ライフサイエンスコーパス: sodium-dependent

1 hibition by L-leucine but not by L-lysine is sodium dependent.

2 transport of nucleosides is proton-, but not sodium-, dependent.

3 Similarly, sodium-dependent [3H]taurocholate uptake into membrane v

4 pendent on GLUT transport of DHA rather than sodium-dependent AA uptake.

5 -AP-sensitive channels were activated during sodium-dependent action potentials and mediated a large

6 of CA1 pyramidal neurons and drive bursts of sodium-dependent action potentials at the soma.

7 ed phase advances, suggest that NPY requires sodium-dependent action potentials within GABAergic neur

8 Blockade of sodium-dependent action potentials within the SCN preven

9 that spread to the synaptic terminal without sodium-dependent action potentials.

10 d in part by potassium channels activated by sodium-dependent action potentials.

11 ry across the plasma membrane in response to sodium-dependent action potentials.

12 Larval motor neurons had a long-lasting, sodium-dependent afterhyperpolarization (AHP) following

13 solution of pulmonary edema by up-regulating sodium-dependent alveolar fluid clearance.

14 The sodium-dependent amino acid systems A and Bo,+ are locat

15 The sodium-dependent amino acid transport systems responsibl

16 area of mouse chromosome 13 that encodes the sodium-dependent amino acid transporter B(0)AT1.

17 and dissipate the electrical gradient of the sodium-dependent amino acid transporters in the proximal

18 melanoma cells, and ascorbate transport was sodium dependent and inhibited by ouabain.

19 to assess dapagliflozin's ability to inhibit sodium-dependent and facilitative glucose transport acti

20 ate homeostasis is mediated by high-affinity sodium-dependent and highly hydrophobic plasma membrane

21 The recovery was sodium-dependent and sensitive to Na(+)/H(+)-exchanger (

22 observed in untreated, diabetic rats of both sodium-dependent and sodium-independent glutamate uptake

23 ism and atomic structure in a broad range of sodium-dependent and sodium-independent secondary transp

24 was ATP- and temperature-dependent, but not sodium-dependent, and was inhibited by disulfonic stilbe

25 annel activity in regulating calcium- versus sodium-dependent AP firing.

26 rther strengthened by the comparable loss of sodium-dependent ascorbate transport activity upon the m

27 er mutations of His51 in hSVCT1, significant sodium-dependent ascorbate transport activity was only o

28 tients with correspondingly decreased apical sodium-dependent BA transporter (ASBT) gene expression.

29 al circulation is primarily dependent upon a sodium-dependent basolateral membrane transporter.

30 ystem substrate-binding protein A)/sbtA (for sodium-dependent bicarbonate transporter A): Delta4 muta

31 sted for their ability to inhibit the apical sodium dependent bile acid transport (ASBT)-mediated upt

32 nvestigate the factors controlling rat liver sodium-dependent bile acid cotransporter (ntcp) gene exp

33 Apical sodium-dependent bile acid transporter (ASBT) (SLC10A2),

34 Expression of the apical sodium-dependent bile acid transporter (ASBT) and the il

35 rlying the transport of bile acids by apical sodium-dependent bile acid transporter (Asbt) are not we

36 cular mechanisms of regulation of the apical sodium-dependent bile acid transporter (ASBT) by inflamm

37 g transmembrane (TM) segment 7 of the apical sodium-dependent bile acid transporter (ASBT) in substra

38 Ileal expression of the apical sodium-dependent bile acid transporter (ASBT) in the rat

39 The rat ileal sodium-dependent bile acid transporter (Asbt) is a polyt

40 The apical sodium-dependent bile acid transporter (Asbt) is respons

41 er membrane by the well characterized apical sodium-dependent bile acid transporter (Asbt) Slc10a2; h

42 The rat ileal apical sodium-dependent bile acid transporter (Asbt) transports

43 The apical sodium-dependent bile acid transporter (ASBT) transports

44 ), spanning residues V127-T149 of the apical sodium-dependent bile acid transporter (ASBT), a key mem

45 rmacological inhibition of the ileal, apical sodium-dependent bile acid transporter (ASBT), blocks pr

46 tokine-mediated repression of the rat apical sodium-dependent bile acid transporter (ASBT).

47 alts is mediated in large part by the apical sodium-dependent bile acid transporter (ASBT).

48 ng reabsorbed in the intestine by the apical sodium-dependent bile acid transporter (ASBT, also known

49 The apical sodium-dependent bile acid transporter (ASBT, SLC10A2) f

50 The apical sodium-dependent bile acid transporter (ASBT, SLC10A2) m

51 A1) expressed in hepatocytes, and the apical sodium-dependent bile acid transporter (ASBT; also known

52 ulation, is mediated primarily by the apical sodium-dependent bile acid transporter (ASBT=SLC10A2).

53 The human apical sodium-dependent bile acid transporter (hASBT) may serve

54 The membrane topology of the human apical sodium-dependent bile acid transporter (hASBT) remains u

55 in substrate interaction of the human apical sodium-dependent bile acid transporter (hASBT, SLC10A2)

56 Human apical sodium-dependent bile acid transporter (hASBT, SLC10A2)

57 The human apical sodium-dependent bile acid transporter (hASBT, SLC10A2)

58 sed mRNA and protein expression of the ileal sodium-dependent bile acid transporter (ISBT) in the int

59 The recent cloning of a human sodium-dependent bile acid transporter (NTCP) permits an

60 f bile salts via up-regulation of the apical sodium-dependent bile acid transporter and diminished ca

61 cholestasis-1 led to up-regulation of apical sodium-dependent bile acid transporter and down-regulati

62 Transport, apical sodium-dependent bile acid transporter and ileal lipid-b

63 tide 1, and the BA transport systems, apical sodium-dependent bile acid transporter and Na(+) -tauroc

64 al hepatobiliary transporters, including the sodium-dependent bile acid transporter gene, ntcp.

65 The apical sodium-dependent bile acid transporter is critical for i

66 Increased ileal apical sodium-dependent bile acid transporter messenger RNA (mR

67 veloped using 3.0 kilobase of the rat apical sodium-dependent bile acid transporter promoter to drive

68 co-2 cells, the activity of the human apical sodium-dependent bile acid transporter promoter was enha

69 s resulted in activation of the human apical sodium-dependent bile acid transporter promoter.

70 The ileal sodium-dependent bile acid transporter reclaims bile aci

71 The apical sodium-dependent bile acid transporter was up-regulated

72 ning extracellular loop (EL) 1 of the apical sodium-dependent bile acid transporter were determined v

73 , and transporters, such as the ileal apical sodium-dependent bile acid transporter, appear to affect

74 sion of bile acid signaling machinery apical sodium-dependent bile acid transporter, FXR, and small h

75 ess mature biliary markers, including apical sodium-dependent bile acid transporter, secretin recepto

76 Interactions among the apical sodium-dependent bile acid transporter, the farnesoid X

77 and messenger RNA and protein for the apical sodium-dependent bile acid transporter, the ileal bile a

78 ding involving multiple residues to describe sodium-dependent bile acid transporter-mediated bile aci

79 Mammalian sodium-dependent bile acid transporters (SBATs) responsi

80 deficient, were stably transfected with the sodium-dependent bile acid transporting polypeptide.

81 liver, tissues that also express the apical sodium-dependent bile acid uptake transporter ASBT (SLC1

82 Sodium-dependent bile salt transport was also measured i

83 By that time, the ileal sodium-dependent bile salt transporter messenger RNA and

84 , multidrug resistance protein 3, and apical sodium-dependent bile salt transporter.

85 Endotoxin inhibits hepatocellular sodium-dependent bile salt uptake by decreasing both exp

86 Apical sodium-dependent BS transporter inhibitors that reduce i

87 During hypoxia, blockade of neurons with sodium-dependent bursting properties abolishes respirato

88 This effect is sodium dependent but not potassium or chloride dependent

89 death of pneumococci specifically required a sodium-dependent calcium influx, as shown using calcium

90 2', 4'-dichlorobenzamil (DCB) inhibits the sodium-dependent calcium transporter (NCX1.1) much more

91 It is caused by mutations in the sodium-dependent carnitine cotransporter OCTN2.

92 dent vitamin C transporter (hSVCT1) mediates sodium-dependent cellular uptake of the essential micron

93 Thus sodium-dependent changes of calcium transport are indire

94 eby demonstrate that cells expressing mutant sodium-dependent citrate transporter have a complete los

95 SLC13A5 encodes a high affinity sodium-dependent citrate transporter, which is expressed

96 d by the AE family of Cl-/HCO3- exchangers), sodium-dependent Cl-/HCO3- exchange, and Na+:HCO3- cotra

97 ient to transmembrane solute transfer in the sodium-dependent co-transporter OCTN2.

98 r GDP, suggesting that they were mediated by sodium-dependent conductances in a G-protein-dependent m

99 C in its reduced form, ascorbate, through a sodium-dependent cotransporter.

100 The sodium-dependent current underlying these oscillations,

101 2 (engulfment and cell motility 2), SLC13A3 (sodium-dependent dicarboxylate transporter member 3), an

102 approach, we isolated a novel member of the sodium-dependent dicarboxylate/sulfate transporter calle

103 We further demonstrate sodium-dependent dimerization of PGRP-Ialpha in solution

104 es with largely diminished capacities of (1) sodium-dependent efflux of vacuolar protons and (2) elic

105 nd the nucleus where it colocalized with the sodium-dependent excitatory amino acid transporter, EAAT

106 corbic acid (AA), is taken up into cells via sodium-dependent facilitated transport.

107 st of these changes; IGF-I in vitro enhanced sodium-dependent glucose absorption but had no other eff

108 e GLUT inhibitor cytochalasin B, but not the sodium-dependent glucose cotransport inhibitor phloridzi

109 Inhibition of the sodium-dependent glucose cotransporter 1 (SGLT1) with ph

110 affinity, Na(+)-coupled, glucose transporter sodium-dependent glucose cotransporter 1, was evaluated

111 al reports of inhibitors directed toward the sodium-dependent glucose cotransporter 2 (SGLT2) as a me

112 osine phosphatase 1B) or by inhibiting renal sodium-dependent glucose cotransporter 2 (SGLT2).

113 The sodium-dependent glucose cotransporter SGLT1 undergoes a

114 Inhibitors of the sodium-dependent glucose cotransporters (SGLT) have appe

115 ChCoT, which was shown to be a member of the sodium-dependent glucose transporter family (SLGT), shou

116 t of restoration of normoglycemia by a novel sodium-dependent glucose transporter inhibitor (T-1095)

117 nal lumen into absorptive enterocytes by the sodium-dependent glucose transporter isoform 1 (SGLT1).

118 the expression of the known facilitative and sodium-dependent glucose transporter isoforms in six dif

119 whose expression, and that of GLUT 2 and the sodium-dependent glucose transporter protein 1 (SGLT1),

120 e Na/Pi cotransporter as mRNA levels for the sodium-dependent glucose transporter were not affected.

121 testinal expression of glucose transporters (sodium-dependent glucose transporter-1 and glucose trans

122 ose is absorbed from the small intestine via sodium-dependent glucose transporter-1 and glucose trans

123 cose absorption and expression of intestinal sodium-dependent glucose transporter-1, glucose transpor

124 critically ill patients, duodenal levels of sodium-dependent glucose transporter-1, glucose transpor

125 jejunum of cecal ligation and puncture mice sodium-dependent glucose transporter-1, glucose transpor

126 sure absolute (human) and relative levels of sodium-dependent glucose transporter-1, glucose transpor

127 rtance of another glucose import system, the sodium-dependent glucose transporters (SGLTs), in pancre

128 and it is removed from the synaptic cleft by sodium-dependent glutamate transport activity.

129 as associated with a progressive decrease in sodium-dependent glutamate transport capacity.

130 kinases C or A modulates the activity of the sodium-dependent glutamate transporter EAAC1.

131 hloride conductance with the properties of a sodium-dependent glutamate transporter has been describe

132 The sodium-dependent glutamate transporter, excitatory amino

133 em and is removed from the synaptic cleft by sodium-dependent glutamate transporters.

134 We found that OLs in culture are capable of sodium-dependent glutamate uptake with a K(m) of 10 +/-

135 and degradation of the EAAC1 transporter, a sodium-dependent glutamate/aspartate transport protein t

136 f different human solid tumor-derived cells, sodium-dependent glutamine transport was characterized i

137 In all cells, regardless of tissue origin, sodium-dependent glutamine transport was mediated almost

138 It is well documented that the sodium dependent, hemicholinium-3 sensitive, high affini

139 Presynaptic uptake of choline via the sodium-dependent, hemicholinium-3-sensitive choline tran

140 Several subtypes of sodium-dependent high affinity (SDHA) glutamate transpor

141 Using [3H]L-glutamate as the tracer, sodium-dependent high affinity glutamate transport was d

142 mutations in SLC5A7 encoding the presynaptic sodium-dependent high-affinity choline transporter 1 (CH

143 The sodium-dependent, high affinity serotonin [5-hydroxytryp

144 tosomes from pleural-pedal ganglia exhibited sodium-dependent, high-affinity Glu transport.

145 we investigated some mechanisms involved in sodium-dependent hypertension of rats exposed to chronic

146 ly validated, showing that proton efflux was sodium-dependent, inhibited by amiloride analogs, and ac

147 Transport was sodium-dependent, inhibited by excess ascorbate, and sim

148 s associated with expression of the type III sodium-dependent inorganic phosphate (Pi) cotransporter

149 ar in sequence to a mammalian brain-specific sodium-dependent inorganic phosphate cotransporter I (BN

150 A CreER(T2) cassette was knocked into the sodium-dependent inorganic phosphate transporter SLC34a1

151 a virus, this receptor is the human type III sodium-dependent inorganic phosphate transporter, SLC20A

152 Mfsd2a is a newly described sodium-dependent lysophosphatidylcholine (LPC) symporter

153 atty acids from the circulation is through a sodium-dependent lysophosphatidylcholine (LPC) transport

154 2A (MFSD2A) was recently characterized as a sodium-dependent lysophosphatidylcholine transporter exp

155 perfused IBDUs absorbed TCA in a saturable, sodium-dependent manner; in addition, TCA absorption was

156 ocycline through a saturable, concentrative, sodium-dependent mechanism with a Michaelis constant (K(

157 nsports ascorbic acid (ASC) inward through a sodium-dependent mechanism.

158 lytoxin by regulating JNK and SEK1 through a sodium-dependent mechanism.

159 There are two sodium-dependent membrane transporters encoded by SLC23A

160 The nucleotide sequence of the cDNA (sodium-dependent multivitamin transporter (SMVT)) predic

161 an cells accumulate biotin by using both the sodium-dependent multivitamin transporter and monocarbox

162 Three transporters, SMVT (sodium-dependent multivitamin transporter for biotin and

163 ched in L-fucose, a competitive inhibitor of sodium-dependent myo-inositol transport.

164 Expression of the M813 receptor murine sodium-dependent myo-inositol transporter 1 (mSMIT1) all

165 sitol, an osmolyte transported into cells by sodium-dependent myo-inositol transporters (SMITs).

166 The sodium-dependent NADH dehydrogenase (Na(+)-NQR) is a key

167 The sodium-dependent NADH dehydrogenase (Na(+)-NQR) is the m

168 T-1 is coded by snf-11 gene, a member of the sodium-dependent neurotransmitter symporter gene family

169 Sodium-dependent neurotransmitter transporters participa

170 and the norepinephrine transporter (NET) are sodium-dependent neurotransmitter transporters responsib

171 thermophilum encodes a protein homologous to sodium-dependent neurotransmitter transporters.

172 System A, the sodium-dependent neutral amino acid transport activity,

173 rter 1 (GLUT-1), taurine transporter (TAUT), sodium-dependent neutral amino acid transporter (SNAT),

174 An ATF4 target gene, SNAT2 (system A sodium-dependent neutral amino acid transporter 2), cont

175 tium formation by interacting with the human sodium-dependent neutral amino acid transporter type 2 (

176 The sodium-dependent neutral amino acid transporter type 2 (

177 these viruses and identified it as the human sodium-dependent neutral amino acid transporter type 2 (

178 lating arsenite-induced ER stress, including sodium-dependent neutral amino acid transporter, SNAT2.

179 g (Kir) potassium channels and activation of sodium-dependent, nonselective cationic channels (NSCCs)

180 the reduction in membrane expression of the sodium-dependent P(i) co-transporters, NPT2a and NPT2c,

181 and for increased expression of the type III sodium-dependent P(i) cotransporter Pit-1 and certain os

182 H 3T3 cells resulted in a marked increase in sodium-dependent P(i) uptake.

183 The sodium-dependent phosphate (Na/P(i)) transporters NaPi-2

184 triction is associated with up-regulation of sodium-dependent phosphate (Na/Pi) cotransport by renal

185 orbol 12-myristate 13-acetate (PMA) enhanced sodium-dependent phosphate (Na/Pi) uptake.

186 than the adenylyl cyclase, pathway mediates sodium-dependent phosphate co-transport in LLC-PK1 cells

187 , where both PTH-stimulated PLC activity and sodium-dependent phosphate co-transport were essentially

188 hosphate levels and seem to be mediated by a sodium-dependent phosphate co-transporter, Pit-1 (Glvr-1

189 evated phosphate on HSMCs were mediated by a sodium-dependent phosphate cotransporter (NPC), as indic

190 parathyroid hormone receptor (PTHR), type II sodium-dependent phosphate cotransporter (Npt2a), and be

191 pyrophosphatase/phosphodiesterase 1 enzyme, sodium-dependent phosphate cotransporter 1 (encoded by t

192 ranscellular mechanism involving the type II sodium-dependent phosphate cotransporter NPT2b (SLC34a2)

193 e have investigated the role of the type III sodium-dependent phosphate cotransporter, Pit-1, in SMC

194 sodium-glucose cotransporter 2, and type IIa sodium-dependent phosphate cotransporter, suggesting api

195 ion and mineralization via the activity of a sodium-dependent phosphate cotransporter.

196 phosphate-induced calcification, implicating sodium-dependent phosphate cotransporters in this proces

197 However, FeLV-B and A-MLV use different sodium-dependent phosphate symporters, Pit1 and Pit2, re

198 decreased Pit-1 mRNA and protein levels and sodium-dependent phosphate transport activity compared w

199 a resulted in a 36.0 +/- 6.3% higher rate of sodium-dependent phosphate transport and a significant i

200 s a phosphaturic factor that suppresses both sodium-dependent phosphate transport and production of 1

201 ibroblast growth factor-23 (FGF-23) inhibits sodium-dependent phosphate transport in brush border mem

202 Basal sodium-dependent phosphate transport was lower in cells

203 thelial cells, sFRP-4 specifically inhibited sodium-dependent phosphate transport.

204 le cells, neither PTH nor dopamine inhibited sodium-dependent phosphate transport.

205 The sodium-dependent phosphate transporter (HOST5/SLC34A2) e

206 protein (LP), and HOST5 codes for a type II sodium-dependent phosphate transporter (SLC34A2).

207 Whereas KoRV-A uses the sodium-dependent phosphate transporter 1 (PiT1) as a rec

208 domain decreased the binding affinity to the sodium-dependent phosphate transporter 2a (Npt2a) as com

209 ing of cellular targets including the Npt2a (sodium-dependent phosphate transporter 2a).

210 Ram-1 acts as both a sodium-dependent phosphate transporter and a receptor fo

211 e leukemia virus (A-MuLV) utilizes the Pit-2 sodium-dependent phosphate transporter as a cell surface

212 ne leukemia virus (A-MuLV) utilizes the PiT2 sodium-dependent phosphate transporter as its cell surfa

213 and 1,25(OH)(2)D, reduced expression of the sodium-dependent phosphate transporter NPT2a in the prox

214 acilitating the internalization of the major sodium-dependent phosphate transporter, Npt2a.

215 mal tubule of the kidney by retrieval of the sodium-dependent phosphate transporters (Npt2a and Npt2c

216 etrovirus serve normal cellular functions as sodium-dependent phosphate transporters (Pit-1 and Pit-2

217 the renal tubule by the action of the apical sodium-dependent phosphate transporters, NaPi-IIa/NaPi-I

218 cells, on the other hand, had lower rates of sodium-dependent phosphate uptake and low phosphate medi

219 Mammalian sodium-dependent Pi cotransporters have been grouped int

220 Interestingly, in the absence of sodium-dependent Pi transport activity, the PiT1-PiT2 he

221 diet-induced hypophosphatemia as well as in sodium-dependent Pi transporter solute carrier family 34

222 igated the involvement of the high-affinity, sodium-dependent Pi transporters PiT1 and PiT2 in mediat

223 lpha, induced PiT-1 expression and increased sodium-dependent Pi uptake by >40% in chondrocytes.

224 ase progression through PiT-1 expression and sodium-dependent Pi uptake mediated by CXCR1 signaling.

225 modulating chondrocyte PiT-1 expression and sodium-dependent Pi uptake, and to assess differential r

226 NTT4 was previously thought to function as a sodium-dependent plasma membrane transporter, recent stu

227 ises from an intrinsic cellular mechanism: a sodium-dependent potassium conductance that causes prolo

228 es the direct activation of an electrogenic, sodium-dependent presynaptic transporter, which supplies

229 ockouts identified 15% and 40% reductions in sodium-dependent proline and leucine transport, respecti

230 Sodium-dependent proton secretion in NHE3(-/-) mice was

231 Here we examined apical membrane PCT sodium-dependent proton secretion of wild-type (NHE3(+/+

232 The residual sodium-dependent proton secretion was inhibited by 100 m

233 Luminal sodium-dependent proton secretion was the same in NHE3(-

234 ) and wild-type mice had comparable rates of sodium-dependent proton secretion.

235 to isolate a 2653 bp cDNA encoding the mouse sodium-dependent, purine nucleoside selective, concentra

236 ntity to the previously cloned rat and human sodium-dependent, purine nucleoside selective, nucleosid

237 demonstrated that NHE3 activity, measured as sodium-dependent recovery of the intracellular pH after

238 tegral membrane proteins responsible for the sodium-dependent reuptake of small-molecule neurotransmi

239 o these facilitated urea transporters, three sodium-dependent, secondary active urea transport mechan

240 more potent and less reversible at blocking sodium-dependent short-circuit current than amiloride.

241 ies demonstrated that palytoxin stimulates a sodium-dependent signaling pathway that activates the c-

242 ults demonstrate that palytoxin stimulates a sodium-dependent signaling pathway that activates the SE

243 ls functionally and physically interact with sodium-dependent solute transporters, including myo-inos

244 ses in PIP2, SMIT1, and likely other related sodium-dependent solute transporters, regulates KCNQ cha

245 sometimes abolished by TTX, suggesting that sodium-dependent spikes play an important role in the tr

246 tions of tetrodotoxin citrate (TTX) to block sodium-dependent spiking; TTX+N-methyl-D-aspartic acid (

247 oposed S2 binding site, respectively, retain sodium-dependent substrate binding in the S1 site simila

248 talline LeuT samples and identify one set of sodium-dependent substrate-specific chemical shifts.

249 The endogenous sodium-dependent succinate transport in Caco-2 cells is

250 ice exhibited increased renal and intestinal sodium-dependent succinate uptake, as well as urinary hy

251 the role of a triad of aromatic residues in sodium-dependent sugar cotransporters (SGLTs).

252 y metabolism that seems to be coupled to the sodium-dependent synthesis of ATP.

253 of the gamma-glutamyl cycle, stimulates the sodium-dependent systems A and Bo,+ by 70 and 20%, respe

254 ene expression of the hepatocyte basolateral sodium-dependent taurocholate cotransporter (Ntcp) to de

255 for the role of the farsenoid X receptor and sodium-dependent taurocholate cotransporting polypeptide

256 nion transporting polypeptide (OATP) 1B1 and sodium-dependent taurocholate cotransporting polypeptide

257 ing of complementary DNAs for the sinusoidal sodium-dependent taurocholate cotransporting polypeptide

258 ition 120, epitope mutants displayed active, sodium-dependent taurocholate uptake.

259 holate cotransporter and markedly diminished sodium-dependent taurocholate uptake.

260 Furthermore, sodium-dependent taurocholic acid uptake was inhibited b

261 y Slc10a1(-/-) hepatocytes showed absence of sodium-dependent taurocholic acid uptake, whereas sodium

262 The voltage dependence of the sodium-dependent transient currents of the Phe-294 mutan

263 D451E exhibited robust sodium-dependent transient currents with a voltage-depen

264 Sodium-dependent transmitter exchange and a transporter-

265 In basolateral plasma membrane vesicles, sodium-dependent transport for bile acids was reduced by

266 Sodium-dependent transport into astrocytes is critical f

267 We find: (a) sodium-dependent transport of ascorbate in mixed neurona

268 l epoxide hydrolase (mEH) is able to mediate sodium-dependent transport of bile acids such as tauroch

269 The transfected MDCK cells also exhibited sodium-dependent transport of cholate at levels 150% of

270 SDCT2 expressed in Xenopus oocytes mediated sodium-dependent transport of di- and tricarboxylates wi

271 enopus oocytes, SDCT1 mediated electrogenic, sodium-dependent transport of most Krebs cycle intermedi

272 ransfected MDCK cells and is able to mediate sodium-dependent transport of taurocholate and cholate.

273 Sodium-dependent transport of taurocholate was shown to

274 ikely presence of a NBMPR-insensitive and/or sodium-dependent transport system of the N2 (cit) type a

275 across the brush border membrane (BBM) via a sodium-dependent transporter, SGLT, and exit across the

276 omologies to an amino acid transporter and a sodium-dependent transporter.

277 quisqualate reach nuclear receptors via both sodium-dependent transporters and cystine glutamate exch

278 Neuronal and glial sodium-dependent transporters are crucial for the contro

279 Sodium-dependent transporters clear extracellular glutam

280 nic nitrogen and phosphorus sources and more sodium-dependent transporters than a model freshwater cy

281 ntified apical targeting motifs in two other sodium-dependent transporters, and we suggest this conse

282 y regulation of transport through at least 2 sodium-dependent transporters: Slc23a1 and Slc23a2 (also

283 Sodium-dependent uptake of (3)H-taurocholate in renal br

284 transport function was assessed by examining sodium-dependent uptake of [3H]-taurocholate (TC) into b

285 Sodium-dependent uptake of bile acids across the hepatic

286 nobiotic metabolism as well as mediating the sodium-dependent uptake of bile acids into the liver, wh

287 ent an important class of proteins mediating sodium-dependent uptake of neurotransmitters from the ex

288 Sodium-dependent uptake of phosphate (Pi) in renal BBMV

289 Polymorphisms in the genes encoding sodium-dependent vitamin C transport proteins are strong

290 The human sodium-dependent vitamin C transporter (hSVCT1) mediates

291 as well as the expression of transcripts for sodium-dependent vitamin C transporter (SVCT)-1 and SVCT

292 In embryos lacking the sodium-dependent vitamin C transporter 2 (SVCT2), very l

293 were shown to have two variants of the human sodium-dependent vitamin C transporter, hSVCT1; one is a

294 n UVA-irradiated lenses from human IDO/human sodium-dependent vitamin C transporter-2 mice, which con

295 ry active transport of ascorbate through the sodium-dependent vitamin C transporters SVCT1 and SVCT2

296 Sodium-dependent vitamin C transporters, SVCT1 and SVCT2

297 Human sodium-dependent vitamin C transporters, SVCT1 and SVCT2

298 Both these high affinity systems were sodium dependent with a Hill coefficient of about 2.0 in