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1 nd Gli2 to transactivate Myf5 in the epaxial somite.
2 required for Myf5 expression in the epaxial somite.
3 on in the anterior compartment of the future somite.
4 l crest cell migration to the center of each somite.
5 tissues initiate differentiation within the somite.
6 presumptive limb myoblasts emigrate from the somite.
7 rapid morphogenetic movements in the amniote somite.
8 to mutually repress each other in the mouse somite.
9 thus full epithelialization of the posterior somite.
10 ut differentiate prematurely adjacent to the somite.
11 sternal precursors do not originate from the somites.
12 rated embryonic mesodermal structures called somites.
13 a mechanism to specify posterior identity of somites.
14 expression of medium/long Vegfa isoforms in somites.
15 streams, or stall shortly after entering the somites.
16 derivatives, this enhancer is not active in somites.
17 are held back in order to give rise to later somites.
18 er, these mutants are able to form organized somites.
19 embryo's posterior body, including the tail somites.
20 ors results in complete absence of posterior somites.
21 s associated with an increased cell death in somites.
22 n ceased after the production of about 15-20 somites.
23 nd distal at 4 somites, and distal only at 6 somites.
24 the protein level of phosphorylated p38a in somites.
25 are only able to form roughly half of their somites.
26 ationship of these two regulators in amniote somites.
27 its the expression of cardiac markers in the somites.
28 the muscles for both systems arise from the somites.
29 romotes myotomal extension directly from the somites.
30 al vertebrae, and reduced number of ribs and somites.
31 ardiac progenitors and cardiomyocytes in the somites.
32 ules but to allow expression in the adjacent somites.
33 ogenitors expressing Pax3 are missing in the somites.
34 a dorsal, hollow nerve cord, a notochord and somites.
35 h the lateral plate mesoderm and also in the somites.
36 gmental de-adhesion and individualization of somites.
37 f the vertebrate embryo's body segments, the somites.
38 o expressed in mesodermal tissues, including somites.
39 mutants exhibited more severe phenotypes in somites.
40 ntial progenitors that normally give rise to somites.
41 oevolutionary diversity in limbs, digits and somites.
42 cells oscillate until they incorporate into somites.
43 egments into bilateral tissue blocks, called somites.
44 neck arise from Pax3-expressing cells in the somites.
47 This allele causes dominant skeletal and somite abnormalities that are distinct from those seen i
49 ate and in vasculogenic regions of the avian somite and are able to promote a vascular endothelial fa
52 ed by canonical Wnt signaling in the epaxial somite and second branchial arch, but not in the limb or
53 rsors migrate across the ventral face of the somite and that junctional adhesion molecules (JAMs) med
54 n (Mb) and Cb by Wnt1 occurs between the one-somite and the six- to eight-somite stages and is solely
55 tures are homologous across vertebrates, how somite and vertebral patterning are connected, and the d
58 from the hypaxial dermomyotome of limb level somites and migrate into the limb bud where they form th
59 ion by miR-206 and miR-133 in the developing somites and miR-124 in the developing central nervous sy
61 nt with the shared origin of spinal cord and somites and the distinct ontogeny of the anterior and po
62 6, which drives the VEGFA expression in both somites and the DLP, sit at the top of the adult haemang
63 rafish mutants; (b) the delayed formation of somites and the formation of 'salt and pepper' patterns
64 in the spinal cord, hypothalamus, pituitary, somites and upper jaw, but that Boc might negatively reg
66 ormation, was upregulated in mutant cervical somites, and conditional ablation of ectodermal Tbx3 exp
69 lial growth factor A (VEGFA) ligand from the somites, and finally for gata2 expression in the DA, but
70 dorsal medial dermomyotome of newly forming somites, and immunohistological studies show that Zic2 p
71 egmentation of the vertebrate body axis into somites, and later vertebrae, relies on a genetic oscill
72 uently converted into the spatial pattern of somites, and Mesp2 plays crucial roles in this conversio
74 p from the sclerotome layer of the metameric somites, and PNS neurons and glia differentiate from neu
76 ver, somite size is reduced and later-formed somites are caudalized, coincident with increased cell d
81 termination wavefront, controlling where new somites are permitted to form along the anteroposterior
85 velopment when the vertebral precursors, the somites, are rhythmically produced from presomitic mesod
87 stream, and that they consequently enter the somites as multiple streams, or stall shortly after ente
88 by expression of Lbx1, are specified in the somite at forelimb level, but endothelial progenitors ar
90 sp quadruple mutant embryos the positions of somite boundaries were clearly determined and morphologi
95 rospectively tracking oscillations of future somite boundary cells, we find that clock reporter signa
96 and that, in zebrafish, the genes regulating somite boundary formation also regulate the development
97 that a gene network previously implicated in somite boundary formation, involving the transcriptional
101 a statistical enrichment of Dvl in notochord-somite boundary-(NSB)-directed protrusions, which is dep
106 regulate the number of premyogenic cells in somites but does not regulate myogenic initiation in the
107 nd spatially restricted to the center of the somite, but that motor axons are dispensable for segment
108 ead and some neck muscles do not derive from somites, but mainly form from mesoderm in the pharyngeal
109 al muscles of body and limb are derived from somites, but most head muscles originate from cranial me
110 for proper patterning of the neural tube and somites by regulating notochord formation, and provide e
111 zebrafish STARS (zSTARS) first occurs in the somites by the 16 somite stage [17 hours post fertilizat
113 gether, our data demonstrate Pax3-expressing somite cells as a source of OSM and SHM fibres, and high
115 of the presomitic mesoderm anterior border, somite chevron morphology and hypaxial myoblast migratio
120 Dll3 specifically disrupts key regulators of somite cycling, we carried out functional analysis to id
121 which results in ectopic Fgf8 expression and somite defects, was recruited near the RARb RARE by RA,
126 ineage origin) with their limb and diaphragm somite-derived counterparts, but are remarkably endowed
127 We conclude that the small proportion of somite-derived endothelial cells in the limb is required
129 o initiate myogenesis in branchial arch- and somite-derived muscles are known, but the comparable ups
130 common hallmarks with the prototypic SCs in somite-derived muscles, they distinctively feature robus
131 ta signaling in the development of occipital somite-derived myogenic progenitors during tongue morpho
133 muscles constitute a transition zone between somite-derived skeletal muscles of the trunk and limbs,
134 se elimination is occurring in most EOMs and somite-derived skeletal muscles, it appears to be dramat
136 Blastopore closure, notochord formation, somite development, neural tube closure, and the formati
139 notochord during early segmentation, in the somites during later segmentation and in the liver at th
143 and Zic1 in transactivating the Myf5 epaxial somite (ES) enhancer in concert with the Myf5 promoter.
145 genitors can be transfated from notochord to somite fate after gastrulation by ectopic expression of
151 h a role for ntl in mesoderm formation, some somites form within the tail region of embryos lacking n
152 ck' activity that governs the periodicity of somite formation and (iii) preserve the composition and
153 wavefront determines the spatial position of somite formation and the somitogenesis clock controls pe
154 segmentation was significantly affected, and somite formation ceased after the production of about 15
158 ranial neural crest cell-streams relative to somite formation is accelerated in rapidly developing fr
160 ranscription factors Ntla and Tbx16 regulate somite formation upstream of this by controlling supply
161 ggest that the segmentation clock may signal somite formation using a phase gradient with a two-somit
164 cs of cells in the tailbud and their role in somite formation, we have analyzed the genetic factors a
165 of miRNAs in mesoderm development including somite formation, we used T (Brachyury)-Cre mouse line t
172 ized by the periodic formation of epithelial somites from the mesenchymal presomitic mesoderm (PSM).
173 ing genes, controls the periodic cleavage of somites from unsegmented presomitic mesoderm during vert
177 ipotent Pax3-positive (Pax3(+)) cells in the somites give rise to skeletal muscle and to cells of the
182 hin2) leads to the formation of asymmetrical somites in mouse embryos, similar to embryos deprived of
184 mental features of the vertebrate body - the somites - in which beta1-integrin activity regulates epi
186 alling promotes myogenesis in the vertebrate somite, in part by raising the activity of muscle regula
187 formation of an ectopic tail, which contains somites, in embryos devoid of nodal signalling, and this
189 le progenitor cells migrate from the lateral somites into the developing vertebrate limb, where they
190 whereby migration of myogenic cells from the somites into the limb bud is followed by their extension
193 l plate mesoderm (LPM) adjacent to occipital somites is a recently identified embryonic source of cer
194 nt embryos, Myf5 expression in newly forming somites is deficient in both sonic hedgehog(-/-) and in
198 n patterns during development of the palate, somite, kidney, and testis, suggesting that it may be an
200 e the ribs have been shown to arise from the somites, little is known about how the two segments are
201 esoderm markers such as Tcf15 and Meox1, and somite markers such as Uncx, but failed to express scler
202 a in zebrafish embryos: (a) the variation in somite measurements across a number of zebrafish mutants
211 We show that a fundamental distinction in somite myogenesis is into medial versus lateral compartm
214 show that RARbeta2 plays a specific role in somite number and size, restriction of the presomitic me
216 xial and abaxial domains and by increases in somite number, not by changes in the function of primaxi
222 nopus skeletal muscle development, including somite organization, migration of hypaxial muscle anlage
223 ecify posterior otic identity between the 10 somite (otic placode) and 20 somite (early otic vesicle)
224 se gastrula between Early Streak (ES) and 12-somite pair (-s) stages (~6.75-9.0 days post coitum, dpc
225 ing phenotypes in the segmentation clock and somite patterning of mutant mice suggest that LFNG prote
226 Less is understood about how RAR regulates somite patterning, rostral-caudal boundary setting, spec
229 regions, which direct Eya1 expression to the somites, pharyngeal pouches, the preplacodal ectoderm (t
230 to ligand and its localization in the trunk somites positions it at the right time and place to resp
231 defects in somite segmentation and a loss in somite posterior polarity leading to fusions of vertebra
234 naling and myogenesis in selected epithelial somite progenitor cells, which rapidly translocate into
235 he somite is between primaxial muscle of the somite proper and abaxial somite-derived migratory muscl
239 Fgf and Hedgehog function between 10 and 20 somites results in symmetrical otic vesicles with neithe
240 REB family in the mesoderm causes defects in somite segmentation and a loss in somite posterior polar
241 ignaling as a means to orchestrate cells for somite segmentation and anterior/posterior patterning.
242 unction of PAPC in chicken embryos disrupted somite segmentation by altering the CDH2-dependent epith
244 e functions of the two Delta proteins in the somite segmentation clock--an explanation that is based
246 on changes in mesoderm development uncovered somite segmentation, a previously unobservable human emb
247 nic mesoderm, while Dicer is dispensable for somite segmentation, it is essential for proper limb bud
248 Knockdown of itgalpha6b resulted in abnormal somite shape, fewer somitic cells, weaker or absent myf5
249 Accordingly, LPM adjacent to the occipital somites should be regarded as posterior cranial mesoderm
252 sion, and leads to a persistent reduction in somite size until at least the independent feeding stage
253 ransactivation of Gli-dependent Myf5 epaxial somite-specific (ES) enhancer activity in 3T3 cells, and
257 is gene, dissections were performed on early somite stage embryos during an eight-hour time window of
259 e endocardial progenitors as early as the 10-somite stage which suggests that Hh signaling is require
260 ls, labeled by lipophilic dye at the 4- to 6-somite stage, to regions of the heart at 20 to 25 somite
261 data, and in situ hybridization analysis of somite-stage embryos, we carried out comparative analyse
263 pancreatic mesenchyme between the 26 and 27 somite stages (approximately 10.0 dpc) and became interm
264 between the one-somite and the six- to eight-somite stages and is solely dependent on Wnt1 function i
265 In our model, specification occurs during somite stages due to varying Hedgehog protein levels, wh
266 ing such that during late gastrula and early somite stages of embryogenesis, Wnt activity must be sup
267 ryos show that Cubn is required during early somite stages to convey survival signals in the developi
269 left wall of the KV between the six- and 12-somite stages, which is coincident with known left-sided
270 ted PE specification role for Tbx5a at early somite stages; this role occurs earlier than, and appear
271 ent and, in particular, in the regulation of somite survival and axial rotation, a crucial developmen
272 id signalling, which is required to maintain somite symmetry by interacting with Fgf8 in the left-rig
275 ween different tissues derived from the same somites that contribute to the structures of the cervica
276 mesoderm treated with Noggin generates many somites that form simultaneously, without cyclic express
277 estricted to the hypaxial region of anterior somites that generate migratory muscle precursors (MMPs)
278 er dorsal and lateral to neural tube and the somites that is normally formed by PNA-binding proteins
279 al skeletal elements arise from the paraxial somites, the appendicular skeleton and sternum arise fro
280 S that was introduced into a subset of chick somites, the progenitors that give rise to dermis and mu
282 motifs of vertebrate bodies and organs, the somites, the skeletons of the paired limbs, and musculos
283 k neural crest along the rostral side of the somites, the ventromedial pathway, the branchial arches,
284 ebrafish development, derive from the caudal somites through an epithelial-mesenchymal transition (EM
285 is non-cell-autonomous, and acts within the somite to bridge the Wnt and Notch signalling pathways.
286 or is required earlier within the developing somite to regulate HSC emergence in a non-cell-autonomou
287 that Notch3 performs a novel role within the somite to regulate the neighboring precursors of hemogen
288 sduction of requisite Notch signals from the somite to the precursors of HSCs, and that these events
290 recursor cells (MPCs) have migrated from the somites to the limb bud and populated the prospective mu
292 is expressed prominently in the brain, eyes, somites, ventral blood island and branchial arches.
294 f11r) and migrate axially across the ventral somite, where Jam2a and the Notch ligands Dlc and Dld ar
295 nic development, skeletal muscles arise from somites, which derive from the presomitic mesoderm (PSM)
296 engrailed-1 (en1) staining in anterior trunk somites, which is dependent on Hedgehog (Hh) signaling.
297 l distribution corresponding to the injected somites, which suggests that blocking GNAS activity in a
298 Zebrafish ortholog of Nogo-B is expressed in somite while expression of zebrafish NgBR is localized i
299 anterior gradient of stable Cdh2 within each somite, while there is a step-like drop in stable Cdh2 a
300 the paraxial mesoderm becomes segmented into somites, within which proliferative muscle progenitors a
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