter (NaPi IIa).

53 panning protein that functions as a type III sodium phosphate cotransporter and as the receptor for a

54 hybridize to the message for the rat kidney sodium phosphate cotransporter NaPi-2 close to the trans

55 evels and decreased kidney membrane type IIa sodium phosphate cotransporter protein, as was the case

56 ranscripts for hBNP-1, a type 1b human brain sodium phosphate cotransporter, and cystic fibrosis tran

57 Human PiT1 (PiT1), another type III sodium phosphate cotransporter, is a highly related prot

58 on of phosphate uptake and PTH regulation of sodium-phosphate cotransporter (NaPi-4) expression were

59 ein (AKAP) by demonstrating that the type II sodium-phosphate cotransporter (NaPi-4) physically assoc

60 ising candidate gene, SLC34A1 encoding renal sodium-phosphate cotransporter 2A (NaPi-IIa), revealed a

61 sive genes, we inadvertently discovered that sodium-phosphate cotransporter 2B (NaPi-2b) mRNA concent

62 med by co-transfecting HEK293 cells with the sodium-phosphate cotransporter and wild type AKAP, a mut

63 in D 1alpha-hydroxylase cytochrome P450, and sodium-phosphate cotransporter mRNA concentrations.

64 ontains SLC34A3, the gene encoding the renal sodium-phosphate cotransporter NaP(i)-IIc.

65 (a) The proximal sodium-phosphate cotransporter NaPi-2 was markedly upreg

66 FGF23 levels and abundant expression of the sodium-phosphate cotransporter Npt2a at the brush border

67 n of recombinant cKL downregulated the renal sodium-phosphate cotransporter Npt2a in alphaKL-null mic

68 bsorption of phosphate by downregulating the sodium-phosphate cotransporter Npt2a.

69 o bound other cellular targets including the sodium-phosphate cotransporter type IIa encoded by the N

70 sis transmembrane conductance regulator, and sodium-phosphate cotransporter type IIa.

71 also measured the expression of aquaporin-2, sodium/phosphate cotransporter (NaPi)-2, paracellin-1, a

72 ssociated with increased renal expression of sodium/phosphate cotransporter 2a (NaPi2a) protein.

73 The PIT1/SLC20A1 protein, a well-described sodium/phosphate cotransporter and retrovirus receptor,

74 mice have increased renal expression of the sodium/phosphate cotransporter NaP(i)2a and of 1- alpha-

75 se in the transport activity of the type IIa sodium/phosphate cotransporter protein (NaPi) despite an

76 ses suggest that increased renal activity of sodium-phosphate cotransporters (NaPi2a) leads to severe

77 xcretion by reducing expression of the renal sodium-phosphate cotransporters NaPi-IIa and NaPi-IIc in

78 PLGA) nanoparticles containing dexamethasone sodium phosphate (DSP) exhibited a size of 200 nm, 8 wt.

79 d 118 consecutive patients given single-dose sodium phosphate for bowel catharsis and 115 consecutive

80 8.6% (627 of 708) of colonic segments in the sodium phosphate group and in 88.1% (608 of 690) in the

81 ntly different between magnesium citrate and sodium phosphate groups (790 HU +/- 216 vs 978 HU +/- 16

82 weak chromatosome protection, but in 250 mM sodium phosphate has a structure very similar to that of

83 The activity of cell-surface sodium-phosphate (Na(+)-P(i)) cotransporters mediates th

84 Apically expressed sodium-phosphate (Na(+)-P(i)) transporters play a critic

85 , 2.60; 95% confidence interval, 1.46-4.63), sodium phosphate (OR, 9.34; 95% confidence interval, 2.1

86 Oral sodium phosphate (OSP) is a commonly used purgative befo

87 t formation, autolyzed more rapidly in 10 mM sodium phosphate (pH 6.8), and, in contrast to strains t

88 In 10 mM sodium phosphate (pH 7), the GI domain, like the globula

89 Likewise, the formation of alanine in 500 mM sodium phosphate (pH 7.0) from 5 mM glyceraldehyde and 1

90 yde and ammonium ion in aqueous solutions of sodium phosphate (pH 7.0) or imidazole-imidazolium chlor

91 Surprisingly, in 10 mM sodium phosphate (pH 7.0), GII is largely unfolded, wher

92 dodecyl sulfate (SDS) and 1-butanol in 10 mM sodium-phosphate (pH 7.2) at a flow rate of 5 mL/min.

93 sensitivity and speed of analysis were 50 mM sodium phosphate, pH 2.3, and 100 mM ammonium formate, p

94 roximately 2 microM at 25 degreesC in 0.1 mM sodium phosphate, pH 7.2.

95 ibrils after incubation (37 degrees C, 10 mM sodium phosphate, pH 7.60), but in contrast to beta-shee

96 embranes prepared by hypotonic lysis in 5 mM sodium phosphate, pH 8.0 (5P8), conditions in which the

97 oscopy, including polyethylene glycol (PEG), sodium phosphate, picosulfate, or oral sulfate solutions

98 Phenstatin was converted to the sodium phosphate prodrug (3d) by a dibenzyl phosphite ph

99 Hydroxyphenstatin was converted to the sodium phosphate prodrug (6e) by a dibenzyl phosphite ph

100 oscopy preceded by bowel cleansing with oral sodium phosphate solution (OSPS) or Visicol.

101 These viruses use the related sodium-phosphate symporters Pit1 and Pit2, respectively,

102 multicenter trial compared 1.0% prednisolone sodium phosphate to placebo in the treatment of bacteria

103 genes with structural similarity to a type 1 sodium phosphate transporter, 12 novel histone genes, an

104 delivery (PBS pH 7.2 and 0.9% saline), and a sodium phosphate vehicle previously shown to enhance the

105 fference seen in the sigmoid colon (2.17 for sodium phosphate vs 2.44 for magnesium citrate; P< 0.01)

106 When sodium phosphate was substituted for beta-glycerol phosp

107 In 10 mM sodium phosphate with 125 mM NaCl, the combinations of s

108 II-L is very similar to GII folded in 250 mM sodium phosphate, with the exception of the substituted

109 1 M triflic acid) and 1150 mV at pH 6 (10 mM sodium phosphate) yielded a red colored solution with a