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1 erleukin-1 receptor antagonist (sIL-1ra) and soluble tumor necrosis factor receptor 1 (sTNF-R1).
2 ble CD14 level, kynurenine/tryptophan ratio, soluble tumor necrosis factor receptor 1 level, high-sen
3 in-6, C-reactive peptide, interleukin-8, and soluble tumor necrosis factor receptor 1 were most diffe
4 leukin (IL)-1 receptor antagonist (sIL-1ra), soluble tumor necrosis factor receptor 1, soluble vascul
5 rkers C-reactive protein, interleukin 6, and soluble tumor necrosis factor receptors 1 and 2 (sTNF-R1
6 or (t-PA), LDL-C, von Willebrand factor, and soluble tumor necrosis factor receptors 1 and 2.
7 and plasma interleukin-6, interleukin-8, and soluble tumor necrosis factor receptor-1 levels were sig
8 y volunteer levels and reduced inflammation (soluble tumor necrosis factor receptor-1), coagulation (
9 uced set of three biomarkers (interleukin-8, soluble tumor necrosis factor receptor-1, and surfactant
10  angiopoietin-1, angiopoietin-2, IL-6, IL-8, soluble tumor necrosis factor receptor-1, soluble vascul
11 nd factor antigen, surfactant protein-D, and soluble tumor necrosis factor receptor-1, which are biom
12 e protein (CRP), interleukin (IL)-6, and the soluble tumor necrosis factor receptor 2 (sTNFR-2) in bl
13 erval [CI], 1.7-4.2; Ptrend = 1.0 x 10(-6)), soluble tumor necrosis factor receptor 2 (sTNFR2; OR, 3.
14 n inflammatory process dominated by markers: soluble tumor necrosis factor receptor 2, soluble interl
15 ed that levels of leptin, interleukin-6, and soluble tumor necrosis factor receptor-2, as well as the
16 uch as interleukin-1 receptor antagonist and soluble tumor necrosis factor receptors, and is protecti
17 levels approximately 2-fold (P=.03), whereas soluble tumor necrosis factor receptor blunted the incre
18             We evaluated etanercept, a human soluble tumor necrosis factor receptor: Fc fusion protei
19                                Etanercept, a soluble tumor necrosis factor receptor, has been shown t
20 soluble interleukin 6 receptor (sIL-6R), and soluble tumor necrosis factor receptor I (sTNF-RI) were
21  T cells, plasma interleukin 6 (IL-6) level, soluble tumor necrosis factor receptor I (sTNFR-I) level
22 a, C4a, C5a), cytokines (IL-2, IL-6, IL-10), soluble tumor necrosis factor receptor I, and platelet a
23 tein, tumor necrosis factor alpha (monomer), soluble tumor necrosis factor receptors I and II (sTNF-R
24 r 1 or its binding protein) or inflammation (soluble tumor necrosis factor receptors I or II).
25 ve protein (r=0.28), interleukin-6 (r=0.22), soluble tumor necrosis factor receptor II (r=0.22), solu
26 s tested for levels of interleukin-6 (IL-6), soluble tumor necrosis factor receptor II (sTNFRII) (as
27 i-CCP) antibodies, interleukin-6 (IL-6), and soluble tumor necrosis factor receptor II (sTNFRII).
28 ected by pregnancy or infection, circulating soluble tumor necrosis factor receptor II was highest in
29       Etanercept, a dimerized version of the soluble tumor necrosis factor receptor II, and inflixima
30 nsitivity C-reactive protein, interleukin 6, soluble tumor necrosis factor receptor II, soluble inter
31 mum plasma levels of interleukin (IL)-10 and soluble tumor necrosis factor receptor-II correlated wit
32 2 receptor, tumor necrosis factor-alpha, and soluble tumor necrosis factor receptors p55 and p75.
33 ted the safety and efficacy of etanercept, a soluble tumor necrosis factor receptor (p75):Fc fusion p
34 ermine whether the addition of etanercept, a soluble tumor necrosis factor receptor (p75):Fc fusion p
35                                         When soluble tumor necrosis factor receptor (sTNFR) I and II
36 To investigate whether circulating levels of soluble tumor necrosis factor receptors (sTNFR) are pred
37 tumor necrosis factor-[alpha], type I and II soluble tumor necrosis factor receptors (sTNFR-I and -II
38  (IL-6), soluble IL-6 receptor (sIL-6R), and soluble tumor necrosis factor receptors (sTNFRI and -II)
39                      Plasma levels of 80-kDa soluble tumor necrosis factor receptors (sTNFRs) were hi
40 s factor-alpha with monoclonal antibodies or soluble tumor necrosis factor receptor substantially red
41                     Plasma concentrations of soluble tumor necrosis factor receptor superfamily 1A (s
42                                    Exogenous soluble tumor necrosis factor receptor (TNFR) has been s
43                            Recombinant human soluble tumor necrosis factor receptor (TNFR:Fc) is a di
44                                              Soluble tumor necrosis factor receptors (TNFRs) are impo
45  of tumor necrosis factor-alpha (TNF-alpha), soluble tumor necrosis factor receptor type I (sTNFR-I),
46 crosis factor receptor type I (sTNFR-I), and soluble tumor necrosis factor receptor type II (sTNFR-II
47 pha (TNF-alpha) concentrations and increased soluble tumor necrosis factor receptor type II (sTNFRII)
48 , such as serum levels of interleukin-10 and soluble tumor necrosis factor receptors types Iota and I
49 ive protein, temperature, interleukin 8, and soluble tumor necrosis factor receptor were also reduced
50  of the other soluble adhesion molecules and soluble tumor necrosis factor receptor were not signific
51           Also, the plasma concentrations of soluble tumor necrosis factor receptors were higher in I
52                                    Levels of soluble tumor necrosis factor receptors were higher in t
53 as selective cyclooxygenase-2 inhibitors and soluble tumor necrosis factor receptor, whose developmen

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