ライフサイエンスコーパス: sodium vanadate

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1 micked by the tyrosine phosphatase inhibitor sodium vanadate (100 microm).

2 In the presence of sodium vanadate, a phosphatase inhibitor, phosphorylated

3 th specific inhibitors of p38 MAP kinase and sodium vanadate, a potent protein-tyrosine phosphatase i

4 Hence, treatment of Bcl-xL cells with sodium vanadate, a tyrosine phosphatase inhibitor, resto

5 Preincubation with okadaic acid and sodium vanadate abrogated adhesion-induced dephosphoryla

6 In addition, sodium vanadate, also an inhibitor of protein phosphatas

7 Sodium vanadate, an inhibitor of phosphotyrosine phospha

8 titive inhibition of alkaline phosphatase by sodium vanadate and sodium arsenate has been examined, a

9 known PTPase inhibitors phenylarsine oxide, sodium vanadate, and iodoacetate also inhibited enzyme a

10 g protein-tyrosine phosphatase inhibition by sodium vanadate both EGFR expressing Chinese hamster ova

11 t of GEO cells with a phosphatase inhibitor (sodium vanadate) caused a dose-dependent increase in ERK

12 phatase, we found that sodium pyrophosphate, sodium vanadate, cyclosporin A, tautomycin, and okadaic

13 e Rho, or the tyrosine phosphatase inhibitor sodium vanadate increased the level of phosphorylation o

14 ion because pretreatment of hepatocytes with sodium vanadate increases (and 25 microM genistein reduc

15 lished by the tyrosine phosphatase inhibitor sodium vanadate, indicating involvement of protein tyros

16 Furthermore, sodium vanadate induced primitive endoderm differentiati

17 Sodium vanadate is an effective agent for the enrichment

18 Sodium vanadate is an effective drug for the enrichment

19 , the phosphotyrosine phosphatase inhibitor, sodium vanadate, or expression of mutationally activated

20 o increased and decreased by the addition of sodium vanadate, respectively, but these changes were el

21 ition of the tyrosine phosphatase inhibitor, sodium vanadate, selectively repressed Nanog transcripti

22 the astrocytes, while both okadaic acid and sodium vanadate significantly reversed these anti-prolif

23 The enzyme activity was inhibited by sodium vanadate, sodium fluoride, N-ethylmaleimide, and

24 c peptides caused a 70-80% inhibition of the sodium vanadate-stimulated MAPK activity, complete inhib

25 ith compounds acting by distinct mechanisms: sodium vanadate (SV), an inhibitor of protein phosphatas

26 ated tyrosine phosphatases were blocked with sodium vanadate, the high-frequency gating remained rela

27 We measured the stiffness of 50 muM sodium vanadate treated bull sperm in the presence of 4

28 We investigated the molecular mechanisms of sodium vanadate (vanadate)-induced nitric oxide (NO) pro

29 Sodium vanadate (Vi) is a phosphate analog that inhibits

30 s-bridge binding to intermediate levels with sodium vanadate (Vi).

31 force to 6% of pCa 4.0 controls with 1.0 mM sodium vanadate (Vi).

32 The effects of sodium vanadate were abrogated by Grb2 deficiency or by

33 minin 5, we found that phosphatase inhibitor sodium vanadate, which blocked the p80 dephosphorylation

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