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1 sed expression of thyroglobulin (Tg) and the sodium/iodide symporter).
2 virus type I thymidine kinase and the human sodium-iodide symporter.
3 yroid-stimulating hormone in stimulating the sodium-iodide symporter.
4 as attributed to decreased expression of the sodium-iodide symporter.
5 egulated expression of thyroglobulin and the sodium/iodide symporter.
8 A levels for thyroglobulin, thyroperoxidase, sodium iodide symporter, and deiodinase type 2, and deio
9 2, sodium hydrogen exchanger 3, aquaporin 7, sodium iodide symporter, and hydrogen potassium adenosin
10 HR expression was required for expression of sodium-iodide symporter but was not required for thyrogl
12 this observation was the lack of detectable sodium-iodide symporter expression in TSHR-KO thyroid gl
18 differentiation, including the loss of human sodium iodide symporter (hNIS) expression, radioactive i
19 carcinoembryonic antigen (CEA) or the human sodium iodide symporter (hNIS) for oncolytic potential i
20 ionuclide accumulation mediated by the human sodium iodide symporter (hNIS) gene in conjunction with
21 ntification of cardiac gene expression after sodium iodide symporter (hNIS) gene transfer in cardiac
22 ic vaccinia virus, GLV-1h153, carrying human sodium iodide symporter (hNIS), in combination with radi
24 man norepinephrine transporter (hNET), human sodium-iodide symporter (hNIS), a human deoxycytidine ki
27 Here we show that a specialized form of the sodium/iodide symporter in the mammary gland mediates ac
28 nal obtained by firefly luciferase and human sodium-iodide symporter labeling of cardiosphere-derived
29 hese results indicate that the mammary gland sodium/iodide symporter may be an essential breast cance
31 virus (VSV)-murine interferon beta (IFNbeta)-sodium iodide symporter (NIS) (VSV-mIFNbeta-NIS) oncolyt
32 second strategy involves transduction of the sodium iodide symporter (NIS) and free radionuclide ther
33 cells with retroviral vectors containing the sodium iodide symporter (NIS) and thyroperoxidase (TPO)
37 cancer cells to express functionally active sodium iodide symporter (NIS) by targeted NIS gene trans
38 ast cancer cells stably expressing the human sodium iodide symporter (NIS) fused to a red fluorescent
40 its binding factor PBF repress expression of sodium iodide symporter (NIS) messenger RNA (mRNA), and
42 cancer cells to express functionally active sodium iodide symporter (NIS) would enable those cells t
43 din-Darby canine kidney cells expressing the sodium iodide symporter (NIS), pendrin, or NIS and pendr
44 ne levels by up-regulating expression of the sodium iodide symporter (NIS), thyroid peroxidase (TPO),
48 t tissue and some breast cancers express the sodium/iodide symporter (NIS) and concentrate iodide.
51 Perchlorate, thiocyanate, and nitrate are sodium/iodide symporter (NIS) inhibitors that block iodi
56 ds to the 3'-untranslated region of PAX8 and sodium/iodide symporter (NIS), leading to impaired prote
57 wn that RET/PTC1 decreases expression of the sodium/iodide symporter (NIS), the molecule that mediate
59 functional role in tumor progression of the sodium/iodide symporter (NIS; aka SLC5A5), which is upre
60 isease, based on decreased expression of the sodium iodide symporter SLC5A5 (NIS), diminished membran
61 Pax-8, thereby suppressing expression of the sodium iodide symporter, thyroid peroxidase, TG, and thy
62 thyroid-specific target genes thyroglobulin, sodium iodide symporter, thyroperoxidase, and thyroid-st
63 the hormonal regulation of the mammary gland sodium/iodide symporter, we demonstrate by scintigraphy
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