ライフサイエンスコーパス: soft drink

1 d with a distilled beverage and a carbonated soft drink.

2 e and after the ingestion of a candy bar and soft drink.

3 , so has their consumption of fast foods and soft drinks.

4 , as well as in commercial orange juices and soft drinks.

5 published methods for the analysis of BVO in soft drinks.

6 tion, brushing frequency, and consumption of soft drinks.

7 P) and di-(2-ethylhexyl) phthalate (DEHP) in soft drinks.

8 ation of SY and Tz in commercially available soft drinks.

9 clude lower consumption of high-GI foods and soft drinks.

10 udies in humans have examined the effects of soft drinks.

11 .62) compared with those who rarely consumed soft drinks.

12 It is also used worldwide to produce soft drinks.

13 (0.77,1.31; P for trend = 0.76) for regular soft drinks.

14 is present in amounts comparable to those in soft drinks.

15 sugar-sweetened, and artificially sweetened soft drink and juice and nectar consumption) and pancrea

16 g insulin resistance, and dietary intakes of soft drinks and alcohol and was positively associated wi

17 affeinated sugar- and artificially sweetened soft drinks and early menarche (defined as menarche age

18 kes in children who consume large amounts of soft drinks and fast foods are not compensated for by in

19 hough food and beverage trends were similar, soft drinks and fruit drinks provided the most HFCS (158

20 Sugar-sweetened beverages like soft drinks and fruit punches contain large amounts of r

21 ction and preconcentration of Cd and Pb from soft drinks and further determination by GF AAS was deve

22 The authors examined the association between soft drinks and juice and the risk of type 2 diabetes am

23 Relatively frequent intake of soft drinks and juice is associated with an increased ri

24 f caffeine and Class IV caramel in cola-type soft drinks and of caffeine, Class III caramel and ribof

25 further applied for analyzing six commercial soft drinks and one was found containing 453.67 ng/mL of

26 Soft drinks and other sweetened beverages may contribute

27 ontrols and were even more likely to consume soft drinks and tea [odds ratio (OR) = 2.01 95% confiden

28 procedure for the analysis of energy drink, soft drink, and chocolate milk samples was demonstrated.

29 Consumption of milk, coffee, tea, soft drinks, and alcohol was based on food frequency dat

30 meat intake, seafood intake, sugar sweetened soft drinks, and consumption of foods high in fructose.

31 tion of sugar-containing drinks (e.g., cola, soft drinks, and energy drinks) and addictive substances

32 ximately 64%), tea ( approximately 16%), and soft drinks ( approximately 18%) predominant sources; en

33 coffee, instant coffee, tea, and caffeinated soft drinks, as well as caffeine intoxication, tolerance

34 quality control of the synthetic colours in soft drinks, as well as to determine whether the levels

35 programme (NDNS RP) from 2008-12 and British Soft Drinks Association annual reports to calculate suga

36 orld application, identification of enhanced soft drinks based on their Ca(2+), Mg(2+), and Zn(2+) ca

37 ication of brominated vegetable oil (BVO) in soft drinks based upon liquid chromatography-electrospra

38 kcal (from 1.0 to 1.6 oz [28.4 to 45.4 g]), soft drinks by 49 kcal (13.1 to 19.9 fl oz [387.4 to 588

39 l, Cu, Cr, Fe and Ni in Brazilian carbonated soft drinks by electrothermal atomic absorption spectrom

40 suggest that high consumption of carbonated soft drinks by young children is a risk indicator for de

41 ntakes of meat products, refined grains, and soft drinks (caloric and noncaloric) were found to be co

42 ctose intake, primarily from added sugars in soft drinks, can induce fatty liver in animals and is ep

43 bowl of milk or bubbles at the surface of a soft drink, clump together as a result of capillary attr

44 ing those years, and now soft drinks provide soft drink consumers 188 kcal/d beyond the energy intake

45 a meta-analysis of the relationship between soft drink consumption and cardiometabolic risk, there w

46 gressed each BMD measure on the frequency of soft drink consumption for men and women after adjustmen

47 ng women who increased their sugar-sweetened soft drink consumption from 1 or fewer drinks per week t

48 Children's soft drink consumption has also increased during those y

49 of metabolic syndrome and its components to soft drink consumption in participants in the Framingham

50 In middle-aged adults, soft drink consumption is associated with a higher preva

51 Soft drink consumption may have adverse effects on bone

52 About 13% of children had a high carbonated soft drink consumption pattern; they also had a signific

53 For each soft drink serving per day, soft drink consumption was significantly associated with

54 : 1.03; 95% CI: 0.99, 1.07), sugar-sweetened soft-drink consumption (HR per 100 g/d: 1.02; 95% CI: 0.

55 Total soft-drink consumption (HR per 100 g/d: 1.03; 95% CI: 0.

56 CI: 0.97, 1.08), and artificially sweetened soft-drink consumption (HR per 100 g/d: 1.04; 95% CI: 0.

57 -based intervention found significantly less soft-drink consumption and prevalence of obese and overw

58 Low-strength evidence showed that reducing soft-drink consumption decreased recurrent symptomatic s

59 Soft-drink consumption does not seem to be associated wi

60 Associations with total soft-drink consumption were adjusted for juice and necta

61 rospective research on whether the intake of soft drinks containing caffeine, a modulator of the fema

62 pushing away 90% of alcohol cues and 10% of soft drink cues, whereas this ratio was 50/50 in the sha

63 rsons (21.6%) [corrected] consuming > or = 1 soft drink/day [corrected] Consumption of > or = 1 soft

64 take was 74 g/d, corresponding to 2.5 sugary soft drinks each day.

65 among overweight than nonoverweight youths; soft drink energy contribution was higher among overweig

66 ives and profits; posters/advertisements for soft drinks, fast food, or candy; use of food coupons as

67 added-sugars intakes derived from carbonated soft drinks fell 26% between 1997 and 2011 (from 23 to 1

68 d were randomised to drink either lager or a soft drink from either a curved or straight-sided glass,

69 specific food items (salty snacks, desserts, soft drinks, fruit drinks, french fries, hamburgers, che

70 Testing with real samples (soft drinks, fruit juices) gave good correlation of the

71 an experimental design with beverage (lager, soft drink), glass (straight, curved) and quantity (6 fl

72 Consumption of soft drinks has been linked to obesity in children and a

73 on patterns were identified: high carbonated soft drinks, high juice, high milk, and high water.

74 e hypothesis that consumption of caffeinated soft drinks in childhood is associated with higher risk

75 verages and is the sole caloric sweetener in soft drinks in the United States.

76 creased effects of the fast food and bottled soft drink industries on this nutrition shift is availab

77 responses by industry to the UK Government's soft drinks industry levy have been seen, but the govern

78 rld, exporting much of its production to the soft drinks industry.

79 igh WC may attenuate the association between soft drink intake and BW gain.

80 Associations between soft drink intake and the annual change (Delta) in body

81 bined may strengthen the association between soft drink intake and WC gain.

82 , which suggests that the use of low-calorie soft drinks is a marker for more general dietary behavio

83 Intake of cola, but not of other carbonated soft drinks, is associated with low BMD in women.

84 The health impact of the soft drinks levy is dependent on its implementation by i

85 INTERPRETATION: The health impact of the soft drinks levy is dependent on its implementation by i

86 ed beverages (SSBs), particularly carbonated soft drinks, may be a key contributor to the epidemic of

87 y pattern score, which consisted of alcohol, soft drinks, meat, coffee, and tea, was positively assoc

88 th those of isocaloric milk and a noncaloric soft drink on changes in total fat mass and ectopic fat

89 the effect of high consumption of carbonated soft drinks on caries in the primary dentition.

90 tivariable adjustment, children who consumed soft drinks once or more per day had significantly narro

91 (strawberry and custard) powder samples and soft drink (orange) samples with satisfactory results.

92 than did those who never or rarely consumed soft drinks (P-trend = 0.03).

93 -sectionally, individuals consuming > or = 1 soft drink per day had a higher prevalence of metabolic

94 rink/day [corrected] Consumption of > or = 1 soft drink per day was associated with increased odds of

95 s, women consuming 1 or more sugar-sweetened soft drinks per day had a relative risk [RR] of type 2 d

96 s modified by 5-year weight gain for > or =2 soft drinks per week among those who gained > or =3 kg (

97 confounders, participants consuming > or =2 soft drinks per week had a relative risk of type 2 diabe

98 gain, except for consumption of low-calorie soft drinks (positive association, P = 0.002) and white

99 Moreover, beverages of 100% fruit juices and soft drinks prepared with mineral water (free of HAAs) d

100 ed on commercial carbonated orange and grape soft drinks produced in Ceara State, Brazil.

101 s also increased during those years, and now soft drinks provide soft drink consumers 188 kcal/d beyo

102 ributed 20-24% of energy across all ages and soft drinks provided 8% of energy in adolescents.

103 d by high intake of sugar-sweetened and diet soft drinks, refined grains, red and processed meat, and

104 .25); nor was consumption of sugar-sweetened soft drinks (RR for 1 serving/d increment: 1.15; 95% CI:

105 For each soft drink serving per day, soft drink consumption was s

106 nly with the GRSWC (per risk allele for each soft drink serving per day: -0.06 kg/y; 95% CI: -0.10, -

107 MI and GRSComplete [per risk allele for each soft drink serving per day: 0.05 cm/y (95% CI: 0.02, 0.0

108 rometric determination of glucose content in soft drinks showing good repeatability (DPR=1.72%, n=50)

109 The consumption of sucrose-sweetened soft drinks (SSSDs) has been associated with obesity, th

110 confirm the composition of food colouring in soft drinks stated on label.

111 nd Brilliant Blue (E133) were extracted from soft drinks using C18 SPE and identified by thin layer c

112 The concentration of food colouring in soft drink was determined by ion-pair high performance l

113 Consumption of artificially sweetened soft drinks was also positively associated with risk of

114 body fat, greater consumption of caffeinated soft drinks was associated with a higher risk of early m

115 Greater consumption of carbohydrates and soft drinks was associated with retinal arteriolar narro

116 The positive association with low-calorie soft drinks was no longer present after adjustment for d

117 on of black tea, green tea, fruit juices, or soft drinks was not associated with risk of cirrhosis de

118 Consumption of noncaffeinated soft drinks was not significantly associated with early

119 on of caffeinated and artificially sweetened soft drinks was positively associated with risk of early

120 taining caffeine such as tea and caffeinated soft drinks were not associated with stroke.

121 Diet soft drinks were not associated with the risk of gout (P

122 us aspects of obesity and the consumption of soft drinks, which are a major part of sugar-sweetened b

123 pplied for the determination of Cd and Pb in soft drinks with different brands and flavours.

124 ed by determination of the dyes in processed soft drinks with satisfactory results (recovery>95% and

125 ully tested on strawberry jam, low-fat milk, soft drink, yogurt and a commercial mixture of steviol g