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1 crotizing myositis, bacteremia, and skin and soft tissue infection.
2 y patterns was examined in a murine model of soft tissue infection.
3 cts of the care of patients with necrotizing soft tissue infection.
4 tants highly attenuated in a murine model of soft tissue infection.
5 ghly attenuated in the murine ulcer model of soft tissue infection.
6 anscriptome of GAS during experimental mouse soft tissue infection.
7 he pathogenesis of streptococcal necrotising soft tissue infection.
8 ted to have a minimal potential for invasive soft tissue infection.
9 lence factor for invasive disease, including soft tissue infection.
10 y and is mainly associated with subcutaneous soft tissue infection.
11 uronic acid capsule as a virulence factor in soft tissue infection.
12 sociated with enhanced virulence in skin and soft tissue infection.
13 lococcus aureus is a major cause of skin and soft tissue infection.
14 ME mutants was assessed in a murine model of soft tissue infection.
15 a less invasive form of superficial skin and soft tissue infection.
16 Haemophilus influenzae is a rare cause of soft tissue infection.
17 d pathogenesis in a murine model of skin and soft tissue infection.
18 cause of community-onset S. aureus skin and soft-tissue infection.
19 sons with community-onset S. aureus skin and soft-tissue infection.
20 luenced by covR also was identified in mouse soft-tissue infection.
21 protein in a murine model of human invasive soft-tissue infection.
22 fasciitis is a rare, but potentially fatal, soft-tissue infection.
23 elative to the odds of PVL-positive skin and soft-tissue infection.
24 tial therapeutic in SAg-mediated necrotizing soft-tissue infection.
25 l infection in a murine model of necrotizing soft-tissue infection.
26 s a role in pathogenesis in a mouse model of soft-tissue infection.
27 ossible, probable, or definite cellulitis or soft tissue infections.
28 hogen causing pulmonary disease and skin and soft tissue infections.
29 using an ongoing pandemic of mostly skin and soft tissue infections.
30 t of which (53%) were pneumonia and skin and soft tissue infections.
31 re pandemic consisting primarily of skin and soft tissue infections.
32 n the differential diagnosis of gingival and soft tissue infections.
33 014 primarily reflected declines in skin and soft tissue infections.
34 throat) and with invasive or "flesh-eating" soft tissue infections.
35 ysteine protease contributes to virulence in soft tissue infections.
36 s aureus (MRSA) is a major cause of skin and soft tissue infections.
37 s invasive disease, most frequently skin and soft tissue infections.
38 SA) causes invasive, drug-resistant skin and soft tissue infections.
39 fections and syndromes-most notably skin and soft tissue infections.
40 ng invasive disease, pneumonia, and skin and soft tissue infections.
41 empirical treatment guidelines for skin and soft-tissue infections.
42 or patients presenting with serious skin and soft-tissue infections.
43 me a predominant cause of childhood skin and soft-tissue infections.
44 eus should be considered a cause of skin and soft-tissue infections.
45 nes, a common agent of pharyngeal, skin, and soft-tissue infections.
46 nes, a common agent of pharyngeal, skin, and soft-tissue infections.
47 tates and has caused an epidemic of skin and soft-tissue infections.
48 a-analysis for Staphylococcus aureus skin or soft-tissue infections.
49 l, predominantly for abdominal, urinary, and soft-tissue infections.
50 aureus, a common cause for serious skin and soft tissues infections.
52 nd against isolates associated with skin and soft tissue infection (68%, compared to 85% susceptible
54 awareness of fungi as a cause of necrotizing soft-tissue infections after a natural disaster is warra
56 ed USA300-0114, is a major cause of skin and soft tissue infections among persons in community settin
57 e most common identifiable cause of skin and soft-tissue infections among patients presenting to emer
59 Cellulitis is a commonly occurring skin and soft tissue infection and one of the most frequently see
60 for virulence in a murine model of invasive soft tissue infection and this attenuation is complement
61 frequent manifestation of S. aureus skin and soft tissue infections and are formed, in part, to conta
62 e opportunist pathogens that cause blood and soft tissue infections and are often resistant to antimi
64 We describe an HIV-infected patient with 24 soft tissue infections and multiple colonization events.
65 ble for severe, rapidly progressive skin and soft tissue infections and native valve endocarditis.
66 le for a wide range of infections, including soft tissue infections and potentially fatal bacteremias
67 ection and plays an important role in severe soft tissue infections and systemic dissemination of GAS
69 A (SPEA), in the pathogenesis of necrotizing soft-tissue infection and toxic shock syndrome resulting
70 es are consistently associated with skin and soft-tissue infections and are comparatively rare in inv
71 nderstanding of the epidemiology of skin and soft-tissue infections and osteoarticular infections is
72 cation are changing the approach to skin and soft-tissue infections and osteoarticular infections.
74 children, NTM cause lymphadenitis, skin and soft tissue infections, and occasionally also lung disea
75 aureus is the most common cause of skin and soft tissue infections, and rapidly emerging antibiotic-
76 roup of patients presenting with necrotizing soft tissue infections, and, based on this analysis, pro
77 mutants were attenuated in a murine model of soft-tissue infection, and analysis of gene expression i
78 ezolid in the setting of pneumonia, skin and soft-tissue infections, and infections due to vancomycin
80 sful management of patients with necrotizing soft tissue infection are early recognition and complete
81 (MyD88) deficiency, staphylococcal skin and soft tissue infections are a leading and potentially lif
85 injection drug use who were readmitted with soft tissue infections at new sites (16.8% of readmissio
86 ism to explain the development of severe GAS soft-tissue infections at the sites of prior minor muscl
87 nt of streptococcal sore throat and invasive soft-tissue infections, attaches to human pharyngeal or
88 ribe an outbreak of Nocardia cyriacigeorgica soft-tissue infections attributable to unlicensed cosmet
89 aureus (CA-MRSA), a major cause of skin and soft tissue infections, became successful so quickly, ov
90 trains are strongly associated with skin and soft-tissue infections, but are comparatively rare in pn
92 We present the first case of human skin and soft tissue infection caused by this species in a patien
93 increasing in frequency is serious bacterial soft tissue infections caused by group A streptococci.
95 idence of infections, such as RTIs; skin and soft-tissue infections; chronic comorbid conditions, inc
96 y is indicated for the treatment of skin and soft-tissue infections, clinicians should consider obtai
99 n immunocompromised patient with an invasive soft tissue infection due to Scedosporium apiospermum wa
100 tered three patients with severe necrotising soft tissue infections due to beta-haemolytic group G st
105 nt of streptococcal sore throat and invasive soft tissue infections, evades internalization and intra
106 e differential diagnosis of chronic skin and soft tissue infections following bee venom acupuncture.
109 ngal pathogen, causing a variety of skin and soft tissue infections in healthy people and more virule
118 rgan transplant recipients, localized fungal soft tissue infection is infrequent, with only 35 cases
119 ezolid has approved indications for skin and soft tissue infections; lower respiratory tract infectio
120 r as adjuncts to antibiotic therapy, for GAS soft tissue infection may contribute to worse outcomes.
122 and 24 h after bacterial challenge in a rat soft tissue infection model resulted in a significant de
123 creased lesion size and severity in a murine soft tissue infection model when compared with parental
124 d with HlaH35L and challenged via a skin and soft tissue infection model, HlaH35L immunization led to
127 B307.27 was significantly decreased in a rat soft-tissue infection model (P<.001) and a rat pneumonia
130 drug-resistant Acinetobacter baumannii in a soft-tissue infection model through light-triggered NO d
131 were subsequently used for challenge in the soft-tissue infection model to determine if the disrupte
134 gnificantly attenuated in the bacteremia and soft tissue infection models, and the mutant strain lack
135 ed the hypothesis that the rat pneumonia and soft-tissue infection models that our laboratory had pre
136 ically significant, with bacteremia (n = 5), soft tissue infections (n = 3) osteomyelitis (n = 2), in
137 in (PVL) are associated with severe skin and soft tissue infections, necrotizing pneumonia, and eye i
139 ortunistic pathogen associated with skin and soft tissue infections, nosocomial pneumonia and sepsis.
145 ervous System Infections, Ocular Infections, Soft Tissue Infections of the Head and Neck, Upper Respi
146 not a critical virulence factor in invasive soft-tissue infection or bacteraemia caused by S. pyogen
148 eraemia, musculoskeletal infection, skin and soft-tissue infection, or colonisation published before
149 Infections, Genital Infections, and Skin and Soft Tissue Infections; or into etiologic agent groups,
151 bacteremia, meningitis, pneumonia, skin and soft tissue infections, peritonitis, and urinary tract i
152 tients with invasive infections, noninvasive soft tissue infections, pharyngitis, and rheumatic fever
153 tients with invasive infections, noninvasive soft tissue infections, pharyngitis, and rheumatic fever
154 adult patients with acute, purulent skin and soft-tissue infections presenting to 11 university-affil
155 uced and (2) many strains of MSSA that cause soft-tissue infections produce higher levels of PVL than
158 roup G streptococcus can produce necrotising soft tissue infections resembling those produced by grou
159 0DeltasaeR/S) in murine models of sepsis and soft-tissue infection revealed that this sensory system
160 smaller series of patients with necrotizing soft tissue infections, similar analyses of risk factors
161 olonization of cases and subsequent skin and soft tissue infection (SSTI) in cases and household cont
162 rrent infection, in which S. aureus skin and soft tissue infection (SSTI) strongly protected against
166 ant Staphylococcus aureus (CO-MRSA) skin and soft tissue infections (SSTI) are associated with SCCmec
167 tained from pediatric patients with skin and soft tissue infections (SSTI) from Taipei did not carry
168 MRSA) is currently a major cause of skin and soft tissue infections (SSTI) in the United States.
169 s (CA-MRSA) is a prevalent cause of skin and soft tissue infections (SSTI), but the association betwe
170 has been associated previously with skin and soft-tissue infection (SSTI) and with carriage of staphy
172 t (ED) visit or hospitalization for skin and soft-tissue infection (SSTI), respiratory infection, int
175 subtropics with S. aureus-positive skin and soft tissue infections (SSTIs) and 124 control patients
176 RSA) lineage causes the majority of skin and soft tissue infections (SSTIs) and is highly associated
177 ccus aureus is the leading cause of skin and soft tissue infections (SSTIs) and mounting antibiotic r
178 n which we identified patients with skin and soft tissue infections (SSTIs) and randomized them to re
179 Recurrent Staphylococcus aureus skin and soft tissue infections (SSTIs) are common despite detect
180 other nontuberculous mycobacterial skin and soft tissue infections (SSTIs) associated with handling
181 empirical therapy for patients with skin and soft tissue infections (SSTIs) caused by molecularly and
184 is causing an increasing number of skin and soft tissue infections (SSTIs) in Denmark and other Euro
185 illin-susceptible, has been causing skin and soft tissue infections (SSTIs) in epidemic proportions a
186 istant Staphylococcus aureus (MRSA) skin and soft tissue infections (SSTIs) in high-risk community se
187 USA300 has led to a high burden of skin and soft tissue infections (SSTIs) in the United States, yet
189 ncreasingly important as a cause of skin and soft tissue infections (SSTIs), particularly abscesses,
190 tive bacteria cause the majority of skin and soft tissue infections (SSTIs), resulting in the most co
191 aureus is the most common cause of skin and soft tissue infections (SSTIs), yet sex bias in suscepti
196 tion coding system, epidemiology of skin and soft-tissue infections (SSTIs) has conflated abscess wit
197 ave increased the overall number of skin and soft-tissue infections (SSTIs), increasing the overall d
198 nds and 136 non-IFI wounds (63 with skin and soft tissue infections [SSTIs] and 73 without) were exam
199 solates from infective endocarditis (IE) and soft tissue infections (STIs), we sought to (1) validate
200 nearly twice as common in community skin and soft tissue infections than in nosocomial bloodstream in
202 n 1750 developed nonlethal necrotic skin and soft tissue infections that healed spontaneously after 1
203 ) is a rapidly progressive, life-threatening soft-tissue infection that is traditionally caused by gr
206 de spectrum of disease ranging from skin and soft tissue infections to life-threatening pneumonia and
207 uman disease ranging from localized skin and soft tissue infections to potentially lethal systemic in
208 ecutive patients with documented necrotizing soft tissue infections, treated at a single institution
209 l infection rates and rates of RTI, skin and soft-tissue infection, urinary tract infection, and bloo
210 The proportion of operations for skin and soft tissue infections was highest during natural disast
211 However, the frequency of serious skin and soft tissue infections was increased in anti-TNF-treated
212 In contrast, the rate of serious skin and soft tissue infections was increased, suggesting an impo
214 pically similar virulence defect in skin and soft tissue infection, we sought to determine the relati
220 of the isolates were recovered from skin and soft tissue infections, whereas the remaining isolates (
221 hil deployment protected mice against lethal soft tissue infection with Streptococcus pyogenes and pr
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