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1 ma ELISPOT assays were used to measure donor-specific reactivity.
2 in both TCRs that enhanced CD4(+) T cell Ag-specific reactivity.
3 e residue were sufficient to disrupt variant-specific reactivity.
4 d in HCV(+) donors who showed strong HCV-NS3-specific reactivity.
5 tested against autologous tumor demonstrated specific reactivity.
6 n of both TCRs in human PBLs demonstrated Ag-specific reactivity against a range of melanoma and nonm
7 Ig) isotype-specific as well as IgG subclass-specific reactivity against Bartonella henselae whole-ce
8 egies for inducing systemic, long-term tumor-specific reactivity among CD4+ T cells will be critical
10 c, autoimmune Th1 cells fostered their organ-specific reactivity, as confirmed in an autoimmune mouse
12 quantify the contributions they make to this specific reactivity by thermodynamic and kinetic analyse
15 , numerous restimulations were required, and specific reactivity could not be generated in many patie
16 ost informative when used to compare residue-specific reactivities in a number of functional states o
17 3alpha chain was identified that enhanced Ag-specific reactivity in gene-modified CD4(+) and CD8(+) T
18 ern blot analysis, MAb CP-33 exhibited genus-specific reactivity in that it recognized the LPSs of C.
20 e 10(5)-10(7) M(-1)s(-1)), despite their low specific reactivity (kinact </= 2.1 x 10(-3) s(-1)), whi
23 codon 273 in P53 may reflect either sequence-specific reactivity of BPDE and/or inefficient repair of
28 rfacial bonding linkage was confirmed by the specific reactivity of the nanoparticles with imine deri
31 be useful clinically for distinguishing CCP-specific reactivity seen in RA from reactivity with both
34 V-transformed B cell lines were selected for specific reactivity to a 20-mer TCR peptide incorporatin
39 94%) were positive in this assay, whereas no specific reactivity was detected in any of the 107 contr
41 T cell activation, migration, and beta cell-specific reactivity were similar in NOD mice of all thre
42 dentity of p116 has been demonstrated by its specific reactivity with anti-Cbl and similarity of phos
43 n a concentration dependent manner, while no specific reactivity with factors X or IXa was observed.
45 ay ionization (ESI) mass spectrometry and by specific reactivity with mAb 8E11, directed to GM2/GM3 d
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