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1 seudomonas syringae and PopP2 from Ralstonia solanacearum.
2 in another P. syringae strain and Ralstonia solanacearum.
3 ulation of flagellum-mediated motility in R. solanacearum.
4 (bp) genes, as well as their orthologs in R. solanacearum.
5 ytopathogenic Xanthomonas spp. and Ralstonia solanacearum.
6 tii, as well as the plant pathogen Ralstonia solanacearum.
7 ulence genes in the plant pathogen Ralstonia solanacearum.
8 unknown function, and 13% were unique to R. solanacearum.
9 nas campestris pv. vesicatoria and Ralstonia solanacearum.
10 ve as sole carbon or nitrogen sources for R. solanacearum.
11 ty was with Ralstonia eutropha and Ralstonia solanacearum.
12 or (VEF) produced by wild-type strains of R. solanacearum.
13 h includes R. eutropha, R. pickettii, and R. solanacearum.
14 operon is the major virulence factor for R. solanacearum.
15 es virulence gene expression in wild-type R. solanacearum.
16 ributes substantially to the virulence of R. solanacearum.
19 ntly showed that, in Ralstonia (Pseudomonas) solanacearum, a phytopathogenic bacterium, acyl-HSL prod
20 Expression of virulence genes in Ralstonia solanacearum, a phytopathogenic bacterium, is controlled
24 red for a wild-type level of virulence in R. solanacearum although its individual role in wilt diseas
29 vity in plant-pathogenic bacteria (Ralstonia solanacearum) and fungi (Cochliobolus heterostrophus) re
30 dule, a fucose-binding lectin from Ralstonia solanacearum, and human norovirus VA387 P particle (24-m
33 ly-related bacteria 'Candidatus Liberibacter solanacearum' (CLso), associated with vegetative disorde
34 e of ipx genes suggests that in its host, R. solanacearum confronts and overcomes a stressful and nut
35 In the phytopathogen Ralstonia (Pseudomonas) solanacearum, control of many virulence genes is partly
41 py revealed that during tomato infection, R. solanacearum forms biofilm-like masses in xylem vessels.
42 dge, this is the first demonstration that R. solanacearum forms biofilms in plant xylem vessels, and
48 n resistance to the plant pathogen Ralstonia solanacearum in microcosms and in tomato plant rhizosphe
49 tor produced by the plant pathogen Ralstonia solanacearum, in complex with inositol hexaphosphate (In
59 Expression of virulence factors in Ralstonia solanacearum is controlled by a complex regulatory netwo
60 vessels of infected plants, we found that R. solanacearum is essentially nonmotile in planta, althoug
61 cyl-HSL-dependent autoinduction system in R. solanacearum is part of a more complex autoregulatory hi
64 ormed by oligomerization as in the Ralstonia solanacearum lectin and not by sequential domains like t
67 an extracellular factor that complements R. solanacearum mutants deficient in production of the 3-OH
69 To locate and infect host plant roots R. solanacearum needs taxis, the ability to move toward mor
72 type rescued DeltaspeC growth, indicating R. solanacearum produced and exported putrescine to xylem s
73 phy, and mass spectroscopy indicated that R. solanacearum produces staphyloferrin B rather than schiz
74 oea stewartii, Erwinia carotovora, Ralstonia solanacearum, Pseudomonas syringae, Pseudomonas aerugino
75 phores present in culture supernatants of R. solanacearum, R. metallidurans, and Bacillus megaterium
77 of the phytopathogen Ralstonia (Pseudomonas) solanacearum, requires the products of at least seven re
79 he gene encoding the catalytic subunit of R. solanacearum's sole assimilatory nitrate reductase, did
83 (IVET), we screened a library of 133 200 R. solanacearum strain K60 promoter fusions and isolated ap
86 olog of HrpB, the master regulator of the R. solanacearum T3SS (T3SS(rso)) and its secreted effectors
90 syringae and the vascular pathogen Ralstonia solanacearum Thus, the GFP strand system can be broadly
91 ntributes significantly to the ability of R. solanacearum to locate and effectively interact with its
94 uggest that nitrate assimilation promotes R. solanacearum virulence by enhancing root attachment, the
95 nvestigate the role of these acyl-HSLs in R. solanacearum virulence gene expression, transposon mutan
97 not a sole carbon or nitrogen source for R. solanacearum, was enriched 76-fold to 37 microM in R. so
98 the growth of a bacterial invader, Ralstonia solanacearum, when introduced into communities comprised
102 ovora, Pectobacterium carotovorum, Ralstonia solanacearum, Xanthomonas campestris, Xanthomonas oryzae
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