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1 in the neuronal GlyR alpha or beta subunits (spasmodic and spastic) were shown to be deficient in the
5 to the clinical and subclinical features of spasmodic dysphonia and may represent the neurochemical
6 YT4 dystonia, a dominantly inherited form of spasmodic dysphonia combined with other focal or general
10 the corticobulbar/corticospinal tract in 20 spasmodic dysphonia patients compared to 20 healthy subj
13 C DeltaBP measures, while longer duration of spasmodic dysphonia was associated with a decrease in ta
16 capacitation, but capacitation of sperm from spasmodic mice for up to 3 h did not result in significa
21 ine at amino acid 52, is responsible for the spasmodic phenotype in mice and alters the ability of gl
25 disease duration >/=3 years, Toronto Western Spasmodic Torticollis Rating Scale [TWSTRS] severity sco
26 muscles was investigated in 24 patients with spasmodic torticollis using small, abrupt 'drops' of the
29 -collic reflex is disrupted in patients with spasmodic torticollis, a finding which corroborates the
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