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1 ma ELISPOT assays were used to measure donor-specific reactivity.
2  in both TCRs that enhanced CD4(+) T cell Ag-specific reactivity.
3 e residue were sufficient to disrupt variant-specific reactivity.
4 d in HCV(+) donors who showed strong HCV-NS3-specific reactivity.
5 tested against autologous tumor demonstrated specific reactivity.
6 n of both TCRs in human PBLs demonstrated Ag-specific reactivity against a range of melanoma and nonm
7 Ig) isotype-specific as well as IgG subclass-specific reactivity against Bartonella henselae whole-ce
8 egies for inducing systemic, long-term tumor-specific reactivity among CD4+ T cells will be critical
9 e adhesive surface and screened toward their specific reactivity and cross-reactivity.
10 c, autoimmune Th1 cells fostered their organ-specific reactivity, as confirmed in an autoimmune mouse
11                         Examination of donor-specific reactivity by means of lymphocyte proliferation
12 quantify the contributions they make to this specific reactivity by thermodynamic and kinetic analyse
13                           We evaluated donor-specific reactivity, chimerism, and antibody production.
14                           This changes their specific reactivity compared to Cr(III) sites in oxygen
15 , numerous restimulations were required, and specific reactivity could not be generated in many patie
16 ost informative when used to compare residue-specific reactivities in a number of functional states o
17 3alpha chain was identified that enhanced Ag-specific reactivity in gene-modified CD4(+) and CD8(+) T
18 ern blot analysis, MAb CP-33 exhibited genus-specific reactivity in that it recognized the LPSs of C.
19                             Absence of donor-specific reactivity is then confirmed by a pretransplant
20 e 10(5)-10(7) M(-1)s(-1)), despite their low specific reactivity (kinact </= 2.1 x 10(-3) s(-1)), whi
21                                              Specific reactivities of an anti-smitin polyclonal antib
22           These data demonstrate the isotype-specific reactivity of antibodies during the autoimmune
23 codon 273 in P53 may reflect either sequence-specific reactivity of BPDE and/or inefficient repair of
24 re are intrinsic differences in the serotype-specific reactivity of CD8(+) T-cell responses.
25             For the purpose of examining the specific reactivity of lymphoid elements in the intestin
26                        More importantly, the specific reactivity of orthoboric groups to diols in cel
27                                          The specific reactivity of porphyrinoids has offered an insi
28 rfacial bonding linkage was confirmed by the specific reactivity of the nanoparticles with imine deri
29                                          The specific reactivity of these constructs was evaluated in
30        This approach is ideal for addressing specific reactivity problems, but its focused nature mig
31  be useful clinically for distinguishing CCP-specific reactivity seen in RA from reactivity with both
32                                         TmpA-specific reactivity showed good correlation with RPR tit
33                                         This specific reactivity suggests that MAb 8H9 may be useful
34 V-transformed B cell lines were selected for specific reactivity to a 20-mer TCR peptide incorporatin
35 ted in the loss of binding to beta 2GP-1 and specific reactivity to prothrombin.
36                          Five samples showed specific reactivity to the alpha3 chain, four to the alp
37              Single-chain Fv antibody tissue-specific reactivity was assessed by immunostaining of sy
38               Following transduction of PBL, specific reactivity was confirmed by cytokine production
39 94%) were positive in this assay, whereas no specific reactivity was detected in any of the 107 contr
40                                          HBV-specific reactivity was evaluated by T-cell proliferatio
41  T cell activation, migration, and beta cell-specific reactivity were similar in NOD mice of all thre
42 dentity of p116 has been demonstrated by its specific reactivity with anti-Cbl and similarity of phos
43 n a concentration dependent manner, while no specific reactivity with factors X or IXa was observed.
44             Conversely, MAb 19e exhibited no specific reactivity with free human CD4.
45 ay ionization (ESI) mass spectrometry and by specific reactivity with mAb 8E11, directed to GM2/GM3 d

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