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2 ble CD14 level, kynurenine/tryptophan ratio, soluble tumor necrosis factor receptor 1 level, high-sen
3 in-6, C-reactive peptide, interleukin-8, and soluble tumor necrosis factor receptor 1 were most diffe
4 leukin (IL)-1 receptor antagonist (sIL-1ra), soluble tumor necrosis factor receptor 1, soluble vascul
5 rkers C-reactive protein, interleukin 6, and soluble tumor necrosis factor receptors 1 and 2 (sTNF-R1
7 and plasma interleukin-6, interleukin-8, and soluble tumor necrosis factor receptor-1 levels were sig
8 y volunteer levels and reduced inflammation (soluble tumor necrosis factor receptor-1), coagulation (
9 uced set of three biomarkers (interleukin-8, soluble tumor necrosis factor receptor-1, and surfactant
10 angiopoietin-1, angiopoietin-2, IL-6, IL-8, soluble tumor necrosis factor receptor-1, soluble vascul
11 nd factor antigen, surfactant protein-D, and soluble tumor necrosis factor receptor-1, which are biom
12 e protein (CRP), interleukin (IL)-6, and the soluble tumor necrosis factor receptor 2 (sTNFR-2) in bl
13 erval [CI], 1.7-4.2; Ptrend = 1.0 x 10(-6)), soluble tumor necrosis factor receptor 2 (sTNFR2; OR, 3.
14 n inflammatory process dominated by markers: soluble tumor necrosis factor receptor 2, soluble interl
15 ed that levels of leptin, interleukin-6, and soluble tumor necrosis factor receptor-2, as well as the
16 uch as interleukin-1 receptor antagonist and soluble tumor necrosis factor receptors, and is protecti
17 levels approximately 2-fold (P=.03), whereas soluble tumor necrosis factor receptor blunted the incre
20 soluble interleukin 6 receptor (sIL-6R), and soluble tumor necrosis factor receptor I (sTNF-RI) were
21 T cells, plasma interleukin 6 (IL-6) level, soluble tumor necrosis factor receptor I (sTNFR-I) level
22 a, C4a, C5a), cytokines (IL-2, IL-6, IL-10), soluble tumor necrosis factor receptor I, and platelet a
23 tein, tumor necrosis factor alpha (monomer), soluble tumor necrosis factor receptors I and II (sTNF-R
25 ve protein (r=0.28), interleukin-6 (r=0.22), soluble tumor necrosis factor receptor II (r=0.22), solu
26 s tested for levels of interleukin-6 (IL-6), soluble tumor necrosis factor receptor II (sTNFRII) (as
27 i-CCP) antibodies, interleukin-6 (IL-6), and soluble tumor necrosis factor receptor II (sTNFRII).
28 ected by pregnancy or infection, circulating soluble tumor necrosis factor receptor II was highest in
30 nsitivity C-reactive protein, interleukin 6, soluble tumor necrosis factor receptor II, soluble inter
31 mum plasma levels of interleukin (IL)-10 and soluble tumor necrosis factor receptor-II correlated wit
32 2 receptor, tumor necrosis factor-alpha, and soluble tumor necrosis factor receptors p55 and p75.
33 ted the safety and efficacy of etanercept, a soluble tumor necrosis factor receptor (p75):Fc fusion p
34 ermine whether the addition of etanercept, a soluble tumor necrosis factor receptor (p75):Fc fusion p
36 To investigate whether circulating levels of soluble tumor necrosis factor receptors (sTNFR) are pred
37 tumor necrosis factor-[alpha], type I and II soluble tumor necrosis factor receptors (sTNFR-I and -II
38 (IL-6), soluble IL-6 receptor (sIL-6R), and soluble tumor necrosis factor receptors (sTNFRI and -II)
40 s factor-alpha with monoclonal antibodies or soluble tumor necrosis factor receptor substantially red
45 of tumor necrosis factor-alpha (TNF-alpha), soluble tumor necrosis factor receptor type I (sTNFR-I),
46 crosis factor receptor type I (sTNFR-I), and soluble tumor necrosis factor receptor type II (sTNFR-II
47 pha (TNF-alpha) concentrations and increased soluble tumor necrosis factor receptor type II (sTNFRII)
48 , such as serum levels of interleukin-10 and soluble tumor necrosis factor receptors types Iota and I
49 ive protein, temperature, interleukin 8, and soluble tumor necrosis factor receptor were also reduced
50 of the other soluble adhesion molecules and soluble tumor necrosis factor receptor were not signific
53 as selective cyclooxygenase-2 inhibitors and soluble tumor necrosis factor receptor, whose developmen
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