ライフサイエンスコーパス: sodium sulfate

1 as induced by oral administration of dextran sodium sulfate.

2 r administration of azoxymethane and dextran sodium sulfate.

3 e after exposure to azoxymethane and dextran sodium sulfate.

4 signaling during colitis induced by dextran sodium sulfate.

5 susceptibility to colitis induced by dextran sodium sulfate.

6 y 3 cycles of oral administration of dextran sodium sulfate.

7 xymethane followed by treatment with dextran sodium sulfate.

8 aused by chemical irritants, such as dextran sodium sulfate.

9 hesis was measured by incorporation of [35S]-sodium sulfate.

10 e to wounding of the epithelium with dextran sodium sulfate.

11 ments are repeated in the presence of 400 mM sodium sulfate.

12 absence and presence of a stabilizing salt, sodium sulfate.

13 ed by the high sodium content of the dextran sodium sulfate.

14 loys treatment with azoxymethane and dextran sodium sulfate.

15 inflammation induced by azoxymethane/dextran sodium sulfate.

16 a mouse model of colitis induced by dextran sodium sulfate.

17 stration of azoxymethane followed by dextran sodium sulfate.

18 l injury following administration of dextran sodium sulfate.

19 sis and epithelial injury induced by dextran sodium sulfate.

20 itis in mice after administration of dextran sodium sulfate.

21 its stability at different concentrations of sodium sulfate.

22 ration with diatomaceous earth and anhydrous sodium sulfate.

23 colitis induced by a single cycle of dextran sodium sulfate administration.

24 presence of the stabilizing additives 0.23 M sodium sulfate and 1 M sodium chloride.

25 ults were found when 0.5g mussel shell, 0.5g sodium sulfate and 5mL ethanol were used.

26 c epithelium after administration of dextran sodium sulfate and azoxymethane than control mice.

27 cer was induced by administration of dextran sodium sulfate and azoxymethane.

28 Here we report that the dextran sodium sulfate and CD4+CD45RBhigh T-cell transfer models

29 We found that in both the dextran sodium sulfate and Citrobacter rodentium models of colit

30 was not observed after ingestion of dextran sodium sulfate and correlated with exacerbation of the m

31 ator (Cftr) knockout mice exposed to dextran sodium sulfate and in vitro in primary cholangiocytes is

32 tis was induced by administration of dextran sodium sulfate, and colitis-associated cancer was induce

33 hobiont challenge, administration of dextran sodium sulfate, and high-fat diets.

34 in C57/Bl6 mice by administration of dextran sodium sulfate, and mice were given 10(8) bacteria for 1

35 ry (ATOFMS) revealed the presence of halite, sodium sulfates, and sodium carbonates that were strongl

36 Similarly, Spns2 deletion reduced dextran sodium sulfate- and oxazolone-induced colitis.

37 nogenesis protocol [azoxymethane and dextran sodium sulfate (AOM-DSS) administration] exhibited a two

38 on tumorigenesis in the azoxymethane-dextran sodium sulfate (AOM-DSS) model of colitis-associated car

39 c-deficient mice in the azoxymethane/dextran sodium sulfate (AOM/DSS) model of colorectal cancer.

40 arcinogenesis using the azoxymethane/dextran sodium sulfate (AOM/DSS) model.

41 ecreased in both the presence and absence of sodium sulfate, as previously reported for a variety of

42 n of a 3.41 mol/kg water aqueous solution of sodium sulfate at 1.54 GPa in a diamond-anvil cell resul

43 (PAA) and Amplon (blend of sulfuric acid and sodium sulfate) at a poultry processing pilot plant scal

44 enomas formed following azoxymethane/dextran sodium sulfate challenge.

45 formation following an azoxymethane/dextran sodium sulfate challenge.

46 We identified and characterized the sodium/sulfate co-transporter (NaS-1; Slc13a1) as an Fxr

47 signaling protected mice from acute dextran sodium sulfate colitis because DR3(-/-) mice showed more

48 (DeltaIEC)) mice develop more severe dextran sodium sulfate colitis due to delayed ulcer healing and

49 issue from Ier3(-/-) mice subject of dextran sodium sulfate colitis exhibit greater Nrf2 activity tha

50 /2(-/-) BM chimera mice with chronic dextran sodium sulfate colitis exhibited delayed ulcer healing,

51 Studies of dextran sodium sulfate colitis in intestinal epithelial-specific

52 Dextran sodium sulfate colitis led to increased levels of inflam

53 e were similarly more susceptible to dextran sodium sulfate colitis, although without mortality and w

54 Using the azoxymethane and dextran sodium sulfate colitis-associated colorectal cancer mode

55 lity and increased susceptibility to dextran sodium sulfate colitis.

56 ins in macrophages protect mice from dextran sodium sulfate-colitis by enhancing 15-PGDH-dependent ox

57 In response to dextran sodium sulfate, colonic infiltration of neutrophils and

58 nt gas, the extract was dried with anhydrous sodium sulfate, concentrated through evaporation, and th

59 of one-dimensional bonded chains observed in sodium sulfate decahydrate (mirabilite).

60 stinal inflammation upon exposure to dextran sodium sulfate, demonstrating a previously unrecognized

61 e mice treated with azoxymethane and dextran sodium sulfate developed approximately 7-10 tumors per m

62 burdens following azoxymethane (AOM)/dextran sodium sulfate (DSS) administration compared with wild-t

63 We administered 2 exogenous agents, dextran sodium sulfate (DSS) and acetic acid, to assess the susc

64 In the dextran sodium sulfate (DSS) and adoptive T-cell transfer models

65 tis and colitis-associated CRC using dextran sodium sulfate (DSS) and azoxymethane (AOM)-DSS experime

66 Dextran sodium sulfate (DSS) and Citrobacter rodentium colitis (

67 mely sensitive to colitis induced by dextran sodium sulfate (DSS) and developed spontaneous ileitis a

68 using two models, administration of dextran sodium sulfate (DSS) and Salmonella enterica subsp. sero

69 nitrobenzene sulfonic acid (TNBS) or dextran sodium sulfate (DSS) and the inflammatory responses were

70 The dextran sodium sulfate (DSS) and trinitrobenzene sulfonic acid (

71 ucosal injury and colitis induced by dextran sodium sulfate (DSS) are ameliorated in epimorphin-/- mi

72 nted diets was assayed after a 7-day dextran sodium sulfate (DSS) challenge by quantitative real-time

73 sessment of mucosal damage in murine dextran sodium sulfate (DSS) colitis and human IBD.

74 DeltaIEC) mice) were subjected to 2% dextran sodium sulfate (DSS) colitis for 7 days.

75 eficient mice and chemically induced dextran sodium sulfate (DSS) colitis have led to inconsistent re

76 he development of azoxymethane (AOM)/dextran sodium sulfate (DSS) colitis-associated cancer (CAC).

77 eoplasia, we compared differences in dextran sodium sulfate (DSS) colitis-associated neoplasia betwee

78 nistering the specific inhibitors in dextran sodium sulfate (DSS) colitis.

79 Dextran sodium sulfate (DSS) exposure induced STAT-6 and NF-kapp

80 Mice given dextran sodium sulfate (DSS) followed by NP-KPV were protected a

81 type littermates by administering 3% dextran sodium sulfate (DSS) for 7 days followed by 2-week recov

82 Mice (C57BL/6) were exposed to 3% dextran sodium sulfate (DSS) for 7 days or 4% DSS for 5 days fol

83 tis was induced by administration of dextran sodium sulfate (DSS) in distilled water.

84 and wild-type mice by administering dextran sodium sulfate (DSS) in drinking water.

85 extracellular osmolarity induced by dextran sodium sulfate (DSS) in vivo Collectively, these finding

86 gammadelta IEL was evaluated in the dextran sodium sulfate (DSS) induced mouse colitis model system.

87 duces the severity of colitis in the dextran sodium sulfate (DSS) model of murine colonic injury.

88 itrobenzene sulfonic acid (TNBS) and dextran sodium sulfate (DSS) models of colitis.

89 Administration of dextran sodium sulfate (DSS) significantly increased intestinal

90 thane followed by multiple rounds of dextran sodium sulfate (DSS) to induce colitis and tumorigenesis

91 Using dextran sodium sulfate (DSS) to induce colitis in mice, we ident

92 ymethane followed by three cycles of dextran sodium sulfate (DSS) to induce colitis-associated cancer

93 Some mice were given dextran sodium sulfate (DSS) to induce colitis.

94 Mice were fed 3% dextran sodium sulfate (DSS) to induce colonic inflammation, wit

95 beta(-/-) mice, and, when exposed to dextran sodium sulfate (DSS) to induce inflammatory bowel diseas

96 tis was induced by administration of dextran sodium sulfate (DSS) to mice or transfer of T cells to l

97 X-2(-/-), and heterozygous mice with dextran sodium sulfate (DSS) to provoke acute colonic inflammati

98 We show that dextran sodium sulfate (DSS) treatment promotes the recruitment

99 microbiota was achieved via low-dose dextran sodium sulfate (DSS) treatment.

100 Dextran sodium sulfate (DSS) was used to induce colitis in C57BL

101 lowed by three 1-week cycles of 2.5% dextran sodium sulfate (DSS) water, each cycle separated by 2 we

102 Dextran sodium sulfate (DSS), 2,4,6-trinitrobenzene sulfonic aci

103 intestinal inflammation elicited by dextran sodium sulfate (DSS), a model of experimental colitis.

104 exposure to the oral innate trigger dextran sodium sulfate (DSS), a nonredundant proinflammatory rol

105 induced in mice by administration of dextran sodium sulfate (DSS), and carcinogenesis was induced by

106 Dextran sodium sulfate (DSS), at doses that resulted in little e

107 ice, or given azoxymethane (AOM) and dextran sodium sulfate (DSS), or 1,2-dimethylhydrazine and DSS,

108 nvironmental epithelial injury using dextran sodium sulfate (DSS), Tfeb (DeltaIEC) mice exhibited exa

109 In acute colitis induced by dextran sodium sulfate (DSS), Vim KO mice develop significantly

110 epithelium with low-molecular-weight dextran sodium sulfate (DSS), which is a well-studied model of m

111 is were induced by administration of dextran sodium sulfate (DSS), with or without azoxymethane (AOM)

112 We used dextran sodium sulfate (DSS)-induced chronic colitis to investig

113 In this article, we show that dextran sodium sulfate (DSS)-induced clinical disease and histol

114 Mucosal repair was assessed after dextran sodium sulfate (DSS)-induced colitis in mice receiving i

115 antioxidants on gut permeability and dextran sodium sulfate (DSS)-induced colitis in mice was tested.

116 l thrombosis that is associated with dextran sodium sulfate (DSS)-induced colitis in mice.

117 ve component of the host response to dextran sodium sulfate (DSS)-induced colitis in the mouse is med

118 Although dextran sodium sulfate (DSS)-induced colitis is a commonly used

119 estinal tract, significantly reduced dextran sodium sulfate (DSS)-induced colitis severity, whereas d

120 alactosylceramide (alpha-GalCer), on dextran sodium sulfate (DSS)-induced colitis were examined.

121 IL-6-deficient (IL-6(-/-)) mice with dextran sodium sulfate (DSS)-induced colitis.

122 caspase-1 were highly susceptible to dextran sodium sulfate (DSS)-induced colitis.

123 f Pirb-/- and wild-type (WT) mice to dextran sodium sulfate (DSS)-induced colitis.

124 at causes enhanced susceptibility to dextran sodium sulfate (DSS)-induced colitis.

125 inflammation or crypt damage during dextran sodium sulfate (DSS)-induced colitis.

126 protect intestinal epithelium during dextran sodium sulfate (DSS)-induced colitis.

127 or 50 mg/kg), in an animal model of dextran sodium sulfate (DSS)-induced colitis.

128 Here we address this issue using a dextran sodium sulfate (DSS)-induced colonic regeneration model.

129 to induce IL-36gamma in response to dextran sodium sulfate (DSS)-induced damage, suggesting that gut

130 pared with wild-type (WT) mice after dextran sodium sulfate (DSS)-induced injury.

131 Using the dextran sodium sulfate (DSS)-induced intestinal epithelial injur

132 Colon was examined after acute dextran sodium sulfate (DSS)-induced mucosal injury or after azo

133 oethylene-o,o') tellurate (AS101) on dextran sodium sulfate (DSS)-induced murine colitis.

134 )Apc(Min/+) mice, azoxymethane (AOM)/dextran sodium sulfate (DSS)-treated mice and de-identified huma

135 ring TNFalpha-siRNA-loaded NPs to 3% dextran sodium sulfate (DSS)-treated mice and investigated the t

136 In comparison with wild-type mice, Dextran Sodium Sulfate (DSS)-treated TRPM8 knockout mice showed

137 alities after oral administration of dextran sodium sulfate (DSS).

138 equently exposed to colonic irritant dextran sodium sulfate (DSS).

139 d IL-8Tg mice given azoxymethane and dextran sodium sulfate (DSS).

140 mical colitis induced by exposure to dextran sodium sulfate (DSS).

141 then subjected to repeated cycles of dextran sodium sulfate (DSS).

142 ollowed by induction of colitis with dextran sodium sulfate (DSS).

143 d the severity of colitis induced by dextran sodium sulfate (DSS).

144 We induced colitis in mice using Dextran Sodium Sulfate (DSS).

145 he mouse model of colitis induced by dextran sodium sulfate (DSS).

146 mipenem before or after receiving 5% dextran sodium sulfate (DSS).

147 by continuous oral administration of dextran sodium sulfate (DSS).

148 Colitis was chemically induced with dextran sodium sulfate (DSS).

149 ion and tissue repair in response to dextran sodium sulfate (DSS).

150 he mouse, which are augmented during dextran sodium sulfate (DSS)/azoxymethane (AOM)-induced CAC.

151 as induced by oral administration of dextran sodium sulfate (DSS, 5 g/dL) to knockout mice, their gen

152 of IFN-gamma to G2A(-/-) mice during dextran sodium sulfate exposure abolished the excess colitic inf

153 methane alone or in combination with dextran sodium sulfate; formation of aberrant crypt foci and col

154 s of sodium sulfate is the highly metastable sodium sulfate heptahydrate (Na(2)SO(4).7H(2)O).

155 ted in the formation of a previously unknown sodium sulfate hydrate, which we have determined by sing

156 is of proteoglycans via incorporation of 35S-sodium sulfate in the culture medium.

157 Acute colitis was induced using 4% dextran sodium sulfate in wild-type mice maintained on Se-defici

158 e models (Salmonella typhimurium and dextran sodium sulfate) in PHB transgenic mice and wild-type lit

159 naturation were observed; for example, 0.4 M sodium sulfate increased the free energy of wild-type SN

160 in altering the onset and relapse of dextran sodium sulfate induced murine colitis.

161 is under normal conditions; however, dextran sodium sulfate-induced (DSS-induced) colitis promoted th

162 In a dextran sodium sulfate-induced acute colitis model, WT mice lost

163 tes, and increases susceptibility to dextran sodium sulfate-induced bowel injury.

164 in mice inhibited azoxymethane- and dextran sodium sulfate-induced CAC, IL-6 expression, STAT3 phosp

165 CDDO-Me suppressed azoxymethane plus dextran sodium sulfate-induced carcinogenesis in wild-type anima

166 in epithelial cells during both the dextran sodium sulfate-induced colitic and the recovery phase.

167 Further, administration of EGCG to dextran sodium sulfate-induced colitic mice significantly reduce

168 Mouse models of dextran sodium sulfate-induced colitis and a T cell transfer mod

169 ts were observed in a mouse model of dextran sodium sulfate-induced colitis and in Caco2-BBE cells tr

170 th trinitrobenzene sulfonic acid- or dextran sodium sulfate-induced colitis and in Il10(-/-) mice.

171 Dextran sodium sulfate-induced colitis and Salmonella typhimuriu

172 bsence of villin predisposes mice to dextran sodium sulfate-induced colitis by promoting apoptosis.

173 od2 results in higher sensitivity to dextran sodium sulfate-induced colitis compared with a single de

174 ignificantly reduced the severity of dextran sodium sulfate-induced colitis in mice.

175 Increased sensitivity to dextran sodium sulfate-induced colitis in Pglyrp3(-/-)Nod2(-/-)

176 ministration of MDP does not prevent dextran sodium sulfate-induced colitis in SAMP mice and that the

177 d ameliorating disease phenotypes in dextran sodium sulfate-induced colitis mice.

178 Treatment of mice with HDACi in a dextran sodium sulfate-induced colitis model resulted in a stron

179 Dextran sodium sulfate-induced colitis model was established in

180 etermined in vitro as well as in the dextran sodium sulfate-induced colitis mouse model.

181 nt model of allergic airway disease, dextran sodium sulfate-induced colitis was significantly reduced

182 1(-/-) mice and CRHR2(-/-) mice with dextran sodium sulfate-induced colitis were analyzed in comparis

183 R (NCM460-NK-1R cells) and mice with dextran sodium sulfate-induced colitis were used.

184 rom TNF exposure, and exhibit severe dextran sodium sulfate-induced colitis, ameliorated by TNF inhib

185 elevated in the colons of mice with dextran sodium sulfate-induced colitis, which was reduced by tre

186 re transferred to CD1 nude mice with dextran sodium sulfate-induced colitis, with or without oral adm

187 , TNF-alpha, and IL-17) in mice with dextran sodium sulfate-induced colitis.

188 while promoting Treg development in dextran sodium sulfate-induced colitis.

189 , it exerted protective functions in dextran sodium sulfate-induced colitis.

190 cking colonic RelA were sensitive to dextran sodium sulfate-induced colitis.

191 oth autoimmune encephalomyelitis and dextran sodium sulfate-induced colitis.

192 enhance or attenuate the severity of dextran sodium sulfate-induced colitis.

193 comparing healthy mice and mice with dextran sodium sulfate-induced colitis.

194 ing are transmissible and exacerbate dextran sodium sulfate-induced colitis.

195 and exaggerated inflammation during dextran sodium sulfate-induced colitis.

196 rfering (si)RNA to C57BL/6 mice with dextran sodium sulfate-induced colitis.

197 lfonic acid (DNBS)-, oxazolone-, and dextran-sodium sulfate-induced colitis.

198 en associated with susceptibility to dextran sodium sulfate-induced colitis.

199 reater incidence of azoxymethane and dextran sodium sulfate-induced colon carcinoma.

200 rotected the colonic epithelium from dextran sodium sulfate-induced damage.

201 e highly susceptible to azoxymethane/dextran sodium sulfate-induced inflammation and suffered from dr

202 than Rag(-/-) mice to development of dextran sodium sulfate-induced intestinal inflammation, indicati

203 tenance of intestinal homeostasis in dextran sodium sulfate-induced intestinal injury.

204 Using a dextran sodium sulfate-induced mouse model of acute colitis, we

205 n in Cftr-knockout mice exposed to a dextran sodium sulfate-induced portal endotoxemia.

206 revention and recovery rat models of dextran-sodium-sulfate-induced colitis.

207 portantly, PPARdelta is required for dextran sodium sulfate induction of proinflammatory mediators, i

208 in TNBS-inflamed guinea pigs, and in dextran sodium sulfate-inflamed mice, treated with a free radica

209 yl dipeptide (to stimulate NOD2), or dextran sodium sulfate; intestinal lamina propria cells were col

210 sis was measured as the incorporation of 35S-sodium sulfate into macromolecules separated from uninco

211 the crystallization of aqueous solutions of sodium sulfate is the highly metastable sodium sulfate h

212 ption of the epithelial barrier with dextran sodium sulfate leads to increased IL-19 expression.

213 colon into the lymphatic system in a dextran sodium sulfate mediated model of inflammatory bowel dise

214 models of colonic inflammation: the dextran sodium sulfate model and multidrug resistance gene 1a-de

215 Here, studies utilizing the murine dextran sodium sulfate model of colitis revealed the crucial rol

216 ENT FINDINGS: Using the azoxymethane-dextran sodium sulfate model, wound healing pathways seem to be

217 ion and colitis-associated cancer in dextran sodium sulfate model.

218 he trinitrobenzene sulfonic acid and dextran sodium sulfate models of colitis, we show the importance

219 litis was induced in mice by oral 5% dextran sodium sulfate or rectal 5% acetic acid, followed by ene

220 OD, and B6.AKR) by administration of dextran sodium sulfate or rectal application of trinitrobenzene

221 MMP-9-/- mice exposed to dextran sodium sulfate or salmonella had a significantly reduced

222 vere colitis after administration of dextran sodium sulfate or trinitrobenzene sulfonate than mice wi

223 Colitis was induced using dextran sodium sulfate or trinitrobenzene sulfonic acid (TNBS).

224 olitis, induced by administration of dextran sodium sulfate or trinitrobenzene sulfonic acid.

225 e signal response was found to be linear for sodium sulfate over the concentration ranges of 0.2-100

226 05% of the total protein population in 20 mM sodium sulfate, respectively.

227 response, induction of colitis with dextran sodium sulfate resulted in a MyD88-dependent serum Ab re

228 s following initiation of refolding in 0.4 M sodium sulfate revealed weak protection in the first bet

229 Addition of sodium sulfate shifts the rate profile to higher denatur

230 in the rectum following injury with dextran sodium sulfate, similarly treated Myd88(-/-) (TLR signal

231 studied the influence of a number of salts (sodium sulfate, sodium fluoride, sodium acetate, and sod

232 ablated all but the first exon of SLC13A1, a sodium/sulfate symporter responsible for regulating seru

233 vere colitis after administration of dextran sodium sulfate than mice infected with LF82-DeltachiA or

234 oncentrations, especially in the presence of sodium sulfate), the kinetics of folding shows evidence

235 This resembles anhydrous sodium sulfate (thenardite) but contrasts with the hepta

236 o Rag(-/-) mice or administration of dextran sodium sulfate to C57BL/6 mice.

237 ced in some mice by addition of 2.5% dextran sodium sulfate to drinking water for 5-9 consecutive day

238 ice were exposed to azoxymethane and dextran sodium sulfate to induce colitis and tumorigenesis.

239 l polyinosinic:polycytidylic acid or dextran sodium sulfate to induce colitis.

240 n of sodium chloride, potassium chloride, or sodium sulfate to leptospiral medium to physiological os

241 tis was induced by administration of dextran sodium sulfate to wild-type and Cav-1(-/-) mice, as well

242 e, or given azoxymethane followed by dextran sodium sulfate, to assess intestinal tumor formation.

243 g azoxymethane injection followed by dextran sodium sulfate treatment in TLR4-deficient or wild-type

244 nuated colon inflammation induced by dextran sodium sulfate treatment or Citrobacter rodentium infect

245 e gut in response to indomethacin or dextran sodium sulfate treatment.

246 uced tumor burden after azoxymethane/dextran sodium sulfate treatment.

247 ation compared with WT animals after dextran sodium sulfate treatment.

248 rcinogenesis induced by azoxymethane/dextran sodium sulfate treatment.

249 ion-driven tumor model (azoxymethane/dextran sodium sulfate), VS28 mice developed a significantly hig

250 njury induced by the toxic substance dextran sodium sulfate was fundamentally altered to include path

251 mon colitis model, administration of dextran sodium sulfate, was hopelessly confounded by the high so

252 alpha-d-glucose and citric acid, along with sodium sulfate, were produced using established and newl

253 human FN promoter, given water or 3% dextran sodium sulfate, were used as animal models of colitis.

254 For example, a sodium sulfate with single, oligomeric vinyl acetate (VA