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1 unction is impaired through gross defects in sperm motility.
2 m tail suggest that CABYR may be involved in sperm motility.
3 mammalian sperm tail and it is essential for sperm motility.
4 ed to male sterility resulting from aberrant sperm motility.
5 An inhibitor blocks the suNCKX activity and sperm motility.
6 llum, suggesting a role in the regulation of sperm motility.
7 other known voltage-gated channels, regulate sperm motility.
8 evealing a critical role for this isoform in sperm motility.
9 lt of significantly reduced sperm output and sperm motility.
10 inhibition of this isoform alone eliminates sperm motility.
11 wever, sperm competition does require normal sperm motility.
12 e fertile and have no significant changes in sperm motility.
13 in the sperm flagellum believed to modulate sperm motility.
14 ture of the fibrous sheath or participate in sperm motility.
15 is important for the initiation of mammalian sperm motility.
16 CO3 (-) is a key factor in the regulation of sperm motility.
17 ncidence of retained nipples and compromised sperm motility.
18 able to fertilize oocytes and exhibited poor sperm motility.
19 onment within the oviduct, thereby affecting sperm motility.
20 paratus have strikingly different effects on sperm motility.
21 1 may play a role in acrosome biogenesis and sperm motility.
22 ility associated with defects in progressive sperm motility.
23 n between superoxide production and enhanced sperm motility.
24 additional cryoprotectants and CAT on fresh sperm motility.
25 exposure for cellular repair and increasing sperm motility.
26 and the brain, and flagella are required for sperm motility.
27 be critical for normal sperm morphology and sperm motility.
28 along the length of the flagellum to support sperm motility.
29 fertility associated with a complete loss of sperm motility.
30 round-headed sperm morphology and no forward sperm motility.
31 to play essential roles in the mechanics of sperm motility.
32 fy lipid regulators required for directional sperm motility.
33 ra block the activity of suPDE5 and increase sperm motility.
34 ed in the mitochondrial capsule and enhances sperm motility.
35 regulation and a novel cellular function in sperm motility.
36 cGMP levels in sperm, which in turn modulate sperm motility.
37 n species known to have cyclic AMP-dependent sperm motility.
38 he major Ca2+ channel (CatSper) required for sperm motility.
39 m cells, reduced sperm counts, and decreased sperm motility.
40 f the coagulum, semenogelin I, also inhibits sperm motility.
42 on of the CATSPER1 channel, which can affect sperm motility, an important determinant in sperm compet
43 ar function driven by selective pressures on sperm motility, an important determinant of male reprodu
45 vating factor (PAF) has been shown to affect sperm motility and acrosomal function, thereby altering
46 s have revealed a wide range of variation in sperm motility and ATP production and that the laborator
47 inase A is thought to play a pivotal role in sperm motility and capacitation, the distinctive biochem
54 uid and in a dramatic but incomplete loss of sperm motility and fertilization capacity, raising the p
55 : 1) cryopreservation of coral sperm reduced sperm motility and fertilization success in half, thus f
57 pha4 deletion results in severe reduction in sperm motility and hyperactivation typical of sperm capa
58 ts also displayed a substantial reduction in sperm motility and infertility, whereas those carrying m
61 y provide help to uncover the causes of poor sperm motility and suggest new approaches for novel trea
62 KA catalytic activity, is a key regulator of sperm motility and that disruption of this interaction u
63 indicates that its role may be conserved in sperm motility and that JAM-A may be a candidate gene fo
67 processes are important in the regulation of sperm motility, and gene targeting was used here to test
68 y lowered daily sperm production, in reduced sperm motility, and in several animals, in sloughing of
73 hat previously observed effects of microG on sperm motility are coupled to changes in phosphorylation
74 aphase II, cellular components essential for sperm motility are partitioned almost exclusively to the
76 tics of protein tyrosine phosphorylation and sperm motility are unaltered in mutant sperm relative to
78 oxidative phosphorylation as ATP sources for sperm motility between mouse species that exhibit signif
79 4-Aminopyridine, a powerful modulator of sperm motility, both raised pHi and mobilized Ca(2+) sto
80 t functional terms, including fertilization, sperm motility, calcium channel regulation, and SNARE pr
81 n of IL17BR, rs1025689, is linked to altered sperm motility characteristics and changes in choline me
82 f sperm intracellular pH and Ca(2+) regulate sperm motility, chemotaxis, capacitation and the acrosom
85 andidate gene for the analysis of idiopathic sperm motility defects resulting in male subfertility in
86 Tpst2-deficient mice have male infertility, sperm motility defects, and possible abnormalities in sp
87 Coupling between protein phosphorylation and sperm motility during activation in microG and at 1 G wa
89 s were infertile and had profound defects in sperm motility, exhibiting sluggish movement without for
90 ins are implicated in Caenorhabditis elegans sperm motility (Fer-1), mammalian skeletal muscle develo
91 ever, there were significant improvements in sperm motility for men with study entry CD4 cell counts
92 r sperm concentration, increased progressive sperm motility generated more pyruvate conversion to lac
94 ess reaction in zebrafish sperm reducing its sperm motility in a concentration dependent manner (P<0.
96 Studies of both survival after sepsis and sperm motility in human populations have shown significa
100 ns the way for convenient bioassays based on sperm motility including at-home motile sperm tests.
101 studies suggest several roles for hAKAP82 in sperm motility, including the regulation of signal trans
106 s imply that most of the energy required for sperm motility is generated by glycolysis rather than ox
107 ity of the fibrous sheath and that effective sperm motility is lost in the absence of AKAP4 because s
113 he major sperm protein (MSP) -based nematode sperm-motility machinery resembles that observed with ri
114 (as shown in mouse) that susAC has a role in sperm motility, most probably through axonemal protein p
116 in intermediate chain presumably involved in sperm motility, originated from complex genetic rearrang
117 itigated stress and maintained viability and sperm motility (P>0.05), whereas superoxide dismutase (S
120 hat this SNP is also associated with altered sperm motility patterns and dysmorphic mitochondrial str
124 ust penetrate both layers in steps requiring sperm motility, sperm surface enzymes, and probably sper
125 n decreased male fertility due to diminished sperm motility; sperm from Chdh(-/-) males have decrease
126 lated and other experiments revealed reduced sperm motility, survival time, and sperm count also cont
127 eostasis, resulting in substantially reduced sperm motility, swimming speed, and HCO3 (-)-enhanced be
128 A concentration was positively correlated to sperm motility, to sperm count, and to the desmosterol-t
129 ffect of 633 nm coherent, red laser light on sperm motility using a novel wavelet-based algorithm tha
135 e relationship between energy metabolism and sperm motility we used dissolution Dynamic Nuclear Polar
137 role in acquisition of normal morphology and sperm motility when faced with hyperosmotic challenges,
138 nderstanding of the link between [Ca2+]i and sperm motility will only be gained by analysis of [Ca2+]
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